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Islet inflammation and ductal proliferation may be linked to increased pancreatitis risk in type 2 diabetes.

JCI insight

Schludi B, Moin ASM, Montemurro C, Gurlo T, Matveyenko AV, Kirakossian D, Dawson DW, Dry SM, Butler PC, Butler AE.
PMID: 28679961
JCI Insight. 2017 Jul 06;2(13). doi: 10.1172/jci.insight.92282. eCollection 2017 Jul 06.

Pancreatitis is more frequent in type 2 diabetes mellitus (T2DM), although the underlying cause is unknown. We tested the hypothesis that ongoing β cell stress and apoptosis in T2DM induces ductal tree proliferation, particularly the pancreatic duct gland (PDG)...

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Schludi B, Moin ASM, Montemurro C, et al. Islet inflammation and ductal proliferation may be linked to increased pancreatitis risk in type 2 diabetes. JCI Insight. 2017;2(13)doi: 10.1172/jci.insight.92282.
Schludi, B., Moin, A. S. M., Montemurro, C., Gurlo, T., Matveyenko, A. V., Kirakossian, D., Dawson, D. W., Dry, S. M., Butler, P. C., & Butler, A. E. (2017). Islet inflammation and ductal proliferation may be linked to increased pancreatitis risk in type 2 diabetes. JCI insight, 2(13), . https://doi.org/10.1172/jci.insight.92282
Schludi, Belinda, et al. "Islet inflammation and ductal proliferation may be linked to increased pancreatitis risk in type 2 diabetes." JCI insight vol. 2,13 (2017). doi: https://doi.org/10.1172/jci.insight.92282
Schludi B, Moin ASM, Montemurro C, Gurlo T, Matveyenko AV, Kirakossian D, Dawson DW, Dry SM, Butler PC, Butler AE. Islet inflammation and ductal proliferation may be linked to increased pancreatitis risk in type 2 diabetes. JCI Insight. 2017 Jul 06;2(13). doi: 10.1172/jci.insight.92282. eCollection 2017 Jul 06. PMID: 28679961; PMCID: PMC5499368.
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Pancreatic Nonhormone Expressing Endocrine Cells in Children With Type 1 Diabetes.

Journal of the Endocrine Society

Md Moin AS, Cory M, Ong A, Choi J, Dhawan S, Butler PC, Butler AE.
PMID: 28782056
J Endocr Soc. 2017 May 01;1(5):385-395. doi: 10.1210/js.2017-00081. Epub 2017 Mar 24.

It has been proposed that the deficit in

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Md Moin AS, Cory M, Ong A, et al. Pancreatic Nonhormone Expressing Endocrine Cells in Children With Type 1 Diabetes. J Endocr Soc. 2017;1(5):385-395doi: 10.1210/js.2017-00081.
Md Moin, A. S., Cory, M., Ong, A., Choi, J., Dhawan, S., Butler, P. C., & Butler, A. E. (2017). Pancreatic Nonhormone Expressing Endocrine Cells in Children With Type 1 Diabetes. Journal of the Endocrine Society, 1(5), 385-395. https://doi.org/10.1210/js.2017-00081
Md Moin, Abu Saleh, et al. "Pancreatic Nonhormone Expressing Endocrine Cells in Children With Type 1 Diabetes." Journal of the Endocrine Society vol. 1,5 (2017): 385-395. doi: https://doi.org/10.1210/js.2017-00081
Md Moin AS, Cory M, Ong A, Choi J, Dhawan S, Butler PC, Butler AE. Pancreatic Nonhormone Expressing Endocrine Cells in Children With Type 1 Diabetes. J Endocr Soc. 2017 May 01;1(5):385-395. doi: 10.1210/js.2017-00081. Epub 2017 Mar 24. PMID: 28782056; PMCID: PMC5542010.
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Recovery of high-quality RNA from laser capture microdissected human and rodent pancreas.

Journal of histotechnology

Butler AE, Matveyenko AV, Kirakossian D, Park J, Gurlo T, Butler PC.
PMID: 27231405
J Histotechnol. 2016;39(2):59-65. doi: 10.1080/01478885.2015.1106073. Epub 2016 May 10.

Laser capture microdissection (LCM) is a powerful method to isolate specific populations of cells for subsequent analysis such as gene expression profiling, for example, microarrays or ribonucleic (RNA)-Seq. This technique has been applied to frozen as well as formalin-fixed,...

