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Showing 1 to 12 of 15 entries
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Central Serotonergic Modulation of Carrageenin-induced Pedal Inflammation in Rats.

Pharmaceutical research

Bhattacharya SK, Das N.
PMID: 24271133
Pharm Res. 1985 Nov;2(6):315-8. doi: 10.1023/A:1016358004907.

Putative central serotonergic modulation of acute peripheral inflammation was investigated in rats, using the carrageenin-induced pedal edema as the experimental model. Serotonin and the serotonin precursor 5-hydroxytryptophan (5HTP) produced a dose-related inhibition of the peripheral edema when given intracerebroventricularly...

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Bhattacharya SK, Das N. Central Serotonergic Modulation of Carrageenin-induced Pedal Inflammation in Rats. Pharm Res. 1985;2(6):315-8doi: 10.1023/A:1016358004907.
Bhattacharya, S. K., & Das, N. (1985). Central Serotonergic Modulation of Carrageenin-induced Pedal Inflammation in Rats. Pharmaceutical research, 2(6), 315-8. https://doi.org/10.1023/A:1016358004907
Bhattacharya, S K, and Das, N. "Central Serotonergic Modulation of Carrageenin-induced Pedal Inflammation in Rats." Pharmaceutical research vol. 2,6 (1985): 315-8. doi: https://doi.org/10.1023/A:1016358004907
Bhattacharya SK, Das N. Central Serotonergic Modulation of Carrageenin-induced Pedal Inflammation in Rats. Pharm Res. 1985 Nov;2(6):315-8. doi: 10.1023/A:1016358004907. PMID: 24271133.
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Evidence of the existence of two different intraneuronal pools from which pharmacological agents can release serotonin.

Neurochemistry international

Mennini T, Borroni E, Samanin R, Garattini S.
PMID: 20487825
Neurochem Int. 1981;3(5):289-94. doi: 10.1016/0197-0186(81)90015-2.

The effect of various drugs on the release of [(3)H]-serotonin from synaptosomes of reserpine-treated rats was compared with that obtained with synaptosomes of untreated animals. The increase in [(3)H]-serotonin release induced by d-fenfluramine was virtually abolished by reserpine; the...

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Mennini T, Borroni E, Samanin R, et al. Evidence of the existence of two different intraneuronal pools from which pharmacological agents can release serotonin. Neurochem Int. 1981;3(5):289-94doi: 10.1016/0197-0186(81)90015-2.
Mennini, T., Borroni, E., Samanin, R., & Garattini, S. (1981). Evidence of the existence of two different intraneuronal pools from which pharmacological agents can release serotonin. Neurochemistry international, 3(5), 289-94. https://doi.org/10.1016/0197-0186(81)90015-2
Mennini, T, et al. "Evidence of the existence of two different intraneuronal pools from which pharmacological agents can release serotonin." Neurochemistry international vol. 3,5 (1981): 289-94. doi: https://doi.org/10.1016/0197-0186(81)90015-2
Mennini T, Borroni E, Samanin R, Garattini S. Evidence of the existence of two different intraneuronal pools from which pharmacological agents can release serotonin. Neurochem Int. 1981;3(5):289-94. doi: 10.1016/0197-0186(81)90015-2. PMID: 20487825.
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In vivo [(3)H]ketanserin binding: effect of modifying synaptic serotonin levels.

Neurochemistry international

Assie MB, Charveron M, Briley M.
PMID: 20501207
Neurochem Int. 1988;12(1):85-9. doi: 10.1016/0197-0186(88)90152-0.

Most antidepressant therapies aim to increase the synaptic concentration of one or more of the monoamines. Synaptic monoamine levels are, however, extremely difficult to measure. In order to estimate synaptic levels of serotonin, an indirect method has been used....

