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Disrupted ER-to-Golgi Trafficking Underlies Anti-HIV Drugs and Alcohol-Induced Cellular Stress and Hepatic Injury.

Hepatology communications

Han H, He Y, Hu J, Lau R, Lee H, Ji C.
PMID: 28626835
Hepatol Commun. 2017 Apr;1(2):122-139. doi: 10.1002/hep4.1030. Epub 2017 Mar 27.

Endoplasmic reticulum (ER) stress and unfolded protein response (UPR) are involved in anti-human immunodeficiency virus (HIV) drugs and alcohol-induced liver disease in a significant number of patients infected with HIV. However, the precise mechanism by which the drugs and...

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Han H, He Y, Hu J, et al. Disrupted ER-to-Golgi Trafficking Underlies Anti-HIV Drugs and Alcohol-Induced Cellular Stress and Hepatic Injury. Hepatol Commun. 2017;1(2):122-139doi: 10.1002/hep4.1030.
Han, H., He, Y., Hu, J., Lau, R., Lee, H., & Ji, C. (2017). Disrupted ER-to-Golgi Trafficking Underlies Anti-HIV Drugs and Alcohol-Induced Cellular Stress and Hepatic Injury. Hepatology communications, 1(2), 122-139. https://doi.org/10.1002/hep4.1030
Han, Hui, et al. "Disrupted ER-to-Golgi Trafficking Underlies Anti-HIV Drugs and Alcohol-Induced Cellular Stress and Hepatic Injury." Hepatology communications vol. 1,2 (2017): 122-139. doi: https://doi.org/10.1002/hep4.1030
Han H, He Y, Hu J, Lau R, Lee H, Ji C. Disrupted ER-to-Golgi Trafficking Underlies Anti-HIV Drugs and Alcohol-Induced Cellular Stress and Hepatic Injury. Hepatol Commun. 2017 Apr;1(2):122-139. doi: 10.1002/hep4.1030. Epub 2017 Mar 27. PMID: 28626835; PMCID: PMC5473515.
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Enhanced expression of glucose-regulated protein 78 correlates with malondialdehyde levels during the formation of liver cirrhosis in rats.

Experimental and therapeutic medicine

Zhang Y, Zhang H, Zhao Z, Lv M, Jia J, Zhang L, Tian X, Chen Y, Li B, Liu M, Han D, Ji C.
PMID: 26668603
Exp Ther Med. 2015 Dec;10(6):2119-2125. doi: 10.3892/etm.2015.2783. Epub 2015 Oct 01.

The aim of the present study was to explore the role of glucose-regulated protein 78 (GRP78) in the development of liver cirrhosis promoted by intestinal endotoxemia in rats. Fifty-one male Wistar rats were randomly divided into the liver cirrhosis...

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Zhang Y, Zhang H, Zhao Z, et al. Enhanced expression of glucose-regulated protein 78 correlates with malondialdehyde levels during the formation of liver cirrhosis in rats. Exp Ther Med. 2015;10(6):2119-2125doi: 10.3892/etm.2015.2783.
Zhang, Y., Zhang, H., Zhao, Z., Lv, M., Jia, J., Zhang, L., Tian, X., Chen, Y., Li, B., Liu, M., Han, D., & Ji, C. (2015). Enhanced expression of glucose-regulated protein 78 correlates with malondialdehyde levels during the formation of liver cirrhosis in rats. Experimental and therapeutic medicine, 10(6), 2119-2125. https://doi.org/10.3892/etm.2015.2783
Zhang, Yun, et al. "Enhanced expression of glucose-regulated protein 78 correlates with malondialdehyde levels during the formation of liver cirrhosis in rats." Experimental and therapeutic medicine vol. 10,6 (2015): 2119-2125. doi: https://doi.org/10.3892/etm.2015.2783
Zhang Y, Zhang H, Zhao Z, Lv M, Jia J, Zhang L, Tian X, Chen Y, Li B, Liu M, Han D, Ji C. Enhanced expression of glucose-regulated protein 78 correlates with malondialdehyde levels during the formation of liver cirrhosis in rats. Exp Ther Med. 2015 Dec;10(6):2119-2125. doi: 10.3892/etm.2015.2783. Epub 2015 Oct 01. PMID: 26668603; PMCID: PMC4665701.
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Mechanisms of alcohol-induced endoplasmic reticulum stress and organ injuries.

Biochemistry research international

Ji C.
PMID: 22110961
Biochem Res Int. 2012;2012:216450. doi: 10.1155/2012/216450. Epub 2011 Oct 26.

