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Imatinib treatment reduces brain injury in a murine model of traumatic brain injury.

Frontiers in cellular neuroscience

Su EJ, Fredriksson L, Kanzawa M, Moore S, Folestad E, Stevenson TK, Nilsson I, Sashindranath M, Schielke GP, Warnock M, Ragsdale M, Mann K, Lawrence AL, Medcalf RL, Eriksson U, Murphy GG, Lawrence DA.
PMID: 26500491
Front Cell Neurosci. 2015 Oct 07;9:385. doi: 10.3389/fncel.2015.00385. eCollection 2015.

Current therapies for Traumatic brain injury (TBI) focus on stabilizing individuals and on preventing further damage from the secondary consequences of TBI. A major complication of TBI is cerebral edema, which can be caused by the loss of blood...

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Su EJ, Fredriksson L, Kanzawa M, et al. Imatinib treatment reduces brain injury in a murine model of traumatic brain injury. Front Cell Neurosci. 2015;9:385doi: 10.3389/fncel.2015.00385.
Su, E. J., Fredriksson, L., Kanzawa, M., Moore, S., Folestad, E., Stevenson, T. K., Nilsson, I., Sashindranath, M., Schielke, G. P., Warnock, M., Ragsdale, M., Mann, K., Lawrence, A. L., Medcalf, R. L., Eriksson, U., Murphy, G. G., & Lawrence, D. A. (2015). Imatinib treatment reduces brain injury in a murine model of traumatic brain injury. Frontiers in cellular neuroscience, 9385. https://doi.org/10.3389/fncel.2015.00385
Su, Enming J, et al. "Imatinib treatment reduces brain injury in a murine model of traumatic brain injury." Frontiers in cellular neuroscience vol. 9 (2015): 385. doi: https://doi.org/10.3389/fncel.2015.00385
Su EJ, Fredriksson L, Kanzawa M, Moore S, Folestad E, Stevenson TK, Nilsson I, Sashindranath M, Schielke GP, Warnock M, Ragsdale M, Mann K, Lawrence AL, Medcalf RL, Eriksson U, Murphy GG, Lawrence DA. Imatinib treatment reduces brain injury in a murine model of traumatic brain injury. Front Cell Neurosci. 2015 Oct 07;9:385. doi: 10.3389/fncel.2015.00385. eCollection 2015. PMID: 26500491; PMCID: PMC4596067.
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Identification of a neurovascular signaling pathway regulating seizures in mice.

Annals of clinical and translational neurology

Fredriksson L, Stevenson TK, Su EJ, Ragsdale M, Moore S, Craciun S, Schielke GP, Murphy GG, Lawrence DA.
PMID: 26273685
Ann Clin Transl Neurol. 2015 Jul;2(7):722-38. doi: 10.1002/acn3.209. Epub 2015 May 01.

OBJECTIVE: A growing body of evidence suggests that increased blood-brain barrier (BBB) permeability can contribute to the development of seizures. The protease tissue plasminogen activator (tPA) has been shown to promote BBB permeability and susceptibility to seizures. In this...

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Fredriksson L, Stevenson TK, Su EJ, et al. Identification of a neurovascular signaling pathway regulating seizures in mice. Ann Clin Transl Neurol. 2015;2(7):722-38doi: 10.1002/acn3.209.
Fredriksson, L., Stevenson, T. K., Su, E. J., Ragsdale, M., Moore, S., Craciun, S., Schielke, G. P., Murphy, G. G., & Lawrence, D. A. (2015). Identification of a neurovascular signaling pathway regulating seizures in mice. Annals of clinical and translational neurology, 2(7), 722-38. https://doi.org/10.1002/acn3.209
Fredriksson, Linda, et al. "Identification of a neurovascular signaling pathway regulating seizures in mice." Annals of clinical and translational neurology vol. 2,7 (2015): 722-38. doi: https://doi.org/10.1002/acn3.209
Fredriksson L, Stevenson TK, Su EJ, Ragsdale M, Moore S, Craciun S, Schielke GP, Murphy GG, Lawrence DA. Identification of a neurovascular signaling pathway regulating seizures in mice. Ann Clin Transl Neurol. 2015 Jul;2(7):722-38. doi: 10.1002/acn3.209. Epub 2015 May 01. PMID: 26273685; PMCID: PMC4531055.
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Low-molecular-weight heparin modulates vein wall fibrotic response in a plasminogen activator inhibitor 1-dependent manner.

Journal of vascular surgery. Venous and lymphatic disorders

Obi AT, Diaz JA, Ballard-Lipka NL, Roelofs KJ, Farris DM, Lawrence DA, Henke PK, Wakefield TW.
PMID: 25419511
J Vasc Surg Venous Lymphat Disord. 2014 Oct 01;2(4):441-450.e1. doi: 10.1016/j.jvsv.2014.02.004.

BACKGROUND: Treatment with low-molecular-weight heparin (LMWH) favorably alters the vein wall response to deep venous thrombosis (DVT), although the mechanisms remain unclear. Previous studies have suggested that LMWH alters the levels of circulating plasminogen activator inhibitor 1 (PAI-1), a...

