Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 14 entries
Sorted by: Best Match Show Resources per page
Notch Inhibition Promotes Differentiation of Liver Progenitor Cells into Hepatocytes via .

Stem cells international

Russell JO, Ko S, Monga SP, Shin D.
PMID: 30992706
Stem Cells Int. 2019 Mar 12;2019:8451282. doi: 10.1155/2019/8451282. eCollection 2019.

Liver regeneration after most forms of injury is mediated through the proliferation of hepatocytes. However, when hepatocyte proliferation is impaired, such as during chronic liver disease, liver progenitor cells (LPCs) arising from the biliary epithelial cell (BEC) compartment can...

Cite
Russell JO, Ko S, Monga SP, et al. Notch Inhibition Promotes Differentiation of Liver Progenitor Cells into Hepatocytes via . Stem Cells Int. 2019;2019:8451282doi: 10.1155/2019/8451282.
Russell, J. O., Ko, S., Monga, S. P., & Shin, D. (2019). Notch Inhibition Promotes Differentiation of Liver Progenitor Cells into Hepatocytes via . Stem cells international, 20198451282. https://doi.org/10.1155/2019/8451282
Russell, Jacquelyn O, et al. "Notch Inhibition Promotes Differentiation of Liver Progenitor Cells into Hepatocytes via ." Stem cells international vol. 2019 (2019): 8451282. doi: https://doi.org/10.1155/2019/8451282
Russell JO, Ko S, Monga SP, Shin D. Notch Inhibition Promotes Differentiation of Liver Progenitor Cells into Hepatocytes via . Stem Cells Int. 2019 Mar 12;2019:8451282. doi: 10.1155/2019/8451282. eCollection 2019. PMID: 30992706; PMCID: PMC6434270.
Copy Download .nbib Format: NLM
Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors.

Cell reports

Molina LM, Zhu J, Li Q, Pradhan-Sundd T, Krutsenko Y, Sayed K, Jenkins N, Vats R, Bhushan B, Ko S, Hu S, Poddar M, Singh S, Tao J, Sundd P, Singhi A, Watkins S, Ma X, Benos PV, Feranchak A, Michalopoulos G, Nejak-Bowen K, Watson A, Bell A, Monga SP.
PMID: 34233187
Cell Rep. 2021 Jul 06;36(1):109310. doi: 10.1016/j.celrep.2021.109310.

Yes-associated protein 1 (YAP1) regulates cell plasticity during liver injury, regeneration, and cancer, but its role in liver development is unknown. We detect YAP1 activity in biliary cells and in cells at the hepatobiliary bifurcation in single-cell RNA sequencing...

Cite
Molina LM, Zhu J, Li Q, et al. Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors. Cell Rep. 2021;36(1):109310doi: 10.1016/j.celrep.2021.109310.
Molina, L. M., Zhu, J., Li, Q., Pradhan-Sundd, T., Krutsenko, Y., Sayed, K., Jenkins, N., Vats, R., Bhushan, B., Ko, S., Hu, S., Poddar, M., Singh, S., Tao, J., Sundd, P., Singhi, A., Watkins, S., Ma, X., Benos, P. V., Feranchak, A., Michalopoulos, G., Nejak-Bowen, K., Watson, A., Bell, A., & Monga, S. P. (2021). Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors. Cell reports, 36(1), 109310. https://doi.org/10.1016/j.celrep.2021.109310
Molina, Laura M, et al. "Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors." Cell reports vol. 36,1 (2021): 109310. doi: https://doi.org/10.1016/j.celrep.2021.109310
Molina LM, Zhu J, Li Q, Pradhan-Sundd T, Krutsenko Y, Sayed K, Jenkins N, Vats R, Bhushan B, Ko S, Hu S, Poddar M, Singh S, Tao J, Sundd P, Singhi A, Watkins S, Ma X, Benos PV, Feranchak A, Michalopoulos G, Nejak-Bowen K, Watson A, Bell A, Monga SP. Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors. Cell Rep. 2021 Jul 06;36(1):109310. doi: 10.1016/j.celrep.2021.109310. PMID: 34233187; PMCID: PMC8280534.
Copy Download .nbib Format: NLM
Nuclear factor erythroid 2-related factor 2 and β-Catenin Coactivation in Hepatocellular Cancer: Biological and Therapeutic Implications.

