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Targeting the Peroxisome Proliferator-Activated Receptor-γ to Counter the Inflammatory Milieu in Obesity.

Diabetes & metabolism journal

Corzo C, Griffin PR.
PMID: 24404510
Diabetes Metab J. 2013 Dec;37(6):395-403. doi: 10.4093/dmj.2013.37.6.395.

Adipose tissue, which was once viewed as a simple organ for storage of triglycerides, is now considered an important endocrine organ. Abnormal adipose tissue mass is associated with defects in endocrine and metabolic functions which are the underlying causes...

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Corzo C, Griffin PR. Targeting the Peroxisome Proliferator-Activated Receptor-γ to Counter the Inflammatory Milieu in Obesity. Diabetes Metab J. 2013;37(6):395-403doi: 10.4093/dmj.2013.37.6.395.
Corzo, C., & Griffin, P. R. (2013). Targeting the Peroxisome Proliferator-Activated Receptor-γ to Counter the Inflammatory Milieu in Obesity. Diabetes & metabolism journal, 37(6), 395-403. https://doi.org/10.4093/dmj.2013.37.6.395
Corzo, Cesar, and Griffin, Patrick R. "Targeting the Peroxisome Proliferator-Activated Receptor-γ to Counter the Inflammatory Milieu in Obesity." Diabetes & metabolism journal vol. 37,6 (2013): 395-403. doi: https://doi.org/10.4093/dmj.2013.37.6.395
Corzo C, Griffin PR. Targeting the Peroxisome Proliferator-Activated Receptor-γ to Counter the Inflammatory Milieu in Obesity. Diabetes Metab J. 2013 Dec;37(6):395-403. doi: 10.4093/dmj.2013.37.6.395. PMID: 24404510; PMCID: PMC3881323.
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Nuclear Receptors Resolve Endoplasmic Reticulum Stress to Improve Hepatic Insulin Resistance.

Diabetes & metabolism journal

Lee JM.
PMID: 28236381
Diabetes Metab J. 2017 Feb;41(1):10-19. doi: 10.4093/dmj.2017.41.1.10.

Chronic endoplasmic reticulum (ER) stress culminating in proteotoxicity contributes to the development of insulin resistance and progression to type 2 diabetes mellitus. Pharmacologic interventions targeting several different nuclear receptors have emerged as potential treatments for insulin resistance. The mechanistic...

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Lee JM. Nuclear Receptors Resolve Endoplasmic Reticulum Stress to Improve Hepatic Insulin Resistance. Diabetes Metab J. 2017;41(1):10-19doi: 10.4093/dmj.2017.41.1.10.
Lee, J. M. (2017). Nuclear Receptors Resolve Endoplasmic Reticulum Stress to Improve Hepatic Insulin Resistance. Diabetes & metabolism journal, 41(1), 10-19. https://doi.org/10.4093/dmj.2017.41.1.10
Lee, Jae Man. "Nuclear Receptors Resolve Endoplasmic Reticulum Stress to Improve Hepatic Insulin Resistance." Diabetes & metabolism journal vol. 41,1 (2017): 10-19. doi: https://doi.org/10.4093/dmj.2017.41.1.10
Lee JM. Nuclear Receptors Resolve Endoplasmic Reticulum Stress to Improve Hepatic Insulin Resistance. Diabetes Metab J. 2017 Feb;41(1):10-19. doi: 10.4093/dmj.2017.41.1.10. PMID: 28236381; PMCID: PMC5328691.
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Bile Acid Nuclear Receptor Farnesoid X Receptor: Therapeutic Target for Nonalcoholic Fatty Liver Disease.

Endocrinology and metabolism (Seoul, Korea)

Kim SG, Kim BK, Kim K, Fang S.
PMID: 28029021
Endocrinol Metab (Seoul). 2016 Dec;31(4):500-504. doi: 10.3803/EnM.2016.31.4.500.

Nonalcoholic fatty liver disease (NAFLD) is one of the causes of fatty liver, occurring when fat is accumulated in the liver without alcohol consumption. NAFLD is the most common liver disorder in advanced countries. NAFLD is a spectrum of...

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Kim SG, Kim BK, Kim K, et al. Bile Acid Nuclear Receptor Farnesoid X Receptor: Therapeutic Target for Nonalcoholic Fatty Liver Disease. Endocrinol Metab (Seoul). 2016;31(4):500-504doi: 10.3803/EnM.2016.31.4.500.
Kim, S. G., Kim, B. K., Kim, K., & Fang, S. (2016). Bile Acid Nuclear Receptor Farnesoid X Receptor: Therapeutic Target for Nonalcoholic Fatty Liver Disease. Endocrinology and metabolism (Seoul, Korea), 31(4), 500-504. https://doi.org/10.3803/EnM.2016.31.4.500
Kim, Sun Gi, et al. "Bile Acid Nuclear Receptor Farnesoid X Receptor: Therapeutic Target for Nonalcoholic Fatty Liver Disease." Endocrinology and metabolism (Seoul, Korea) vol. 31,4 (2016): 500-504. doi: https://doi.org/10.3803/EnM.2016.31.4.500
Kim SG, Kim BK, Kim K, Fang S. Bile Acid Nuclear Receptor Farnesoid X Receptor: Therapeutic Target for Nonalcoholic Fatty Liver Disease. Endocrinol Metab (Seoul). 2016 Dec;31(4):500-504. doi: 10.3803/EnM.2016.31.4.500. PMID: 28029021; PMCID: PMC5195824.
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Receptor-Mediated Muscle Homeostasis as a Target for Sarcopenia Therapeutics.

