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The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats.

The Journal of nutritional biochemistry

Ramos LV, da Costa THM, Arruda SF.
PMID: 34653600
J Nutr Biochem. 2021 Oct 12;100:108881. doi: 10.1016/j.jnutbio.2021.108881. Epub 2021 Oct 12.

Coffee effects on glucose homeostasis in obesity remain controversial. We investigated whether coffee mitigates the negative effects on glucose metabolism induced by a high-fat diet and the interrelationships with redox-inflammatory responses. Rats were treated with: control (CT-); coffee (CT+)...

Cite
Ramos LV, da Costa THM, Arruda SF. The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. J Nutr Biochem. 2021;100:108881doi: 10.1016/j.jnutbio.2021.108881.
Ramos, L. V., da Costa, T. H. M., & Arruda, S. F. (2021). The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. The Journal of nutritional biochemistry, 100108881. https://doi.org/10.1016/j.jnutbio.2021.108881
Ramos, Larissa Valadares, et al. "The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats." The Journal of nutritional biochemistry vol. 100 (2021): 108881. doi: https://doi.org/10.1016/j.jnutbio.2021.108881
Ramos LV, da Costa THM, Arruda SF. The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. J Nutr Biochem. 2021 Oct 12;100:108881. doi: 10.1016/j.jnutbio.2021.108881. Epub 2021 Oct 12. PMID: 34653600.
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Glycolysis Rate-Limiting Enzymes: Novel Potential Regulators of Rheumatoid Arthritis Pathogenesis.

Frontiers in immunology

Zuo J, Tang J, Lu M, Zhou Z, Li Y, Tian H, Liu E, Gao B, Liu T, Shao P.
PMID: 34899740
Front Immunol. 2021 Nov 24;12:779787. doi: 10.3389/fimmu.2021.779787. eCollection 2021.

Rheumatoid arthritis (RA) is a classic autoimmune disease characterized by uncontrolled synovial proliferation, pannus formation, cartilage injury, and bone destruction. The specific pathogenesis of RA, a chronic inflammatory disease, remains unclear. However, both key glycolysis rate-limiting enzymes, hexokinase-II (HK-II),...

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Zuo J, Tang J, Lu M, et al. Glycolysis Rate-Limiting Enzymes: Novel Potential Regulators of Rheumatoid Arthritis Pathogenesis. Front Immunol. 2021;12:779787doi: 10.3389/fimmu.2021.779787.
Zuo, J., Tang, J., Lu, M., Zhou, Z., Li, Y., Tian, H., Liu, E., Gao, B., Liu, T., & Shao, P. (2021). Glycolysis Rate-Limiting Enzymes: Novel Potential Regulators of Rheumatoid Arthritis Pathogenesis. Frontiers in immunology, 12779787. https://doi.org/10.3389/fimmu.2021.779787
Zuo, Jianlin, et al. "Glycolysis Rate-Limiting Enzymes: Novel Potential Regulators of Rheumatoid Arthritis Pathogenesis." Frontiers in immunology vol. 12 (2021): 779787. doi: https://doi.org/10.3389/fimmu.2021.779787
Zuo J, Tang J, Lu M, Zhou Z, Li Y, Tian H, Liu E, Gao B, Liu T, Shao P. Glycolysis Rate-Limiting Enzymes: Novel Potential Regulators of Rheumatoid Arthritis Pathogenesis. Front Immunol. 2021 Nov 24;12:779787. doi: 10.3389/fimmu.2021.779787. eCollection 2021. PMID: 34899740; PMCID: PMC8651870.
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PFKP facilitates ATG4B phosphorylation during amino acid deprivation-induced autophagy.

Cellular signalling

Li X, Sun L, Yan G, Yan X.
PMID: 33607258
Cell Signal. 2021 Jun;82:109956. doi: 10.1016/j.cellsig.2021.109956. Epub 2021 Feb 16.

ATG4B facilitates autophagy by promoting autophagosome maturation through the reversible lipidation and delipidation of LC3. Recent reports have shown that phosphorylation of ATG4B regulates its activity and LC3 processing, leading to modulate autophagy activity. However, the mechanism about how...

