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Showing 1 to 12 of 17 entries
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Stabilizing Ryanodine Receptors Improves Left Ventricular Function in Juvenile Dogs With Duchenne Muscular Dystrophy.

Journal of the American College of Cardiology

Cazorla O, Barthélémy I, Su JB, Meli AC, Chetboul V, Scheuermann V, Gouni V, Anglerot C, Richard S, Blot S, Ghaleh B, Lacampagne A.
PMID: 34886965
J Am Coll Cardiol. 2021 Dec 14;78(24):2439-2453. doi: 10.1016/j.jacc.2021.10.014.

BACKGROUND: Duchenne muscular dystrophy is associated with progressive deterioration in left ventricular (LV) function. The golden retriever muscular dystrophy (GRMD) dog model recapitulates the pathology and clinical manifestations of Duchenne muscular dystrophy. Importantly, they develop progressive LV dysfunction starting...

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Cazorla O, Barthélémy I, Su JB, et al. Stabilizing Ryanodine Receptors Improves Left Ventricular Function in Juvenile Dogs With Duchenne Muscular Dystrophy. J Am Coll Cardiol. 2021;78(24):2439-2453doi: 10.1016/j.jacc.2021.10.014.
Cazorla, O., Barthélémy, I., Su, J. B., Meli, A. C., Chetboul, V., Scheuermann, V., Gouni, V., Anglerot, C., Richard, S., Blot, S., Ghaleh, B., & Lacampagne, A. (2021). Stabilizing Ryanodine Receptors Improves Left Ventricular Function in Juvenile Dogs With Duchenne Muscular Dystrophy. Journal of the American College of Cardiology, 78(24), 2439-2453. https://doi.org/10.1016/j.jacc.2021.10.014
Cazorla, Olivier, et al. "Stabilizing Ryanodine Receptors Improves Left Ventricular Function in Juvenile Dogs With Duchenne Muscular Dystrophy." Journal of the American College of Cardiology vol. 78,24 (2021): 2439-2453. doi: https://doi.org/10.1016/j.jacc.2021.10.014
Cazorla O, Barthélémy I, Su JB, Meli AC, Chetboul V, Scheuermann V, Gouni V, Anglerot C, Richard S, Blot S, Ghaleh B, Lacampagne A. Stabilizing Ryanodine Receptors Improves Left Ventricular Function in Juvenile Dogs With Duchenne Muscular Dystrophy. J Am Coll Cardiol. 2021 Dec 14;78(24):2439-2453. doi: 10.1016/j.jacc.2021.10.014. PMID: 34886965.
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Expression and regulation of ryanodine receptor/calcium release channels.

Trends in cardiovascular medicine

Marks AR.
PMID: 21232286
Trends Cardiovasc Med. 1996 May;6(4):130-5. doi: 10.1016/1050-1738(96)00035-7.

Intracellular calcium release channels on the endoplasmic or sarcoplasmic reticula (ryanodine receptors, RyR, and inositol 1,4,5-trisphosphate receptors, IP(3)R) comprise a unique family of molecules that are structurally and functionally distinct from all other known ion channels. These channels play...

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Marks AR. Expression and regulation of ryanodine receptor/calcium release channels. Trends Cardiovasc Med. 1996;6(4):130-5doi: 10.1016/1050-1738(96)00035-7.
Marks, A. R. (1996). Expression and regulation of ryanodine receptor/calcium release channels. Trends in cardiovascular medicine, 6(4), 130-5. https://doi.org/10.1016/1050-1738(96)00035-7
Marks, A R. "Expression and regulation of ryanodine receptor/calcium release channels." Trends in cardiovascular medicine vol. 6,4 (1996): 130-5. doi: https://doi.org/10.1016/1050-1738(96)00035-7
Marks AR. Expression and regulation of ryanodine receptor/calcium release channels. Trends Cardiovasc Med. 1996 May;6(4):130-5. doi: 10.1016/1050-1738(96)00035-7. PMID: 21232286.
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Atomic force microscopy study of the rabbit skeletal muscle ryanodine receptors in different functional states.

Science in China. Series C, Life sciences

Wei Q, Cheng X, Chen K, Hu J, Li M, Zhu P.
PMID: 18759045
Sci China C Life Sci. 2002 Jun;45(3):225-36. doi: 10.1360/02yc9025.

Atomic force microscope was applied to investigate the effect of extrinsic phospholipid on the structure of rabbit skeletal muscle ryanodine receptor/calcium release channel (RyR1). In addition, in the presence of extrinsic phospholipid, the height and elasticity of the RyR1s...

