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Catechins and Sialic Acid Attenuate Helicobacter pylori-Triggered Epithelial Caspase-1 Activity and Eradicate Helicobacter pylori Infection.

Evidence-based complementary and alternative medicine : eCAM

Yang JC, Yang HC, Shun CT, Wang TH, Chien CT, Kao JY.
PMID: 23653660
Evid Based Complement Alternat Med. 2013;2013:248585. doi: 10.1155/2013/248585. Epub 2013 Apr 11.

The inflammasome/caspase-1 signaling pathway in immune cells plays a critical role in bacterial pathogenesis; however, the regulation of this pathway in the gastric epithelium during Helicobacter pylori infection is yet to be elucidated. Here, we investigated the effect of...

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Yang JC, Yang HC, Shun CT, et al. Catechins and Sialic Acid Attenuate Helicobacter pylori-Triggered Epithelial Caspase-1 Activity and Eradicate Helicobacter pylori Infection. Evid Based Complement Alternat Med. 2013;2013:248585doi: 10.1155/2013/248585.
Yang, J. C., Yang, H. C., Shun, C. T., Wang, T. H., Chien, C. T., & Kao, J. Y. (2013). Catechins and Sialic Acid Attenuate Helicobacter pylori-Triggered Epithelial Caspase-1 Activity and Eradicate Helicobacter pylori Infection. Evidence-based complementary and alternative medicine : eCAM, 2013248585. https://doi.org/10.1155/2013/248585
Yang, Jyh-Chin, et al. "Catechins and Sialic Acid Attenuate Helicobacter pylori-Triggered Epithelial Caspase-1 Activity and Eradicate Helicobacter pylori Infection." Evidence-based complementary and alternative medicine : eCAM vol. 2013 (2013): 248585. doi: https://doi.org/10.1155/2013/248585
Yang JC, Yang HC, Shun CT, Wang TH, Chien CT, Kao JY. Catechins and Sialic Acid Attenuate Helicobacter pylori-Triggered Epithelial Caspase-1 Activity and Eradicate Helicobacter pylori Infection. Evid Based Complement Alternat Med. 2013;2013:248585. doi: 10.1155/2013/248585. Epub 2013 Apr 11. PMID: 23653660; PMCID: PMC3638598.
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Functional Characterization of Inflammatory Bowel Disease-Associated Gut Dysbiosis in Gnotobiotic Mice.

Cellular and molecular gastroenterology and hepatology

Nagao-Kitamoto H, Shreiner AB, Gillilland MG, Kitamoto S, Ishii C, Hirayama A, Kuffa P, El-Zaatari M, Grasberger H, Seekatz AM, Higgins PD, Young VB, Fukuda S, Kao JY, Kamada N.
PMID: 27795980
Cell Mol Gastroenterol Hepatol. 2016 Mar 03;2(4):468-481. doi: 10.1016/j.jcmgh.2016.02.003. eCollection 2016 Jul.

BACKGROUND & AIMS: Gut dysbiosis is closely involved in the pathogenesis of inflammatory bowel disease (IBD). However, it remains unclear whether IBD-associated gut dysbiosis contributes to disease pathogenesis or is merely secondary to intestinal inflammation. We established a humanized...

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Nagao-Kitamoto H, Shreiner AB, Gillilland MG, et al. Functional Characterization of Inflammatory Bowel Disease-Associated Gut Dysbiosis in Gnotobiotic Mice. Cell Mol Gastroenterol Hepatol. 2016;2(4):468-481doi: 10.1016/j.jcmgh.2016.02.003.
Nagao-Kitamoto, H., Shreiner, A. B., Gillilland, M. G., Kitamoto, S., Ishii, C., Hirayama, A., Kuffa, P., El-Zaatari, M., Grasberger, H., Seekatz, A. M., Higgins, P. D., Young, V. B., Fukuda, S., Kao, J. Y., & Kamada, N. (2016). Functional Characterization of Inflammatory Bowel Disease-Associated Gut Dysbiosis in Gnotobiotic Mice. Cellular and molecular gastroenterology and hepatology, 2(4), 468-481. https://doi.org/10.1016/j.jcmgh.2016.02.003
Nagao-Kitamoto, Hiroko, et al. "Functional Characterization of Inflammatory Bowel Disease-Associated Gut Dysbiosis in Gnotobiotic Mice." Cellular and molecular gastroenterology and hepatology vol. 2,4 (2016): 468-481. doi: https://doi.org/10.1016/j.jcmgh.2016.02.003
Nagao-Kitamoto H, Shreiner AB, Gillilland MG, Kitamoto S, Ishii C, Hirayama A, Kuffa P, El-Zaatari M, Grasberger H, Seekatz AM, Higgins PD, Young VB, Fukuda S, Kao JY, Kamada N. Functional Characterization of Inflammatory Bowel Disease-Associated Gut Dysbiosis in Gnotobiotic Mice. Cell Mol Gastroenterol Hepatol. 2016 Mar 03;2(4):468-481. doi: 10.1016/j.jcmgh.2016.02.003. eCollection 2016 Jul. PMID: 27795980; PMCID: PMC5042563.
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