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Showing 1 to 12 of 12 entries
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Screening circular RNA expression patterns following focal cerebral ischemia in mice.

Oncotarget

Liu C, Zhang C, Yang J, Geng X, Du H, Ji X, Zhao H.
PMID: 29156814
Oncotarget. 2017 Sep 23;8(49):86535-86547. doi: 10.18632/oncotarget.21238. eCollection 2017 Oct 17.

Circular RNAs (circRNAs) have been demonstrated to act as microRNA (miRNA) sponges and they play important roles in regulating gene expression through a circRNA-miRNA-gene pathway. The specific roles of circRNAs in the pathogenesis of cerebral ischemia, however, are still...

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Liu C, Zhang C, Yang J, et al. Screening circular RNA expression patterns following focal cerebral ischemia in mice. Oncotarget. 2017;8(49):86535-86547doi: 10.18632/oncotarget.21238.
Liu, C., Zhang, C., Yang, J., Geng, X., Du, H., Ji, X., & Zhao, H. (2017). Screening circular RNA expression patterns following focal cerebral ischemia in mice. Oncotarget, 8(49), 86535-86547. https://doi.org/10.18632/oncotarget.21238
Liu, Cuiying, et al. "Screening circular RNA expression patterns following focal cerebral ischemia in mice." Oncotarget vol. 8,49 (2017): 86535-86547. doi: https://doi.org/10.18632/oncotarget.21238
Liu C, Zhang C, Yang J, Geng X, Du H, Ji X, Zhao H. Screening circular RNA expression patterns following focal cerebral ischemia in mice. Oncotarget. 2017 Sep 23;8(49):86535-86547. doi: 10.18632/oncotarget.21238. eCollection 2017 Oct 17. PMID: 29156814; PMCID: PMC5689704.
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Using hormetic strategies to improve ischemic preconditioning and postconditioning against stroke.

International journal of physiology, pathophysiology and pharmacology

Zhao H, Joo S, Xie W, Ji X.
PMID: 23750305
Int J Physiol Pathophysiol Pharmacol. 2013 May 27;5(2):61-72. Print 2013.

Both ischemic preconditioning (IPreC) and ischemic postconditioning (IPostC) trigger endogenous neuroprotective mechanisms in cerebral ischemia. IPreC is defined as a brief ischemia that protects against a subsequent severe ischemia, while IPostC refers to a series of brief cerebral blood...

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Zhao H, Joo S, Xie W, et al. Using hormetic strategies to improve ischemic preconditioning and postconditioning against stroke. Int J Physiol Pathophysiol Pharmacol. 2013;5(2):61-72
Zhao, H., Joo, S., Xie, W., & Ji, X. (2013). Using hormetic strategies to improve ischemic preconditioning and postconditioning against stroke. International journal of physiology, pathophysiology and pharmacology, 5(2), 61-72.
Zhao, Heng, et al. "Using hormetic strategies to improve ischemic preconditioning and postconditioning against stroke." International journal of physiology, pathophysiology and pharmacology vol. 5,2 (2013): 61-72.
Zhao H, Joo S, Xie W, Ji X. Using hormetic strategies to improve ischemic preconditioning and postconditioning against stroke. Int J Physiol Pathophysiol Pharmacol. 2013 May 27;5(2):61-72. Print 2013. PMID: 23750305; PMCID: PMC3669735.
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Ischemic postconditioning for stroke treatment: current experimental advances and future directions.

Conditioning medicine

Chen H, Shen J, Zhao H.
PMID: 34396060
Cond Med. 2020 Apr;3(2):104-115. Epub 2020 May 05.

Ischemic postconditioning (IPostC) protects against brain injury induced by stroke and is a potential strategy for ischemic stroke treatment. Understanding its underlying mechanisms and potential hurdles is essential for clinical translation. In this review article, we will summarize the...

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Chen H, Shen J, Zhao H. Ischemic postconditioning for stroke treatment: current experimental advances and future directions. Cond Med. 2020;3(2):104-115
Chen, H., Shen, J., & Zhao, H. (2020). Ischemic postconditioning for stroke treatment: current experimental advances and future directions. Conditioning medicine, 3(2), 104-115.
Chen, Hansen, et al. "Ischemic postconditioning for stroke treatment: current experimental advances and future directions." Conditioning medicine vol. 3,2 (2020): 104-115.
Chen H, Shen J, Zhao H. Ischemic postconditioning for stroke treatment: current experimental advances and future directions. Cond Med. 2020 Apr;3(2):104-115. Epub 2020 May 05. PMID: 34396060; PMCID: PMC8360401.
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CD4 T cell deficiency attenuates ischemic stroke, inhibits oxidative stress, and enhances Akt/mTOR survival signaling pathways in mice.

