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The inflammatory cytokine tumor necrosis factor modulates the expression of Salmonella typhimurium effector proteins.

Journal of inflammation (London, England)

Ma J, Zhang YG, Xia Y, Sun J.
PMID: 20704730
J Inflamm (Lond). 2010 Aug 12;7:42. doi: 10.1186/1476-9255-7-42.

Tumor necrosis factor alpha (TNF-alpha)is a host inflammatory factor. Bacteria increase TNF-alpha expression in a variety of human diseases including infectious diseases, inflammatory bowel diseases, and cancer. It is unknown, however, how TNF-alpha directly modulates bacterial protein expression during...

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Ma J, Zhang YG, Xia Y, et al. The inflammatory cytokine tumor necrosis factor modulates the expression of Salmonella typhimurium effector proteins. J Inflamm (Lond). 2010;7:42doi: 10.1186/1476-9255-7-42.
Ma, J., Zhang, Y. G., Xia, Y., & Sun, J. (2010). The inflammatory cytokine tumor necrosis factor modulates the expression of Salmonella typhimurium effector proteins. Journal of inflammation (London, England), 742. https://doi.org/10.1186/1476-9255-7-42
Ma, Jun, et al. "The inflammatory cytokine tumor necrosis factor modulates the expression of Salmonella typhimurium effector proteins." Journal of inflammation (London, England) vol. 7 (2010): 42. doi: https://doi.org/10.1186/1476-9255-7-42
Ma J, Zhang YG, Xia Y, Sun J. The inflammatory cytokine tumor necrosis factor modulates the expression of Salmonella typhimurium effector proteins. J Inflamm (Lond). 2010 Aug 12;7:42. doi: 10.1186/1476-9255-7-42. PMID: 20704730; PMCID: PMC2925363.
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Probiotics, nuclear receptor signaling, and anti-inflammatory pathways.

Gastroenterology research and practice

Yoon SS, Sun J.
PMID: 21808643
Gastroenterol Res Pract. 2011;2011:971938. doi: 10.1155/2011/971938. Epub 2011 Jul 26.

There is increased investigation of the human microbiome as it relates to health and disease. Dysbiosis is implicated in various clinical conditions including inflammatory bowel disease (IBD). Probiotics have been explored as a potential treatment for IBD and other...

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Yoon SS, Sun J. Probiotics, nuclear receptor signaling, and anti-inflammatory pathways. Gastroenterol Res Pract. 2011;2011:971938doi: 10.1155/2011/971938.
Yoon, S. S., & Sun, J. (2011). Probiotics, nuclear receptor signaling, and anti-inflammatory pathways. Gastroenterology research and practice, 2011971938. https://doi.org/10.1155/2011/971938
Yoon, Sonia S, and Sun, Jun. "Probiotics, nuclear receptor signaling, and anti-inflammatory pathways." Gastroenterology research and practice vol. 2011 (2011): 971938. doi: https://doi.org/10.1155/2011/971938
Yoon SS, Sun J. Probiotics, nuclear receptor signaling, and anti-inflammatory pathways. Gastroenterol Res Pract. 2011;2011:971938. doi: 10.1155/2011/971938. Epub 2011 Jul 26. PMID: 21808643; PMCID: PMC3144714.
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Enteric bacteria and cancer stem cells.

Cancers

Sun J.
PMID: 21297903
Cancers (Basel). 2010;3(1):285-97. doi: 10.3390/cancers3010285.

Intestinal bacteria can contribute to cell proliferation and cancer development, particularly in chronic infectious diseases in which bacteria and/or bacterial components might interfere with cell function. The number of microbial cells within the gut lumen is estimated to be...

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Sun J. Enteric bacteria and cancer stem cells. Cancers (Basel). 2010;3(1):285-97doi: 10.3390/cancers3010285.
Sun, J. (2010). Enteric bacteria and cancer stem cells. Cancers, 3(1), 285-97. https://doi.org/10.3390/cancers3010285
Sun, Jun. "Enteric bacteria and cancer stem cells." Cancers vol. 3,1 (2010): 285-97. doi: https://doi.org/10.3390/cancers3010285
Sun J. Enteric bacteria and cancer stem cells. Cancers (Basel). 2010;3(1):285-97. doi: 10.3390/cancers3010285. PMID: 21297903; PMCID: PMC3033586.
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Vitamin D, Vitamin D Receptor, and Tissue Barriers.

Tissue barriers

Zhang YG, Wu S, Sun J.
PMID: 24358453
Tissue Barriers. 2013 Jan 01;1(1). doi: 10.4161/tisb.23118.

Tissue barriers are critical in the pathogenesis of human diseases, such as atopic dermatitis, inflammatory bowel diseases, and various cancers. Preserving or restoring barrier functions of the epithelia cells is a therapeutic strategy to prevent and treat the illness....