Hepatology (Baltimore, Md.)

Tao J, Krutsenko Y, Moghe A, Singh S, Poddar M, Bell A, Oertel M, Singhi AD, Geller D, Chen X, Lujambio A, Liu S, Monga SP.
PMID: 33529367
Hepatology. 2021 Aug;74(2):741-759. doi: 10.1002/hep.31730. Epub 2021 Jun 21.

BACKGROUND AND AIMS: HCC remains a major unmet clinical need. Although activating catenin beta-1 (CTNNB1) mutations are observed in prominent subsets of HCC cases, these by themselves are insufficient for hepatocarcinogenesis. Coexpression of mutant CTNNB1 with clinically relevant co-occurrence...

Cite
Tao J, Krutsenko Y, Moghe A, et al. Nuclear factor erythroid 2-related factor 2 and β-Catenin Coactivation in Hepatocellular Cancer: Biological and Therapeutic Implications. Hepatology. 2021;74(2):741-759doi: 10.1002/hep.31730.
Tao, J., Krutsenko, Y., Moghe, A., Singh, S., Poddar, M., Bell, A., Oertel, M., Singhi, A. D., Geller, D., Chen, X., Lujambio, A., Liu, S., & Monga, S. P. (2021). Nuclear factor erythroid 2-related factor 2 and β-Catenin Coactivation in Hepatocellular Cancer: Biological and Therapeutic Implications. Hepatology (Baltimore, Md.), 74(2), 741-759. https://doi.org/10.1002/hep.31730
Tao, Junyan, et al. "Nuclear factor erythroid 2-related factor 2 and β-Catenin Coactivation in Hepatocellular Cancer: Biological and Therapeutic Implications." Hepatology (Baltimore, Md.) vol. 74,2 (2021): 741-759. doi: https://doi.org/10.1002/hep.31730
Tao J, Krutsenko Y, Moghe A, Singh S, Poddar M, Bell A, Oertel M, Singhi AD, Geller D, Chen X, Lujambio A, Liu S, Monga SP. Nuclear factor erythroid 2-related factor 2 and β-Catenin Coactivation in Hepatocellular Cancer: Biological and Therapeutic Implications. Hepatology. 2021 Aug;74(2):741-759. doi: 10.1002/hep.31730. Epub 2021 Jun 21. PMID: 33529367; PMCID: PMC8326305.
Copy Download .nbib Format: NLM
Endothelial Wnts regulate β-catenin signaling in murine liver zonation and regeneration: A sequel to the Wnt-Wnt situation.

Hepatology communications

Preziosi M, Okabe H, Poddar M, Singh S, Monga SP.
PMID: 30027142
Hepatol Commun. 2018 Jun 21;2(7):845-860. doi: 10.1002/hep4.1196. eCollection 2018 Jul.

β-Catenin in hepatocytes, under the control of Wnts, regulates pericentral gene expression. It also contributes to liver regeneration (LR) after partial hepatectomy (PH) by regulating cyclin-D1 gene expression as shown in the β-catenin and Wnt coreceptors low-density lipoprotein receptor-related...

Cite
Preziosi M, Okabe H, Poddar M, et al. Endothelial Wnts regulate β-catenin signaling in murine liver zonation and regeneration: A sequel to the Wnt-Wnt situation. Hepatol Commun. 2018;2(7):845-860doi: 10.1002/hep4.1196.
Preziosi, M., Okabe, H., Poddar, M., Singh, S., & Monga, S. P. (2018). Endothelial Wnts regulate β-catenin signaling in murine liver zonation and regeneration: A sequel to the Wnt-Wnt situation. Hepatology communications, 2(7), 845-860. https://doi.org/10.1002/hep4.1196
Preziosi, Morgan, et al. "Endothelial Wnts regulate β-catenin signaling in murine liver zonation and regeneration: A sequel to the Wnt-Wnt situation." Hepatology communications vol. 2,7 (2018): 845-860. doi: https://doi.org/10.1002/hep4.1196
Preziosi M, Okabe H, Poddar M, Singh S, Monga SP. Endothelial Wnts regulate β-catenin signaling in murine liver zonation and regeneration: A sequel to the Wnt-Wnt situation. Hepatol Commun. 2018 Jun 21;2(7):845-860. doi: 10.1002/hep4.1196. eCollection 2018 Jul. PMID: 30027142; PMCID: PMC6049069.
Copy Download .nbib Format: NLM
Nuclear factor erythroid 2-related factor 2 and β-Catenin Coactivation in Hepatocellular Cancer: Biological and Therapeutic Implications.

