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Showing 1 to 7 of 7 entries
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Mitochondrial mutations and polymorphisms in psychiatric disorders.

Frontiers in genetics

Sequeira A, Martin MV, Rollins B, Moon EA, Bunney WE, Macciardi F, Lupoli S, Smith EN, Kelsoe J, Magnan CN, van Oven M, Baldi P, Wallace DC, Vawter MP.
PMID: 22723804
Front Genet. 2012 Jun 20;3:103. doi: 10.3389/fgene.2012.00103. eCollection 2012.

Mitochondrial deficiencies with unknown causes have been observed in schizophrenia (SZ) and bipolar disorder (BD) in imaging and postmortem studies. Polymorphisms and somatic mutations in mitochondrial DNA (mtDNA) were investigated as potential causes with next generation sequencing of mtDNA...

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Sequeira A, Martin MV, Rollins B, et al. Mitochondrial mutations and polymorphisms in psychiatric disorders. Front Genet. 2012;3:103doi: 10.3389/fgene.2012.00103.
Sequeira, A., Martin, M. V., Rollins, B., Moon, E. A., Bunney, W. E., Macciardi, F., Lupoli, S., Smith, E. N., Kelsoe, J., Magnan, C. N., van Oven, M., Baldi, P., Wallace, D. C., & Vawter, M. P. (2012). Mitochondrial mutations and polymorphisms in psychiatric disorders. Frontiers in genetics, 3103. https://doi.org/10.3389/fgene.2012.00103
Sequeira, Adolfo, et al. "Mitochondrial mutations and polymorphisms in psychiatric disorders." Frontiers in genetics vol. 3 (2012): 103. doi: https://doi.org/10.3389/fgene.2012.00103
Sequeira A, Martin MV, Rollins B, Moon EA, Bunney WE, Macciardi F, Lupoli S, Smith EN, Kelsoe J, Magnan CN, van Oven M, Baldi P, Wallace DC, Vawter MP. Mitochondrial mutations and polymorphisms in psychiatric disorders. Front Genet. 2012 Jun 20;3:103. doi: 10.3389/fgene.2012.00103. eCollection 2012. PMID: 22723804; PMCID: PMC3379031.
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Circadian polymorphisms associated with affective disorders.

Journal of circadian rhythms

Kripke DF, Nievergelt CM, Joo E, Shekhtman T, Kelsoe JR.
PMID: 19166596
J Circadian Rhythms. 2009 Jan 23;7:2. doi: 10.1186/1740-3391-7-2.

BACKGROUND: Clinical symptoms of affective disorders, their response to light treatment, and sensitivity to other circadian interventions indicate that the circadian system has a role in mood disorders. Possibly the mechanisms involve circadian seasonal and photoperiodic mechanisms. Since genetic...

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Kripke DF, Nievergelt CM, Joo E, et al. Circadian polymorphisms associated with affective disorders. J Circadian Rhythms. 2009;7:2doi: 10.1186/1740-3391-7-2.
Kripke, D. F., Nievergelt, C. M., Joo, E., Shekhtman, T., & Kelsoe, J. R. (2009). Circadian polymorphisms associated with affective disorders. Journal of circadian rhythms, 72. https://doi.org/10.1186/1740-3391-7-2
Kripke, Daniel F, et al. "Circadian polymorphisms associated with affective disorders." Journal of circadian rhythms vol. 7 (2009): 2. doi: https://doi.org/10.1186/1740-3391-7-2
Kripke DF, Nievergelt CM, Joo E, Shekhtman T, Kelsoe JR. Circadian polymorphisms associated with affective disorders. J Circadian Rhythms. 2009 Jan 23;7:2. doi: 10.1186/1740-3391-7-2. PMID: 19166596; PMCID: PMC2661876.
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Converging Evidence for Epistasis between ANK3 and Potassium Channel Gene KCNQ2 in Bipolar Disorder.

Frontiers in genetics

Judy JT, Seifuddin F, Pirooznia M, Mahon PB, Jancic D, Goes FS, Schulze T, Cichon S, Noethen M, Rietschel M, Depaulo JR, Potash JB, Zandi PP.
PMID: 23730306
Front Genet. 2013 May 17;4:87. doi: 10.3389/fgene.2013.00087. eCollection 2013.

Genome-wide association studies (GWAS) have implicated ANK3 as a susceptibility gene for bipolar disorder (BP). We examined whether epistasis with ANK3 may contribute to the "missing heritability" in BP. We first identified via the STRING database 14 genes encoding...

