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Myocardial electrotonic response to submaximal exercise in dogs with healed myocardial infarctions: evidence for β-adrenoceptor mediated enhanced coupling during exercise testing.

Frontiers in physiology

Del Rio CL, Clymer BD, Billman GE.
PMID: 25698976
Front Physiol. 2015 Feb 05;6:25. doi: 10.3389/fphys.2015.00025. eCollection 2015.

INTRODUCTION: Autonomic neural activation during cardiac stress testing is an established risk-stratification tool in post-myocardial infarction (MI) patients. However, autonomic activation can also modulate myocardial electrotonic coupling, a known factor to contribute to the genesis of arrhythmias. The present...

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Del Rio CL, Clymer BD, Billman GE. Myocardial electrotonic response to submaximal exercise in dogs with healed myocardial infarctions: evidence for β-adrenoceptor mediated enhanced coupling during exercise testing. Front Physiol. 2015;6:25doi: 10.3389/fphys.2015.00025.
Del Rio, C. L., Clymer, B. D., & Billman, G. E. (2015). Myocardial electrotonic response to submaximal exercise in dogs with healed myocardial infarctions: evidence for β-adrenoceptor mediated enhanced coupling during exercise testing. Frontiers in physiology, 625. https://doi.org/10.3389/fphys.2015.00025
Del Rio, Carlos L, et al. "Myocardial electrotonic response to submaximal exercise in dogs with healed myocardial infarctions: evidence for β-adrenoceptor mediated enhanced coupling during exercise testing." Frontiers in physiology vol. 6 (2015): 25. doi: https://doi.org/10.3389/fphys.2015.00025
Del Rio CL, Clymer BD, Billman GE. Myocardial electrotonic response to submaximal exercise in dogs with healed myocardial infarctions: evidence for β-adrenoceptor mediated enhanced coupling during exercise testing. Front Physiol. 2015 Feb 05;6:25. doi: 10.3389/fphys.2015.00025. eCollection 2015. PMID: 25698976; PMCID: PMC4318283.
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Endurance exercise training reduces cardiac sodium/calcium exchanger expression in animals susceptible to ventricular fibrillation.

Frontiers in physiology

Kukielka M, Holycross BJ, Billman GE.
PMID: 21423413
Front Physiol. 2011 Feb 14;2:3. doi: 10.3389/fphys.2011.00003. eCollection 2011.

AIM: Increased sodium/calcium exchanger activity (NCX1, an important regulator of cardiomyocyte cystolic calcium) may provoke arrhythmias. Exercise training can decrease NCX1 expression in animals with heart failure improving cytosolic calcium regulation, and could thereby reduce the risk for ventricular...

Cite
Kukielka M, Holycross BJ, Billman GE. Endurance exercise training reduces cardiac sodium/calcium exchanger expression in animals susceptible to ventricular fibrillation. Front Physiol. 2011;2:3doi: 10.3389/fphys.2011.00003.
Kukielka, M., Holycross, B. J., & Billman, G. E. (2011). Endurance exercise training reduces cardiac sodium/calcium exchanger expression in animals susceptible to ventricular fibrillation. Frontiers in physiology, 23. https://doi.org/10.3389/fphys.2011.00003
Kukielka, Monica, et al. "Endurance exercise training reduces cardiac sodium/calcium exchanger expression in animals susceptible to ventricular fibrillation." Frontiers in physiology vol. 2 (2011): 3. doi: https://doi.org/10.3389/fphys.2011.00003
Kukielka M, Holycross BJ, Billman GE. Endurance exercise training reduces cardiac sodium/calcium exchanger expression in animals susceptible to ventricular fibrillation. Front Physiol. 2011 Feb 14;2:3. doi: 10.3389/fphys.2011.00003. eCollection 2011. PMID: 21423413; PMCID: PMC3059610.
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