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Li X, Sun L, Yan G, et al. PFKP facilitates ATG4B phosphorylation during amino acid deprivation-induced autophagy. Cell Signal. 2021;82:109956doi: 10.1016/j.cellsig.2021.109956.
Li, X., Sun, L., Yan, G., & Yan, X. (2021). PFKP facilitates ATG4B phosphorylation during amino acid deprivation-induced autophagy. Cellular signalling, 82109956. https://doi.org/10.1016/j.cellsig.2021.109956
Li, Xiuzhi, et al. "PFKP facilitates ATG4B phosphorylation during amino acid deprivation-induced autophagy." Cellular signalling vol. 82 (2021): 109956. doi: https://doi.org/10.1016/j.cellsig.2021.109956
Li X, Sun L, Yan G, Yan X. PFKP facilitates ATG4B phosphorylation during amino acid deprivation-induced autophagy. Cell Signal. 2021 Jun;82:109956. doi: 10.1016/j.cellsig.2021.109956. Epub 2021 Feb 16. PMID: 33607258.
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The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats.

The Journal of nutritional biochemistry

Ramos LV, da Costa THM, Arruda SF.
PMID: 34653600
J Nutr Biochem. 2021 Oct 12;100:108881. doi: 10.1016/j.jnutbio.2021.108881. Epub 2021 Oct 12.

Coffee effects on glucose homeostasis in obesity remain controversial. We investigated whether coffee mitigates the negative effects on glucose metabolism induced by a high-fat diet and the interrelationships with redox-inflammatory responses. Rats were treated with: control (CT-); coffee (CT+)...

Cite
Ramos LV, da Costa THM, Arruda SF. The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. J Nutr Biochem. 2021;100:108881doi: 10.1016/j.jnutbio.2021.108881.
Ramos, L. V., da Costa, T. H. M., & Arruda, S. F. (2021). The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. The Journal of nutritional biochemistry, 100108881. https://doi.org/10.1016/j.jnutbio.2021.108881
Ramos, Larissa Valadares, et al. "The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats." The Journal of nutritional biochemistry vol. 100 (2021): 108881. doi: https://doi.org/10.1016/j.jnutbio.2021.108881
Ramos LV, da Costa THM, Arruda SF. The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. J Nutr Biochem. 2021 Oct 12;100:108881. doi: 10.1016/j.jnutbio.2021.108881. Epub 2021 Oct 12. PMID: 34653600.
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Normobaric oxygen therapy attenuates hyperglycolysis in ischemic stroke.

Neural regeneration research

Cheng Z, Li FW, Stone CR, Elkin K, Peng CY, Bardhi R, Geng XK, Ding YC.
PMID: 33269745
Neural Regen Res. 2021 Jun;16(6):1017-1023. doi: 10.4103/1673-5374.300452.

Normobaric oxygen therapy has gained attention as a simple and convenient means of achieving neuroprotection against the pathogenic cascade initiated by acute ischemic stroke. The mechanisms underlying the neuroprotective efficacy of normobaric oxygen therapy, however, have not been fully...

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Cheng Z, Li FW, Stone CR, et al. Normobaric oxygen therapy attenuates hyperglycolysis in ischemic stroke. Neural Regen Res. 2021;16(6):1017-1023doi: 10.4103/1673-5374.300452.
Cheng, Z., Li, F. W., Stone, C. R., Elkin, K., Peng, C. Y., Bardhi, R., Geng, X. K., & Ding, Y. C. (2021). Normobaric oxygen therapy attenuates hyperglycolysis in ischemic stroke. Neural regeneration research, 16(6), 1017-1023. https://doi.org/10.4103/1673-5374.300452
Cheng, Zhe, et al. "Normobaric oxygen therapy attenuates hyperglycolysis in ischemic stroke." Neural regeneration research vol. 16,6 (2021): 1017-1023. doi: https://doi.org/10.4103/1673-5374.300452
Cheng Z, Li FW, Stone CR, Elkin K, Peng CY, Bardhi R, Geng XK, Ding YC. Normobaric oxygen therapy attenuates hyperglycolysis in ischemic stroke. Neural Regen Res. 2021 Jun;16(6):1017-1023. doi: 10.4103/1673-5374.300452. PMID: 33269745; PMCID: PMC8224134.
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Selective activation of PFKL suppresses the phagocytic oxidative burst.