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Assie MB, Charveron M, Briley M. In vivo [(3)H]ketanserin binding: effect of modifying synaptic serotonin levels. Neurochem Int. 1988;12(1):85-9doi: 10.1016/0197-0186(88)90152-0.
Assie, M. B., Charveron, M., & Briley, M. (1988). In vivo [(3)H]ketanserin binding: effect of modifying synaptic serotonin levels. Neurochemistry international, 12(1), 85-9. https://doi.org/10.1016/0197-0186(88)90152-0
Assie, M B, et al. "In vivo [(3)H]ketanserin binding: effect of modifying synaptic serotonin levels." Neurochemistry international vol. 12,1 (1988): 85-9. doi: https://doi.org/10.1016/0197-0186(88)90152-0
Assie MB, Charveron M, Briley M. In vivo [(3)H]ketanserin binding: effect of modifying synaptic serotonin levels. Neurochem Int. 1988;12(1):85-9. doi: 10.1016/0197-0186(88)90152-0. PMID: 20501207.
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Fluoxetine, a selective serotonin reuptake inhibitor used clinically, improves bladder function in a mouse model of moderate spinal cord injury.

Neural regeneration research

Ma L, Tang JY, Zhou JY, Zhu C, Zhang X, Zhou P, Yu Q, Wang Y, Gu XJ.
PMID: 33642399
Neural Regen Res. 2021 Oct;16(10):2093-2098. doi: 10.4103/1673-5374.308667.

After spinal cord injury, the upward conduction of the spinal cord is lost, resulting in the loss of micturition control, which manifests as detrusor sphincter dyssynergia and insufficient micturition. Studies have shown that serotonergic axons play important roles in...

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Ma L, Tang JY, Zhou JY, et al. Fluoxetine, a selective serotonin reuptake inhibitor used clinically, improves bladder function in a mouse model of moderate spinal cord injury. Neural Regen Res. 2021;16(10):2093-2098doi: 10.4103/1673-5374.308667.
Ma, L., Tang, J. Y., Zhou, J. Y., Zhu, C., Zhang, X., Zhou, P., Yu, Q., Wang, Y., & Gu, X. J. (2021). Fluoxetine, a selective serotonin reuptake inhibitor used clinically, improves bladder function in a mouse model of moderate spinal cord injury. Neural regeneration research, 16(10), 2093-2098. https://doi.org/10.4103/1673-5374.308667
Ma, Long, et al. "Fluoxetine, a selective serotonin reuptake inhibitor used clinically, improves bladder function in a mouse model of moderate spinal cord injury." Neural regeneration research vol. 16,10 (2021): 2093-2098. doi: https://doi.org/10.4103/1673-5374.308667
Ma L, Tang JY, Zhou JY, Zhu C, Zhang X, Zhou P, Yu Q, Wang Y, Gu XJ. Fluoxetine, a selective serotonin reuptake inhibitor used clinically, improves bladder function in a mouse model of moderate spinal cord injury. Neural Regen Res. 2021 Oct;16(10):2093-2098. doi: 10.4103/1673-5374.308667. PMID: 33642399; PMCID: PMC8343320.
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Rapid changes in functional responsiveness of the 5-HT system after single-dose and multiple-dose treatment with antidepressants: effects of maprotiline and oxaprotiline and its enantiomers.

Journal of psychopharmacology (Oxford, England)

Delini-Stula A, Mogilnicka E.
PMID: 22156305
J Psychopharmacol. 1989 Jan;3(1):7-13. doi: 10.1177/026988118900300102.

Functional responsiveness to 5-HTP (L-5-hydroxytryptophan) and quipazine 24 h after one single dose and 24 h and 72 h after repeated administration (2.5 and 10 mg/kg twice daily for 7 days) of maprotiline, oxaprotiline and its (+) and (-)...

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Delini-Stula A, Mogilnicka E. Rapid changes in functional responsiveness of the 5-HT system after single-dose and multiple-dose treatment with antidepressants: effects of maprotiline and oxaprotiline and its enantiomers. J Psychopharmacol. 1989;3(1):7-13doi: 10.1177/026988118900300102.
Delini-Stula, A., & Mogilnicka, E. (1989). Rapid changes in functional responsiveness of the 5-HT system after single-dose and multiple-dose treatment with antidepressants: effects of maprotiline and oxaprotiline and its enantiomers. Journal of psychopharmacology (Oxford, England), 3(1), 7-13. https://doi.org/10.1177/026988118900300102
Delini-Stula, A, and Mogilnicka, E. "Rapid changes in functional responsiveness of the 5-HT system after single-dose and multiple-dose treatment with antidepressants: effects of maprotiline and oxaprotiline and its enantiomers." Journal of psychopharmacology (Oxford, England) vol. 3,1 (1989): 7-13. doi: https://doi.org/10.1177/026988118900300102
Delini-Stula A, Mogilnicka E. Rapid changes in functional responsiveness of the 5-HT system after single-dose and multiple-dose treatment with antidepressants: effects of maprotiline and oxaprotiline and its enantiomers. J Psychopharmacol. 1989 Jan;3(1):7-13. doi: 10.1177/026988118900300102. PMID: 22156305.
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Regulation of Basal and nursing-induced secretion of growth hormone in the neonatal rat: the involvement of serotonergic, muscarinic cholinergic, alpha-adrenergic, somatostatin and growth hormone-releasing hormone systems.