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Obi AT, Diaz JA, Ballard-Lipka NL, et al. Low-molecular-weight heparin modulates vein wall fibrotic response in a plasminogen activator inhibitor 1-dependent manner. J Vasc Surg Venous Lymphat Disord. 2014;2(4):441-450.e1doi: 10.1016/j.jvsv.2014.02.004.
Obi, A. T., Diaz, J. A., Ballard-Lipka, N. L., Roelofs, K. J., Farris, D. M., Lawrence, D. A., Henke, P. K., & Wakefield, T. W. (2014). Low-molecular-weight heparin modulates vein wall fibrotic response in a plasminogen activator inhibitor 1-dependent manner. Journal of vascular surgery. Venous and lymphatic disorders, 2(4), 441-450.e1. https://doi.org/10.1016/j.jvsv.2014.02.004
Obi, Andrea T, et al. "Low-molecular-weight heparin modulates vein wall fibrotic response in a plasminogen activator inhibitor 1-dependent manner." Journal of vascular surgery. Venous and lymphatic disorders vol. 2,4 (2014): 441-450.e1. doi: https://doi.org/10.1016/j.jvsv.2014.02.004
Obi AT, Diaz JA, Ballard-Lipka NL, Roelofs KJ, Farris DM, Lawrence DA, Henke PK, Wakefield TW. Low-molecular-weight heparin modulates vein wall fibrotic response in a plasminogen activator inhibitor 1-dependent manner. J Vasc Surg Venous Lymphat Disord. 2014 Oct 01;2(4):441-450.e1. doi: 10.1016/j.jvsv.2014.02.004. PMID: 25419511; PMCID: PMC4235166.
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Editorial: The Role of the Plasminogen Activating System in Neurobiology.

Frontiers in cellular neuroscience

Medcalf RL, Lawrence DA.
PMID: 27757075
Front Cell Neurosci. 2016 Oct 04;10:222. doi: 10.3389/fncel.2016.00222. eCollection 2016.

No abstract available.

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Medcalf RL, Lawrence DA. Editorial: The Role of the Plasminogen Activating System in Neurobiology. Front Cell Neurosci. 2016;10:222doi: 10.3389/fncel.2016.00222.
Medcalf, R. L., & Lawrence, D. A. (2016). Editorial: The Role of the Plasminogen Activating System in Neurobiology. Frontiers in cellular neuroscience, 10222. https://doi.org/10.3389/fncel.2016.00222
Medcalf, Robert L, and Lawrence, Daniel A. "Editorial: The Role of the Plasminogen Activating System in Neurobiology." Frontiers in cellular neuroscience vol. 10 (2016): 222. doi: https://doi.org/10.3389/fncel.2016.00222
Medcalf RL, Lawrence DA. Editorial: The Role of the Plasminogen Activating System in Neurobiology. Front Cell Neurosci. 2016 Oct 04;10:222. doi: 10.3389/fncel.2016.00222. eCollection 2016. PMID: 27757075; PMCID: PMC5048060.
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Neuroserpin Differentiates Between Forms of Tissue Type Plasminogen Activator via pH Dependent Deacylation.

Frontiers in cellular neuroscience

Carlson KS, Nguyen L, Schwartz K, Lawrence DA, Schwartz BS.
PMID: 27378851
Front Cell Neurosci. 2016 Jun 15;10:154. doi: 10.3389/fncel.2016.00154. eCollection 2016.

Tissue-type plasminogen activator (t-PA), initially characterized for its critical role in fibrinolysis, also has key functions in both physiologic and pathologic processes in the CNS. Neuroserpin (NSP) is a t-PA specific serine protease inhibitor (serpin) found almost exclusively in...

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Carlson KS, Nguyen L, Schwartz K, et al. Neuroserpin Differentiates Between Forms of Tissue Type Plasminogen Activator via pH Dependent Deacylation. Front Cell Neurosci. 2016;10:154doi: 10.3389/fncel.2016.00154.
Carlson, K. S., Nguyen, L., Schwartz, K., Lawrence, D. A., & Schwartz, B. S. (2016). Neuroserpin Differentiates Between Forms of Tissue Type Plasminogen Activator via pH Dependent Deacylation. Frontiers in cellular neuroscience, 10154. https://doi.org/10.3389/fncel.2016.00154
Carlson, Karen-Sue B, et al. "Neuroserpin Differentiates Between Forms of Tissue Type Plasminogen Activator via pH Dependent Deacylation." Frontiers in cellular neuroscience vol. 10 (2016): 154. doi: https://doi.org/10.3389/fncel.2016.00154
Carlson KS, Nguyen L, Schwartz K, Lawrence DA, Schwartz BS. Neuroserpin Differentiates Between Forms of Tissue Type Plasminogen Activator via pH Dependent Deacylation. Front Cell Neurosci. 2016 Jun 15;10:154. doi: 10.3389/fncel.2016.00154. eCollection 2016. PMID: 27378851; PMCID: PMC4908126.
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Showing 1 to 5 of 5 entries