Cell

Amara N, Cooper MP, Voronkova MA, Webb BA, Lynch EM, Kollman JM, Ma T, Yu K, Lai Z, Sangaraju D, Kayagaki N, Newton K, Bogyo M, Staben ST, Dixit VM.
PMID: 34320407
Cell. 2021 Aug 19;184(17):4480-4494.e15. doi: 10.1016/j.cell.2021.07.004. Epub 2021 Jul 27.

In neutrophils, nicotinamide adenine dinucleotide phosphate (NADPH) generated via the pentose phosphate pathway fuels NADPH oxidase NOX2 to produce reactive oxygen species for killing invading pathogens. However, excessive NOX2 activity can exacerbate inflammation, as in acute respiratory distress syndrome...

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Amara N, Cooper MP, Voronkova MA, et al. Selective activation of PFKL suppresses the phagocytic oxidative burst. Cell. 2021;184(17):4480-4494.e15doi: 10.1016/j.cell.2021.07.004.
Amara, N., Cooper, M. P., Voronkova, M. A., Webb, B. A., Lynch, E. M., Kollman, J. M., Ma, T., Yu, K., Lai, Z., Sangaraju, D., Kayagaki, N., Newton, K., Bogyo, M., Staben, S. T., & Dixit, V. M. (2021). Selective activation of PFKL suppresses the phagocytic oxidative burst. Cell, 184(17), 4480-4494.e15. https://doi.org/10.1016/j.cell.2021.07.004
Amara, Neri, et al. "Selective activation of PFKL suppresses the phagocytic oxidative burst." Cell vol. 184,17 (2021): 4480-4494.e15. doi: https://doi.org/10.1016/j.cell.2021.07.004
Amara N, Cooper MP, Voronkova MA, Webb BA, Lynch EM, Kollman JM, Ma T, Yu K, Lai Z, Sangaraju D, Kayagaki N, Newton K, Bogyo M, Staben ST, Dixit VM. Selective activation of PFKL suppresses the phagocytic oxidative burst. Cell. 2021 Aug 19;184(17):4480-4494.e15. doi: 10.1016/j.cell.2021.07.004. Epub 2021 Jul 27. PMID: 34320407.
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The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats.

The Journal of nutritional biochemistry

Ramos LV, da Costa THM, Arruda SF.
PMID: 34653600
J Nutr Biochem. 2022 Feb;100:108881. doi: 10.1016/j.jnutbio.2021.108881. Epub 2021 Oct 12.

Coffee effects on glucose homeostasis in obesity remain controversial. We investigated whether coffee mitigates the negative effects on glucose metabolism induced by a high-fat diet and the interrelationships with redox-inflammatory responses. Rats were treated with: control (CT-); coffee (CT+)...

Cite
Ramos LV, da Costa THM, Arruda SF. The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. J Nutr Biochem. 2021;100:108881doi: 10.1016/j.jnutbio.2021.108881.
Ramos, L. V., da Costa, T. H. M., & Arruda, S. F. (2022). The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. The Journal of nutritional biochemistry, 100108881. https://doi.org/10.1016/j.jnutbio.2021.108881
Ramos, Larissa Valadares, et al. "The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats." The Journal of nutritional biochemistry vol. 100 (2022): 108881. doi: https://doi.org/10.1016/j.jnutbio.2021.108881
Ramos LV, da Costa THM, Arruda SF. The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. J Nutr Biochem. 2022 Feb;100:108881. doi: 10.1016/j.jnutbio.2021.108881. Epub 2021 Oct 12. PMID: 34653600.
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The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats.

The Journal of nutritional biochemistry

Ramos LV, da Costa THM, Arruda SF.
PMID: 34653600
J Nutr Biochem. 2021 Oct 12;100:108881. doi: 10.1016/j.jnutbio.2021.108881. Epub 2021 Oct 12.

Coffee effects on glucose homeostasis in obesity remain controversial. We investigated whether coffee mitigates the negative effects on glucose metabolism induced by a high-fat diet and the interrelationships with redox-inflammatory responses. Rats were treated with: control (CT-); coffee (CT+)...