Journal of neuroendocrinology

Kacsóh B, Grosvenor CE.
PMID: 19215503
J Neuroendocrinol. 1991 Oct 01;3(5):529-37. doi: 10.1111/j.1365-2826.1991.tb00314.x.

Abstract Various neural factors are involved in the suckling-induced increase in serum growth hormone (GH) levels in neonatal rats, and, in the present study the serotonergic, cholinergic, somatostatin and GH-releasing hormone (GHRH) systems were investigated. The serotonin (5-HT) precursor...

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Kacsóh B, Grosvenor CE. Regulation of Basal and nursing-induced secretion of growth hormone in the neonatal rat: the involvement of serotonergic, muscarinic cholinergic, alpha-adrenergic, somatostatin and growth hormone-releasing hormone systems. J Neuroendocrinol. 1991;3(5):529-37doi: 10.1111/j.1365-2826.1991.tb00314.x.
Kacsóh, B., & Grosvenor, C. E. (1991). Regulation of Basal and nursing-induced secretion of growth hormone in the neonatal rat: the involvement of serotonergic, muscarinic cholinergic, alpha-adrenergic, somatostatin and growth hormone-releasing hormone systems. Journal of neuroendocrinology, 3(5), 529-37. https://doi.org/10.1111/j.1365-2826.1991.tb00314.x
Kacsóh, B, and Grosvenor, C E. "Regulation of Basal and nursing-induced secretion of growth hormone in the neonatal rat: the involvement of serotonergic, muscarinic cholinergic, alpha-adrenergic, somatostatin and growth hormone-releasing hormone systems." Journal of neuroendocrinology vol. 3,5 (1991): 529-37. doi: https://doi.org/10.1111/j.1365-2826.1991.tb00314.x
Kacsóh B, Grosvenor CE. Regulation of Basal and nursing-induced secretion of growth hormone in the neonatal rat: the involvement of serotonergic, muscarinic cholinergic, alpha-adrenergic, somatostatin and growth hormone-releasing hormone systems. J Neuroendocrinol. 1991 Oct 01;3(5):529-37. doi: 10.1111/j.1365-2826.1991.tb00314.x. PMID: 19215503.
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Expression level of long non-coding RNA colon adenocarcinoma hypermethylated serves as a novel prognostic biomarker in patients with thyroid carcinoma.

Bioscience reports

Xiao Y, Tu Y, Li Y.
PMID: 33792624
Biosci Rep. 2021 Apr 30;41(4). doi: 10.1042/BSR20210284.

The present study attempts to identify the prognostic value and potential mechanism of action of colorectal adenocarcinoma hypermethylated (CAHM) in thyroid carcinoma (THCA) by using the RNA sequencing (RNA-seq) dataset from The Cancer Genome Atlas (TCGA). The functional mechanism...

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Xiao Y, Tu Y, Li Y. Expression level of long non-coding RNA colon adenocarcinoma hypermethylated serves as a novel prognostic biomarker in patients with thyroid carcinoma. Biosci Rep. 2021;41(4)doi: 10.1042/BSR20210284.
Xiao, Y., Tu, Y., & Li, Y. (2021). Expression level of long non-coding RNA colon adenocarcinoma hypermethylated serves as a novel prognostic biomarker in patients with thyroid carcinoma. Bioscience reports, 41(4), . https://doi.org/10.1042/BSR20210284
Xiao, Yong, et al. "Expression level of long non-coding RNA colon adenocarcinoma hypermethylated serves as a novel prognostic biomarker in patients with thyroid carcinoma." Bioscience reports vol. 41,4 (2021). doi: https://doi.org/10.1042/BSR20210284
Xiao Y, Tu Y, Li Y. Expression level of long non-coding RNA colon adenocarcinoma hypermethylated serves as a novel prognostic biomarker in patients with thyroid carcinoma. Biosci Rep. 2021 Apr 30;41(4). doi: 10.1042/BSR20210284. PMID: 33792624; PMCID: PMC8056003.
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Acute intrathecal administration of quipazine elicits air-stepping behavior.

