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"Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis.

Cell metabolism

Febbraio MA, Karin M.
PMID: 34619076
Cell Metab. 2021 Dec 07;33(12):2316-2328. doi: 10.1016/j.cmet.2021.09.004. Epub 2021 Oct 06.

Glucose and fructose are closely related simple sugars, but fructose has been associated more closely with metabolic disease. Until the 1960s, the major dietary source of fructose was fruit, but subsequently, high-fructose corn syrup (HFCS) became a dominant component...

Cite
Febbraio MA, Karin M. "Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis. Cell Metab. 2021;33(12):2316-2328doi: 10.1016/j.cmet.2021.09.004.
Febbraio, M. A., & Karin, M. (2021). "Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis. Cell metabolism, 33(12), 2316-2328. https://doi.org/10.1016/j.cmet.2021.09.004
Febbraio, Mark A, and Karin, Michael. ""Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis." Cell metabolism vol. 33,12 (2021): 2316-2328. doi: https://doi.org/10.1016/j.cmet.2021.09.004
Febbraio MA, Karin M. "Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis. Cell Metab. 2021 Dec 07;33(12):2316-2328. doi: 10.1016/j.cmet.2021.09.004. Epub 2021 Oct 06. PMID: 34619076; PMCID: PMC8665123.
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"Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis.

Cell metabolism

Febbraio MA, Karin M.
PMID: 34619076
Cell Metab. 2021 Dec 07;33(12):2316-2328. doi: 10.1016/j.cmet.2021.09.004. Epub 2021 Oct 06.

Glucose and fructose are closely related simple sugars, but fructose has been associated more closely with metabolic disease. Until the 1960s, the major dietary source of fructose was fruit, but subsequently, high-fructose corn syrup (HFCS) became a dominant component...

Cite
Febbraio MA, Karin M. "Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis. Cell Metab. 2021;33(12):2316-2328doi: 10.1016/j.cmet.2021.09.004.
Febbraio, M. A., & Karin, M. (2021). "Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis. Cell metabolism, 33(12), 2316-2328. https://doi.org/10.1016/j.cmet.2021.09.004
Febbraio, Mark A, and Karin, Michael. ""Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis." Cell metabolism vol. 33,12 (2021): 2316-2328. doi: https://doi.org/10.1016/j.cmet.2021.09.004
Febbraio MA, Karin M. "Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis. Cell Metab. 2021 Dec 07;33(12):2316-2328. doi: 10.1016/j.cmet.2021.09.004. Epub 2021 Oct 06. PMID: 34619076; PMCID: PMC8665123.
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STARD1 promotes NASH-driven HCC by sustaining the generation of bile acids through the alternative mitochondrial pathway.

Journal of hepatology

Conde de la Rosa L, Garcia-Ruiz C, Vallejo C, Baulies A, Nuñez S, Monte MJ, Marin JJG, Baila-Rueda L, Cenarro A, Civeira F, Fuster J, Garcia-Valdecasas JC, Ferrer J, Karin M, Ribas V, Fernandez-Checa JC.
PMID: 33515644
J Hepatol. 2021 Jun;74(6):1429-1441. doi: 10.1016/j.jhep.2021.01.028. Epub 2021 Jan 27.

BACKGROUND & AIMS: Besides their physiological role in bile formation and fat digestion, bile acids (BAs) synthesised from cholesterol in hepatocytes act as signalling molecules that modulate hepatocellular carcinoma (HCC). Trafficking of cholesterol to mitochondria through steroidogenic acute regulatory...

Cite
Conde de la Rosa L, Garcia-Ruiz C, Vallejo C, et al. STARD1 promotes NASH-driven HCC by sustaining the generation of bile acids through the alternative mitochondrial pathway. J Hepatol. 2021;74(6):1429-1441doi: 10.1016/j.jhep.2021.01.028.
Conde de la Rosa, L., Garcia-Ruiz, C., Vallejo, C., Baulies, A., Nuñez, S., Monte, M. J., Marin, J. J. G., Baila-Rueda, L., Cenarro, A., Civeira, F., Fuster, J., Garcia-Valdecasas, J. C., Ferrer, J., Karin, M., Ribas, V., & Fernandez-Checa, J. C. (2021). STARD1 promotes NASH-driven HCC by sustaining the generation of bile acids through the alternative mitochondrial pathway. Journal of hepatology, 74(6), 1429-1441. https://doi.org/10.1016/j.jhep.2021.01.028
Conde de la Rosa, Laura, et al. "STARD1 promotes NASH-driven HCC by sustaining the generation of bile acids through the alternative mitochondrial pathway." Journal of hepatology vol. 74,6 (2021): 1429-1441. doi: https://doi.org/10.1016/j.jhep.2021.01.028
Conde de la Rosa L, Garcia-Ruiz C, Vallejo C, Baulies A, Nuñez S, Monte MJ, Marin JJG, Baila-Rueda L, Cenarro A, Civeira F, Fuster J, Garcia-Valdecasas JC, Ferrer J, Karin M, Ribas V, Fernandez-Checa JC. STARD1 promotes NASH-driven HCC by sustaining the generation of bile acids through the alternative mitochondrial pathway. J Hepatol. 2021 Jun;74(6):1429-1441. doi: 10.1016/j.jhep.2021.01.028. Epub 2021 Jan 27. PMID: 33515644; PMCID: PMC8573791.
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"Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis.

