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Febuxostat attenuates ER stress mediated kidney injury in a rat model of hyperuricemic nephropathy.

Oncotarget

He L, Fan Y, Xiao W, Chen T, Wen J, Dong Y, Wang Y, Li S, Xue R, Zheng L, He JC, Wang N.
PMID: 29340054
Oncotarget. 2017 Nov 30;8(67):111295-111308. doi: 10.18632/oncotarget.22784. eCollection 2017 Dec 19.

Hyperuricemia contributes to kidney tubular injury and kidney fibrosis. However, the underlying mechanism remains unclear. Here we examined the role of RTN1A, a novel endoplasmic reticulum (ER)-associated protein and ER stress in hyperuricemic nephropathy. We first found the expression...

Cite
He L, Fan Y, Xiao W, et al. Febuxostat attenuates ER stress mediated kidney injury in a rat model of hyperuricemic nephropathy. Oncotarget. 2017;8(67):111295-111308doi: 10.18632/oncotarget.22784.
He, L., Fan, Y., Xiao, W., Chen, T., Wen, J., Dong, Y., Wang, Y., Li, S., Xue, R., Zheng, L., He, J. C., & Wang, N. (2017). Febuxostat attenuates ER stress mediated kidney injury in a rat model of hyperuricemic nephropathy. Oncotarget, 8(67), 111295-111308. https://doi.org/10.18632/oncotarget.22784
He, Li, et al. "Febuxostat attenuates ER stress mediated kidney injury in a rat model of hyperuricemic nephropathy." Oncotarget vol. 8,67 (2017): 111295-111308. doi: https://doi.org/10.18632/oncotarget.22784
He L, Fan Y, Xiao W, Chen T, Wen J, Dong Y, Wang Y, Li S, Xue R, Zheng L, He JC, Wang N. Febuxostat attenuates ER stress mediated kidney injury in a rat model of hyperuricemic nephropathy. Oncotarget. 2017 Nov 30;8(67):111295-111308. doi: 10.18632/oncotarget.22784. eCollection 2017 Dec 19. PMID: 29340054; PMCID: PMC5762322.
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The beneficial role of retinoids in glomerular disease.

Frontiers in medicine

Mallipattu SK, He JC.
PMID: 25853135
Front Med (Lausanne). 2015 Mar 23;2:16. doi: 10.3389/fmed.2015.00016. eCollection 2015.

The primary etiology of CKD is a direct consequence of initial dysfunction and injury of the glomerulus, the main filtration system. Podocytes are terminally differentiated epithelial cells in the glomerulus, whose major function is the maintenance of this renal...

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Mallipattu SK, He JC. The beneficial role of retinoids in glomerular disease. Front Med (Lausanne). 2015;2:16doi: 10.3389/fmed.2015.00016.
Mallipattu, S. K., & He, J. C. (2015). The beneficial role of retinoids in glomerular disease. Frontiers in medicine, 216. https://doi.org/10.3389/fmed.2015.00016
Mallipattu, Sandeep K, and He, John Cijiang. "The beneficial role of retinoids in glomerular disease." Frontiers in medicine vol. 2 (2015): 16. doi: https://doi.org/10.3389/fmed.2015.00016
Mallipattu SK, He JC. The beneficial role of retinoids in glomerular disease. Front Med (Lausanne). 2015 Mar 23;2:16. doi: 10.3389/fmed.2015.00016. eCollection 2015. PMID: 25853135; PMCID: PMC4370041.
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SIRT1 Is a Potential Drug Target for Treatment of Diabetic Kidney Disease.

Frontiers in endocrinology

Zhong Y, Lee K, He JC.
PMID: 30386303
Front Endocrinol (Lausanne). 2018 Oct 17;9:624. doi: 10.3389/fendo.2018.00624. eCollection 2018.

Multiple studies have demonstrated a critical role of Sirtuin-1 (SIRT1) deacetylase in protecting kidney cells from cellular stresses. A protective role of SIRT1 has been reported in both podocytes and renal tubular cells in multiple kidney disease settings, including...

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Zhong Y, Lee K, He JC. SIRT1 Is a Potential Drug Target for Treatment of Diabetic Kidney Disease. Front Endocrinol (Lausanne). 2018;9:624doi: 10.3389/fendo.2018.00624.
Zhong, Y., Lee, K., & He, J. C. (2018). SIRT1 Is a Potential Drug Target for Treatment of Diabetic Kidney Disease. Frontiers in endocrinology, 9624. https://doi.org/10.3389/fendo.2018.00624
Zhong, Yifei, et al. "SIRT1 Is a Potential Drug Target for Treatment of Diabetic Kidney Disease." Frontiers in endocrinology vol. 9 (2018): 624. doi: https://doi.org/10.3389/fendo.2018.00624
Zhong Y, Lee K, He JC. SIRT1 Is a Potential Drug Target for Treatment of Diabetic Kidney Disease. Front Endocrinol (Lausanne). 2018 Oct 17;9:624. doi: 10.3389/fendo.2018.00624. eCollection 2018. PMID: 30386303; PMCID: PMC6199382.
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