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Showing 1 to 12 of 18 entries
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CCL2/ACKR2 interaction participate in breast cancer metastasis especially in patients with altered lipid metabolism.

Medical hypotheses

Zhong X, Wang X, Sun Q.
PMID: 34861532
Med Hypotheses. 2021 Nov 09;158:110734. doi: 10.1016/j.mehy.2021.110734. Epub 2021 Nov 09.

Breast cancer is the most prevalent cancer worldwide and metastasis accounts for the majority of associated deaths. Altered lipid metabolism has been proved to be involved with breast cancer, yet the underlying mechanism in lipid metabolism mediated metastasis in...

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Zhong X, Wang X, Sun Q. CCL2/ACKR2 interaction participate in breast cancer metastasis especially in patients with altered lipid metabolism. Med Hypotheses. 2021;158:110734doi: 10.1016/j.mehy.2021.110734.
Zhong, X., Wang, X., & Sun, Q. (2021). CCL2/ACKR2 interaction participate in breast cancer metastasis especially in patients with altered lipid metabolism. Medical hypotheses, 158110734. https://doi.org/10.1016/j.mehy.2021.110734
Zhong, Xiaoying, et al. "CCL2/ACKR2 interaction participate in breast cancer metastasis especially in patients with altered lipid metabolism." Medical hypotheses vol. 158 (2021): 110734. doi: https://doi.org/10.1016/j.mehy.2021.110734
Zhong X, Wang X, Sun Q. CCL2/ACKR2 interaction participate in breast cancer metastasis especially in patients with altered lipid metabolism. Med Hypotheses. 2021 Nov 09;158:110734. doi: 10.1016/j.mehy.2021.110734. Epub 2021 Nov 09. PMID: 34861532.
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Chemokine (C-C motif) Ligand 28 and Its Receptor CCR10: Expression and Function at the Maternal-Conceptus Interface in Pigs.

Biology of reproduction

Choi Y, Seo H, Han J, Yoo I, Kim J, Ka H.
PMID: 27580990
Biol Reprod. 2016 Oct 01;95(4):84. doi: 10.1095/biolreprod.116.141903. Epub 2016 Aug 31.

Many chemokines are present at the maternal-fetal interface and play important roles in the establishment and maintenance of pregnancy. Our study demonstrates that a chemokine, chemokine (C-C motif) ligand 28 (CCL28), is expressed in the uterine endometrium during early...

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Choi Y, Seo H, Han J, et al. Chemokine (C-C motif) Ligand 28 and Its Receptor CCR10: Expression and Function at the Maternal-Conceptus Interface in Pigs. Biol Reprod. 2016;95(4):84doi: 10.1095/biolreprod.116.141903.
Choi, Y., Seo, H., Han, J., Yoo, I., Kim, J., & Ka, H. (2016). Chemokine (C-C motif) Ligand 28 and Its Receptor CCR10: Expression and Function at the Maternal-Conceptus Interface in Pigs. Biology of reproduction, 95(4), 84. https://doi.org/10.1095/biolreprod.116.141903
Choi, Yohan, et al. "Chemokine (C-C motif) Ligand 28 and Its Receptor CCR10: Expression and Function at the Maternal-Conceptus Interface in Pigs." Biology of reproduction vol. 95,4 (2016): 84. doi: https://doi.org/10.1095/biolreprod.116.141903
Choi Y, Seo H, Han J, Yoo I, Kim J, Ka H. Chemokine (C-C motif) Ligand 28 and Its Receptor CCR10: Expression and Function at the Maternal-Conceptus Interface in Pigs. Biol Reprod. 2016 Oct 01;95(4):84. doi: 10.1095/biolreprod.116.141903. Epub 2016 Aug 31. PMID: 27580990.
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Immune Modulation in Heart Failure: the Promise of Novel Biologics.

Current treatment options in cardiovascular medicine

Alvarez P, Briasoulis A.
PMID: 29541873
Curr Treat Options Cardiovasc Med. 2018 Mar 15;20(3):26. doi: 10.1007/s11936-018-0617-z.

PURPOSE OF REVIEW: Immune system activation plays a central role in heart failure progression. Large-scale immune modulatory clinical trials targeting tumor necrosis factor-α and broad spectrum immune modulation have been negative. The objective of this review is to highlight...