Hepatology (Baltimore, Md.)

Tao J, Krutsenko Y, Moghe A, Singh S, Poddar M, Bell A, Oertel M, Singhi AD, Geller D, Chen X, Lujambio A, Liu S, Monga SP.
PMID: 33529367
Hepatology. 2021 Aug;74(2):741-759. doi: 10.1002/hep.31730. Epub 2021 Jun 21.

BACKGROUND AND AIMS: HCC remains a major unmet clinical need. Although activating catenin beta-1 (CTNNB1) mutations are observed in prominent subsets of HCC cases, these by themselves are insufficient for hepatocarcinogenesis. Coexpression of mutant CTNNB1 with clinically relevant co-occurrence...

Cite
Tao J, Krutsenko Y, Moghe A, et al. Nuclear factor erythroid 2-related factor 2 and β-Catenin Coactivation in Hepatocellular Cancer: Biological and Therapeutic Implications. Hepatology. 2021;74(2):741-759doi: 10.1002/hep.31730.
Tao, J., Krutsenko, Y., Moghe, A., Singh, S., Poddar, M., Bell, A., Oertel, M., Singhi, A. D., Geller, D., Chen, X., Lujambio, A., Liu, S., & Monga, S. P. (2021). Nuclear factor erythroid 2-related factor 2 and β-Catenin Coactivation in Hepatocellular Cancer: Biological and Therapeutic Implications. Hepatology (Baltimore, Md.), 74(2), 741-759. https://doi.org/10.1002/hep.31730
Tao, Junyan, et al. "Nuclear factor erythroid 2-related factor 2 and β-Catenin Coactivation in Hepatocellular Cancer: Biological and Therapeutic Implications." Hepatology (Baltimore, Md.) vol. 74,2 (2021): 741-759. doi: https://doi.org/10.1002/hep.31730
Tao J, Krutsenko Y, Moghe A, Singh S, Poddar M, Bell A, Oertel M, Singhi AD, Geller D, Chen X, Lujambio A, Liu S, Monga SP. Nuclear factor erythroid 2-related factor 2 and β-Catenin Coactivation in Hepatocellular Cancer: Biological and Therapeutic Implications. Hepatology. 2021 Aug;74(2):741-759. doi: 10.1002/hep.31730. Epub 2021 Jun 21. PMID: 33529367; PMCID: PMC8326305.
Copy Download .nbib Format: NLM
Wnt/-Catenin Signaling and Liver Regeneration: Circuit, Biology, and Opportunities.

Gene expression

Hu S, Monga SP.
PMID: 33472727
Gene Expr. 2021 Jun 11;20(3):189-199. doi: 10.3727/105221621X16111780348794. Epub 2021 Jan 20.

The liver is uniquely bestowed with an ability to regenerate following a surgical or toxicant insult. One of the most researched models to demonstrate the regenerative potential of this organ is the partial hepatectomy model, where two thirds of...

Cite
Hu S, Monga SP. Wnt/-Catenin Signaling and Liver Regeneration: Circuit, Biology, and Opportunities. Gene Expr. 2021;20(3):189-199doi: 10.3727/105221621X16111780348794.
Hu, S., & Monga, S. P. (2021). Wnt/-Catenin Signaling and Liver Regeneration: Circuit, Biology, and Opportunities. Gene expression, 20(3), 189-199. https://doi.org/10.3727/105221621X16111780348794
Hu, Shikai, and Monga, Satdarshan P. "Wnt/-Catenin Signaling and Liver Regeneration: Circuit, Biology, and Opportunities." Gene expression vol. 20,3 (2021): 189-199. doi: https://doi.org/10.3727/105221621X16111780348794
Hu S, Monga SP. Wnt/-Catenin Signaling and Liver Regeneration: Circuit, Biology, and Opportunities. Gene Expr. 2021 Jun 11;20(3):189-199. doi: 10.3727/105221621X16111780348794. Epub 2021 Jan 20. PMID: 33472727; PMCID: PMC8201651.
Copy Download .nbib Format: NLM
Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors.

