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Angiotensin II-Superoxide Signaling and Arterial Baroreceptor Function in Type-1 Diabetes Mellitus.

Journal of diabetes & metabolism

Li YL.
PMID: 24567847
J Diabetes Metab. 2013;1-6. doi: 10.4172/2155-6156.S12-001.

Diabetes is a major world health problem. Growing evidence from both clinical trials and animal experiments has clearly confirmed that arterial baroreflex dysfunction is a feature of type 1 diabetes, which links to prognosis and mortality of the type...

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Li YL. Angiotensin II-Superoxide Signaling and Arterial Baroreceptor Function in Type-1 Diabetes Mellitus. J Diabetes Metab. 2013;1-6doi: 10.4172/2155-6156.S12-001.
Li, Y. L. (2013). Angiotensin II-Superoxide Signaling and Arterial Baroreceptor Function in Type-1 Diabetes Mellitus. Journal of diabetes & metabolism, 1-6. https://doi.org/10.4172/2155-6156.S12-001
Li, Yu-Long. "Angiotensin II-Superoxide Signaling and Arterial Baroreceptor Function in Type-1 Diabetes Mellitus." Journal of diabetes & metabolism vol. (2013): 1-6. doi: https://doi.org/10.4172/2155-6156.S12-001
Li YL. Angiotensin II-Superoxide Signaling and Arterial Baroreceptor Function in Type-1 Diabetes Mellitus. J Diabetes Metab. 2013;1-6. doi: 10.4172/2155-6156.S12-001. PMID: 24567847; PMCID: PMC3932051.
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Elevated angiotensin II in rat nodose ganglia primes diabetes-blunted arterial baroreflex sensitivity: involvement of NADPH oxidase-derived superoxide.

Journal of diabetes & metabolism

Li YL.
PMID: 22308229
J Diabetes Metab. 2011 Sep 08;2(6). doi: 10.4172/2155-6156.1000135.

Clinical trials and experimental animal studies have confirmed the contribution of arterial baroreflex impairment in causing excess morbidity and mortality in type-1 diabetes. Our previous study has shown that angiotensin II (Ang II)-NADPH oxidase-superoxide signaling is associated with the...

Cite
Li YL. Elevated angiotensin II in rat nodose ganglia primes diabetes-blunted arterial baroreflex sensitivity: involvement of NADPH oxidase-derived superoxide. J Diabetes Metab. 2011;2(6)doi: 10.4172/2155-6156.1000135.
Li, Y. L. (2011). Elevated angiotensin II in rat nodose ganglia primes diabetes-blunted arterial baroreflex sensitivity: involvement of NADPH oxidase-derived superoxide. Journal of diabetes & metabolism, 2(6), . https://doi.org/10.4172/2155-6156.1000135
Li, Yu-Long. "Elevated angiotensin II in rat nodose ganglia primes diabetes-blunted arterial baroreflex sensitivity: involvement of NADPH oxidase-derived superoxide." Journal of diabetes & metabolism vol. 2,6 (2011). doi: https://doi.org/10.4172/2155-6156.1000135
Li YL. Elevated angiotensin II in rat nodose ganglia primes diabetes-blunted arterial baroreflex sensitivity: involvement of NADPH oxidase-derived superoxide. J Diabetes Metab. 2011 Sep 08;2(6). doi: 10.4172/2155-6156.1000135. PMID: 22308229; PMCID: PMC3269316.
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Angiotensin II-superoxide-NFκB signaling and aortic baroreceptor dysfunction in chronic heart failure.

Frontiers in neuroscience

Zhang D, Muelleman RL, Li YL.
PMID: 26528122
Front Neurosci. 2015 Oct 16;9:382. doi: 10.3389/fnins.2015.00382. eCollection 2015.

Chronic heart failure (CHF) affects approximately 5.7 million people in the United States. Increasing evidence from both clinical and experimental studies indicates that the sensitivity of arterial baroreflex is blunted in the CHF state, which is a predictive risk...

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Zhang D, Muelleman RL, Li YL. Angiotensin II-superoxide-NFκB signaling and aortic baroreceptor dysfunction in chronic heart failure. Front Neurosci. 2015;9:382doi: 10.3389/fnins.2015.00382.
Zhang, D., Muelleman, R. L., & Li, Y. L. (2015). Angiotensin II-superoxide-NFκB signaling and aortic baroreceptor dysfunction in chronic heart failure. Frontiers in neuroscience, 9382. https://doi.org/10.3389/fnins.2015.00382
Zhang, Dongze, et al. "Angiotensin II-superoxide-NFκB signaling and aortic baroreceptor dysfunction in chronic heart failure." Frontiers in neuroscience vol. 9 (2015): 382. doi: https://doi.org/10.3389/fnins.2015.00382
Zhang D, Muelleman RL, Li YL. Angiotensin II-superoxide-NFκB signaling and aortic baroreceptor dysfunction in chronic heart failure. Front Neurosci. 2015 Oct 16;9:382. doi: 10.3389/fnins.2015.00382. eCollection 2015. PMID: 26528122; PMCID: PMC4607814.
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Macrophage depletion in stellate ganglia alleviates cardiac sympathetic overactivation and ventricular arrhythmogenesis by attenuating neuroinflammation in heart failure.

Basic research in cardiology

Zhang D, Hu W, Tu H, Hackfort BT, Duan B, Xiong W, Wadman MC, Li YL.
PMID: 33884509
Basic Res Cardiol. 2021 Apr 21;116(1):28. doi: 10.1007/s00395-021-00871-x.

Cardiac sympathetic overactivation is involved in arrhythmogenesis in patients with chronic heart failure (CHF). Inflammatory infiltration in the stellate ganglion (SG) is a critical factor for cardiac sympathoexcitation in patients with ventricular arrhythmias. This study aims to investigate if...

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Zhang D, Hu W, Tu H, et al. Macrophage depletion in stellate ganglia alleviates cardiac sympathetic overactivation and ventricular arrhythmogenesis by attenuating neuroinflammation in heart failure. Basic Res Cardiol. 2021;116(1):28doi: 10.1007/s00395-021-00871-x.
Zhang, D., Hu, W., Tu, H., Hackfort, B. T., Duan, B., Xiong, W., Wadman, M. C., & Li, Y. L. (2021). Macrophage depletion in stellate ganglia alleviates cardiac sympathetic overactivation and ventricular arrhythmogenesis by attenuating neuroinflammation in heart failure. Basic research in cardiology, 116(1), 28. https://doi.org/10.1007/s00395-021-00871-x
Zhang, Dongze, et al. "Macrophage depletion in stellate ganglia alleviates cardiac sympathetic overactivation and ventricular arrhythmogenesis by attenuating neuroinflammation in heart failure." Basic research in cardiology vol. 116,1 (2021): 28. doi: https://doi.org/10.1007/s00395-021-00871-x
Zhang D, Hu W, Tu H, Hackfort BT, Duan B, Xiong W, Wadman MC, Li YL. Macrophage depletion in stellate ganglia alleviates cardiac sympathetic overactivation and ventricular arrhythmogenesis by attenuating neuroinflammation in heart failure. Basic Res Cardiol. 2021 Apr 21;116(1):28. doi: 10.1007/s00395-021-00871-x. PMID: 33884509; PMCID: PMC8060235.
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Showing 1 to 4 of 4 entries