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Alvarez P, Briasoulis A. Immune Modulation in Heart Failure: the Promise of Novel Biologics. Curr Treat Options Cardiovasc Med. 2018;20(3):26doi: 10.1007/s11936-018-0617-z.
Alvarez, P., & Briasoulis, A. (2018). Immune Modulation in Heart Failure: the Promise of Novel Biologics. Current treatment options in cardiovascular medicine, 20(3), 26. https://doi.org/10.1007/s11936-018-0617-z
Alvarez, Paulino, and Briasoulis, Alexandros. "Immune Modulation in Heart Failure: the Promise of Novel Biologics." Current treatment options in cardiovascular medicine vol. 20,3 (2018): 26. doi: https://doi.org/10.1007/s11936-018-0617-z
Alvarez P, Briasoulis A. Immune Modulation in Heart Failure: the Promise of Novel Biologics. Curr Treat Options Cardiovasc Med. 2018 Mar 15;20(3):26. doi: 10.1007/s11936-018-0617-z. PMID: 29541873.
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Human papillomavirus-driven immune deviation: challenge and novel opportunity for immunotherapy.

Therapeutic advances in vaccines

Smola S, Trimble C, Stern PL.
PMID: 28794879
Ther Adv Vaccines. 2017 Jun;5(3):69-82. doi: 10.1177/2051013617717914. Epub 2017 Jul 05.

It is now recognized that the immune system can be a key component of restraint and control during the neoplastic process. Human papillomavirus (HPV)-associated cancers of the anogenital tract and oropharynx represent a significant clinical problem but there is...

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Smola S, Trimble C, Stern PL. Human papillomavirus-driven immune deviation: challenge and novel opportunity for immunotherapy. Ther Adv Vaccines. 2017;5(3):69-82doi: 10.1177/2051013617717914.
Smola, S., Trimble, C., & Stern, P. L. (2017). Human papillomavirus-driven immune deviation: challenge and novel opportunity for immunotherapy. Therapeutic advances in vaccines, 5(3), 69-82. https://doi.org/10.1177/2051013617717914
Smola, Sigrun, et al. "Human papillomavirus-driven immune deviation: challenge and novel opportunity for immunotherapy." Therapeutic advances in vaccines vol. 5,3 (2017): 69-82. doi: https://doi.org/10.1177/2051013617717914
Smola S, Trimble C, Stern PL. Human papillomavirus-driven immune deviation: challenge and novel opportunity for immunotherapy. Ther Adv Vaccines. 2017 Jun;5(3):69-82. doi: 10.1177/2051013617717914. Epub 2017 Jul 05. PMID: 28794879; PMCID: PMC5524244.
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Comparison of gene expression profiling in sarcomas and mesenchymal stem cells identifies tumorigenic pathways in chemically induced rat sarcoma model.

ISRN oncology

Honoki K, Fujii H, Tohma Y, Tsujiuchi T, Kido A, Tsukamoto S, Mori T, Tanaka Y.
PMID: 22852096
ISRN Oncol. 2012;2012:909453. doi: 10.5402/2012/909453. Epub 2012 Jul 17.

Mesenchymal stem cells (MSCs) are believed to be the cell of origin for most sarcomas including osteosarcoma and malignant fibrous histiocytoma (MFH/UPS). To identify the signaling pathways involved in sarcoma pathogenesis, we compared gene expression profiles in rat osteosarcoma...

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Honoki K, Fujii H, Tohma Y, et al. Comparison of gene expression profiling in sarcomas and mesenchymal stem cells identifies tumorigenic pathways in chemically induced rat sarcoma model. ISRN Oncol. 2012;2012:909453doi: 10.5402/2012/909453.
Honoki, K., Fujii, H., Tohma, Y., Tsujiuchi, T., Kido, A., Tsukamoto, S., Mori, T., & Tanaka, Y. (2012). Comparison of gene expression profiling in sarcomas and mesenchymal stem cells identifies tumorigenic pathways in chemically induced rat sarcoma model. ISRN oncology, 2012909453. https://doi.org/10.5402/2012/909453
Honoki, Kanya, et al. "Comparison of gene expression profiling in sarcomas and mesenchymal stem cells identifies tumorigenic pathways in chemically induced rat sarcoma model." ISRN oncology vol. 2012 (2012): 909453. doi: https://doi.org/10.5402/2012/909453
Honoki K, Fujii H, Tohma Y, Tsujiuchi T, Kido A, Tsukamoto S, Mori T, Tanaka Y. Comparison of gene expression profiling in sarcomas and mesenchymal stem cells identifies tumorigenic pathways in chemically induced rat sarcoma model. ISRN Oncol. 2012;2012:909453. doi: 10.5402/2012/909453. Epub 2012 Jul 17. PMID: 22852096; PMCID: PMC3407640.
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Association of chemokine and chemokine receptor expression with the invasion and metastasis of lung carcinoma.