Cell reports

Molina LM, Zhu J, Li Q, Pradhan-Sundd T, Krutsenko Y, Sayed K, Jenkins N, Vats R, Bhushan B, Ko S, Hu S, Poddar M, Singh S, Tao J, Sundd P, Singhi A, Watkins S, Ma X, Benos PV, Feranchak A, Michalopoulos G, Nejak-Bowen K, Watson A, Bell A, Monga SP.
PMID: 34233187
Cell Rep. 2021 Jul 06;36(1):109310. doi: 10.1016/j.celrep.2021.109310.

Yes-associated protein 1 (YAP1) regulates cell plasticity during liver injury, regeneration, and cancer, but its role in liver development is unknown. We detect YAP1 activity in biliary cells and in cells at the hepatobiliary bifurcation in single-cell RNA sequencing...

Cite
Molina LM, Zhu J, Li Q, et al. Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors. Cell Rep. 2021;36(1):109310doi: 10.1016/j.celrep.2021.109310.
Molina, L. M., Zhu, J., Li, Q., Pradhan-Sundd, T., Krutsenko, Y., Sayed, K., Jenkins, N., Vats, R., Bhushan, B., Ko, S., Hu, S., Poddar, M., Singh, S., Tao, J., Sundd, P., Singhi, A., Watkins, S., Ma, X., Benos, P. V., Feranchak, A., Michalopoulos, G., Nejak-Bowen, K., Watson, A., Bell, A., & Monga, S. P. (2021). Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors. Cell reports, 36(1), 109310. https://doi.org/10.1016/j.celrep.2021.109310
Molina, Laura M, et al. "Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors." Cell reports vol. 36,1 (2021): 109310. doi: https://doi.org/10.1016/j.celrep.2021.109310
Molina LM, Zhu J, Li Q, Pradhan-Sundd T, Krutsenko Y, Sayed K, Jenkins N, Vats R, Bhushan B, Ko S, Hu S, Poddar M, Singh S, Tao J, Sundd P, Singhi A, Watkins S, Ma X, Benos PV, Feranchak A, Michalopoulos G, Nejak-Bowen K, Watson A, Bell A, Monga SP. Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors. Cell Rep. 2021 Jul 06;36(1):109310. doi: 10.1016/j.celrep.2021.109310. PMID: 34233187; PMCID: PMC8280534.
Copy Download .nbib Format: NLM
WNT7B Regulates Cholangiocyte Proliferation and Function During Murine Cholestasis.

Hepatology communications

Kosar K, Cornuet P, Singh S, Lee E, Liu S, Gayden J, Sato T, Freyberg Z, Arteel G, Nejak-Bowen K.
PMID: 34558852
Hepatol Commun. 2021 Dec;5(12):2019-2034. doi: 10.1002/hep4.1784. Epub 2021 Aug 25.

We previously identified an up-regulation of specific Wnt proteins in the cholangiocyte compartment during cholestatic liver injury and found that mice lacking Wnt secretion from hepatocytes and cholangiocytes showed fewer proliferating cholangiocytes and high mortality in response to a...

Cite
Kosar K, Cornuet P, Singh S, et al. WNT7B Regulates Cholangiocyte Proliferation and Function During Murine Cholestasis. Hepatol Commun. 2021;5(12):2019-2034doi: 10.1002/hep4.1784.
Kosar, K., Cornuet, P., Singh, S., Lee, E., Liu, S., Gayden, J., Sato, T., Freyberg, Z., Arteel, G., & Nejak-Bowen, K. (2021). WNT7B Regulates Cholangiocyte Proliferation and Function During Murine Cholestasis. Hepatology communications, 5(12), 2019-2034. https://doi.org/10.1002/hep4.1784
Kosar, Karis, et al. "WNT7B Regulates Cholangiocyte Proliferation and Function During Murine Cholestasis." Hepatology communications vol. 5,12 (2021): 2019-2034. doi: https://doi.org/10.1002/hep4.1784
Kosar K, Cornuet P, Singh S, Lee E, Liu S, Gayden J, Sato T, Freyberg Z, Arteel G, Nejak-Bowen K. WNT7B Regulates Cholangiocyte Proliferation and Function During Murine Cholestasis. Hepatol Commun. 2021 Dec;5(12):2019-2034. doi: 10.1002/hep4.1784. Epub 2021 Aug 25. PMID: 34558852; PMCID: PMC8631094.
Copy Download .nbib Format: NLM
Wnt/beta-catenin signaling in hepatic organogenesis.

