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Growth Hormone-Releaser Diet Attenuates Cognitive Dysfunction in Klotho Mutant Mice via Insulin-Like Growth Factor-1 Receptor Activation in a Genetic Aging Model.

Endocrinology and metabolism (Seoul, Korea)

Park SJ, Chung YH, Lee JH, Dang DK, Nam Y, Jeong JH, Kim YS, Nabeshima T, Shin EJ, Kim HC.
PMID: 25309793
Endocrinol Metab (Seoul). 2014 Sep;29(3):336-48. doi: 10.3803/EnM.2014.29.3.336. Epub 2014 Sep 25.

BACKGROUND: It has been recognized that a defect in klotho gene expression accelerates the degeneration of multiple age-sensitive traits. Accumulating evidence indicates that aging is associated with declines in cognitive function and the activity of growth hormone (GH)/insulin-like growth...

Cite
Park SJ, Chung YH, Lee JH, et al. Growth Hormone-Releaser Diet Attenuates Cognitive Dysfunction in Klotho Mutant Mice via Insulin-Like Growth Factor-1 Receptor Activation in a Genetic Aging Model. Endocrinol Metab (Seoul). 2014;29(3):336-48doi: 10.3803/EnM.2014.29.3.336.
Park, S. J., Chung, Y. H., Lee, J. H., Dang, D. K., Nam, Y., Jeong, J. H., Kim, Y. S., Nabeshima, T., Shin, E. J., & Kim, H. C. (2014). Growth Hormone-Releaser Diet Attenuates Cognitive Dysfunction in Klotho Mutant Mice via Insulin-Like Growth Factor-1 Receptor Activation in a Genetic Aging Model. Endocrinology and metabolism (Seoul, Korea), 29(3), 336-48. https://doi.org/10.3803/EnM.2014.29.3.336
Park, Seok Joo, et al. "Growth Hormone-Releaser Diet Attenuates Cognitive Dysfunction in Klotho Mutant Mice via Insulin-Like Growth Factor-1 Receptor Activation in a Genetic Aging Model." Endocrinology and metabolism (Seoul, Korea) vol. 29,3 (2014): 336-48. doi: https://doi.org/10.3803/EnM.2014.29.3.336
Park SJ, Chung YH, Lee JH, Dang DK, Nam Y, Jeong JH, Kim YS, Nabeshima T, Shin EJ, Kim HC. Growth Hormone-Releaser Diet Attenuates Cognitive Dysfunction in Klotho Mutant Mice via Insulin-Like Growth Factor-1 Receptor Activation in a Genetic Aging Model. Endocrinol Metab (Seoul). 2014 Sep;29(3):336-48. doi: 10.3803/EnM.2014.29.3.336. Epub 2014 Sep 25. PMID: 25309793; PMCID: PMC4192803.
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