Behavioural pharmacology

Swann-Thomsen HE, Viall DD, Brumley MR.
PMID: 33595953
Behav Pharmacol. 2021 Jun 01;32(4):259-264. doi: 10.1097/FBP.0000000000000608.

Serotonin plays a pivotal role in the initiation and modulation of locomotor behavior in the intact animal, as well as following spinal cord injury. Quipazine, a serotonin 2 receptor agonist, has been used successfully to initiate and restore motor...

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Swann-Thomsen HE, Viall DD, Brumley MR. Acute intrathecal administration of quipazine elicits air-stepping behavior. Behav Pharmacol. 2021;32(4):259-264doi: 10.1097/FBP.0000000000000608.
Swann-Thomsen, H. E., Viall, D. D., & Brumley, M. R. (2021). Acute intrathecal administration of quipazine elicits air-stepping behavior. Behavioural pharmacology, 32(4), 259-264. https://doi.org/10.1097/FBP.0000000000000608
Swann-Thomsen, Hillary E, et al. "Acute intrathecal administration of quipazine elicits air-stepping behavior." Behavioural pharmacology vol. 32,4 (2021): 259-264. doi: https://doi.org/10.1097/FBP.0000000000000608
Swann-Thomsen HE, Viall DD, Brumley MR. Acute intrathecal administration of quipazine elicits air-stepping behavior. Behav Pharmacol. 2021 Jun 01;32(4):259-264. doi: 10.1097/FBP.0000000000000608. PMID: 33595953; PMCID: PMC8119288.
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Chlorimipramine, fenfluramine and quipazine decrease 5-hydroxytryptamine synthesis in discrete rat brain nuclei.

Neurochemistry international

Nielsen JA.
PMID: 20493070
Neurochem Int. 1986;8(3):403-12. doi: 10.1016/0197-0186(86)90014-8.

A procedure for studying 5-hydroxytryptamine synthesis by determining the rate of accumulation of 5-hydroxytryptophan after administering m-hydroxybenzylhydrazine, an inhibitor of aromatic-l-amino acid decarboxylase, and large doses of l-tryptophan was characterized. The utility of this method as an index of...

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Nielsen JA. Chlorimipramine, fenfluramine and quipazine decrease 5-hydroxytryptamine synthesis in discrete rat brain nuclei. Neurochem Int. 1986;8(3):403-12doi: 10.1016/0197-0186(86)90014-8.
Nielsen, J. A. (1986). Chlorimipramine, fenfluramine and quipazine decrease 5-hydroxytryptamine synthesis in discrete rat brain nuclei. Neurochemistry international, 8(3), 403-12. https://doi.org/10.1016/0197-0186(86)90014-8
Nielsen, J A. "Chlorimipramine, fenfluramine and quipazine decrease 5-hydroxytryptamine synthesis in discrete rat brain nuclei." Neurochemistry international vol. 8,3 (1986): 403-12. doi: https://doi.org/10.1016/0197-0186(86)90014-8
Nielsen JA. Chlorimipramine, fenfluramine and quipazine decrease 5-hydroxytryptamine synthesis in discrete rat brain nuclei. Neurochem Int. 1986;8(3):403-12. doi: 10.1016/0197-0186(86)90014-8. PMID: 20493070.
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Alterations in dopamine and 5-hydroxytryptamine (5-HT) function during barbiturate dependence and withdrawal in mice.

Journal of psychopharmacology (Oxford, England)

Gray PL, Dawbarn D, Taberner PV.
PMID: 22158891
J Psychopharmacol. 1987 Jan;1(2):101-8. doi: 10.1177/026988118700100207.