Cite
Ramos LV, da Costa THM, Arruda SF. The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. J Nutr Biochem. 2021;100:108881doi: 10.1016/j.jnutbio.2021.108881.
Ramos, L. V., da Costa, T. H. M., & Arruda, S. F. (2021). The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. The Journal of nutritional biochemistry, 100108881. https://doi.org/10.1016/j.jnutbio.2021.108881
Ramos, Larissa Valadares, et al. "The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats." The Journal of nutritional biochemistry vol. 100 (2021): 108881. doi: https://doi.org/10.1016/j.jnutbio.2021.108881
Ramos LV, da Costa THM, Arruda SF. The effect of coffee consumption on glucose homeostasis and redox-inflammatory responses in high-fat diet-induced obese rats. J Nutr Biochem. 2021 Oct 12;100:108881. doi: 10.1016/j.jnutbio.2021.108881. Epub 2021 Oct 12. PMID: 34653600.
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Inhibition of 6-phosphofructo-2-kinase (PFKFB3) induces autophagy as a survival mechanism.

Cancer & metabolism

Klarer AC, O'Neal J, Imbert-Fernandez Y, Clem A, Ellis SR, Clark J, Clem B, Chesney J, Telang S.
PMID: 24451478
Cancer Metab. 2014 Jan 23;2(1):2. doi: 10.1186/2049-3002-2-2.

BACKGROUND: Unlike glycolytic enzymes that directly catabolize glucose to pyruvate, the family of 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatases (PFKFBs) control the conversion of fructose-6-phosphate to and from fructose-2,6-bisphosphate, a key regulator of the glycolytic enzyme phosphofructokinase-1 (PFK-1). One family member, PFKFB3, has been...

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Klarer AC, O'Neal J, Imbert-Fernandez Y, et al. Inhibition of 6-phosphofructo-2-kinase (PFKFB3) induces autophagy as a survival mechanism. Cancer Metab. 2014;2(1):2doi: 10.1186/2049-3002-2-2.
Klarer, A. C., O'Neal, J., Imbert-Fernandez, Y., Clem, A., Ellis, S. R., Clark, J., Clem, B., Chesney, J., & Telang, S. (2014). Inhibition of 6-phosphofructo-2-kinase (PFKFB3) induces autophagy as a survival mechanism. Cancer & metabolism, 2(1), 2. https://doi.org/10.1186/2049-3002-2-2
Klarer, Alden C, et al. "Inhibition of 6-phosphofructo-2-kinase (PFKFB3) induces autophagy as a survival mechanism." Cancer & metabolism vol. 2,1 (2014): 2. doi: https://doi.org/10.1186/2049-3002-2-2
Klarer AC, O'Neal J, Imbert-Fernandez Y, Clem A, Ellis SR, Clark J, Clem B, Chesney J, Telang S. Inhibition of 6-phosphofructo-2-kinase (PFKFB3) induces autophagy as a survival mechanism. Cancer Metab. 2014 Jan 23;2(1):2. doi: 10.1186/2049-3002-2-2. PMID: 24451478; PMCID: PMC3913946.
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Induction of erythroid differentiation is associated with inhibition of glycolysis and a decrease in fructose 2,6-bisphosphate levels.

International journal of oncology

Scher B, Chen J, Fuksina I, Waxman S, Scher W.
PMID: 21528315
Int J Oncol. 1997 Nov;11(5):1135-40. doi: 10.3892/ijo.11.5.1135.

The fraction of glucose metabolized to lactate is dramatically reduced during erythroid differentiation of mouse erythroleukemia (MEL) cells induced by dimethyl sulfoxide (DMSO), hexamethylene bisacetamide (HMBA), or sodium butyrate treatment. In order to determine the mechanism of the reduction...

