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PPARγ: no SirT, no service.

Circulation research

Quelle FW, Sigmund CD.
PMID: 23371897
Circ Res. 2013 Feb 01;112(3):411-4. doi: 10.1161/CIRCRESAHA.113.300870.

No abstract available.

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Quelle FW, Sigmund CD. PPARγ: no SirT, no service. Circ Res. 2013;112(3):411-4doi: 10.1161/CIRCRESAHA.113.300870.
Quelle, F. W., & Sigmund, C. D. (2013). PPARγ: no SirT, no service. Circulation research, 112(3), 411-4. https://doi.org/10.1161/CIRCRESAHA.113.300870
Quelle, Frederick W, and Sigmund, Curt D. "PPARγ: no SirT, no service." Circulation research vol. 112,3 (2013): 411-4. doi: https://doi.org/10.1161/CIRCRESAHA.113.300870
Quelle FW, Sigmund CD. PPARγ: no SirT, no service. Circ Res. 2013 Feb 01;112(3):411-4. doi: 10.1161/CIRCRESAHA.113.300870. PMID: 23371897; PMCID: PMC3580855.
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Pregnant mice lacking indoleamine 2,3-dioxygenase exhibit preeclampsia phenotypes.

Physiological reports

Santillan MK, Pelham CJ, Ketsawatsomkron P, Santillan DA, Davis DR, Devor EJ, Gibson-Corley KN, Scroggins SM, Grobe JL, Yang B, Hunter SK, Sigmund CD.
PMID: 25602015
Physiol Rep. 2015 Jan 19;3(1). doi: 10.14814/phy2.12257. Print 2015 Jan 01.

Preeclampsia is a cardiovascular disorder of late pregnancy that is, commonly characterized by hypertension, renal structural damage and dysfunction, and fetal growth restriction. Prevailing etiologic models of this disorder include T-cell dysfunction as an initiating cause of preeclampsia. Indoleamine...

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Santillan MK, Pelham CJ, Ketsawatsomkron P, et al. Pregnant mice lacking indoleamine 2,3-dioxygenase exhibit preeclampsia phenotypes. Physiol Rep. 2015;3(1)doi: 10.14814/phy2.12257.
Santillan, M. K., Pelham, C. J., Ketsawatsomkron, P., Santillan, D. A., Davis, D. R., Devor, E. J., Gibson-Corley, K. N., Scroggins, S. M., Grobe, J. L., Yang, B., Hunter, S. K., & Sigmund, C. D. (2015). Pregnant mice lacking indoleamine 2,3-dioxygenase exhibit preeclampsia phenotypes. Physiological reports, 3(1), . https://doi.org/10.14814/phy2.12257
Santillan, Mark K, et al. "Pregnant mice lacking indoleamine 2,3-dioxygenase exhibit preeclampsia phenotypes." Physiological reports vol. 3,1 (2015). doi: https://doi.org/10.14814/phy2.12257
Santillan MK, Pelham CJ, Ketsawatsomkron P, Santillan DA, Davis DR, Devor EJ, Gibson-Corley KN, Scroggins SM, Grobe JL, Yang B, Hunter SK, Sigmund CD. Pregnant mice lacking indoleamine 2,3-dioxygenase exhibit preeclampsia phenotypes. Physiol Rep. 2015 Jan 19;3(1). doi: 10.14814/phy2.12257. Print 2015 Jan 01. PMID: 25602015; PMCID: PMC4387753.
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Inflaming hypothalamic neurons raises blood pressure.

Cell metabolism

Rahmouni K, Davisson RL, Sigmund CD.
PMID: 21723497
Cell Metab. 2011 Jul 06;14(1):3-4. doi: 10.1016/j.cmet.2011.06.006.

Obesity and hypertension are strongly associated, and neural dysfunction has been implicated in both. The hypothalamus integrates signals regulating blood pressure and energy homeostasis. A recent paper in Nature Medicine (Purkayastha et al., 2011) suggests that obesity and hypertension...

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Rahmouni K, Davisson RL, Sigmund CD. Inflaming hypothalamic neurons raises blood pressure. Cell Metab. 2011;14(1):3-4doi: 10.1016/j.cmet.2011.06.006.
Rahmouni, K., Davisson, R. L., & Sigmund, C. D. (2011). Inflaming hypothalamic neurons raises blood pressure. Cell metabolism, 14(1), 3-4. https://doi.org/10.1016/j.cmet.2011.06.006
Rahmouni, Kamal, et al. "Inflaming hypothalamic neurons raises blood pressure." Cell metabolism vol. 14,1 (2011): 3-4. doi: https://doi.org/10.1016/j.cmet.2011.06.006
Rahmouni K, Davisson RL, Sigmund CD. Inflaming hypothalamic neurons raises blood pressure. Cell Metab. 2011 Jul 06;14(1):3-4. doi: 10.1016/j.cmet.2011.06.006. PMID: 21723497; PMCID: PMC3131053.
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