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Histone demethylase LSD1 is critical for endochondral ossification during bone fracture healing.

Science advances

Sun J, Feng H, Xing W, Han Y, Suo J, Yallowitz AR, Qian N, Shi Y, Greenblatt MB, Zou W.
PMID: 33148658
Sci Adv. 2020 Nov 04;6(45). doi: 10.1126/sciadv.aaz1410. Print 2020 Nov.

Bone fracture is repaired predominantly through endochondral ossification. However, the regulation of endochondral ossification by key factors during fracture healing remains largely enigmatic. Here, we identify histone modification enzyme LSD1 as a critical factor regulating endochondral ossification during bone...

Cite
Sun J, Feng H, Xing W, et al. Histone demethylase LSD1 is critical for endochondral ossification during bone fracture healing. Sci Adv. 2020;6(45)doi: 10.1126/sciadv.aaz1410.
Sun, J., Feng, H., Xing, W., Han, Y., Suo, J., Yallowitz, A. R., Qian, N., Shi, Y., Greenblatt, M. B., & Zou, W. (2020). Histone demethylase LSD1 is critical for endochondral ossification during bone fracture healing. Science advances, 6(45), . https://doi.org/10.1126/sciadv.aaz1410
Sun, Jun, et al. "Histone demethylase LSD1 is critical for endochondral ossification during bone fracture healing." Science advances vol. 6,45 (2020). doi: https://doi.org/10.1126/sciadv.aaz1410
Sun J, Feng H, Xing W, Han Y, Suo J, Yallowitz AR, Qian N, Shi Y, Greenblatt MB, Zou W. Histone demethylase LSD1 is critical for endochondral ossification during bone fracture healing. Sci Adv. 2020 Nov 04;6(45). doi: 10.1126/sciadv.aaz1410. Print 2020 Nov. PMID: 33148658; PMCID: PMC7673679.
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Osteoclasts are not a source of SLIT3.

Bone research

Li N, Inoue K, Sun J, Niu Y, Lalani S, Yallowitz A, Yang X, Zhang C, Shen R, Zhao B, Xu R, Greenblatt MB.
PMID: 32133214
Bone Res. 2020 Feb 19;8:11. doi: 10.1038/s41413-020-0086-3. eCollection 2020.

The axon guidance cue SLIT3 was identified as an osteoanabolic agent in two recent reports. However, these reports conflict in their nomination of osteoblasts versus osteoclasts as the key producers of skeletal SLIT3 and additionally offer conflicting data on...

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Li N, Inoue K, Sun J, et al. Osteoclasts are not a source of SLIT3. Bone Res. 2020;8:11doi: 10.1038/s41413-020-0086-3.
Li, N., Inoue, K., Sun, J., Niu, Y., Lalani, S., Yallowitz, A., Yang, X., Zhang, C., Shen, R., Zhao, B., Xu, R., & Greenblatt, M. B. (2020). Osteoclasts are not a source of SLIT3. Bone research, 811. https://doi.org/10.1038/s41413-020-0086-3
Li, Na, et al. "Osteoclasts are not a source of SLIT3." Bone research vol. 8 (2020): 11. doi: https://doi.org/10.1038/s41413-020-0086-3
Li N, Inoue K, Sun J, Niu Y, Lalani S, Yallowitz A, Yang X, Zhang C, Shen R, Zhao B, Xu R, Greenblatt MB. Osteoclasts are not a source of SLIT3. Bone Res. 2020 Feb 19;8:11. doi: 10.1038/s41413-020-0086-3. eCollection 2020. PMID: 32133214; PMCID: PMC7031526.
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Commentary.

Clinical chemistry

Greenblatt MB.
PMID: 29592906
Clin Chem. 2018 Apr;64(4):642-643. doi: 10.1373/clinchem.2017.280776.

No abstract available.

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Greenblatt MB. Commentary. Clin Chem. 2018;64(4):642-643doi: 10.1373/clinchem.2017.280776.
Greenblatt, M. B. (2018). Commentary. Clinical chemistry, 64(4), 642-643. https://doi.org/10.1373/clinchem.2017.280776
Greenblatt, Matthew B. "Commentary." Clinical chemistry vol. 64,4 (2018): 642-643. doi: https://doi.org/10.1373/clinchem.2017.280776
Greenblatt MB. Commentary. Clin Chem. 2018 Apr;64(4):642-643. doi: 10.1373/clinchem.2017.280776. PMID: 29592906.
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H3K36 methyltransferase NSD1 regulates chondrocyte differentiation for skeletal development and fracture repair.

Bone research

Shao R, Zhang Z, Xu Z, Ouyang H, Wang L, Ouyang H, Greenblatt M, Chen X, Zou W.
PMID: 34099628
Bone Res. 2021 Jun 07;9(1):30. doi: 10.1038/s41413-021-00148-y.

Chondrocyte differentiation is a critical process for endochondral ossification, which is responsible for long bone development and fracture repair. Considerable progress has been made in understanding the transcriptional control of chondrocyte differentiation; however, epigenetic regulation of chondrocyte differentiation remains...