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Scher B, Chen J, Fuksina I, et al. Induction of erythroid differentiation is associated with inhibition of glycolysis and a decrease in fructose 2,6-bisphosphate levels. Int J Oncol. 1997;11(5):1135-40doi: 10.3892/ijo.11.5.1135.
Scher, B., Chen, J., Fuksina, I., Waxman, S., & Scher, W. (1997). Induction of erythroid differentiation is associated with inhibition of glycolysis and a decrease in fructose 2,6-bisphosphate levels. International journal of oncology, 11(5), 1135-40. https://doi.org/10.3892/ijo.11.5.1135
Scher, B, et al. "Induction of erythroid differentiation is associated with inhibition of glycolysis and a decrease in fructose 2,6-bisphosphate levels." International journal of oncology vol. 11,5 (1997): 1135-40. doi: https://doi.org/10.3892/ijo.11.5.1135
Scher B, Chen J, Fuksina I, Waxman S, Scher W. Induction of erythroid differentiation is associated with inhibition of glycolysis and a decrease in fructose 2,6-bisphosphate levels. Int J Oncol. 1997 Nov;11(5):1135-40. doi: 10.3892/ijo.11.5.1135. PMID: 21528315.
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Poly(ADP-ribose) polymerase regulates glycolytic activity in kidney proximal tubule epithelial cells.

Anatomy & cell biology

Song H, Yoon SP, Kim J.
PMID: 27382509
Anat Cell Biol. 2016 Jun;49(2):79-87. doi: 10.5115/acb.2016.49.2.79. Epub 2016 Jun 24.

After renal injury, selective damage occurs in the proximal tubules as a result of inhibition of glycolysis. The molecular mechanism of damage is not known. Poly(ADP-ribose) polymerase (PARP) activation plays a critical role of proximal tubular cell death in...

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Song H, Yoon SP, Kim J. Poly(ADP-ribose) polymerase regulates glycolytic activity in kidney proximal tubule epithelial cells. Anat Cell Biol. 2016;49(2):79-87doi: 10.5115/acb.2016.49.2.79.
Song, H., Yoon, S. P., & Kim, J. (2016). Poly(ADP-ribose) polymerase regulates glycolytic activity in kidney proximal tubule epithelial cells. Anatomy & cell biology, 49(2), 79-87. https://doi.org/10.5115/acb.2016.49.2.79
Song, Hana, et al. "Poly(ADP-ribose) polymerase regulates glycolytic activity in kidney proximal tubule epithelial cells." Anatomy & cell biology vol. 49,2 (2016): 79-87. doi: https://doi.org/10.5115/acb.2016.49.2.79
Song H, Yoon SP, Kim J. Poly(ADP-ribose) polymerase regulates glycolytic activity in kidney proximal tubule epithelial cells. Anat Cell Biol. 2016 Jun;49(2):79-87. doi: 10.5115/acb.2016.49.2.79. Epub 2016 Jun 24. PMID: 27382509; PMCID: PMC4927434.
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Wnt signaling promotes tumor development in part through phosphofructokinase 1 platelet isoform upregulation.

Oncology reports

Jeon SM, Lim JS, Park SH, Lee JH.
PMID: 34515327
Oncol Rep. 2021 Nov;46(5). doi: 10.3892/or.2021.8185. Epub 2021 Sep 13.

The activation of Wnt signaling has been detected in various types of human cancer and has been shown to be associated with cancer development. In the present study, it was revealed that Wnt signaling induced the expression of phosphofructokinase...

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Jeon SM, Lim JS, Park SH, et al. Wnt signaling promotes tumor development in part through phosphofructokinase 1 platelet isoform upregulation. Oncol Rep. 2021;46(5)doi: 10.3892/or.2021.8185.
Jeon, S. M., Lim, J. S., Park, S. H., & Lee, J. H. (2021). Wnt signaling promotes tumor development in part through phosphofructokinase 1 platelet isoform upregulation. Oncology reports, 46(5), . https://doi.org/10.3892/or.2021.8185
Jeon, So Mi, et al. "Wnt signaling promotes tumor development in part through phosphofructokinase 1 platelet isoform upregulation." Oncology reports vol. 46,5 (2021). doi: https://doi.org/10.3892/or.2021.8185
Jeon SM, Lim JS, Park SH, Lee JH. Wnt signaling promotes tumor development in part through phosphofructokinase 1 platelet isoform upregulation. Oncol Rep. 2021 Nov;46(5). doi: 10.3892/or.2021.8185. Epub 2021 Sep 13. PMID: 34515327.
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Showing 1 to 12 of 41 entries