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Showing 1 to 11 of 11 entries
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Cell therapy for cerebral hemorrhage: Five year follow-up report.

Experimental and therapeutic medicine

Chang Z, Mao G, Sun L, Ao Q, Gu Y, Liu Y.
PMID: 28101148
Exp Ther Med. 2016 Dec;12(6):3535-3540. doi: 10.3892/etm.2016.3811. Epub 2016 Oct 18.

The aim of the study was to examine treatment of cerebral hemorrhages with bone-marrow or human umbilical cord-derived mesenchymal stem cells (BMSCs or Hu-MSCs) and conventional surgical approaches, and determine and compare the effectiveness, feasibility, safety and reproducibility of...

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Chang Z, Mao G, Sun L, et al. Cell therapy for cerebral hemorrhage: Five year follow-up report. Exp Ther Med. 2016;12(6):3535-3540doi: 10.3892/etm.2016.3811.
Chang, Z., Mao, G., Sun, L., Ao, Q., Gu, Y., & Liu, Y. (2016). Cell therapy for cerebral hemorrhage: Five year follow-up report. Experimental and therapeutic medicine, 12(6), 3535-3540. https://doi.org/10.3892/etm.2016.3811
Chang, Zhitian, et al. "Cell therapy for cerebral hemorrhage: Five year follow-up report." Experimental and therapeutic medicine vol. 12,6 (2016): 3535-3540. doi: https://doi.org/10.3892/etm.2016.3811
Chang Z, Mao G, Sun L, Ao Q, Gu Y, Liu Y. Cell therapy for cerebral hemorrhage: Five year follow-up report. Exp Ther Med. 2016 Dec;12(6):3535-3540. doi: 10.3892/etm.2016.3811. Epub 2016 Oct 18. PMID: 28101148; PMCID: PMC5228203.
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High-level SAE2 promotes malignant phenotype and predicts outcome in gastric cancer.

American journal of cancer research

Shao DF, Wang XH, Li ZY, Xing XF, Cheng XJ, Guo T, Du H, Hu Y, Dong B, Ding N, Li L, Li S, Li QD, Wen XZ, Zhang LH, Ji JF.
PMID: 25628926
Am J Cancer Res. 2014 Dec 15;5(1):140-54. eCollection 2015.

BACKGROUND: The SUMO pathway has been shown to play an important role in tumorigenesis. This report analyzed the involvement of the sole SUMO-Activating Enzyme Subunit 2 (SAE2) in human gastric cancer (GC) progression and prognosis.METHODS: Expression of SAE2 was...

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Shao DF, Wang XH, Li ZY, et al. High-level SAE2 promotes malignant phenotype and predicts outcome in gastric cancer. Am J Cancer Res. 2014;5(1):140-54
Shao, D. F., Wang, X. H., Li, Z. Y., Xing, X. F., Cheng, X. J., Guo, T., Du, H., Hu, Y., Dong, B., Ding, N., Li, L., Li, S., Li, Q. D., Wen, X. Z., Zhang, L. H., & Ji, J. F. (2015). High-level SAE2 promotes malignant phenotype and predicts outcome in gastric cancer. American journal of cancer research, 5(1), 140-54.
Shao, Duan-Fang, et al. "High-level SAE2 promotes malignant phenotype and predicts outcome in gastric cancer." American journal of cancer research vol. 5,1 (2015): 140-54.
Shao DF, Wang XH, Li ZY, Xing XF, Cheng XJ, Guo T, Du H, Hu Y, Dong B, Ding N, Li L, Li S, Li QD, Wen XZ, Zhang LH, Ji JF. High-level SAE2 promotes malignant phenotype and predicts outcome in gastric cancer. Am J Cancer Res. 2014 Dec 15;5(1):140-54. eCollection 2015. PMID: 25628926; PMCID: PMC4300690.
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Bacteria may have multiple replication origins.

Frontiers in microbiology

Gao F.
PMID: 25941523
Front Microbiol. 2015 Apr 20;6:324. doi: 10.3389/fmicb.2015.00324. eCollection 2015.

No abstract available.

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Gao F. Bacteria may have multiple replication origins. Front Microbiol. 2015;6:324doi: 10.3389/fmicb.2015.00324.
Gao, F. (2015). Bacteria may have multiple replication origins. Frontiers in microbiology, 6324. https://doi.org/10.3389/fmicb.2015.00324
Gao, Feng. "Bacteria may have multiple replication origins." Frontiers in microbiology vol. 6 (2015): 324. doi: https://doi.org/10.3389/fmicb.2015.00324
Gao F. Bacteria may have multiple replication origins. Front Microbiol. 2015 Apr 20;6:324. doi: 10.3389/fmicb.2015.00324. eCollection 2015. PMID: 25941523; PMCID: PMC4403523.
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Editorial: DNA Replication Origins in Microbial Genomes.

