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Showing 1 to 12 of 28 entries
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Multi-focal HIFU reduces cavitation in mild-hyperthermia.

Journal of therapeutic ultrasound

Chaplin V, Caskey CF.
PMID: 28413682
J Ther Ultrasound. 2017 Apr 13;5:12. doi: 10.1186/s40349-017-0089-8. eCollection 2017.

BACKGROUND: Mild-hyperthermia therapy (40-45 °C) with high-intensity focused ultrasound (HIFU) is a technique being considered in a number of different treatments such as thermally activated drug delivery, immune-stimulation, and as a chemotherapy adjuvant. Mechanical damage and loss of cell...

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Chaplin V, Caskey CF. Multi-focal HIFU reduces cavitation in mild-hyperthermia. J Ther Ultrasound. 2017;5:12doi: 10.1186/s40349-017-0089-8.
Chaplin, V., & Caskey, C. F. (2017). Multi-focal HIFU reduces cavitation in mild-hyperthermia. Journal of therapeutic ultrasound, 512. https://doi.org/10.1186/s40349-017-0089-8
Chaplin, Vandiver, and Caskey, Charles F. "Multi-focal HIFU reduces cavitation in mild-hyperthermia." Journal of therapeutic ultrasound vol. 5 (2017): 12. doi: https://doi.org/10.1186/s40349-017-0089-8
Chaplin V, Caskey CF. Multi-focal HIFU reduces cavitation in mild-hyperthermia. J Ther Ultrasound. 2017 Apr 13;5:12. doi: 10.1186/s40349-017-0089-8. eCollection 2017. PMID: 28413682; PMCID: PMC5390440.
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Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment.

Molecular metabolism

Hill AA, Anderson-Baucum EK, Kennedy AJ, Webb CD, Yull FE, Hasty AH.
PMID: 26779432
Mol Metab. 2015 Jul 28;4(10):665-77. doi: 10.1016/j.molmet.2015.07.005. eCollection 2015 Oct.

OBJECTIVE: Macrophage accumulation in adipose tissue (AT) during obesity contributes to inflammation and insulin resistance. Recruitment of monocytes to obese AT has been the most studied mechanism explaining this accumulation. However, recent evidence suggests that recruitment-independent mechanisms may also...

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Hill AA, Anderson-Baucum EK, Kennedy AJ, et al. Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment. Mol Metab. 2015;4(10):665-77doi: 10.1016/j.molmet.2015.07.005.
Hill, A. A., Anderson-Baucum, E. K., Kennedy, A. J., Webb, C. D., Yull, F. E., & Hasty, A. H. (2015). Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment. Molecular metabolism, 4(10), 665-77. https://doi.org/10.1016/j.molmet.2015.07.005
Hill, Andrea A, et al. "Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment." Molecular metabolism vol. 4,10 (2015): 665-77. doi: https://doi.org/10.1016/j.molmet.2015.07.005
Hill AA, Anderson-Baucum EK, Kennedy AJ, Webb CD, Yull FE, Hasty AH. Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment. Mol Metab. 2015 Jul 28;4(10):665-77. doi: 10.1016/j.molmet.2015.07.005. eCollection 2015 Oct. PMID: 26779432; PMCID: PMC4588436.
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MTG16 is a tumor suppressor in colitis-associated carcinoma.

JCI insight

McDonough EM, Barrett CW, Parang B, Mittal MK, Smith JJ, Bradley AM, Choksi YA, Coburn LA, Short SP, Thompson JJ, Zhang B, Poindexter SV, Fischer MA, Chen X, Li J, Revetta FL, Naik R, Washington MK, Rosen MJ, Hiebert SW, Wilson KT, Williams CS.
PMID: 28814670
JCI Insight. 2017 Aug 17;2(16). doi: 10.1172/jci.insight.78210. eCollection 2017 Aug 17.

MTG16 is a member of the myeloid translocation gene (MTG) family of transcriptional corepressors. While MTGs were originally identified in chromosomal translocations in acute myeloid leukemia, recent studies have uncovered a role in intestinal biology. For example, Mtg16-/- mice...