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Judy JT, Seifuddin F, Pirooznia M, et al. Converging Evidence for Epistasis between ANK3 and Potassium Channel Gene KCNQ2 in Bipolar Disorder. Front Genet. 2013;4:87doi: 10.3389/fgene.2013.00087.
Judy, J. T., Seifuddin, F., Pirooznia, M., Mahon, P. B., Jancic, D., Goes, F. S., Schulze, T., Cichon, S., Noethen, M., Rietschel, M., Depaulo, J. R., Potash, J. B., & Zandi, P. P. (2013). Converging Evidence for Epistasis between ANK3 and Potassium Channel Gene KCNQ2 in Bipolar Disorder. Frontiers in genetics, 487. https://doi.org/10.3389/fgene.2013.00087
Judy, Jennifer Toolan, et al. "Converging Evidence for Epistasis between ANK3 and Potassium Channel Gene KCNQ2 in Bipolar Disorder." Frontiers in genetics vol. 4 (2013): 87. doi: https://doi.org/10.3389/fgene.2013.00087
Judy JT, Seifuddin F, Pirooznia M, Mahon PB, Jancic D, Goes FS, Schulze T, Cichon S, Noethen M, Rietschel M, Depaulo JR, Potash JB, Zandi PP. Converging Evidence for Epistasis between ANK3 and Potassium Channel Gene KCNQ2 in Bipolar Disorder. Front Genet. 2013 May 17;4:87. doi: 10.3389/fgene.2013.00087. eCollection 2013. PMID: 23730306; PMCID: PMC3656345.
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Symptom profiles and illness course among Anabaptist and Non-Anabaptist adults with major mood disorders.

International journal of bipolar disorders

Gill KE, Cardenas SA, Kassem L, Schulze TG, McMahon FJ.
PMID: 27734417
Int J Bipolar Disord. 2016 Dec;4(1):21. doi: 10.1186/s40345-016-0062-4. Epub 2016 Oct 12.

BACKGROUND: Anabaptists comprise large and growing Amish and Mennonite populations with a unique genetic heritage and cultural background. Little is known about the symptoms and course of major mood disorders in Anabaptists. Even less is known about the impact...

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Gill KE, Cardenas SA, Kassem L, et al. Symptom profiles and illness course among Anabaptist and Non-Anabaptist adults with major mood disorders. Int J Bipolar Disord. 2016;4(1):21doi: 10.1186/s40345-016-0062-4.
Gill, K. E., Cardenas, S. A., Kassem, L., Schulze, T. G., & McMahon, F. J. (2016). Symptom profiles and illness course among Anabaptist and Non-Anabaptist adults with major mood disorders. International journal of bipolar disorders, 4(1), 21. https://doi.org/10.1186/s40345-016-0062-4
Gill, Kelly E, et al. "Symptom profiles and illness course among Anabaptist and Non-Anabaptist adults with major mood disorders." International journal of bipolar disorders vol. 4,1 (2016): 21. doi: https://doi.org/10.1186/s40345-016-0062-4
Gill KE, Cardenas SA, Kassem L, Schulze TG, McMahon FJ. Symptom profiles and illness course among Anabaptist and Non-Anabaptist adults with major mood disorders. Int J Bipolar Disord. 2016 Dec;4(1):21. doi: 10.1186/s40345-016-0062-4. Epub 2016 Oct 12. PMID: 27734417; PMCID: PMC5061680.
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A significant risk locus on 19q13 for bipolar disorder identified using a combined genome-wide linkage and copy number variation analysis.

BioData mining

Lekman M, Karlsson R, Graae L, Hössjer O, Kockum I.
PMID: 26692414
BioData Min. 2015 Dec 18;8:42. doi: 10.1186/s13040-015-0076-y. eCollection 2015.

BACKGROUND: The genetic background to bipolar disorder (BPD) has been attributed to different genetic and genomic risk factors. In the present study we hypothesized that inherited copy number variations (CNVs) contribute to susceptibility of BPD. We screened 637 BP-pedigrees...