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Butler AE, Matveyenko AV, Kirakossian D, et al. Recovery of high-quality RNA from laser capture microdissected human and rodent pancreas. J Histotechnol. 2016;39(2):59-65doi: 10.1080/01478885.2015.1106073.
Butler, A. E., Matveyenko, A. V., Kirakossian, D., Park, J., Gurlo, T., & Butler, P. C. (2016). Recovery of high-quality RNA from laser capture microdissected human and rodent pancreas. Journal of histotechnology, 39(2), 59-65. https://doi.org/10.1080/01478885.2015.1106073
Butler, Alexandra E, et al. "Recovery of high-quality RNA from laser capture microdissected human and rodent pancreas." Journal of histotechnology vol. 39,2 (2016): 59-65. doi: https://doi.org/10.1080/01478885.2015.1106073
Butler AE, Matveyenko AV, Kirakossian D, Park J, Gurlo T, Butler PC. Recovery of high-quality RNA from laser capture microdissected human and rodent pancreas. J Histotechnol. 2016;39(2):59-65. doi: 10.1080/01478885.2015.1106073. Epub 2016 May 10. PMID: 27231405; PMCID: PMC4878453.
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Evaluation of immunohistochemical staining for glucagon in human pancreatic tissue.

Journal of histotechnology

Gurlo T, Butler PC, Butler AE.
PMID: 27182095
J Histotechnol. 2016;39(1):8-16. doi: 10.1179/2046023615Y.0000000013. Epub 2016 Mar 17.

Immunohistochemistry (IHC) and immunofluorescence (IF) staining techniques are important diagnostic tools of anatomic pathology in the clinical setting and widely used analytical tools in research laboratories. In diabetes research, they are routinely used for the assessment of beta- and...

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Gurlo T, Butler PC, Butler AE. Evaluation of immunohistochemical staining for glucagon in human pancreatic tissue. J Histotechnol. 2016;39(1):8-16doi: 10.1179/2046023615Y.0000000013.
Gurlo, T., Butler, P. C., & Butler, A. E. (2016). Evaluation of immunohistochemical staining for glucagon in human pancreatic tissue. Journal of histotechnology, 39(1), 8-16. https://doi.org/10.1179/2046023615Y.0000000013
Gurlo, Tatyana, et al. "Evaluation of immunohistochemical staining for glucagon in human pancreatic tissue." Journal of histotechnology vol. 39,1 (2016): 8-16. doi: https://doi.org/10.1179/2046023615Y.0000000013
Gurlo T, Butler PC, Butler AE. Evaluation of immunohistochemical staining for glucagon in human pancreatic tissue. J Histotechnol. 2016;39(1):8-16. doi: 10.1179/2046023615Y.0000000013. Epub 2016 Mar 17. PMID: 27182095; PMCID: PMC4863713.
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Down Syndrome-Associated Diabetes Is Not Due To a Congenital Deficiency in .

Journal of the Endocrine Society

Butler AE, Sacks W, Rizza RA, Butler PC.
PMID: 28782055
J Endocr Soc. 2017 Jan 01;1(1):39-45. doi: 10.1210/js.2016-1042. Epub 2017 Jan 12.

AIMS/HYPOTHESIS: We sought to establish whether the increased incidence of diabetes associated with Down syndrome was due to a congenital deficit in METHODS: The pancreas was obtained at autopsy from nondiabetic subjects with Down syndrome (n = 29) and...

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Butler AE, Sacks W, Rizza RA, et al. Down Syndrome-Associated Diabetes Is Not Due To a Congenital Deficiency in . J Endocr Soc. 2017;1(1):39-45doi: 10.1210/js.2016-1042.
Butler, A. E., Sacks, W., Rizza, R. A., & Butler, P. C. (2017). Down Syndrome-Associated Diabetes Is Not Due To a Congenital Deficiency in . Journal of the Endocrine Society, 1(1), 39-45. https://doi.org/10.1210/js.2016-1042
Butler, Alexandra E, et al. "Down Syndrome-Associated Diabetes Is Not Due To a Congenital Deficiency in ." Journal of the Endocrine Society vol. 1,1 (2017): 39-45. doi: https://doi.org/10.1210/js.2016-1042
Butler AE, Sacks W, Rizza RA, Butler PC. Down Syndrome-Associated Diabetes Is Not Due To a Congenital Deficiency in . J Endocr Soc. 2017 Jan 01;1(1):39-45. doi: 10.1210/js.2016-1042. Epub 2017 Jan 12. PMID: 28782055; PMCID: PMC5542008.
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Showing 1 to 5 of 5 entries