Chinese neurosurgical journal

Zhang H, Xiong X, Gu L, Xie W, Zhao H.
PMID: 32832192
Chin Neurosurg J. 2018;4. doi: 10.1186/s41016-018-0140-9. Epub 2018 Nov 08.

BACKGROUND: Inhibition of CD4 T cells reduces stroke-induced infarction by inhibiting neuroinflammation in the ischemic brain in experimental stroke. Nevertheless, little is known about its effects on neuronal survival signaling pathways. In this study, we investigated the effects of...

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Zhang H, Xiong X, Gu L, et al. CD4 T cell deficiency attenuates ischemic stroke, inhibits oxidative stress, and enhances Akt/mTOR survival signaling pathways in mice. Chin Neurosurg J. 2018;4doi: 10.1186/s41016-018-0140-9.
Zhang, H., Xiong, X., Gu, L., Xie, W., & Zhao, H. (2018). CD4 T cell deficiency attenuates ischemic stroke, inhibits oxidative stress, and enhances Akt/mTOR survival signaling pathways in mice. Chinese neurosurgical journal, 4. https://doi.org/10.1186/s41016-018-0140-9
Zhang, Hongfei, et al. "CD4 T cell deficiency attenuates ischemic stroke, inhibits oxidative stress, and enhances Akt/mTOR survival signaling pathways in mice." Chinese neurosurgical journal vol. 4 (2018). doi: https://doi.org/10.1186/s41016-018-0140-9
Zhang H, Xiong X, Gu L, Xie W, Zhao H. CD4 T cell deficiency attenuates ischemic stroke, inhibits oxidative stress, and enhances Akt/mTOR survival signaling pathways in mice. Chin Neurosurg J. 2018;4. doi: 10.1186/s41016-018-0140-9. Epub 2018 Nov 08. PMID: 32832192; PMCID: PMC7398241.
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Increased Brain-Specific MiR-9 and MiR-124 in the Serum Exosomes of Acute Ischemic Stroke Patients.

PloS one

Ji Q, Ji Y, Peng J, Zhou X, Chen X, Zhao H, Xu T, Chen L, Xu Y.
PMID: 27661079
PLoS One. 2016 Sep 23;11(9):e0163645. doi: 10.1371/journal.pone.0163645. eCollection 2016.

The aims of this study were to examine the alternation in serum exosome concentrations and the levels of serum exosomal miR-9 and miR-124, two brain-specific miRNAs, in acute ischemic stroke (AIS) patients and to explore the predictive values of...

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Ji Q, Ji Y, Peng J, et al. Increased Brain-Specific MiR-9 and MiR-124 in the Serum Exosomes of Acute Ischemic Stroke Patients. PLoS One. 2016;11(9):e0163645doi: 10.1371/journal.pone.0163645.
Ji, Q., Ji, Y., Peng, J., Zhou, X., Chen, X., Zhao, H., Xu, T., Chen, L., & Xu, Y. (2016). Increased Brain-Specific MiR-9 and MiR-124 in the Serum Exosomes of Acute Ischemic Stroke Patients. PloS one, 11(9), e0163645. https://doi.org/10.1371/journal.pone.0163645
Ji, Qiuhong, et al. "Increased Brain-Specific MiR-9 and MiR-124 in the Serum Exosomes of Acute Ischemic Stroke Patients." PloS one vol. 11,9 (2016): e0163645. doi: https://doi.org/10.1371/journal.pone.0163645
Ji Q, Ji Y, Peng J, Zhou X, Chen X, Zhao H, Xu T, Chen L, Xu Y. Increased Brain-Specific MiR-9 and MiR-124 in the Serum Exosomes of Acute Ischemic Stroke Patients. PLoS One. 2016 Sep 23;11(9):e0163645. doi: 10.1371/journal.pone.0163645. eCollection 2016. PMID: 27661079; PMCID: PMC5035015.
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An insult-inducible vector system activated by hypoxia and oxidative stress for neuronal gene therapy.

Translational stroke research

Cheng MY, Lee IP, Jin M, Sun G, Zhao H, Steinberg GK, Sapolsky RM.
PMID: 21603078
Transl Stroke Res. 2011 Mar 01;2(1):92-100. doi: 10.1007/s12975-010-0060-2.

Gene therapy has demonstrated the protective potential of a variety of genes against stroke. However, conventional gene therapy vectors are limited due to the inability to temporally control their expression, which can sometimes lead to deleterious side effects. Thus,...

