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Obesity and inflammation markers in relation to leukocyte telomere length in a cross-sectional study of persons with Barrett's esophagus.

BMC obesity

Hardikar S, Song X, Risques RA, Montine TJ, Duggan C, Blount PL, Reid BJ, Anderson GL, Kratz M, White E, Vaughan TL.
PMID: 26380096
BMC Obes. 2015 Sep 10;2:32. doi: 10.1186/s40608-015-0063-3. eCollection 2015.

BACKGROUND: Telomere shortening is associated with increasing age, male gender and lifestyle factors such as obesity and smoking. Inflammation has also been implicated in cellular senescence and may promote telomere shortening in chronic conditions such as obesity and diabetes....

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Hardikar S, Song X, Risques RA, et al. Obesity and inflammation markers in relation to leukocyte telomere length in a cross-sectional study of persons with Barrett's esophagus. BMC Obes. 2015;2:32doi: 10.1186/s40608-015-0063-3.
Hardikar, S., Song, X., Risques, R. A., Montine, T. J., Duggan, C., Blount, P. L., Reid, B. J., Anderson, G. L., Kratz, M., White, E., & Vaughan, T. L. (2015). Obesity and inflammation markers in relation to leukocyte telomere length in a cross-sectional study of persons with Barrett's esophagus. BMC obesity, 232. https://doi.org/10.1186/s40608-015-0063-3
Hardikar, Sheetal, et al. "Obesity and inflammation markers in relation to leukocyte telomere length in a cross-sectional study of persons with Barrett's esophagus." BMC obesity vol. 2 (2015): 32. doi: https://doi.org/10.1186/s40608-015-0063-3
Hardikar S, Song X, Risques RA, Montine TJ, Duggan C, Blount PL, Reid BJ, Anderson GL, Kratz M, White E, Vaughan TL. Obesity and inflammation markers in relation to leukocyte telomere length in a cross-sectional study of persons with Barrett's esophagus. BMC Obes. 2015 Sep 10;2:32. doi: 10.1186/s40608-015-0063-3. eCollection 2015. PMID: 26380096; PMCID: PMC4566310.
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Associations between null mutations in GSTT1 and GSTM1, the GSTP1 Ile(105)Val polymorphism, and mortality in breast cancer survivors.

SpringerPlus

Duggan C, Ballard-Barbash R, Baumgartner RN, Baumgartner KB, Bernstein L, McTiernan A.
PMID: 24083102
Springerplus. 2013 Sep 11;2:450. doi: 10.1186/2193-1801-2-450. eCollection 2013.

PURPOSE: Here we assessed associations between null mutations in glutathione-S-transferase (GST)T1 and GSTM1 genes, and the rs1695 polymorphism in GSTP1 (Ile(105)Val), and risk of breast cancer-specific (n=45) and all-cause (n=99) mortality in a multiethnic, prospective cohort of 533 women...

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Duggan C, Ballard-Barbash R, Baumgartner RN, et al. Associations between null mutations in GSTT1 and GSTM1, the GSTP1 Ile(105)Val polymorphism, and mortality in breast cancer survivors. Springerplus. 2013;2:450doi: 10.1186/2193-1801-2-450.
Duggan, C., Ballard-Barbash, R., Baumgartner, R. N., Baumgartner, K. B., Bernstein, L., & McTiernan, A. (2013). Associations between null mutations in GSTT1 and GSTM1, the GSTP1 Ile(105)Val polymorphism, and mortality in breast cancer survivors. SpringerPlus, 2450. https://doi.org/10.1186/2193-1801-2-450
Duggan, Catherine, et al. "Associations between null mutations in GSTT1 and GSTM1, the GSTP1 Ile(105)Val polymorphism, and mortality in breast cancer survivors." SpringerPlus vol. 2 (2013): 450. doi: https://doi.org/10.1186/2193-1801-2-450
Duggan C, Ballard-Barbash R, Baumgartner RN, Baumgartner KB, Bernstein L, McTiernan A. Associations between null mutations in GSTT1 and GSTM1, the GSTP1 Ile(105)Val polymorphism, and mortality in breast cancer survivors. Springerplus. 2013 Sep 11;2:450. doi: 10.1186/2193-1801-2-450. eCollection 2013. PMID: 24083102; PMCID: PMC3786079.
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Physiology of sedentary behavior and its relationship to health outcomes.