Alcohol is readily distributed throughout the body in the blood stream and crosses biological membranes, which affect virtually all biological processes inside the cell. Excessive alcohol consumption induces numerous pathological stress responses, part of which is endoplasmic reticulum (ER)...

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Ji C. Mechanisms of alcohol-induced endoplasmic reticulum stress and organ injuries. Biochem Res Int. 2011;2012:216450doi: 10.1155/2012/216450.
Ji, C. (2012). Mechanisms of alcohol-induced endoplasmic reticulum stress and organ injuries. Biochemistry research international, 2012216450. https://doi.org/10.1155/2012/216450
Ji, Cheng. "Mechanisms of alcohol-induced endoplasmic reticulum stress and organ injuries." Biochemistry research international vol. 2012 (2012): 216450. doi: https://doi.org/10.1155/2012/216450
Ji C. Mechanisms of alcohol-induced endoplasmic reticulum stress and organ injuries. Biochem Res Int. 2012;2012:216450. doi: 10.1155/2012/216450. Epub 2011 Oct 26. PMID: 22110961; PMCID: PMC3205771.
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New Insights into the Pathogenesis of Alcohol-Induced ER Stress and Liver Diseases.

International journal of hepatology

Ji C.
PMID: 24868470
Int J Hepatol. 2014;2014:513787. doi: 10.1155/2014/513787. Epub 2014 Apr 29.

Alcohol-induced liver disease increasingly contributes to human mortality worldwide. Alcohol-induced endoplasmic reticulum (ER) stress and disruption of cellular protein homeostasis have recently been established as a significant mechanism contributing to liver diseases. The alcohol-induced ER stress occurs not only...

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Ji C. New Insights into the Pathogenesis of Alcohol-Induced ER Stress and Liver Diseases. Int J Hepatol. 2014;2014:513787doi: 10.1155/2014/513787.
Ji, C. (2014). New Insights into the Pathogenesis of Alcohol-Induced ER Stress and Liver Diseases. International journal of hepatology, 2014513787. https://doi.org/10.1155/2014/513787
Ji, Cheng. "New Insights into the Pathogenesis of Alcohol-Induced ER Stress and Liver Diseases." International journal of hepatology vol. 2014 (2014): 513787. doi: https://doi.org/10.1155/2014/513787
Ji C. New Insights into the Pathogenesis of Alcohol-Induced ER Stress and Liver Diseases. Int J Hepatol. 2014;2014:513787. doi: 10.1155/2014/513787. Epub 2014 Apr 29. PMID: 24868470; PMCID: PMC4020372.
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Altered methylation and expression of ER-associated degradation factors in long-term alcohol and constitutive ER stress-induced murine hepatic tumors.

Frontiers in genetics

Han H, Hu J, Lau MY, Feng M, Petrovic LM, Ji C.
PMID: 24198826
Front Genet. 2013 Oct 31;4:224. doi: 10.3389/fgene.2013.00224. eCollection 2013.

Mortality from liver cancer in humans is increasingly attributable to heavy or long-term alcohol consumption. The mechanisms by which alcohol exerts its carcinogenic effect are not well understood. In this study, the role of alcohol-induced endoplasmic reticulum (ER) stress...

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Han H, Hu J, Lau MY, et al. Altered methylation and expression of ER-associated degradation factors in long-term alcohol and constitutive ER stress-induced murine hepatic tumors. Front Genet. 2013;4:224doi: 10.3389/fgene.2013.00224.
Han, H., Hu, J., Lau, M. Y., Feng, M., Petrovic, L. M., & Ji, C. (2013). Altered methylation and expression of ER-associated degradation factors in long-term alcohol and constitutive ER stress-induced murine hepatic tumors. Frontiers in genetics, 4224. https://doi.org/10.3389/fgene.2013.00224
Han, Hui, et al. "Altered methylation and expression of ER-associated degradation factors in long-term alcohol and constitutive ER stress-induced murine hepatic tumors." Frontiers in genetics vol. 4 (2013): 224. doi: https://doi.org/10.3389/fgene.2013.00224
Han H, Hu J, Lau MY, Feng M, Petrovic LM, Ji C. Altered methylation and expression of ER-associated degradation factors in long-term alcohol and constitutive ER stress-induced murine hepatic tumors. Front Genet. 2013 Oct 31;4:224. doi: 10.3389/fgene.2013.00224. eCollection 2013. PMID: 24198826; PMCID: PMC3813967.
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Showing 1 to 5 of 5 entries