Frontiers in microbiology

Gao F.
PMID: 26779176
Front Microbiol. 2016 Jan 08;6:1545. doi: 10.3389/fmicb.2015.01545. eCollection 2015.

No abstract available.

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Gao F. Editorial: DNA Replication Origins in Microbial Genomes. Front Microbiol. 2016;6:1545doi: 10.3389/fmicb.2015.01545.
Gao, F. (2015). Editorial: DNA Replication Origins in Microbial Genomes. Frontiers in microbiology, 61545. https://doi.org/10.3389/fmicb.2015.01545
Gao, Feng. "Editorial: DNA Replication Origins in Microbial Genomes." Frontiers in microbiology vol. 6 (2015): 1545. doi: https://doi.org/10.3389/fmicb.2015.01545
Gao F. Editorial: DNA Replication Origins in Microbial Genomes. Front Microbiol. 2016 Jan 08;6:1545. doi: 10.3389/fmicb.2015.01545. eCollection 2015. PMID: 26779176; PMCID: PMC4705448.
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The construction of an interfacial valve-based microfluidic chip for thermotaxis evaluation of human sperm.

Biomicrofluidics

Li Z, Liu W, Qiu T, Xie L, Chen W, Liu R, Lu Y, Mitchelson K, Wang J, Qiao J, Cheng J.
PMID: 24803958
Biomicrofluidics. 2014 Mar 05;8(2):024102. doi: 10.1063/1.4866851. eCollection 2014 Mar.

Thermotaxis has been demonstrated to be an important criterion for sperm evaluation, yet clinical assessment of thermotaxis capacity is currently lacking. In this article, the on-chip thermotaxis evaluation of human sperm is presented for the first time using an...

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Li Z, Liu W, Qiu T, et al. The construction of an interfacial valve-based microfluidic chip for thermotaxis evaluation of human sperm. Biomicrofluidics. 2014;8(2):024102doi: 10.1063/1.4866851.
Li, Z., Liu, W., Qiu, T., Xie, L., Chen, W., Liu, R., Lu, Y., Mitchelson, K., Wang, J., Qiao, J., & Cheng, J. (2014). The construction of an interfacial valve-based microfluidic chip for thermotaxis evaluation of human sperm. Biomicrofluidics, 8(2), 024102. https://doi.org/10.1063/1.4866851
Li, Zhuoqi, et al. "The construction of an interfacial valve-based microfluidic chip for thermotaxis evaluation of human sperm." Biomicrofluidics vol. 8,2 (2014): 024102. doi: https://doi.org/10.1063/1.4866851
Li Z, Liu W, Qiu T, Xie L, Chen W, Liu R, Lu Y, Mitchelson K, Wang J, Qiao J, Cheng J. The construction of an interfacial valve-based microfluidic chip for thermotaxis evaluation of human sperm. Biomicrofluidics. 2014 Mar 05;8(2):024102. doi: 10.1063/1.4866851. eCollection 2014 Mar. PMID: 24803958; PMCID: PMC3987097.
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Recent advances in the genome-wide study of DNA replication origins in yeast.

Frontiers in microbiology

Peng C, Luo H, Zhang X, Gao F.
PMID: 25745419
Front Microbiol. 2015 Feb 19;6:117. doi: 10.3389/fmicb.2015.00117. eCollection 2015.

DNA replication, one of the central events in the cell cycle, is the basis of biological inheritance. In order to be duplicated, a DNA double helix must be opened at defined sites, which are called DNA replication origins (ORIs)....

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Peng C, Luo H, Zhang X, et al. Recent advances in the genome-wide study of DNA replication origins in yeast. Front Microbiol. 2015;6:117doi: 10.3389/fmicb.2015.00117.
Peng, C., Luo, H., Zhang, X., & Gao, F. (2015). Recent advances in the genome-wide study of DNA replication origins in yeast. Frontiers in microbiology, 6117. https://doi.org/10.3389/fmicb.2015.00117
Peng, Chong, et al. "Recent advances in the genome-wide study of DNA replication origins in yeast." Frontiers in microbiology vol. 6 (2015): 117. doi: https://doi.org/10.3389/fmicb.2015.00117
Peng C, Luo H, Zhang X, Gao F. Recent advances in the genome-wide study of DNA replication origins in yeast. Front Microbiol. 2015 Feb 19;6:117. doi: 10.3389/fmicb.2015.00117. eCollection 2015. PMID: 25745419; PMCID: PMC4333867.
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Hepatoma-Derived Growth Factor Secreted from Mesenchymal Stem Cells Reduces Myocardial Ischemia-Reperfusion Injury.