Oncology letters

Liu Y, Wu BQ, Geng H, Xu ML, Zhong HH.
PMID: 26622670
Oncol Lett. 2015 Sep;10(3):1315-1322. doi: 10.3892/ol.2015.3402. Epub 2015 Jun 22.

The chemokine system has been reported to be utilized and manipulated by tumor cells in order to promote local tumor growth and distant dissemination. The present study aimed to investigate the expression of three chemokine ligand-receptor axes in lung...

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Liu Y, Wu BQ, Geng H, et al. Association of chemokine and chemokine receptor expression with the invasion and metastasis of lung carcinoma. Oncol Lett. 2015;10(3):1315-1322doi: 10.3892/ol.2015.3402.
Liu, Y., Wu, B. Q., Geng, H., Xu, M. L., & Zhong, H. H. (2015). Association of chemokine and chemokine receptor expression with the invasion and metastasis of lung carcinoma. Oncology letters, 10(3), 1315-1322. https://doi.org/10.3892/ol.2015.3402
Liu, Yan, et al. "Association of chemokine and chemokine receptor expression with the invasion and metastasis of lung carcinoma." Oncology letters vol. 10,3 (2015): 1315-1322. doi: https://doi.org/10.3892/ol.2015.3402
Liu Y, Wu BQ, Geng H, Xu ML, Zhong HH. Association of chemokine and chemokine receptor expression with the invasion and metastasis of lung carcinoma. Oncol Lett. 2015 Sep;10(3):1315-1322. doi: 10.3892/ol.2015.3402. Epub 2015 Jun 22. PMID: 26622670; PMCID: PMC4533290.
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ADAM17 orchestrates Interleukin-6, TNFα and EGF-R signaling in inflammation and cancer.

Biochimica et biophysica acta. Molecular cell research

Schumacher N, Rose-John S.
PMID: 34610348
Biochim Biophys Acta Mol Cell Res. 2022 Jan;1869(1):119141. doi: 10.1016/j.bbamcr.2021.119141. Epub 2021 Oct 02.

It was realized in the 1990s that some membrane proteins such as TNFα, both TNF receptors, ligands of the EGF-R and the Interleukin-6 receptor are proteolytically cleaved and are shed from the cell membrane as soluble proteins. The major...

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Schumacher N, Rose-John S. ADAM17 orchestrates Interleukin-6, TNFα and EGF-R signaling in inflammation and cancer. Biochim Biophys Acta Mol Cell Res. 2021;1869(1):119141doi: 10.1016/j.bbamcr.2021.119141.
Schumacher, N., & Rose-John, S. (2022). ADAM17 orchestrates Interleukin-6, TNFα and EGF-R signaling in inflammation and cancer. Biochimica et biophysica acta. Molecular cell research, 1869(1), 119141. https://doi.org/10.1016/j.bbamcr.2021.119141
Schumacher, Neele, and Rose-John, Stefan. "ADAM17 orchestrates Interleukin-6, TNFα and EGF-R signaling in inflammation and cancer." Biochimica et biophysica acta. Molecular cell research vol. 1869,1 (2022): 119141. doi: https://doi.org/10.1016/j.bbamcr.2021.119141
Schumacher N, Rose-John S. ADAM17 orchestrates Interleukin-6, TNFα and EGF-R signaling in inflammation and cancer. Biochim Biophys Acta Mol Cell Res. 2022 Jan;1869(1):119141. doi: 10.1016/j.bbamcr.2021.119141. Epub 2021 Oct 02. PMID: 34610348.
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Receptor biology and signal transduction.

Current opinion in gastroenterology

Ding XQ, Ding WQ, Miller LJ.
PMID: 17031193
Curr Opin Gastroenterol. 2001 Sep;17(5):410-5. doi: 10.1097/00001574-200109000-00002.