Organogenesis

Nejak-Bowen K, Monga SP.
PMID: 19279720
Organogenesis. 2008 Apr;4(2):92-9. doi: 10.4161/org.4.2.5855.

Wnt/beta-catenin signaling has come to the forefront of liver biology in recent years. This pathway regulates key pathophysiological events inherent to the liver including development, regeneration and cancer, by dictating several biological processes such as proliferation, apoptosis, differentiation, adhesion,...

Cite
Nejak-Bowen K, Monga SP. Wnt/beta-catenin signaling in hepatic organogenesis. Organogenesis. 2008;4(2):92-9doi: 10.4161/org.4.2.5855.
Nejak-Bowen, K., & Monga, S. P. (2008). Wnt/beta-catenin signaling in hepatic organogenesis. Organogenesis, 4(2), 92-9. https://doi.org/10.4161/org.4.2.5855
Nejak-Bowen, Kari, and Monga, Satdarshan Ps. "Wnt/beta-catenin signaling in hepatic organogenesis." Organogenesis vol. 4,2 (2008): 92-9. doi: https://doi.org/10.4161/org.4.2.5855
Nejak-Bowen K, Monga SP. Wnt/beta-catenin signaling in hepatic organogenesis. Organogenesis. 2008 Apr;4(2):92-9. doi: 10.4161/org.4.2.5855. PMID: 19279720; PMCID: PMC2634254.
Copy Download .nbib Format: NLM
Yes-Associated Protein Is Crucial for Constitutive Androstane Receptor-Driven Hepatocyte Proliferation But Not for Induction of Drug Metabolism Genes in Mice.

Hepatology (Baltimore, Md.)

Bhushan B, Molina L, Koral K, Stoops JW, Mars WM, Banerjee S, Orr A, Paranjpe S, Monga SP, Locker J, Michalopoulos GK.
PMID: 32794202
Hepatology. 2021 May;73(5):2005-2022. doi: 10.1002/hep.31521.

BACKGROUND AND AIMS: Constitutive androstane receptor (CAR) agonists, such as 1,4-bis [2-(3,5-dichloropyridyloxy)] benzene (TCPOBOP), are known to cause robust hepatocyte proliferation and hepatomegaly in mice along with induction of drug metabolism genes without any associated liver injury. Yes-associated protein...

Cite
Bhushan B, Molina L, Koral K, et al. Yes-Associated Protein Is Crucial for Constitutive Androstane Receptor-Driven Hepatocyte Proliferation But Not for Induction of Drug Metabolism Genes in Mice. Hepatology. 2021;73(5):2005-2022doi: 10.1002/hep.31521.
Bhushan, B., Molina, L., Koral, K., Stoops, J. W., Mars, W. M., Banerjee, S., Orr, A., Paranjpe, S., Monga, S. P., Locker, J., & Michalopoulos, G. K. (2021). Yes-Associated Protein Is Crucial for Constitutive Androstane Receptor-Driven Hepatocyte Proliferation But Not for Induction of Drug Metabolism Genes in Mice. Hepatology (Baltimore, Md.), 73(5), 2005-2022. https://doi.org/10.1002/hep.31521
Bhushan, Bharat, et al. "Yes-Associated Protein Is Crucial for Constitutive Androstane Receptor-Driven Hepatocyte Proliferation But Not for Induction of Drug Metabolism Genes in Mice." Hepatology (Baltimore, Md.) vol. 73,5 (2021): 2005-2022. doi: https://doi.org/10.1002/hep.31521
Bhushan B, Molina L, Koral K, Stoops JW, Mars WM, Banerjee S, Orr A, Paranjpe S, Monga SP, Locker J, Michalopoulos GK. Yes-Associated Protein Is Crucial for Constitutive Androstane Receptor-Driven Hepatocyte Proliferation But Not for Induction of Drug Metabolism Genes in Mice. Hepatology. 2021 May;73(5):2005-2022. doi: 10.1002/hep.31521. PMID: 32794202; PMCID: PMC7885729.
Copy Download .nbib Format: NLM
Yes-Associated Protein Is Crucial for Constitutive Androstane Receptor-Driven Hepatocyte Proliferation But Not for Induction of Drug Metabolism Genes in Mice.