Medicine and science in sports and exercise

Thyfault JP, Du M, Kraus WE, Levine JA, Booth FW.
PMID: 25222820
Med Sci Sports Exerc. 2015 Jun;47(6):1301-5. doi: 10.1249/MSS.0000000000000518.

PURPOSE: This article reports on the findings and recommendations of the "Physiology of Sedentary Behavior and Its Relationship to Health Outcomes" group, a part of a larger workshop entitled Sedentary Behavior: Identifying Research Priorities sponsored by the National Heart,...

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Thyfault JP, Du M, Kraus WE, et al. Physiology of sedentary behavior and its relationship to health outcomes. Med Sci Sports Exerc. 2015;47(6):1301-5doi: 10.1249/MSS.0000000000000518.
Thyfault, J. P., Du, M., Kraus, W. E., Levine, J. A., & Booth, F. W. (2015). Physiology of sedentary behavior and its relationship to health outcomes. Medicine and science in sports and exercise, 47(6), 1301-5. https://doi.org/10.1249/MSS.0000000000000518
Thyfault, John P, et al. "Physiology of sedentary behavior and its relationship to health outcomes." Medicine and science in sports and exercise vol. 47,6 (2015): 1301-5. doi: https://doi.org/10.1249/MSS.0000000000000518
Thyfault JP, Du M, Kraus WE, Levine JA, Booth FW. Physiology of sedentary behavior and its relationship to health outcomes. Med Sci Sports Exerc. 2015 Jun;47(6):1301-5. doi: 10.1249/MSS.0000000000000518. PMID: 25222820; PMCID: PMC4362885.
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Randomized trial of exercise in sedentary middle aged women: effects on quality of life.

The international journal of behavioral nutrition and physical activity

Bowen DJ, Fesinmeyer MD, Yasui Y, Tworoger S, Ulrich CM, Irwin ML, Rudolph RE, LaCroix KL, Schwartz RR, McTiernan A.
PMID: 17020620
Int J Behav Nutr Phys Act. 2006 Oct 04;3:34. doi: 10.1186/1479-5868-3-34.

Increasing physical activity is currently considered to be a possible prevention strategy for cancer, obesity, and cardiovascular disease, either alone or in combination with dietary changes. This paper presents results of a randomized trial of moderate-to-vigorous intensity exercise in...

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Bowen DJ, Fesinmeyer MD, Yasui Y, et al. Randomized trial of exercise in sedentary middle aged women: effects on quality of life. Int J Behav Nutr Phys Act. 2006;3:34doi: 10.1186/1479-5868-3-34.
Bowen, D. J., Fesinmeyer, M. D., Yasui, Y., Tworoger, S., Ulrich, C. M., Irwin, M. L., Rudolph, R. E., LaCroix, K. L., Schwartz, R. R., & McTiernan, A. (2006). Randomized trial of exercise in sedentary middle aged women: effects on quality of life. The international journal of behavioral nutrition and physical activity, 334. https://doi.org/10.1186/1479-5868-3-34
Bowen, Deborah J, et al. "Randomized trial of exercise in sedentary middle aged women: effects on quality of life." The international journal of behavioral nutrition and physical activity vol. 3 (2006): 34. doi: https://doi.org/10.1186/1479-5868-3-34
Bowen DJ, Fesinmeyer MD, Yasui Y, Tworoger S, Ulrich CM, Irwin ML, Rudolph RE, LaCroix KL, Schwartz RR, McTiernan A. Randomized trial of exercise in sedentary middle aged women: effects on quality of life. Int J Behav Nutr Phys Act. 2006 Oct 04;3:34. doi: 10.1186/1479-5868-3-34. PMID: 17020620; PMCID: PMC1618856.
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Phospholipase A2G1B polymorphisms and risk of colorectal neoplasia.