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Zhang YG, Wu S, Sun J. Vitamin D, Vitamin D Receptor, and Tissue Barriers. Tissue Barriers. 2013;1(1)doi: 10.4161/tisb.23118.
Zhang, Y. G., Wu, S., & Sun, J. (2013). Vitamin D, Vitamin D Receptor, and Tissue Barriers. Tissue barriers, 1(1), . https://doi.org/10.4161/tisb.23118
Zhang, Yong-Guo, et al. "Vitamin D, Vitamin D Receptor, and Tissue Barriers." Tissue barriers vol. 1,1 (2013). doi: https://doi.org/10.4161/tisb.23118
Zhang YG, Wu S, Sun J. Vitamin D, Vitamin D Receptor, and Tissue Barriers. Tissue Barriers. 2013 Jan 01;1(1). doi: 10.4161/tisb.23118. PMID: 24358453; PMCID: PMC3865708.
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Bacterial protein AvrA stabilizes intestinal epithelial tight junctions via blockage of the C-Jun N-terminal kinase pathway.

Tissue barriers

Zhang Y, Wu S, Ma J, Xia Y, Ai X, Sun J.
PMID: 25838979
Tissue Barriers. 2015 Apr 03;3(1):e972849. doi: 10.4161/21688362.2014.972849. eCollection 2015.

The Salmonella type III secretory system secretes virulence proteins, called effectors. Effectors are responsible for the alteration of tight junctions (TJ) and epithelial functions in intestinal infection and inflammation. In a previous study, we have demonstrated that a bacterial...

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Zhang Y, Wu S, Ma J, et al. Bacterial protein AvrA stabilizes intestinal epithelial tight junctions via blockage of the C-Jun N-terminal kinase pathway. Tissue Barriers. 2015;3(1):e972849doi: 10.4161/21688362.2014.972849.
Zhang, Y., Wu, S., Ma, J., Xia, Y., Ai, X., & Sun, J. (2015). Bacterial protein AvrA stabilizes intestinal epithelial tight junctions via blockage of the C-Jun N-terminal kinase pathway. Tissue barriers, 3(1), e972849. https://doi.org/10.4161/21688362.2014.972849
Zhang, Yongguo, et al. "Bacterial protein AvrA stabilizes intestinal epithelial tight junctions via blockage of the C-Jun N-terminal kinase pathway." Tissue barriers vol. 3,1 (2015): e972849. doi: https://doi.org/10.4161/21688362.2014.972849
Zhang Y, Wu S, Ma J, Xia Y, Ai X, Sun J. Bacterial protein AvrA stabilizes intestinal epithelial tight junctions via blockage of the C-Jun N-terminal kinase pathway. Tissue Barriers. 2015 Apr 03;3(1):e972849. doi: 10.4161/21688362.2014.972849. eCollection 2015. PMID: 25838979; PMCID: PMC4372022.
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Leaky intestine and impaired microbiome in an amyotrophic lateral sclerosis mouse model.

Physiological reports

Wu S, Yi J, Zhang YG, Zhou J, Sun J.
PMID: 25847918
Physiol Rep. 2015 Apr;3(4). doi: 10.14814/phy2.12356.

Emerging evidence has demonstrated that intestinal homeostasis and the microbiome play essential roles in neurological diseases, such as Parkinson's disease. Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disease characterized by a progressive loss of motor neurons and muscle...

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Wu S, Yi J, Zhang YG, et al. Leaky intestine and impaired microbiome in an amyotrophic lateral sclerosis mouse model. Physiol Rep. 2015;3(4)doi: 10.14814/phy2.12356.
Wu, S., Yi, J., Zhang, Y. G., Zhou, J., & Sun, J. (2015). Leaky intestine and impaired microbiome in an amyotrophic lateral sclerosis mouse model. Physiological reports, 3(4), . https://doi.org/10.14814/phy2.12356
Wu, Shaoping, et al. "Leaky intestine and impaired microbiome in an amyotrophic lateral sclerosis mouse model." Physiological reports vol. 3,4 (2015). doi: https://doi.org/10.14814/phy2.12356
Wu S, Yi J, Zhang YG, Zhou J, Sun J. Leaky intestine and impaired microbiome in an amyotrophic lateral sclerosis mouse model. Physiol Rep. 2015 Apr;3(4). doi: 10.14814/phy2.12356. PMID: 25847918; PMCID: PMC4425962.
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Salmonella-infected crypt-derived intestinal organoid culture system for host-bacterial interactions.

Physiological reports

Zhang YG, Wu S, Xia Y, Sun J.
PMID: 25214524
Physiol Rep. 2014 Sep 11;2(9). doi: 10.14814/phy2.12147. Print 2014 Sep 01.