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Cheng MY, Lee IP, Jin M, et al. An insult-inducible vector system activated by hypoxia and oxidative stress for neuronal gene therapy. Transl Stroke Res. 2011;2(1):92-100doi: 10.1007/s12975-010-0060-2.
Cheng, M. Y., Lee, I. P., Jin, M., Sun, G., Zhao, H., Steinberg, G. K., & Sapolsky, R. M. (2011). An insult-inducible vector system activated by hypoxia and oxidative stress for neuronal gene therapy. Translational stroke research, 2(1), 92-100. https://doi.org/10.1007/s12975-010-0060-2
Cheng, Michelle Y, et al. "An insult-inducible vector system activated by hypoxia and oxidative stress for neuronal gene therapy." Translational stroke research vol. 2,1 (2011): 92-100. doi: https://doi.org/10.1007/s12975-010-0060-2
Cheng MY, Lee IP, Jin M, Sun G, Zhao H, Steinberg GK, Sapolsky RM. An insult-inducible vector system activated by hypoxia and oxidative stress for neuronal gene therapy. Transl Stroke Res. 2011 Mar 01;2(1):92-100. doi: 10.1007/s12975-010-0060-2. PMID: 21603078; PMCID: PMC3097421.
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Lithium treatment reduces brain injury induced by focal ischemia with partial reperfusion and the protective mechanisms dispute the importance of akt activity.

Aging and disease

Takahashi T, Steinberg GK, Zhao H.
PMID: 22724081
Aging Dis. 2012 Jun;3(3):226-33. Epub 2012 Feb 08.

Lithium is a mood stabilizer shown to have neuroprotective effects against several chronic and acute neuronal injuries, including stroke. However, it is unknown whether lithium treatment protects against brain injury post-stroke in a rat model of permanent distal middle...

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Takahashi T, Steinberg GK, Zhao H. Lithium treatment reduces brain injury induced by focal ischemia with partial reperfusion and the protective mechanisms dispute the importance of akt activity. Aging Dis. 2012;3(3):226-33
Takahashi, T., Steinberg, G. K., & Zhao, H. (2012). Lithium treatment reduces brain injury induced by focal ischemia with partial reperfusion and the protective mechanisms dispute the importance of akt activity. Aging and disease, 3(3), 226-33.
Takahashi, Tetsuya, et al. "Lithium treatment reduces brain injury induced by focal ischemia with partial reperfusion and the protective mechanisms dispute the importance of akt activity." Aging and disease vol. 3,3 (2012): 226-33.
Takahashi T, Steinberg GK, Zhao H. Lithium treatment reduces brain injury induced by focal ischemia with partial reperfusion and the protective mechanisms dispute the importance of akt activity. Aging Dis. 2012 Jun;3(3):226-33. Epub 2012 Feb 08. PMID: 22724081; PMCID: PMC3375079.
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The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats.

Translational stroke research

Gao X, Zhang H, Steinberg G, Zhao H.
PMID: 21804899
Transl Stroke Res. 2010 Sep;1(3):202-9. doi: 10.1007/s12975-010-0017-5.

Although the protective mechanisms of delayed ischemic preconditioning have received extensive studies, few have addressed the mechanisms associated with rapid ischemic postconditioning. We investigated whether ischemic tolerance induced by rapid preconditioning is regulated by the Akt survival signaling pathway....

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Gao X, Zhang H, Steinberg G, et al. The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats. Transl Stroke Res. 2010;1(3):202-9doi: 10.1007/s12975-010-0017-5.
Gao, X., Zhang, H., Steinberg, G., & Zhao, H. (2010). The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats. Translational stroke research, 1(3), 202-9. https://doi.org/10.1007/s12975-010-0017-5
Gao, Xuwen, et al. "The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats." Translational stroke research vol. 1,3 (2010): 202-9. doi: https://doi.org/10.1007/s12975-010-0017-5
Gao X, Zhang H, Steinberg G, Zhao H. The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats. Transl Stroke Res. 2010 Sep;1(3):202-9. doi: 10.1007/s12975-010-0017-5. PMID: 21804899; PMCID: PMC3144475.
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The underlying mechanisms involved in the protective effects of ischemic postconditioning.

Conditioning medicine

Xie R, Li J, Zhao H.
PMID: 29782624
Cond Med. 2018;1(2):73-79.

Cerebral ischemic postconditioning (PostC) refers to a series of brief ischemia and reperfusion (I/R) cycles applied at the onset of reperfusion following an ischemic event. PostC has been shown to have neuroprotective effects, and represents a promising clinical strategy...

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Xie R, Li J, Zhao H. The underlying mechanisms involved in the protective effects of ischemic postconditioning. Cond Med. 2018;1(2):73-79
Xie, R., Li, J., & Zhao, H. (2018). The underlying mechanisms involved in the protective effects of ischemic postconditioning. Conditioning medicine, 1(2), 73-79.
Xie, Rong, et al. "The underlying mechanisms involved in the protective effects of ischemic postconditioning." Conditioning medicine vol. 1,2 (2018): 73-79.
Xie R, Li J, Zhao H. The underlying mechanisms involved in the protective effects of ischemic postconditioning. Cond Med. 2018;1(2):73-79. PMID: 29782624; PMCID: PMC5959054.
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Editorial: Stem Cells and Aging.