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Lekman M, Karlsson R, Graae L, et al. A significant risk locus on 19q13 for bipolar disorder identified using a combined genome-wide linkage and copy number variation analysis. BioData Min. 2015;8:42doi: 10.1186/s13040-015-0076-y.
Lekman, M., Karlsson, R., Graae, L., Hössjer, O., & Kockum, I. (2015). A significant risk locus on 19q13 for bipolar disorder identified using a combined genome-wide linkage and copy number variation analysis. BioData mining, 842. https://doi.org/10.1186/s13040-015-0076-y
Lekman, Magnus, et al. "A significant risk locus on 19q13 for bipolar disorder identified using a combined genome-wide linkage and copy number variation analysis." BioData mining vol. 8 (2015): 42. doi: https://doi.org/10.1186/s13040-015-0076-y
Lekman M, Karlsson R, Graae L, Hössjer O, Kockum I. A significant risk locus on 19q13 for bipolar disorder identified using a combined genome-wide linkage and copy number variation analysis. BioData Min. 2015 Dec 18;8:42. doi: 10.1186/s13040-015-0076-y. eCollection 2015. PMID: 26692414; PMCID: PMC4683747.
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Variants in Ion Channel Genes Link Phenotypic Features of Bipolar Illness to Specific Neurobiological Process Domains.

Molecular neuropsychiatry

Balaraman Y, Lahiri DK, Nurnberger JI.
PMID: 27602355
Mol Neuropsychiatry. 2015 May;1(1):23-35. doi: 10.1159/000371886. Epub 2015 Feb 20.

Recent advances in genome-wide association studies are pointing towards a major role for voltage-gated ion channels in neuropsychiatric disorders and, in particular, bipolar disorder (BD). The phenotype of BD is complex, with symptoms during mood episodes and deficits persisting...

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Balaraman Y, Lahiri DK, Nurnberger JI. Variants in Ion Channel Genes Link Phenotypic Features of Bipolar Illness to Specific Neurobiological Process Domains. Mol Neuropsychiatry. 2015;1(1):23-35doi: 10.1159/000371886.
Balaraman, Y., Lahiri, D. K., & Nurnberger, J. I. (2015). Variants in Ion Channel Genes Link Phenotypic Features of Bipolar Illness to Specific Neurobiological Process Domains. Molecular neuropsychiatry, 1(1), 23-35. https://doi.org/10.1159/000371886
Balaraman, Yokesh, et al. "Variants in Ion Channel Genes Link Phenotypic Features of Bipolar Illness to Specific Neurobiological Process Domains." Molecular neuropsychiatry vol. 1,1 (2015): 23-35. doi: https://doi.org/10.1159/000371886
Balaraman Y, Lahiri DK, Nurnberger JI. Variants in Ion Channel Genes Link Phenotypic Features of Bipolar Illness to Specific Neurobiological Process Domains. Mol Neuropsychiatry. 2015 May;1(1):23-35. doi: 10.1159/000371886. Epub 2015 Feb 20. PMID: 27602355; PMCID: PMC4996004.
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Evaluation of potential novel variations and their interactions related to bipolar disorders: analysis of genome-wide association study data.

Neuropsychiatric disease and treatment

Acikel C, Aydin Son Y, Celik C, Gul H.
PMID: 27920536
Neuropsychiatr Dis Treat. 2016 Nov 24;12:2997-3004. doi: 10.2147/NDT.S112558. eCollection 2016.

BACKGROUND: Multifactor dimensionality reduction (MDR) is a nonparametric approach that can be used to detect relevant interactions between single-nucleotide polymorphisms (SNPs). The aim of this study was to build the best genomic model based on SNP associations and to...

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Acikel C, Aydin Son Y, Celik C, et al. Evaluation of potential novel variations and their interactions related to bipolar disorders: analysis of genome-wide association study data. Neuropsychiatr Dis Treat. 2016;12:2997-3004doi: 10.2147/NDT.S112558.
Acikel, C., Aydin Son, Y., Celik, C., & Gul, H. (2016). Evaluation of potential novel variations and their interactions related to bipolar disorders: analysis of genome-wide association study data. Neuropsychiatric disease and treatment, 122997-3004. https://doi.org/10.2147/NDT.S112558
Acikel, Cengizhan, et al. "Evaluation of potential novel variations and their interactions related to bipolar disorders: analysis of genome-wide association study data." Neuropsychiatric disease and treatment vol. 12 (2016): 2997-3004. doi: https://doi.org/10.2147/NDT.S112558
Acikel C, Aydin Son Y, Celik C, Gul H. Evaluation of potential novel variations and their interactions related to bipolar disorders: analysis of genome-wide association study data. Neuropsychiatr Dis Treat. 2016 Nov 24;12:2997-3004. doi: 10.2147/NDT.S112558. eCollection 2016. PMID: 27920536; PMCID: PMC5127431.
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Showing 1 to 7 of 7 entries