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McDonough EM, Barrett CW, Parang B, et al. MTG16 is a tumor suppressor in colitis-associated carcinoma. JCI Insight. 2017;2(16)doi: 10.1172/jci.insight.78210.
McDonough, E. M., Barrett, C. W., Parang, B., Mittal, M. K., Smith, J. J., Bradley, A. M., Choksi, Y. A., Coburn, L. A., Short, S. P., Thompson, J. J., Zhang, B., Poindexter, S. V., Fischer, M. A., Chen, X., Li, J., Revetta, F. L., Naik, R., Washington, M. K., Rosen, M. J., Hiebert, S. W., Wilson, K. T., & Williams, C. S. (2017). MTG16 is a tumor suppressor in colitis-associated carcinoma. JCI insight, 2(16), . https://doi.org/10.1172/jci.insight.78210
McDonough, Elizabeth M, et al. "MTG16 is a tumor suppressor in colitis-associated carcinoma." JCI insight vol. 2,16 (2017). doi: https://doi.org/10.1172/jci.insight.78210
McDonough EM, Barrett CW, Parang B, Mittal MK, Smith JJ, Bradley AM, Choksi YA, Coburn LA, Short SP, Thompson JJ, Zhang B, Poindexter SV, Fischer MA, Chen X, Li J, Revetta FL, Naik R, Washington MK, Rosen MJ, Hiebert SW, Wilson KT, Williams CS. MTG16 is a tumor suppressor in colitis-associated carcinoma. JCI Insight. 2017 Aug 17;2(16). doi: 10.1172/jci.insight.78210. eCollection 2017 Aug 17. PMID: 28814670; PMCID: PMC5621889.
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Integrin αvβ3 Signaling in Tumor-Induced Bone Disease.

Cancers

Kwakwa KA, Sterling JA.
PMID: 28698458
Cancers (Basel). 2017 Jul 08;9(7). doi: 10.3390/cancers9070084.

Tumor-induced bone disease is common among patients with advanced solid cancers, especially those with breast, prostate, and lung malignancies. The tendency of these cancers to metastasize to bone and induce bone destruction is, in part, due to alterations in...

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Kwakwa KA, Sterling JA. Integrin αvβ3 Signaling in Tumor-Induced Bone Disease. Cancers (Basel). 2017;9(7)doi: 10.3390/cancers9070084.
Kwakwa, K. A., & Sterling, J. A. (2017). Integrin αvβ3 Signaling in Tumor-Induced Bone Disease. Cancers, 9(7), . https://doi.org/10.3390/cancers9070084
Kwakwa, Kristin A, and Sterling, Julie A. "Integrin αvβ3 Signaling in Tumor-Induced Bone Disease." Cancers vol. 9,7 (2017). doi: https://doi.org/10.3390/cancers9070084
Kwakwa KA, Sterling JA. Integrin αvβ3 Signaling in Tumor-Induced Bone Disease. Cancers (Basel). 2017 Jul 08;9(7). doi: 10.3390/cancers9070084. PMID: 28698458; PMCID: PMC5532620.
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Basement membrane ultrastructure and component localization data from uterine tissues during early mouse pregnancy.

Data in brief

Jones-Paris CR, Paria S, Berg T, Saus J, Bhave G, Paria BC, Hudson BG.
PMID: 27896299
Data Brief. 2016 Nov 05;9:931-939. doi: 10.1016/j.dib.2016.10.033. eCollection 2016 Dec.

Basement membranes (BMs) are specialized extracellular scaffolds that provide architecture and modulate cell behaviors in tissues, such as fat, muscle, endothelium, endometrium, and decidua. Properties of BMs are maintained in homeostasis for most adult tissues. However, BM ultrastructure, composition,...

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Jones-Paris CR, Paria S, Berg T, et al. Basement membrane ultrastructure and component localization data from uterine tissues during early mouse pregnancy. Data Brief. 2016;9:931-939doi: 10.1016/j.dib.2016.10.033.
Jones-Paris, C. R., Paria, S., Berg, T., Saus, J., Bhave, G., Paria, B. C., & Hudson, B. G. (2016). Basement membrane ultrastructure and component localization data from uterine tissues during early mouse pregnancy. Data in brief, 9931-939. https://doi.org/10.1016/j.dib.2016.10.033
Jones-Paris, Celestial R, et al. "Basement membrane ultrastructure and component localization data from uterine tissues during early mouse pregnancy." Data in brief vol. 9 (2016): 931-939. doi: https://doi.org/10.1016/j.dib.2016.10.033
Jones-Paris CR, Paria S, Berg T, Saus J, Bhave G, Paria BC, Hudson BG. Basement membrane ultrastructure and component localization data from uterine tissues during early mouse pregnancy. Data Brief. 2016 Nov 05;9:931-939. doi: 10.1016/j.dib.2016.10.033. eCollection 2016 Dec. PMID: 27896299; PMCID: PMC5118616.
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Therapeutic inhibition of Mcl-1 blocks cell survival in estrogen receptor-positive breast cancers.