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Shao R, Zhang Z, Xu Z, et al. H3K36 methyltransferase NSD1 regulates chondrocyte differentiation for skeletal development and fracture repair. Bone Res. 2021;9(1):30doi: 10.1038/s41413-021-00148-y.
Shao, R., Zhang, Z., Xu, Z., Ouyang, H., Wang, L., Ouyang, H., Greenblatt, M., Chen, X., & Zou, W. (2021). H3K36 methyltransferase NSD1 regulates chondrocyte differentiation for skeletal development and fracture repair. Bone research, 9(1), 30. https://doi.org/10.1038/s41413-021-00148-y
Shao, Rui, et al. "H3K36 methyltransferase NSD1 regulates chondrocyte differentiation for skeletal development and fracture repair." Bone research vol. 9,1 (2021): 30. doi: https://doi.org/10.1038/s41413-021-00148-y
Shao R, Zhang Z, Xu Z, Ouyang H, Wang L, Ouyang H, Greenblatt M, Chen X, Zou W. H3K36 methyltransferase NSD1 regulates chondrocyte differentiation for skeletal development and fracture repair. Bone Res. 2021 Jun 07;9(1):30. doi: 10.1038/s41413-021-00148-y. PMID: 34099628; PMCID: PMC8185073.
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RNA-seq Analysis of Peri-Implant Tissue Shows Differences in Immune, Notch, Wnt, and Angiogenesis Pathways in Aged Versus Young Mice.

JBMR plus

Turajane K, Ji G, Chinenov Y, Chao M, Ayturk U, Suhardi VJ, Greenblatt MB, Ivashkiv LB, Bostrom MP, Yang X.
PMID: 34761143
JBMR Plus. 2021 Sep 09;5(11):e10535. doi: 10.1002/jbm4.10535. eCollection 2021 Nov.

The number of total joint replacements (TJRs) in the United States is increasing annually. Cementless implants are intended to improve upon traditional cemented implants by allowing bone growth directly on the surface to improve implant longevity. One major complication...

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Turajane K, Ji G, Chinenov Y, et al. RNA-seq Analysis of Peri-Implant Tissue Shows Differences in Immune, Notch, Wnt, and Angiogenesis Pathways in Aged Versus Young Mice. JBMR Plus. 2021;5(11):e10535doi: 10.1002/jbm4.10535.
Turajane, K., Ji, G., Chinenov, Y., Chao, M., Ayturk, U., Suhardi, V. J., Greenblatt, M. B., Ivashkiv, L. B., Bostrom, M. P., & Yang, X. (2021). RNA-seq Analysis of Peri-Implant Tissue Shows Differences in Immune, Notch, Wnt, and Angiogenesis Pathways in Aged Versus Young Mice. JBMR plus, 5(11), e10535. https://doi.org/10.1002/jbm4.10535
Turajane, Kathleen, et al. "RNA-seq Analysis of Peri-Implant Tissue Shows Differences in Immune, Notch, Wnt, and Angiogenesis Pathways in Aged Versus Young Mice." JBMR plus vol. 5,11 (2021): e10535. doi: https://doi.org/10.1002/jbm4.10535
Turajane K, Ji G, Chinenov Y, Chao M, Ayturk U, Suhardi VJ, Greenblatt MB, Ivashkiv LB, Bostrom MP, Yang X. RNA-seq Analysis of Peri-Implant Tissue Shows Differences in Immune, Notch, Wnt, and Angiogenesis Pathways in Aged Versus Young Mice. JBMR Plus. 2021 Sep 09;5(11):e10535. doi: 10.1002/jbm4.10535. eCollection 2021 Nov. PMID: 34761143; PMCID: PMC8567488.
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Identification of candidate regulators of mandibular bone loss in FcγRIIB.

Scientific reports

Sakunrangsit N, Pholtaisong J, Sucharitakul J, Wanna-Udom S, Prombutara P, Pisitkun P, Leelahavanichkul A, Aporntewan C, Greenblatt MB, Lotinun S.
PMID: 34548536
Sci Rep. 2021 Sep 21;11(1):18726. doi: 10.1038/s41598-021-98108-3.

Patients with systemic lupus erythematosus (SLE) have increased inflammatory cytokines, leading to periodontitis and alveolar bone loss. However, the mechanisms driving this phenomenon are still unknown. Here, we have identified novel therapeutic targets for and mediators of lupus-mediated bone...

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Sakunrangsit N, Pholtaisong J, Sucharitakul J, et al. Identification of candidate regulators of mandibular bone loss in FcγRIIB. Sci Rep. 2021;11(1):18726doi: 10.1038/s41598-021-98108-3.
Sakunrangsit, N., Pholtaisong, J., Sucharitakul, J., Wanna-Udom, S., Prombutara, P., Pisitkun, P., Leelahavanichkul, A., Aporntewan, C., Greenblatt, M. B., & Lotinun, S. (2021). Identification of candidate regulators of mandibular bone loss in FcγRIIB. Scientific reports, 11(1), 18726. https://doi.org/10.1038/s41598-021-98108-3
Sakunrangsit, Nithidol, et al. "Identification of candidate regulators of mandibular bone loss in FcγRIIB." Scientific reports vol. 11,1 (2021): 18726. doi: https://doi.org/10.1038/s41598-021-98108-3
Sakunrangsit N, Pholtaisong J, Sucharitakul J, Wanna-Udom S, Prombutara P, Pisitkun P, Leelahavanichkul A, Aporntewan C, Greenblatt MB, Lotinun S. Identification of candidate regulators of mandibular bone loss in FcγRIIB. Sci Rep. 2021 Sep 21;11(1):18726. doi: 10.1038/s41598-021-98108-3. PMID: 34548536; PMCID: PMC8455620.
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Showing 1 to 6 of 6 entries