International journal of molecular epidemiology and genetics

Abbenhardt C, Poole EM, Kulmacz RJ, Xiao L, Curtin K, Galbraith RL, Duggan D, Hsu L, Makar KW, Caan BJ, Koepl L, Owen RW, Scherer D, Carlson CS, Potter JD, Slattery ML, Ulrich CM.
PMID: 24046806
Int J Mol Epidemiol Genet. 2013 Sep 12;4(3):140-9. eCollection 2013.

Pancreatic phospholipase A2, product of PLA2G1B, catalyzes the release of fatty acids from dietary phospholipids.Diet is the ultimate source of arachidonic acid in cellular phospholipids, precursor of eicosanoid signaling molecules, linked to inflammation, cell proliferation and colorectal carcinogenesis. We...

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Abbenhardt C, Poole EM, Kulmacz RJ, et al. Phospholipase A2G1B polymorphisms and risk of colorectal neoplasia. Int J Mol Epidemiol Genet. 2013;4(3):140-9
Abbenhardt, C., Poole, E. M., Kulmacz, R. J., Xiao, L., Curtin, K., Galbraith, R. L., Duggan, D., Hsu, L., Makar, K. W., Caan, B. J., Koepl, L., Owen, R. W., Scherer, D., Carlson, C. S., Potter, J. D., Slattery, M. L., & Ulrich, C. M. (2013). Phospholipase A2G1B polymorphisms and risk of colorectal neoplasia. International journal of molecular epidemiology and genetics, 4(3), 140-9.
Abbenhardt, Clare, et al. "Phospholipase A2G1B polymorphisms and risk of colorectal neoplasia." International journal of molecular epidemiology and genetics vol. 4,3 (2013): 140-9.
Abbenhardt C, Poole EM, Kulmacz RJ, Xiao L, Curtin K, Galbraith RL, Duggan D, Hsu L, Makar KW, Caan BJ, Koepl L, Owen RW, Scherer D, Carlson CS, Potter JD, Slattery ML, Ulrich CM. Phospholipase A2G1B polymorphisms and risk of colorectal neoplasia. Int J Mol Epidemiol Genet. 2013 Sep 12;4(3):140-9. eCollection 2013. PMID: 24046806; PMCID: PMC3773565.
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Genetic variability in EGFR, Src and HER2 and risk of colorectal adenoma and cancer.

International journal of molecular epidemiology and genetics

Poole EM, Curtin K, Hsu L, Kulmacz RJ, Duggan DJ, Makar KW, Xiao L, Carlson CS, Slattery ML, Caan BJ, Potter JD, Ulrich CM.
PMID: 22199994
Int J Mol Epidemiol Genet. 2011;2(4):300-15. Epub 2011 Dec 03.

The EGFR signaling pathway is involved in carcinogenesis at multiple sites, particularly colorectal cancer, and is a target of colorectal cancer chemotherapy. EGFR signaling is linked to pro-carcinogenic mechanisms, including cell proliferation, survival, angiogenesis, and more recently prostaglandin synthesis....

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Poole EM, Curtin K, Hsu L, et al. Genetic variability in EGFR, Src and HER2 and risk of colorectal adenoma and cancer. Int J Mol Epidemiol Genet. 2011;2(4):300-15
Poole, E. M., Curtin, K., Hsu, L., Kulmacz, R. J., Duggan, D. J., Makar, K. W., Xiao, L., Carlson, C. S., Slattery, M. L., Caan, B. J., Potter, J. D., & Ulrich, C. M. (2011). Genetic variability in EGFR, Src and HER2 and risk of colorectal adenoma and cancer. International journal of molecular epidemiology and genetics, 2(4), 300-15.
Poole, Elizabeth M, et al. "Genetic variability in EGFR, Src and HER2 and risk of colorectal adenoma and cancer." International journal of molecular epidemiology and genetics vol. 2,4 (2011): 300-15.
Poole EM, Curtin K, Hsu L, Kulmacz RJ, Duggan DJ, Makar KW, Xiao L, Carlson CS, Slattery ML, Caan BJ, Potter JD, Ulrich CM. Genetic variability in EGFR, Src and HER2 and risk of colorectal adenoma and cancer. Int J Mol Epidemiol Genet. 2011;2(4):300-15. Epub 2011 Dec 03. PMID: 22199994; PMCID: PMC3244110.
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Showing 1 to 6 of 6 entries