Oncotarget

Williams MM, Elion DL, Rahman B, Hicks DJ, Sanchez V, Cook RS.
PMID: 31595181
Oncotarget. 2019 Oct 09;10(52):5389-5402. doi: 10.18632/oncotarget.27070. eCollection 2019 Sep 10.

Cancers often overexpress anti-apoptotic Bcl-2 proteins for cell death evasion, a recognized hallmark of cancer progression. While estrogen receptor (ER)-α+ breast cancers express high levels of three anti-apoptotic Bcl-2 family members (Bcl-2, Bcl-xL, and Mcl-1), pharmacological inhibition of Bcl-2...

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Williams MM, Elion DL, Rahman B, et al. Therapeutic inhibition of Mcl-1 blocks cell survival in estrogen receptor-positive breast cancers. Oncotarget. 2019;10(52):5389-5402doi: 10.18632/oncotarget.27070.
Williams, M. M., Elion, D. L., Rahman, B., Hicks, D. J., Sanchez, V., & Cook, R. S. (2019). Therapeutic inhibition of Mcl-1 blocks cell survival in estrogen receptor-positive breast cancers. Oncotarget, 10(52), 5389-5402. https://doi.org/10.18632/oncotarget.27070
Williams, Michelle M, et al. "Therapeutic inhibition of Mcl-1 blocks cell survival in estrogen receptor-positive breast cancers." Oncotarget vol. 10,52 (2019): 5389-5402. doi: https://doi.org/10.18632/oncotarget.27070
Williams MM, Elion DL, Rahman B, Hicks DJ, Sanchez V, Cook RS. Therapeutic inhibition of Mcl-1 blocks cell survival in estrogen receptor-positive breast cancers. Oncotarget. 2019 Oct 09;10(52):5389-5402. doi: 10.18632/oncotarget.27070. eCollection 2019 Sep 10. PMID: 31595181; PMCID: PMC6739218.
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Interleukin-6 Deficiency Attenuates Retinal Ganglion Cell Axonopathy and Glaucoma-Related Vision Loss.

Frontiers in neuroscience

Echevarria FD, Formichella CR, Sappington RM.
PMID: 28620279
Front Neurosci. 2017 May 31;11:318. doi: 10.3389/fnins.2017.00318. eCollection 2017.

The pleotropic cytokine interleukin-6 (IL-6) is implicated in retinal ganglion cell (RGC) survival and degeneration, including that associated with glaucoma. IL-6 protects RGCs from pressure-induced apoptosis

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Echevarria FD, Formichella CR, Sappington RM. Interleukin-6 Deficiency Attenuates Retinal Ganglion Cell Axonopathy and Glaucoma-Related Vision Loss. Front Neurosci. 2017;11:318doi: 10.3389/fnins.2017.00318.
Echevarria, F. D., Formichella, C. R., & Sappington, R. M. (2017). Interleukin-6 Deficiency Attenuates Retinal Ganglion Cell Axonopathy and Glaucoma-Related Vision Loss. Frontiers in neuroscience, 11318. https://doi.org/10.3389/fnins.2017.00318
Echevarria, Franklin D, et al. "Interleukin-6 Deficiency Attenuates Retinal Ganglion Cell Axonopathy and Glaucoma-Related Vision Loss." Frontiers in neuroscience vol. 11 (2017): 318. doi: https://doi.org/10.3389/fnins.2017.00318
Echevarria FD, Formichella CR, Sappington RM. Interleukin-6 Deficiency Attenuates Retinal Ganglion Cell Axonopathy and Glaucoma-Related Vision Loss. Front Neurosci. 2017 May 31;11:318. doi: 10.3389/fnins.2017.00318. eCollection 2017. PMID: 28620279; PMCID: PMC5450377.
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Insulin detemir attenuates food intake, body weight gain and fat mass gain in diet-induced obese Sprague-Dawley rats.

Nutrition & diabetes

Rojas JM, Printz RL, Niswender KD.
PMID: 23449422
Nutr Diabetes. 2011 Jul 04;1:e10. doi: 10.1038/nutd.2011.6.

OBJECTIVE: Initiation and intensification of insulin therapy commonly causes weight gain, a barrier to therapy. A contrasting body of evidence indicates that insulin functions as an adiposity negative feedback signal and reduces food intake, weight gain and adiposity via...