Mice were rendered barbiturate-dependent by chronic feeding with barbital-con taining food. Brain dopamine turnover was significantly increased in barbital withdrawal, whereas 5-hydroxytryptamine (5-HT) turnover was significantly decreased. Severity of with drawal was assessed by measuring the convulsions following a...

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Gray PL, Dawbarn D, Taberner PV. Alterations in dopamine and 5-hydroxytryptamine (5-HT) function during barbiturate dependence and withdrawal in mice. J Psychopharmacol. 1987;1(2):101-8doi: 10.1177/026988118700100207.
Gray, P. L., Dawbarn, D., & Taberner, P. V. (1987). Alterations in dopamine and 5-hydroxytryptamine (5-HT) function during barbiturate dependence and withdrawal in mice. Journal of psychopharmacology (Oxford, England), 1(2), 101-8. https://doi.org/10.1177/026988118700100207
Gray, P L, et al. "Alterations in dopamine and 5-hydroxytryptamine (5-HT) function during barbiturate dependence and withdrawal in mice." Journal of psychopharmacology (Oxford, England) vol. 1,2 (1987): 101-8. doi: https://doi.org/10.1177/026988118700100207
Gray PL, Dawbarn D, Taberner PV. Alterations in dopamine and 5-hydroxytryptamine (5-HT) function during barbiturate dependence and withdrawal in mice. J Psychopharmacol. 1987 Jan;1(2):101-8. doi: 10.1177/026988118700100207. PMID: 22158891.
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Discriminative stimulus effects of amphetamine and pentobarbitone separately and as mixtures in rats.

Behavioural pharmacology

Mariathasan EA, Garcha HS, Stolerman IP.
PMID: 11224083
Behav Pharmacol. 1991 Nov;2(4):405-415.

The discriminative effect of a mixture of (+)-amphetamine plus pentobarbitone was investigated to characterize behavioural control by a compound interoceptive stimulus. A two-bar drug discrimination procedure with food reinforcement was used. The role of training dose was assessed by...

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Mariathasan EA, Garcha HS, Stolerman IP. Discriminative stimulus effects of amphetamine and pentobarbitone separately and as mixtures in rats. Behav Pharmacol. 1991;2(4):405-415
Mariathasan, E. A., Garcha, H. S., & Stolerman, I. P. (1991). Discriminative stimulus effects of amphetamine and pentobarbitone separately and as mixtures in rats. Behavioural pharmacology, 2(4), 405-415.
Mariathasan, E.A., et al. "Discriminative stimulus effects of amphetamine and pentobarbitone separately and as mixtures in rats." Behavioural pharmacology vol. 2,4 (1991): 405-415.
Mariathasan EA, Garcha HS, Stolerman IP. Discriminative stimulus effects of amphetamine and pentobarbitone separately and as mixtures in rats. Behav Pharmacol. 1991 Nov;2(4):405-415. PMID: 11224083.
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Discrimination of a drug mixture in rats: role of training dose, and specificity.

Behavioural pharmacology

Garcha HS, Stolerman IP.
PMID: 11175384
Behav Pharmacol. 1989;1(1):25-31. doi: 10.1097/00008877-198900110-00004.

Many drugs produce compound discriminative stimuli with at least two elements; in contrast, the present study examines discrimination of a mixture of two drugs and tests the role of training dose in, and the specificity of, such a discrimination....

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Garcha HS, Stolerman IP. Discrimination of a drug mixture in rats: role of training dose, and specificity. Behav Pharmacol. 1989;1(1):25-31doi: 10.1097/00008877-198900110-00004.
Garcha, H. S., & Stolerman, I. P. (1989). Discrimination of a drug mixture in rats: role of training dose, and specificity. Behavioural pharmacology, 1(1), 25-31. https://doi.org/10.1097/00008877-198900110-00004
Garcha, H.S., and Stolerman, I.P.. "Discrimination of a drug mixture in rats: role of training dose, and specificity." Behavioural pharmacology vol. 1,1 (1989): 25-31. doi: https://doi.org/10.1097/00008877-198900110-00004
Garcha HS, Stolerman IP. Discrimination of a drug mixture in rats: role of training dose, and specificity. Behav Pharmacol. 1989;1(1):25-31. doi: 10.1097/00008877-198900110-00004. PMID: 11175384.
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Showing 1 to 12 of 15 entries