Endocrinology and metabolism (Seoul, Korea)

Yoon JH, Kwon KS.
PMID: 34218646
Endocrinol Metab (Seoul). 2021 Jun;36(3):478-490. doi: 10.3803/EnM.2021.1081. Epub 2021 Jun 28.

Sarcopenia is a disease characterized by age-related decline of skeletal muscle mass and function. The molecular mechanisms of the pathophysiology of sarcopenia form a complex network due to the involvement of multiple interconnected signaling pathways. Therefore, signaling receptors are...

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Yoon JH, Kwon KS. Receptor-Mediated Muscle Homeostasis as a Target for Sarcopenia Therapeutics. Endocrinol Metab (Seoul). 2021;36(3):478-490doi: 10.3803/EnM.2021.1081.
Yoon, J. H., & Kwon, K. S. (2021). Receptor-Mediated Muscle Homeostasis as a Target for Sarcopenia Therapeutics. Endocrinology and metabolism (Seoul, Korea), 36(3), 478-490. https://doi.org/10.3803/EnM.2021.1081
Yoon, Jong Hyeon, and Kwon, Ki-Sun. "Receptor-Mediated Muscle Homeostasis as a Target for Sarcopenia Therapeutics." Endocrinology and metabolism (Seoul, Korea) vol. 36,3 (2021): 478-490. doi: https://doi.org/10.3803/EnM.2021.1081
Yoon JH, Kwon KS. Receptor-Mediated Muscle Homeostasis as a Target for Sarcopenia Therapeutics. Endocrinol Metab (Seoul). 2021 Jun;36(3):478-490. doi: 10.3803/EnM.2021.1081. Epub 2021 Jun 28. PMID: 34218646; PMCID: PMC8258343.
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Transcriptional regulation of pyruvate dehydrogenase kinase.

Diabetes & metabolism journal

Jeong JY, Jeoung NH, Park KG, Lee IK.
PMID: 23130316
Diabetes Metab J. 2012 Oct;36(5):328-35. doi: 10.4093/dmj.2012.36.5.328. Epub 2012 Oct 18.

The pyruvate dehydrogenase complex (PDC) activity is crucial to maintains blood glucose and ATP levels, which largely depends on the phosphorylation status by pyruvate dehydrogenase kinase (PDK) isoenzymes. Although it has been reported that PDC is phosphorylated and inactivated...

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Jeong JY, Jeoung NH, Park KG, et al. Transcriptional regulation of pyruvate dehydrogenase kinase. Diabetes Metab J. 2012;36(5):328-35doi: 10.4093/dmj.2012.36.5.328.
Jeong, J. Y., Jeoung, N. H., Park, K. G., & Lee, I. K. (2012). Transcriptional regulation of pyruvate dehydrogenase kinase. Diabetes & metabolism journal, 36(5), 328-35. https://doi.org/10.4093/dmj.2012.36.5.328
Jeong, Ji Yun, et al. "Transcriptional regulation of pyruvate dehydrogenase kinase." Diabetes & metabolism journal vol. 36,5 (2012): 328-35. doi: https://doi.org/10.4093/dmj.2012.36.5.328
Jeong JY, Jeoung NH, Park KG, Lee IK. Transcriptional regulation of pyruvate dehydrogenase kinase. Diabetes Metab J. 2012 Oct;36(5):328-35. doi: 10.4093/dmj.2012.36.5.328. Epub 2012 Oct 18. PMID: 23130316; PMCID: PMC3486978.
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The Role of Thyroid Hormone in the Regulation of Cerebellar Development.

Endocrinology and metabolism (Seoul, Korea)

Ishii S, Amano I, Koibuchi N.
PMID: 34365775
Endocrinol Metab (Seoul). 2021 Aug;36(4):703-716. doi: 10.3803/EnM.2021.1150. Epub 2021 Aug 09.

The proper organized expression of specific genes in time and space is responsible for the organogenesis of the central nervous system including the cerebellum. The epigenetic regulation of gene expression is tightly regulated by an intrinsic intracellular genetic program,...

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Ishii S, Amano I, Koibuchi N. The Role of Thyroid Hormone in the Regulation of Cerebellar Development. Endocrinol Metab (Seoul). 2021;36(4):703-716doi: 10.3803/EnM.2021.1150.
Ishii, S., Amano, I., & Koibuchi, N. (2021). The Role of Thyroid Hormone in the Regulation of Cerebellar Development. Endocrinology and metabolism (Seoul, Korea), 36(4), 703-716. https://doi.org/10.3803/EnM.2021.1150
Ishii, Sumiyasu, et al. "The Role of Thyroid Hormone in the Regulation of Cerebellar Development." Endocrinology and metabolism (Seoul, Korea) vol. 36,4 (2021): 703-716. doi: https://doi.org/10.3803/EnM.2021.1150
Ishii S, Amano I, Koibuchi N. The Role of Thyroid Hormone in the Regulation of Cerebellar Development. Endocrinol Metab (Seoul). 2021 Aug;36(4):703-716. doi: 10.3803/EnM.2021.1150. Epub 2021 Aug 09. PMID: 34365775; PMCID: PMC8419606.
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Showing 1 to 6 of 6 entries