Stem cells international

Zhou Y, Chen P, Liu Q, Wang Y, Zhang L, Wu R, Chen J, Yu H, Zhu W, Hu X, Wang JA.
PMID: 29358952
Stem Cells Int. 2017;2017:1096980. doi: 10.1155/2017/1096980. Epub 2017 Nov 14.

OBJECTIVES: The present study aimed to explore the major factors that account for the beneficial effects of mesenchymal stem cells (MSCs).METHODS: Using isobaric tags for relative and absolute quantitation method, hepatoma-derived growth factor (HDGF) was identified as an important...

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Zhou Y, Chen P, Liu Q, et al. Hepatoma-Derived Growth Factor Secreted from Mesenchymal Stem Cells Reduces Myocardial Ischemia-Reperfusion Injury. Stem Cells Int. 2017;2017:1096980doi: 10.1155/2017/1096980.
Zhou, Y., Chen, P., Liu, Q., Wang, Y., Zhang, L., Wu, R., Chen, J., Yu, H., Zhu, W., Hu, X., & Wang, J. A. (2017). Hepatoma-Derived Growth Factor Secreted from Mesenchymal Stem Cells Reduces Myocardial Ischemia-Reperfusion Injury. Stem cells international, 20171096980. https://doi.org/10.1155/2017/1096980
Zhou, Yu, et al. "Hepatoma-Derived Growth Factor Secreted from Mesenchymal Stem Cells Reduces Myocardial Ischemia-Reperfusion Injury." Stem cells international vol. 2017 (2017): 1096980. doi: https://doi.org/10.1155/2017/1096980
Zhou Y, Chen P, Liu Q, Wang Y, Zhang L, Wu R, Chen J, Yu H, Zhu W, Hu X, Wang JA. Hepatoma-Derived Growth Factor Secreted from Mesenchymal Stem Cells Reduces Myocardial Ischemia-Reperfusion Injury. Stem Cells Int. 2017;2017:1096980. doi: 10.1155/2017/1096980. Epub 2017 Nov 14. PMID: 29358952; PMCID: PMC5735317.
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High-level SAE2 promotes malignant phenotype and predicts outcome in gastric cancer.

American journal of cancer research

Shao DF, Wang XH, Li ZY, Xing XF, Cheng XJ, Guo T, Du H, Hu Y, Dong B, Ding N, Li L, Li S, Li QD, Wen XZ, Zhang LH, Ji JF.
PMID: 25973299
Am J Cancer Res. 2015 Jan 15;5(2):589-602. eCollection 2015.

BACKGROUND: The SUMO pathway has been shown to play an important role in tumorigenesis. This report analyzed the involvement of the sole SUMO-Activating Enzyme Subunit 2 (SAE2) in human gastric cancer (GC) progression and prognosis.METHODS: Expression of SAE2 was...

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Shao DF, Wang XH, Li ZY, et al. High-level SAE2 promotes malignant phenotype and predicts outcome in gastric cancer. Am J Cancer Res. 2015;5(2):589-602
Shao, D. F., Wang, X. H., Li, Z. Y., Xing, X. F., Cheng, X. J., Guo, T., Du, H., Hu, Y., Dong, B., Ding, N., Li, L., Li, S., Li, Q. D., Wen, X. Z., Zhang, L. H., & Ji, J. F. (2015). High-level SAE2 promotes malignant phenotype and predicts outcome in gastric cancer. American journal of cancer research, 5(2), 589-602.
Shao, Duan-Fang, et al. "High-level SAE2 promotes malignant phenotype and predicts outcome in gastric cancer." American journal of cancer research vol. 5,2 (2015): 589-602.
Shao DF, Wang XH, Li ZY, Xing XF, Cheng XJ, Guo T, Du H, Hu Y, Dong B, Ding N, Li L, Li S, Li QD, Wen XZ, Zhang LH, Ji JF. High-level SAE2 promotes malignant phenotype and predicts outcome in gastric cancer. Am J Cancer Res. 2015 Jan 15;5(2):589-602. eCollection 2015. PMID: 25973299; PMCID: PMC4396028.
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Identification of the Replication Origins from Cyanothece ATCC 51142 and Their Interactions with the DnaA Protein: From In Silico to In Vitro Studies.

Frontiers in microbiology

Huang H, Song CC, Yang ZL, Dong Y, Hu YZ, Gao F.
PMID: 26696980
Front Microbiol. 2015 Dec 10;6:1370. doi: 10.3389/fmicb.2015.01370. eCollection 2015.

Based on the complete genome of Cyanothece ATCC 51142, the oriCs of both the circular and linear chromosomes in Cyanothece ATCC 51142 have been predicted by utilizing a web-based system Ori-Finder. Here, we provide experimental support for the results...