This year has witnessed substantial advances in receptor biology and signal transduction that are relevant to the function and regulation of the healthy pancreas and to the pathogenesis and potential therapy of pancreatitis and pancreatic carcinoma. There has been...

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Ding XQ, Ding WQ, Miller LJ. Receptor biology and signal transduction. Curr Opin Gastroenterol. 2001;17(5):410-5doi: 10.1097/00001574-200109000-00002.
Ding, X. Q., Ding, W. Q., & Miller, L. J. (2001). Receptor biology and signal transduction. Current opinion in gastroenterology, 17(5), 410-5. https://doi.org/10.1097/00001574-200109000-00002
Ding, X Q, et al. "Receptor biology and signal transduction." Current opinion in gastroenterology vol. 17,5 (2001): 410-5. doi: https://doi.org/10.1097/00001574-200109000-00002
Ding XQ, Ding WQ, Miller LJ. Receptor biology and signal transduction. Curr Opin Gastroenterol. 2001 Sep;17(5):410-5. doi: 10.1097/00001574-200109000-00002. PMID: 17031193.
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Targeting Angiogenesis in Rheumatoid Arthritis.

Current rheumatology reviews

Szekanecz Z, Koch AE.
PMID: 20706557
Curr Rheumatol Rev. 2008 Nov 01;4(4):298-303. doi: 10.2174/157339708786263942.

Angiogenesis, the development of new capillaries, is a crucial process in health and disease. The perpetuation of neovascularization in rheumatoid arthritis is highly involved in leukocyte extravasation into the synovium and pannus formation. Numerous soluble and cell surface-bound angiogenic...

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Szekanecz Z, Koch AE. Targeting Angiogenesis in Rheumatoid Arthritis. Curr Rheumatol Rev. 2008;4(4):298-303doi: 10.2174/157339708786263942.
Szekanecz, Z., & Koch, A. E. (2008). Targeting Angiogenesis in Rheumatoid Arthritis. Current rheumatology reviews, 4(4), 298-303. https://doi.org/10.2174/157339708786263942
Szekanecz, Zoltán, and Koch, Alisa E. "Targeting Angiogenesis in Rheumatoid Arthritis." Current rheumatology reviews vol. 4,4 (2008): 298-303. doi: https://doi.org/10.2174/157339708786263942
Szekanecz Z, Koch AE. Targeting Angiogenesis in Rheumatoid Arthritis. Curr Rheumatol Rev. 2008 Nov 01;4(4):298-303. doi: 10.2174/157339708786263942. PMID: 20706557; PMCID: PMC2919854.
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Chemerin acts via CMKLR1 and GPR1 to stimulate migration and invasion of gastric cancer cells: putative role of decreased TIMP-1 and TIMP-2.

Oncotarget

Kumar JD, Aolymat I, Tiszlavicz L, Reisz Z, Garalla HM, Beynon R, Simpson D, Dockray GJ, Varro A.
PMID: 30719206
Oncotarget. 2019 Jan 04;10(2):98-112. doi: 10.18632/oncotarget.26414. eCollection 2019 Jan 04.

The chemokine-like peptide, chemerin, stimulates chemotaxis in several cell types. In this study we examined the expression of putative chemerin receptors in gastric cancer and the action of chemerin on cancer cell migration and invasion. Immunohistochemical studies of gastric...

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Kumar JD, Aolymat I, Tiszlavicz L, et al. Chemerin acts via CMKLR1 and GPR1 to stimulate migration and invasion of gastric cancer cells: putative role of decreased TIMP-1 and TIMP-2. Oncotarget. 2019;10(2):98-112doi: 10.18632/oncotarget.26414.
Kumar, J. D., Aolymat, I., Tiszlavicz, L., Reisz, Z., Garalla, H. M., Beynon, R., Simpson, D., Dockray, G. J., & Varro, A. (2019). Chemerin acts via CMKLR1 and GPR1 to stimulate migration and invasion of gastric cancer cells: putative role of decreased TIMP-1 and TIMP-2. Oncotarget, 10(2), 98-112. https://doi.org/10.18632/oncotarget.26414
Kumar, J Dinesh, et al. "Chemerin acts via CMKLR1 and GPR1 to stimulate migration and invasion of gastric cancer cells: putative role of decreased TIMP-1 and TIMP-2." Oncotarget vol. 10,2 (2019): 98-112. doi: https://doi.org/10.18632/oncotarget.26414
Kumar JD, Aolymat I, Tiszlavicz L, Reisz Z, Garalla HM, Beynon R, Simpson D, Dockray GJ, Varro A. Chemerin acts via CMKLR1 and GPR1 to stimulate migration and invasion of gastric cancer cells: putative role of decreased TIMP-1 and TIMP-2. Oncotarget. 2019 Jan 04;10(2):98-112. doi: 10.18632/oncotarget.26414. eCollection 2019 Jan 04. PMID: 30719206; PMCID: PMC6349446.
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Effective Treatments for Bladder Cancer Affecting CXCL9/CXCL10/CXCL11/CXCR3 Axis: A Review.