The in vitro analysis of bacterial-epithelial interactions in the intestine has been hampered by a lack of suitable intestinal epithelium culture systems. Here, we report a new experimental model using an organoid culture system to study pathophysiology of bacterial-epithelial...

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Zhang YG, Wu S, Xia Y, et al. Salmonella-infected crypt-derived intestinal organoid culture system for host-bacterial interactions. Physiol Rep. 2014;2(9)doi: 10.14814/phy2.12147.
Zhang, Y. G., Wu, S., Xia, Y., & Sun, J. (2014). Salmonella-infected crypt-derived intestinal organoid culture system for host-bacterial interactions. Physiological reports, 2(9), . https://doi.org/10.14814/phy2.12147
Zhang, Yong-Guo, et al. "Salmonella-infected crypt-derived intestinal organoid culture system for host-bacterial interactions." Physiological reports vol. 2,9 (2014). doi: https://doi.org/10.14814/phy2.12147
Zhang YG, Wu S, Xia Y, Sun J. Salmonella-infected crypt-derived intestinal organoid culture system for host-bacterial interactions. Physiol Rep. 2014 Sep 11;2(9). doi: 10.14814/phy2.12147. Print 2014 Sep 01. PMID: 25214524; PMCID: PMC4270227.
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Suppressed autophagy flux in skeletal muscle of an amyotrophic lateral sclerosis mouse model during disease progression.

Physiological reports

Xiao Y, Ma C, Yi J, Wu S, Luo G, Xu X, Lin PH, Sun J, Zhou J.
PMID: 25602021
Physiol Rep. 2015 Jan 19;3(1). doi: 10.14814/phy2.12271. Print 2015 Jan 01.

Accumulation of abnormal protein inclusions is implicated in motor neuron degeneration in amyotrophic lateral sclerosis (ALS). Autophagy, an intracellular process targeting misfolded proteins and damaged organelles for lysosomal degradation, plays crucial roles in survival and diseased conditions. Efforts were...

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Xiao Y, Ma C, Yi J, et al. Suppressed autophagy flux in skeletal muscle of an amyotrophic lateral sclerosis mouse model during disease progression. Physiol Rep. 2015;3(1)doi: 10.14814/phy2.12271.
Xiao, Y., Ma, C., Yi, J., Wu, S., Luo, G., Xu, X., Lin, P. H., Sun, J., & Zhou, J. (2015). Suppressed autophagy flux in skeletal muscle of an amyotrophic lateral sclerosis mouse model during disease progression. Physiological reports, 3(1), . https://doi.org/10.14814/phy2.12271
Xiao, Yajuan, et al. "Suppressed autophagy flux in skeletal muscle of an amyotrophic lateral sclerosis mouse model during disease progression." Physiological reports vol. 3,1 (2015). doi: https://doi.org/10.14814/phy2.12271
Xiao Y, Ma C, Yi J, Wu S, Luo G, Xu X, Lin PH, Sun J, Zhou J. Suppressed autophagy flux in skeletal muscle of an amyotrophic lateral sclerosis mouse model during disease progression. Physiol Rep. 2015 Jan 19;3(1). doi: 10.14814/phy2.12271. Print 2015 Jan 01. PMID: 25602021; PMCID: PMC4387765.
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Probiotic Lactobacillus rhamnosus GG mono-association suppresses human rotavirus-induced autophagy in the gnotobiotic piglet intestine.

Gut pathogens

Wu S, Yuan L, Zhang Y, Liu F, Li G, Wen K, Kocher J, Yang X, Sun J.
PMID: 23924832
Gut Pathog. 2013 Aug 07;5(1):22. doi: 10.1186/1757-4749-5-22.

BACKGROUND: Human rotavirus (HRV) is the most important cause of severe diarrhea in infants and young children. Probiotic Lactobacillus rhamnosus GG (LGG) reduces rotavirus infection and diarrhea. However, the molecular mechanisms of LGG-mediated protection from rotavirus infection are poorly...