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Rojas JM, Printz RL, Niswender KD. Insulin detemir attenuates food intake, body weight gain and fat mass gain in diet-induced obese Sprague-Dawley rats. Nutr Diabetes. 2011;1:e10doi: 10.1038/nutd.2011.6.
Rojas, J. M., Printz, R. L., & Niswender, K. D. (2011). Insulin detemir attenuates food intake, body weight gain and fat mass gain in diet-induced obese Sprague-Dawley rats. Nutrition & diabetes, 1e10. https://doi.org/10.1038/nutd.2011.6
Rojas, J M, et al. "Insulin detemir attenuates food intake, body weight gain and fat mass gain in diet-induced obese Sprague-Dawley rats." Nutrition & diabetes vol. 1 (2011): e10. doi: https://doi.org/10.1038/nutd.2011.6
Rojas JM, Printz RL, Niswender KD. Insulin detemir attenuates food intake, body weight gain and fat mass gain in diet-induced obese Sprague-Dawley rats. Nutr Diabetes. 2011 Jul 04;1:e10. doi: 10.1038/nutd.2011.6. PMID: 23449422; PMCID: PMC3302138.
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The Role of Hedgehog Signaling in Tumor Induced Bone Disease.

Cancers

Cannonier SA, Sterling JA.
PMID: 26343726
Cancers (Basel). 2015 Aug 26;7(3):1658-83. doi: 10.3390/cancers7030856.

Despite significant progress in cancer treatments, tumor induced bone disease continues to cause significant morbidities. While tumors show distinct mutations and clinical characteristics, they behave similarly once they establish in bone. Tumors can metastasize to bone from distant sites...

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Cannonier SA, Sterling JA. The Role of Hedgehog Signaling in Tumor Induced Bone Disease. Cancers (Basel). 2015;7(3):1658-83doi: 10.3390/cancers7030856.
Cannonier, S. A., & Sterling, J. A. (2015). The Role of Hedgehog Signaling in Tumor Induced Bone Disease. Cancers, 7(3), 1658-83. https://doi.org/10.3390/cancers7030856
Cannonier, Shellese A, and Sterling, Julie A. "The Role of Hedgehog Signaling in Tumor Induced Bone Disease." Cancers vol. 7,3 (2015): 1658-83. doi: https://doi.org/10.3390/cancers7030856
Cannonier SA, Sterling JA. The Role of Hedgehog Signaling in Tumor Induced Bone Disease. Cancers (Basel). 2015 Aug 26;7(3):1658-83. doi: 10.3390/cancers7030856. PMID: 26343726; PMCID: PMC4586789.
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Assembly and Turnover of Caveolae: What Do We Really Know?.

Frontiers in cell and developmental biology

Han B, Copeland CA, Tiwari A, Kenworthy AK.
PMID: 27446919
Front Cell Dev Biol. 2016 Jun 27;4:68. doi: 10.3389/fcell.2016.00068. eCollection 2016.

In addition to containing highly dynamic nanoscale domains, the plasma membranes of many cell types are decorated with caveolae, flask-shaped domains enriched in the structural protein caveolin-1 (Cav1). The importance of caveolae in numerous cellular functions and processes has...

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Han B, Copeland CA, Tiwari A, et al. Assembly and Turnover of Caveolae: What Do We Really Know?. Front Cell Dev Biol. 2016;4:68doi: 10.3389/fcell.2016.00068.
Han, B., Copeland, C. A., Tiwari, A., & Kenworthy, A. K. (2016). Assembly and Turnover of Caveolae: What Do We Really Know?. Frontiers in cell and developmental biology, 468. https://doi.org/10.3389/fcell.2016.00068
Han, Bing, et al. "Assembly and Turnover of Caveolae: What Do We Really Know?." Frontiers in cell and developmental biology vol. 4 (2016): 68. doi: https://doi.org/10.3389/fcell.2016.00068
Han B, Copeland CA, Tiwari A, Kenworthy AK. Assembly and Turnover of Caveolae: What Do We Really Know?. Front Cell Dev Biol. 2016 Jun 27;4:68. doi: 10.3389/fcell.2016.00068. eCollection 2016. PMID: 27446919; PMCID: PMC4921483.
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Stressor-dependent Alterations in Glycoprotein 130: Implications for Glial Cell Reactivity, Cytokine Signaling and Ganglion Cell Health in Glaucoma.

Journal of clinical & experimental ophthalmology

Echevarria F, Walker C, Abella S, Won M, Sappington R.
PMID: 25018894
J Clin Exp Ophthalmol. 2013 Jun 30;4(3). doi: 10.4172/2155-9570.1000286.

OBJECTIVE: The interleukin-6 (IL-6) family of cytokines is associated with retinal ganglion cell (RGC) survival and glial reactivity in glaucoma. The purpose of this study was to evaluate glaucoma-related changes in glycoprotein-130 (gp130), the common signal transducer of the...