Frontiers in aging neuroscience

Borlongan CV, Steinberg GK.
PMID: 34079451
Front Aging Neurosci. 2021 May 17;13:690613. doi: 10.3389/fnagi.2021.690613. eCollection 2021.

No abstract available.

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Borlongan CV, Steinberg GK. Editorial: Stem Cells and Aging. Front Aging Neurosci. 2021;13:690613doi: 10.3389/fnagi.2021.690613.
Borlongan, C. V., & Steinberg, G. K. (2021). Editorial: Stem Cells and Aging. Frontiers in aging neuroscience, 13690613. https://doi.org/10.3389/fnagi.2021.690613
Borlongan, Cesar V, and Steinberg, Gary K. "Editorial: Stem Cells and Aging." Frontiers in aging neuroscience vol. 13 (2021): 690613. doi: https://doi.org/10.3389/fnagi.2021.690613
Borlongan CV, Steinberg GK. Editorial: Stem Cells and Aging. Front Aging Neurosci. 2021 May 17;13:690613. doi: 10.3389/fnagi.2021.690613. eCollection 2021. PMID: 34079451; PMCID: PMC8165171.
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The Protective Effects of Ischemic Postconditioning against Stroke: From Rapid to Delayed and Remote Postconditioning.

The open drug discovery journal

Zhao H.
PMID: 22053169
Open Drug Discov J. 2011 Dec 24;5:138-147. doi: 10.2174/1877381801002010138.

The author reviews the protective effects of ischemic postconditioning, a recently emerging strategy with broad implications in the search for new treatments in stroke and myocardial ischemic injury. Ischemic postconditioning, which refers to a series of brief ischemia and...

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Zhao H. The Protective Effects of Ischemic Postconditioning against Stroke: From Rapid to Delayed and Remote Postconditioning. Open Drug Discov J. 2011;5:138-147doi: 10.2174/1877381801002010138.
Zhao, H. (2011). The Protective Effects of Ischemic Postconditioning against Stroke: From Rapid to Delayed and Remote Postconditioning. The open drug discovery journal, 5138-147. https://doi.org/10.2174/1877381801002010138
Zhao, Heng. "The Protective Effects of Ischemic Postconditioning against Stroke: From Rapid to Delayed and Remote Postconditioning." The open drug discovery journal vol. 5 (2011): 138-147. doi: https://doi.org/10.2174/1877381801002010138
Zhao H. The Protective Effects of Ischemic Postconditioning against Stroke: From Rapid to Delayed and Remote Postconditioning. Open Drug Discov J. 2011 Dec 24;5:138-147. doi: 10.2174/1877381801002010138. PMID: 22053169; PMCID: PMC3204606.
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Limited Therapeutic Time Windows of Mild-to-Moderate Hypothermia in a Focal Ischemia Model in Rat.

Stroke research and treatment

Zhao H, Steinberg G.
PMID: 21876846
Stroke Res Treat. 2011;2011:131834. doi: 10.4061/2011/131834. Epub 2011 Aug 16.

Although many studies have shown the great potential of induced hypothermia in stroke treatment, we recognize that there are limitations to the protective effects of hypothermia even in the laboratory. Here, we review our experiments on the protective effects...

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Zhao H, Steinberg G. Limited Therapeutic Time Windows of Mild-to-Moderate Hypothermia in a Focal Ischemia Model in Rat. Stroke Res Treat. 2011;2011:131834doi: 10.4061/2011/131834.
Zhao, H., & Steinberg, G. (2011). Limited Therapeutic Time Windows of Mild-to-Moderate Hypothermia in a Focal Ischemia Model in Rat. Stroke research and treatment, 2011131834. https://doi.org/10.4061/2011/131834
Zhao, Heng, and Steinberg, Gary. "Limited Therapeutic Time Windows of Mild-to-Moderate Hypothermia in a Focal Ischemia Model in Rat." Stroke research and treatment vol. 2011 (2011): 131834. doi: https://doi.org/10.4061/2011/131834
Zhao H, Steinberg G. Limited Therapeutic Time Windows of Mild-to-Moderate Hypothermia in a Focal Ischemia Model in Rat. Stroke Res Treat. 2011;2011:131834. doi: 10.4061/2011/131834. Epub 2011 Aug 16. PMID: 21876846; PMCID: PMC3159378.
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Showing 1 to 12 of 12 entries