Oman medical journal

Nazari A, Ahmadi Z, Hassanshahi G, Abbasifard M, Taghipour Z, Falahati-Pour SK, Khorramdelazad H.
PMID: 32181005
Oman Med J. 2020 Mar 11;35(2):e103. doi: 10.5001/omj.2020.21. eCollection 2020 Mar.

Bladder cancer (BC) originates mainly from the epithelial compartment of the bladder, which is defined as transitional cell carcinoma or urothelial cell carcinoma. About 70% of patients with BC will survive five years from diagnosis. Previous studies revealed that...

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Nazari A, Ahmadi Z, Hassanshahi G, et al. Effective Treatments for Bladder Cancer Affecting CXCL9/CXCL10/CXCL11/CXCR3 Axis: A Review. Oman Med J. 2020;35(2):e103doi: 10.5001/omj.2020.21.
Nazari, A., Ahmadi, Z., Hassanshahi, G., Abbasifard, M., Taghipour, Z., Falahati-Pour, S. K., & Khorramdelazad, H. (2020). Effective Treatments for Bladder Cancer Affecting CXCL9/CXCL10/CXCL11/CXCR3 Axis: A Review. Oman medical journal, 35(2), e103. https://doi.org/10.5001/omj.2020.21
Nazari, Alireza, et al. "Effective Treatments for Bladder Cancer Affecting CXCL9/CXCL10/CXCL11/CXCR3 Axis: A Review." Oman medical journal vol. 35,2 (2020): e103. doi: https://doi.org/10.5001/omj.2020.21
Nazari A, Ahmadi Z, Hassanshahi G, Abbasifard M, Taghipour Z, Falahati-Pour SK, Khorramdelazad H. Effective Treatments for Bladder Cancer Affecting CXCL9/CXCL10/CXCL11/CXCR3 Axis: A Review. Oman Med J. 2020 Mar 11;35(2):e103. doi: 10.5001/omj.2020.21. eCollection 2020 Mar. PMID: 32181005; PMCID: PMC7064791.
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Sensory neuron-associated macrophages as novel modulators of neuropathic pain.

Pain reports

Silva CEA, Guimarães RM, Cunha TM.
PMID: 33981924
Pain Rep. 2021 Mar 09;6(1):e873. doi: 10.1097/PR9.0000000000000873. eCollection 2021.

The peripheral nervous system comprises an infinity of neural networks that act in the communication between the central nervous system and the most diverse tissues of the body. Along with the extension of the primary sensory neurons (axons and...

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Silva CEA, Guimarães RM, Cunha TM. Sensory neuron-associated macrophages as novel modulators of neuropathic pain. Pain Rep. 2021;6(1):e873doi: 10.1097/PR9.0000000000000873.
Silva, C. E. A., Guimarães, R. M., & Cunha, T. M. (2021). Sensory neuron-associated macrophages as novel modulators of neuropathic pain. Pain reports, 6(1), e873. https://doi.org/10.1097/PR9.0000000000000873
Silva, Conceição Elidianne Aníbal, et al. "Sensory neuron-associated macrophages as novel modulators of neuropathic pain." Pain reports vol. 6,1 (2021): e873. doi: https://doi.org/10.1097/PR9.0000000000000873
Silva CEA, Guimarães RM, Cunha TM. Sensory neuron-associated macrophages as novel modulators of neuropathic pain. Pain Rep. 2021 Mar 09;6(1):e873. doi: 10.1097/PR9.0000000000000873. eCollection 2021. PMID: 33981924; PMCID: PMC8108583.
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Showing 1 to 12 of 18 entries