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Echevarria F, Walker C, Abella S, et al. Stressor-dependent Alterations in Glycoprotein 130: Implications for Glial Cell Reactivity, Cytokine Signaling and Ganglion Cell Health in Glaucoma. J Clin Exp Ophthalmol. 2013;4(3)doi: 10.4172/2155-9570.1000286.
Echevarria, F., Walker, C., Abella, S., Won, M., & Sappington, R. (2013). Stressor-dependent Alterations in Glycoprotein 130: Implications for Glial Cell Reactivity, Cytokine Signaling and Ganglion Cell Health in Glaucoma. Journal of clinical & experimental ophthalmology, 4(3), . https://doi.org/10.4172/2155-9570.1000286
Echevarria, Fd, et al. "Stressor-dependent Alterations in Glycoprotein 130: Implications for Glial Cell Reactivity, Cytokine Signaling and Ganglion Cell Health in Glaucoma." Journal of clinical & experimental ophthalmology vol. 4,3 (2013). doi: https://doi.org/10.4172/2155-9570.1000286
Echevarria F, Walker C, Abella S, Won M, Sappington R. Stressor-dependent Alterations in Glycoprotein 130: Implications for Glial Cell Reactivity, Cytokine Signaling and Ganglion Cell Health in Glaucoma. J Clin Exp Ophthalmol. 2013 Jun 30;4(3). doi: 10.4172/2155-9570.1000286. PMID: 25018894; PMCID: PMC4091850.
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Erratum to: A multi-stage genome-wide association study of uterine fibroids in African Americans.

Human genetics

Hellwege JN, Jeff JM, Wise LA, Gallagher CS, Wellons M, Hartmann KE, Jones SF, Torstenson ES, Dickinson S, Ruiz-Narváez EA, Rohland N, Allen A, Reich D, Tandon A, Pasaniuc B, Mancuso N, Im HK, Hinds DA, Palmer JR, Rosenberg L, Denny JC, Roden DM, Stewart EA, Morton CC, Kenny EE, Edwards TL, Velez Edwards DR.
PMID: 28975356
Hum Genet. 2017 Nov;136(11):1497-1498. doi: 10.1007/s00439-017-1846-z.

The article "A multi-stage genome-wide association study of uterine fibroids in African Americans", written by Jacklyn N. Hellwege, was originally published Online First without open access. After publication in volume 136, issue 10, page 1363-1373 the author decided to...

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Hellwege JN, Jeff JM, Wise LA, et al. Erratum to: A multi-stage genome-wide association study of uterine fibroids in African Americans. Hum Genet. 2017;136(11):1497-1498doi: 10.1007/s00439-017-1846-z.
Hellwege, J. N., Jeff, J. M., Wise, L. A., Gallagher, C. S., Wellons, M., Hartmann, K. E., Jones, S. F., Torstenson, E. S., Dickinson, S., Ruiz-Narváez, E. A., Rohland, N., Allen, A., Reich, D., Tandon, A., Pasaniuc, B., Mancuso, N., Im, H. K., Hinds, D. A., Palmer, J. R., Rosenberg, L., Denny, J. C., Roden, D. M., Stewart, E. A., Morton, C. C., Kenny, E. E., Edwards, T. L., & Velez Edwards, D. R. (2017). Erratum to: A multi-stage genome-wide association study of uterine fibroids in African Americans. Human genetics, 136(11), 1497-1498. https://doi.org/10.1007/s00439-017-1846-z
Hellwege, Jacklyn N, et al. "Erratum to: A multi-stage genome-wide association study of uterine fibroids in African Americans." Human genetics vol. 136,11 (2017): 1497-1498. doi: https://doi.org/10.1007/s00439-017-1846-z
Hellwege JN, Jeff JM, Wise LA, Gallagher CS, Wellons M, Hartmann KE, Jones SF, Torstenson ES, Dickinson S, Ruiz-Narváez EA, Rohland N, Allen A, Reich D, Tandon A, Pasaniuc B, Mancuso N, Im HK, Hinds DA, Palmer JR, Rosenberg L, Denny JC, Roden DM, Stewart EA, Morton CC, Kenny EE, Edwards TL, Velez Edwards DR. Erratum to: A multi-stage genome-wide association study of uterine fibroids in African Americans. Hum Genet. 2017 Nov;136(11):1497-1498. doi: 10.1007/s00439-017-1846-z. PMID: 28975356; PMCID: PMC5909202.
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Showing 1 to 12 of 28 entries