Hepatology (Baltimore, Md.)

Bhushan B, Molina L, Koral K, Stoops JW, Mars WM, Banerjee S, Orr A, Paranjpe S, Monga SP, Locker J, Michalopoulos GK.
PMID: 32794202
Hepatology. 2021 May;73(5):2005-2022. doi: 10.1002/hep.31521.

BACKGROUND AND AIMS: Constitutive androstane receptor (CAR) agonists, such as 1,4-bis [2-(3,5-dichloropyridyloxy)] benzene (TCPOBOP), are known to cause robust hepatocyte proliferation and hepatomegaly in mice along with induction of drug metabolism genes without any associated liver injury. Yes-associated protein...

Cite
Bhushan B, Molina L, Koral K, et al. Yes-Associated Protein Is Crucial for Constitutive Androstane Receptor-Driven Hepatocyte Proliferation But Not for Induction of Drug Metabolism Genes in Mice. Hepatology. 2021;73(5):2005-2022doi: 10.1002/hep.31521.
Bhushan, B., Molina, L., Koral, K., Stoops, J. W., Mars, W. M., Banerjee, S., Orr, A., Paranjpe, S., Monga, S. P., Locker, J., & Michalopoulos, G. K. (2021). Yes-Associated Protein Is Crucial for Constitutive Androstane Receptor-Driven Hepatocyte Proliferation But Not for Induction of Drug Metabolism Genes in Mice. Hepatology (Baltimore, Md.), 73(5), 2005-2022. https://doi.org/10.1002/hep.31521
Bhushan, Bharat, et al. "Yes-Associated Protein Is Crucial for Constitutive Androstane Receptor-Driven Hepatocyte Proliferation But Not for Induction of Drug Metabolism Genes in Mice." Hepatology (Baltimore, Md.) vol. 73,5 (2021): 2005-2022. doi: https://doi.org/10.1002/hep.31521
Bhushan B, Molina L, Koral K, Stoops JW, Mars WM, Banerjee S, Orr A, Paranjpe S, Monga SP, Locker J, Michalopoulos GK. Yes-Associated Protein Is Crucial for Constitutive Androstane Receptor-Driven Hepatocyte Proliferation But Not for Induction of Drug Metabolism Genes in Mice. Hepatology. 2021 May;73(5):2005-2022. doi: 10.1002/hep.31521. PMID: 32794202; PMCID: PMC7885729.
Copy Download .nbib Format: NLM
Commentary: Ghrelin promotes midbrain neural stem cells differentiation to dopaminergic neurons through the Wnt/β-catenin pathway.

Frontiers in cellular neuroscience

Gayden JD, Freyberg Z.
PMID: 32973452
Front Cell Neurosci. 2020 Aug 21;14:248. doi: 10.3389/fncel.2020.00248. eCollection 2020.

No abstract available.

Cite
Gayden JD, Freyberg Z. Commentary: Ghrelin promotes midbrain neural stem cells differentiation to dopaminergic neurons through the Wnt/β-catenin pathway. Front Cell Neurosci. 2020;14:248doi: 10.3389/fncel.2020.00248.
Gayden, J. D., & Freyberg, Z. (2020). Commentary: Ghrelin promotes midbrain neural stem cells differentiation to dopaminergic neurons through the Wnt/β-catenin pathway. Frontiers in cellular neuroscience, 14248. https://doi.org/10.3389/fncel.2020.00248
Gayden, Jenesis D, and Freyberg, Zachary. "Commentary: Ghrelin promotes midbrain neural stem cells differentiation to dopaminergic neurons through the Wnt/β-catenin pathway." Frontiers in cellular neuroscience vol. 14 (2020): 248. doi: https://doi.org/10.3389/fncel.2020.00248
Gayden JD, Freyberg Z. Commentary: Ghrelin promotes midbrain neural stem cells differentiation to dopaminergic neurons through the Wnt/β-catenin pathway. Front Cell Neurosci. 2020 Aug 21;14:248. doi: 10.3389/fncel.2020.00248. eCollection 2020. PMID: 32973452; PMCID: PMC7471653.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 14 entries