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Wu S, Yuan L, Zhang Y, et al. Probiotic Lactobacillus rhamnosus GG mono-association suppresses human rotavirus-induced autophagy in the gnotobiotic piglet intestine. Gut Pathog. 2013;5(1):22doi: 10.1186/1757-4749-5-22.
Wu, S., Yuan, L., Zhang, Y., Liu, F., Li, G., Wen, K., Kocher, J., Yang, X., & Sun, J. (2013). Probiotic Lactobacillus rhamnosus GG mono-association suppresses human rotavirus-induced autophagy in the gnotobiotic piglet intestine. Gut pathogens, 5(1), 22. https://doi.org/10.1186/1757-4749-5-22
Wu, Shaoping, et al. "Probiotic Lactobacillus rhamnosus GG mono-association suppresses human rotavirus-induced autophagy in the gnotobiotic piglet intestine." Gut pathogens vol. 5,1 (2013): 22. doi: https://doi.org/10.1186/1757-4749-5-22
Wu S, Yuan L, Zhang Y, Liu F, Li G, Wen K, Kocher J, Yang X, Sun J. Probiotic Lactobacillus rhamnosus GG mono-association suppresses human rotavirus-induced autophagy in the gnotobiotic piglet intestine. Gut Pathog. 2013 Aug 07;5(1):22. doi: 10.1186/1757-4749-5-22. PMID: 23924832; PMCID: PMC3750464.
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The Vitamin D Receptor, Inflammatory Bowel Diseases, and Colon Cancer.

Current colorectal cancer reports

Lu R, Wu S, Xia Y, Sun J.
PMID: 23814529
Curr Colorectal Cancer Rep. 2012 Mar 01;8(1):57-65. doi: 10.1007/s11888-011-0114-1.

The nuclear receptor is an emerging therapeutic target in various human diseases. Vitamin D receptor (VDR), a nuclear receptor, mediates the biological functions of vitamin D. Classically, vitamin D is recognized as an essential contributor to mineral and bone...

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Lu R, Wu S, Xia Y, et al. The Vitamin D Receptor, Inflammatory Bowel Diseases, and Colon Cancer. Curr Colorectal Cancer Rep. 2012;8(1):57-65doi: 10.1007/s11888-011-0114-1.
Lu, R., Wu, S., Xia, Y., & Sun, J. (2012). The Vitamin D Receptor, Inflammatory Bowel Diseases, and Colon Cancer. Current colorectal cancer reports, 8(1), 57-65. https://doi.org/10.1007/s11888-011-0114-1
Lu, Rong, et al. "The Vitamin D Receptor, Inflammatory Bowel Diseases, and Colon Cancer." Current colorectal cancer reports vol. 8,1 (2012): 57-65. doi: https://doi.org/10.1007/s11888-011-0114-1
Lu R, Wu S, Xia Y, Sun J. The Vitamin D Receptor, Inflammatory Bowel Diseases, and Colon Cancer. Curr Colorectal Cancer Rep. 2012 Mar 01;8(1):57-65. doi: 10.1007/s11888-011-0114-1. PMID: 23814529; PMCID: PMC3693087.
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Exploring gut microbes in human health and disease: Pushing the envelope.

Genes & diseases

Sun J, Chang EB.
PMID: 25642449
Genes Dis. 2014 Dec;1(2):132-139. doi: 10.1016/j.gendis.2014.08.001.

Humans have coevolved with their microbes over thousands of years, but this relationship, is now being dramatically affected by shifts in the collective human microbiome resulting from changes in the environment and societal norms. Resulting perturbations of intestinal host-microbe...

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Sun J, Chang EB. Exploring gut microbes in human health and disease: Pushing the envelope. Genes Dis. 2014;1(2):132-139doi: 10.1016/j.gendis.2014.08.001.
Sun, J., & Chang, E. B. (2014). Exploring gut microbes in human health and disease: Pushing the envelope. Genes & diseases, 1(2), 132-139. https://doi.org/10.1016/j.gendis.2014.08.001
Sun, Jun, and Chang, Eugene B. "Exploring gut microbes in human health and disease: Pushing the envelope." Genes & diseases vol. 1,2 (2014): 132-139. doi: https://doi.org/10.1016/j.gendis.2014.08.001
Sun J, Chang EB. Exploring gut microbes in human health and disease: Pushing the envelope. Genes Dis. 2014 Dec;1(2):132-139. doi: 10.1016/j.gendis.2014.08.001. PMID: 25642449; PMCID: PMC4310008.
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[No title available]

Diseases (Basel, Switzerland)

Zha L, Garrett S, Sun J.
PMID: 30857369
Diseases. 2019 Mar 10;7(1). doi: 10.3390/diseases7010028.

No abstract available.

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Zha L, Garrett S, Sun J. . Diseases. 2019;7(1)doi: 10.3390/diseases7010028.
Zha, L., Garrett, S., & Sun, J. (2019). . Diseases (Basel, Switzerland), 7(1), . https://doi.org/10.3390/diseases7010028
Zha, Lang, et al. "." Diseases (Basel, Switzerland) vol. 7,1 (2019). doi: https://doi.org/10.3390/diseases7010028
Zha L, Garrett S, Sun J. . Diseases. 2019 Mar 10;7(1). doi: 10.3390/diseases7010028. PMID: 30857369; PMCID: PMC6473780.
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Showing 1 to 12 of 12 entries