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The effect of whole-body radiation on blood levels of gastrointestinal peptides in the rat.

International journal of clinical and experimental medicine

Katanyutanon S, Wu R, Wang P.
PMID: 19079678
Int J Clin Exp Med. 2008;1(4):332-7. Epub 2008 Oct 15.

Radiation-induced injury may occur in various incidents as well as the terrorist radiation exposure scenario. The digestive tract is among the most radiosensitive organs in the body and its function, which is partly regulated by gastrointestinal (GI) peptides, can...

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Katanyutanon S, Wu R, Wang P. The effect of whole-body radiation on blood levels of gastrointestinal peptides in the rat. Int J Clin Exp Med. 2008;1(4):332-7
Katanyutanon, S., Wu, R., & Wang, P. (2008). The effect of whole-body radiation on blood levels of gastrointestinal peptides in the rat. International journal of clinical and experimental medicine, 1(4), 332-7.
Katanyutanon, Sakdhisapol, et al. "The effect of whole-body radiation on blood levels of gastrointestinal peptides in the rat." International journal of clinical and experimental medicine vol. 1,4 (2008): 332-7.
Katanyutanon S, Wu R, Wang P. The effect of whole-body radiation on blood levels of gastrointestinal peptides in the rat. Int J Clin Exp Med. 2008;1(4):332-7. Epub 2008 Oct 15. PMID: 19079678; PMCID: PMC2596326.
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Sepsis-induced inflammation is exacerbated in an animal model of type 2 diabetes.

International journal of clinical and experimental medicine

Jacob A, Steinberg ML, Yang J, Dong W, Ji Y, Wang P.
PMID: 19079684
Int J Clin Exp Med. 2008;1(1):22-31. Epub 2008 Jan 10.

Hyperglycemia is common in critically ill patients and pronounced hyperglycemia may lead to complications which include severe infections, polyneuropathy, multiple organ failure and death in such patients. Sustained hyperglycemia is generally observed in patients with Type 2 diabetes. To...

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Jacob A, Steinberg ML, Yang J, et al. Sepsis-induced inflammation is exacerbated in an animal model of type 2 diabetes. Int J Clin Exp Med. 2008;1(1):22-31
Jacob, A., Steinberg, M. L., Yang, J., Dong, W., Ji, Y., & Wang, P. (2008). Sepsis-induced inflammation is exacerbated in an animal model of type 2 diabetes. International journal of clinical and experimental medicine, 1(1), 22-31.
Jacob, Asha, et al. "Sepsis-induced inflammation is exacerbated in an animal model of type 2 diabetes." International journal of clinical and experimental medicine vol. 1,1 (2008): 22-31.
Jacob A, Steinberg ML, Yang J, Dong W, Ji Y, Wang P. Sepsis-induced inflammation is exacerbated in an animal model of type 2 diabetes. Int J Clin Exp Med. 2008;1(1):22-31. Epub 2008 Jan 10. PMID: 19079684; PMCID: PMC2596333.
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The role of hepatic cytochrome P-450 in sepsis.

International journal of clinical and experimental medicine

Jacob A, Zhou M, Wu R, Wang P.
PMID: 19918313
Int J Clin Exp Med. 2009 Aug 25;2(3):203-11.

Severe sepsis is a common, expensive, and fatal condition with as many deaths annually as those from acute myocardial infarctions. The average cost per case seems to exceed $22,000. The increased morbidity and mortality attributed to sepsis could be...

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Jacob A, Zhou M, Wu R, et al. The role of hepatic cytochrome P-450 in sepsis. Int J Clin Exp Med. 2009;2(3):203-11
Jacob, A., Zhou, M., Wu, R., & Wang, P. (2009). The role of hepatic cytochrome P-450 in sepsis. International journal of clinical and experimental medicine, 2(3), 203-11.
Jacob, Asha, et al. "The role of hepatic cytochrome P-450 in sepsis." International journal of clinical and experimental medicine vol. 2,3 (2009): 203-11.
Jacob A, Zhou M, Wu R, Wang P. The role of hepatic cytochrome P-450 in sepsis. Int J Clin Exp Med. 2009 Aug 25;2(3):203-11. PMID: 19918313; PMCID: PMC2770183.
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Suppression of PGC-1alpha by Ethanol: Implications of Its Role in Alcohol Induced Liver Injury.

International journal of clinical and experimental medicine

Chaung WW, Jacob A, Ji Y, Wang P.
PMID: 19079670
Int J Clin Exp Med. 2008;1(2):161-70. Epub 2008 Mar 21.