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Huang H, Song CC, Yang ZL, et al. Identification of the Replication Origins from Cyanothece ATCC 51142 and Their Interactions with the DnaA Protein: From In Silico to In Vitro Studies. Front Microbiol. 2015;6:1370doi: 10.3389/fmicb.2015.01370.
Huang, H., Song, C. C., Yang, Z. L., Dong, Y., Hu, Y. Z., & Gao, F. (2015). Identification of the Replication Origins from Cyanothece ATCC 51142 and Their Interactions with the DnaA Protein: From In Silico to In Vitro Studies. Frontiers in microbiology, 61370. https://doi.org/10.3389/fmicb.2015.01370
Huang, He, et al. "Identification of the Replication Origins from Cyanothece ATCC 51142 and Their Interactions with the DnaA Protein: From In Silico to In Vitro Studies." Frontiers in microbiology vol. 6 (2015): 1370. doi: https://doi.org/10.3389/fmicb.2015.01370
Huang H, Song CC, Yang ZL, Dong Y, Hu YZ, Gao F. Identification of the Replication Origins from Cyanothece ATCC 51142 and Their Interactions with the DnaA Protein: From In Silico to In Vitro Studies. Front Microbiol. 2015 Dec 10;6:1370. doi: 10.3389/fmicb.2015.01370. eCollection 2015. PMID: 26696980; PMCID: PMC4674748.
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AMPK-mediated cardioprotection of atorvastatin relates to the reduction of apoptosis and activation of autophagy in infarcted rat hearts.

American journal of translational research

Li Q, Dong QT, Yang YJ, Tian XQ, Jin C, Huang PS, Jiang LP, Chen GH.
PMID: 27830000
Am J Transl Res. 2016 Oct 15;8(10):4160-4171. eCollection 2016.

Atorvastatin (ATV) has an important pro-survival role in cardiomyocytes after acute myocardial infarction (AMI). The objectives of this study were to: 1) determine whether ATV could affect autophagy of cardiomyocytes via the AMP-activated protein kinase (AMPK)/mammalian target of rapamycin...

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Li Q, Dong QT, Yang YJ, et al. AMPK-mediated cardioprotection of atorvastatin relates to the reduction of apoptosis and activation of autophagy in infarcted rat hearts. Am J Transl Res. 2016;8(10):4160-4171
Li, Q., Dong, Q. T., Yang, Y. J., Tian, X. Q., Jin, C., Huang, P. S., Jiang, L. P., & Chen, G. H. (2016). AMPK-mediated cardioprotection of atorvastatin relates to the reduction of apoptosis and activation of autophagy in infarcted rat hearts. American journal of translational research, 8(10), 4160-4171.
Li, Qing, et al. "AMPK-mediated cardioprotection of atorvastatin relates to the reduction of apoptosis and activation of autophagy in infarcted rat hearts." American journal of translational research vol. 8,10 (2016): 4160-4171.
Li Q, Dong QT, Yang YJ, Tian XQ, Jin C, Huang PS, Jiang LP, Chen GH. AMPK-mediated cardioprotection of atorvastatin relates to the reduction of apoptosis and activation of autophagy in infarcted rat hearts. Am J Transl Res. 2016 Oct 15;8(10):4160-4171. eCollection 2016. PMID: 27830000; PMCID: PMC5095309.
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Identification of Alzheimer's Disease-Related Genes Based on Data Integration Method.

Frontiers in genetics

Hu Y, Zhao T, Zang T, Zhang Y, Cheng L.
PMID: 30740125
Front Genet. 2019 Jan 25;9:703. doi: 10.3389/fgene.2018.00703. eCollection 2018.

Alzheimer disease (AD) is the fourth major cause of death in the elderly following cancer, heart disease and cerebrovascular disease. Finding candidate causal genes can help in the design of Gene targeted drugs and effectively reduce the risk of...

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Hu Y, Zhao T, Zang T, et al. Identification of Alzheimer's Disease-Related Genes Based on Data Integration Method. Front Genet. 2019;9:703doi: 10.3389/fgene.2018.00703.
Hu, Y., Zhao, T., Zang, T., Zhang, Y., & Cheng, L. (2018). Identification of Alzheimer's Disease-Related Genes Based on Data Integration Method. Frontiers in genetics, 9703. https://doi.org/10.3389/fgene.2018.00703
Hu, Yang, et al. "Identification of Alzheimer's Disease-Related Genes Based on Data Integration Method." Frontiers in genetics vol. 9 (2018): 703. doi: https://doi.org/10.3389/fgene.2018.00703
Hu Y, Zhao T, Zang T, Zhang Y, Cheng L. Identification of Alzheimer's Disease-Related Genes Based on Data Integration Method. Front Genet. 2019 Jan 25;9:703. doi: 10.3389/fgene.2018.00703. eCollection 2018. PMID: 30740125; PMCID: PMC6355707.
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