Cell metabolism

Febbraio MA, Karin M.
PMID: 34619076
Cell Metab. 2021 Dec 07;33(12):2316-2328. doi: 10.1016/j.cmet.2021.09.004. Epub 2021 Oct 06.

Glucose and fructose are closely related simple sugars, but fructose has been associated more closely with metabolic disease. Until the 1960s, the major dietary source of fructose was fruit, but subsequently, high-fructose corn syrup (HFCS) became a dominant component...

Cite
Febbraio MA, Karin M. "Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis. Cell Metab. 2021;33(12):2316-2328doi: 10.1016/j.cmet.2021.09.004.
Febbraio, M. A., & Karin, M. (2021). "Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis. Cell metabolism, 33(12), 2316-2328. https://doi.org/10.1016/j.cmet.2021.09.004
Febbraio, Mark A, and Karin, Michael. ""Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis." Cell metabolism vol. 33,12 (2021): 2316-2328. doi: https://doi.org/10.1016/j.cmet.2021.09.004
Febbraio MA, Karin M. "Sweet death": Fructose as a metabolic toxin that targets the gut-liver axis. Cell Metab. 2021 Dec 07;33(12):2316-2328. doi: 10.1016/j.cmet.2021.09.004. Epub 2021 Oct 06. PMID: 34619076; PMCID: PMC8665123.
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From Liver Fat to Cancer: Perils of the Western Diet.

Cancers

Kim JY, He F, Karin M.
PMID: 33806428
Cancers (Basel). 2021 Mar 04;13(5). doi: 10.3390/cancers13051095.

Hepatocellular carcinoma (HCC), the most common type of primary liver cancer provides the prototypical example of an obesity-related cancer. The obesity epidemic gave rise to an enormous increase in the incidence of non-alcoholic fatty liver disease (NAFLD), a condition...

Cite
Kim JY, He F, Karin M. From Liver Fat to Cancer: Perils of the Western Diet. Cancers (Basel). 2021;13(5)doi: 10.3390/cancers13051095.
Kim, J. Y., He, F., & Karin, M. (2021). From Liver Fat to Cancer: Perils of the Western Diet. Cancers, 13(5), . https://doi.org/10.3390/cancers13051095
Kim, Ju Youn, et al. "From Liver Fat to Cancer: Perils of the Western Diet." Cancers vol. 13,5 (2021). doi: https://doi.org/10.3390/cancers13051095
Kim JY, He F, Karin M. From Liver Fat to Cancer: Perils of the Western Diet. Cancers (Basel). 2021 Mar 04;13(5). doi: 10.3390/cancers13051095. PMID: 33806428; PMCID: PMC7961422.
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STARD1 promotes NASH-driven HCC by sustaining the generation of bile acids through the alternative mitochondrial pathway.

Journal of hepatology

Conde de la Rosa L, Garcia-Ruiz C, Vallejo C, Baulies A, Nuñez S, Monte MJ, Marin JJG, Baila-Rueda L, Cenarro A, Civeira F, Fuster J, Garcia-Valdecasas JC, Ferrer J, Karin M, Ribas V, Fernandez-Checa JC.
PMID: 33515644
J Hepatol. 2021 Jun;74(6):1429-1441. doi: 10.1016/j.jhep.2021.01.028. Epub 2021 Jan 27.

BACKGROUND & AIMS: Besides their physiological role in bile formation and fat digestion, bile acids (BAs) synthesised from cholesterol in hepatocytes act as signalling molecules that modulate hepatocellular carcinoma (HCC). Trafficking of cholesterol to mitochondria through steroidogenic acute regulatory...

Cite
Conde de la Rosa L, Garcia-Ruiz C, Vallejo C, et al. STARD1 promotes NASH-driven HCC by sustaining the generation of bile acids through the alternative mitochondrial pathway. J Hepatol. 2021;74(6):1429-1441doi: 10.1016/j.jhep.2021.01.028.
Conde de la Rosa, L., Garcia-Ruiz, C., Vallejo, C., Baulies, A., Nuñez, S., Monte, M. J., Marin, J. J. G., Baila-Rueda, L., Cenarro, A., Civeira, F., Fuster, J., Garcia-Valdecasas, J. C., Ferrer, J., Karin, M., Ribas, V., & Fernandez-Checa, J. C. (2021). STARD1 promotes NASH-driven HCC by sustaining the generation of bile acids through the alternative mitochondrial pathway. Journal of hepatology, 74(6), 1429-1441. https://doi.org/10.1016/j.jhep.2021.01.028
Conde de la Rosa, Laura, et al. "STARD1 promotes NASH-driven HCC by sustaining the generation of bile acids through the alternative mitochondrial pathway." Journal of hepatology vol. 74,6 (2021): 1429-1441. doi: https://doi.org/10.1016/j.jhep.2021.01.028
Conde de la Rosa L, Garcia-Ruiz C, Vallejo C, Baulies A, Nuñez S, Monte MJ, Marin JJG, Baila-Rueda L, Cenarro A, Civeira F, Fuster J, Garcia-Valdecasas JC, Ferrer J, Karin M, Ribas V, Fernandez-Checa JC. STARD1 promotes NASH-driven HCC by sustaining the generation of bile acids through the alternative mitochondrial pathway. J Hepatol. 2021 Jun;74(6):1429-1441. doi: 10.1016/j.jhep.2021.01.028. Epub 2021 Jan 27. PMID: 33515644; PMCID: PMC8573791.
Copy Download .nbib Format: NLM
Showing 1 to 6 of 6 entries