Ethanol has been known to cause injury to the liver and other tissues; however the molecular factors responsible for alcohol induced liver injury has not been fully understood. Recent studies indicate that reactive oxygen species (ROS) may play an...

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Chaung WW, Jacob A, Ji Y, et al. Suppression of PGC-1alpha by Ethanol: Implications of Its Role in Alcohol Induced Liver Injury. Int J Clin Exp Med. 2008;1(2):161-70
Chaung, W. W., Jacob, A., Ji, Y., & Wang, P. (2008). Suppression of PGC-1alpha by Ethanol: Implications of Its Role in Alcohol Induced Liver Injury. International journal of clinical and experimental medicine, 1(2), 161-70.
Chaung, Wayne W, et al. "Suppression of PGC-1alpha by Ethanol: Implications of Its Role in Alcohol Induced Liver Injury." International journal of clinical and experimental medicine vol. 1,2 (2008): 161-70.
Chaung WW, Jacob A, Ji Y, Wang P. Suppression of PGC-1alpha by Ethanol: Implications of Its Role in Alcohol Induced Liver Injury. Int J Clin Exp Med. 2008;1(2):161-70. Epub 2008 Mar 21. PMID: 19079670; PMCID: PMC2596315.
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Mechanism of the inhibitory effect of ghrelin in sepsis.

Hepatic medicine : evidence and research

Jacob A, Wu R, Zhou M, Coppa GF, Wang P.
PMID: 24367207
Hepat Med. 2010 Feb 23;2:33-8. doi: 10.2147/hmer.s7187.

Sepsis and septic shock are the leading causes of death in intensive care units. Approximately 40%-70% of the mortality is associated with severe sepsis and septic shock. Systemic antibiotic usage, surgical intervention, aggressive fluid resuscitation and careful monitoring are...

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Jacob A, Wu R, Zhou M, et al. Mechanism of the inhibitory effect of ghrelin in sepsis. Hepat Med. 2010;2:33-8doi: 10.2147/hmer.s7187.
Jacob, A., Wu, R., Zhou, M., Coppa, G. F., & Wang, P. (2010). Mechanism of the inhibitory effect of ghrelin in sepsis. Hepatic medicine : evidence and research, 233-8. https://doi.org/10.2147/hmer.s7187
Jacob, Asha, et al. "Mechanism of the inhibitory effect of ghrelin in sepsis." Hepatic medicine : evidence and research vol. 2 (2010): 33-8. doi: https://doi.org/10.2147/hmer.s7187
Jacob A, Wu R, Zhou M, Coppa GF, Wang P. Mechanism of the inhibitory effect of ghrelin in sepsis. Hepat Med. 2010 Feb 23;2:33-8. doi: 10.2147/hmer.s7187. PMID: 24367207; PMCID: PMC3846870.
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Mechanism of the Anti-inflammatory Effect of Curcumin: PPAR-gamma Activation.

PPAR research

Jacob A, Wu R, Zhou M, Wang P.
PMID: 18274631
PPAR Res. 2007;2007:89369. doi: 10.1155/2007/89369.

Curcumin, the phytochemical component in turmeric, is used as a dietary spice and a topical ointment for the treatment of inflammation in India for centuries. Curcumin (diferuloylmethane) is relatively insoluble in water, but dissolves in acetone, dimethylsulphoxide, and ethanol....

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Jacob A, Wu R, Zhou M, et al. Mechanism of the Anti-inflammatory Effect of Curcumin: PPAR-gamma Activation. PPAR Res. 2007;2007:89369doi: 10.1155/2007/89369.
Jacob, A., Wu, R., Zhou, M., & Wang, P. (2007). Mechanism of the Anti-inflammatory Effect of Curcumin: PPAR-gamma Activation. PPAR research, 200789369. https://doi.org/10.1155/2007/89369
Jacob, Asha, et al. "Mechanism of the Anti-inflammatory Effect of Curcumin: PPAR-gamma Activation." PPAR research vol. 2007 (2007): 89369. doi: https://doi.org/10.1155/2007/89369
Jacob A, Wu R, Zhou M, Wang P. Mechanism of the Anti-inflammatory Effect of Curcumin: PPAR-gamma Activation. PPAR Res. 2007;2007:89369. doi: 10.1155/2007/89369. PMID: 18274631; PMCID: PMC2234255.
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Showing 1 to 6 of 6 entries