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Wei Q, Cheng X, Chen K, et al. Atomic force microscopy study of the rabbit skeletal muscle ryanodine receptors in different functional states. Sci China C Life Sci. 2002;45(3):225-36doi: 10.1360/02yc9025.
Wei, Q., Cheng, X., Chen, K., Hu, J., Li, M., & Zhu, P. (2002). Atomic force microscopy study of the rabbit skeletal muscle ryanodine receptors in different functional states. Science in China. Series C, Life sciences, 45(3), 225-36. https://doi.org/10.1360/02yc9025
Wei, Qingqing, et al. "Atomic force microscopy study of the rabbit skeletal muscle ryanodine receptors in different functional states." Science in China. Series C, Life sciences vol. 45,3 (2002): 225-36. doi: https://doi.org/10.1360/02yc9025
Wei Q, Cheng X, Chen K, Hu J, Li M, Zhu P. Atomic force microscopy study of the rabbit skeletal muscle ryanodine receptors in different functional states. Sci China C Life Sci. 2002 Jun;45(3):225-36. doi: 10.1360/02yc9025. PMID: 18759045.
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Moving in the right direction: elucidating the mechanisms of interaction between flecainide and the cardiac ryanodine receptor.

British journal of pharmacology

Bannister ML, MacLeod KT, George CH.
PMID: 34698387
Br J Pharmacol. 2021 Oct 26; doi: 10.1111/bph.15718. Epub 2021 Oct 26.

Flecainide is used to treat catecholaminergic polymorphic ventricular tachycardia (CPVT), an arrhythmia caused by disrupted cellular Ca

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Bannister ML, MacLeod KT, George CH. Moving in the right direction: elucidating the mechanisms of interaction between flecainide and the cardiac ryanodine receptor. Br J Pharmacol. 2021;doi: 10.1111/bph.15718.
Bannister, M. L., MacLeod, K. T., & George, C. H. (2021). Moving in the right direction: elucidating the mechanisms of interaction between flecainide and the cardiac ryanodine receptor. British journal of pharmacology, . https://doi.org/10.1111/bph.15718
Bannister, Mark L, et al. "Moving in the right direction: elucidating the mechanisms of interaction between flecainide and the cardiac ryanodine receptor." British journal of pharmacology vol. (2021). doi: https://doi.org/10.1111/bph.15718
Bannister ML, MacLeod KT, George CH. Moving in the right direction: elucidating the mechanisms of interaction between flecainide and the cardiac ryanodine receptor. Br J Pharmacol. 2021 Oct 26; doi: 10.1111/bph.15718. Epub 2021 Oct 26. PMID: 34698387.
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Ryanodine receptor calcium release channels in trophoblasts and their role in cell migration.

Biochimica et biophysica acta. Molecular cell research

Zheng L, Lindsay A, McSweeney K, Aplin J, Forbes K, Smith S, Tunwell R, Mackrill JJ.
PMID: 34624436
Biochim Biophys Acta Mol Cell Res. 2022 Jan;1869(1):119139. doi: 10.1016/j.bbamcr.2021.119139. Epub 2021 Oct 06.

Trophoblasts are specialized epithelial cells of the placenta that are involved in invasion, communication and the exchange of materials between the mother and fetus. Cytoplasmic Ca

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Zheng L, Lindsay A, McSweeney K, et al. Ryanodine receptor calcium release channels in trophoblasts and their role in cell migration. Biochim Biophys Acta Mol Cell Res. 2021;1869(1):119139doi: 10.1016/j.bbamcr.2021.119139.
Zheng, L., Lindsay, A., McSweeney, K., Aplin, J., Forbes, K., Smith, S., Tunwell, R., & Mackrill, J. J. (2022). Ryanodine receptor calcium release channels in trophoblasts and their role in cell migration. Biochimica et biophysica acta. Molecular cell research, 1869(1), 119139. https://doi.org/10.1016/j.bbamcr.2021.119139
Zheng, Limian, et al. "Ryanodine receptor calcium release channels in trophoblasts and their role in cell migration." Biochimica et biophysica acta. Molecular cell research vol. 1869,1 (2022): 119139. doi: https://doi.org/10.1016/j.bbamcr.2021.119139
Zheng L, Lindsay A, McSweeney K, Aplin J, Forbes K, Smith S, Tunwell R, Mackrill JJ. Ryanodine receptor calcium release channels in trophoblasts and their role in cell migration. Biochim Biophys Acta Mol Cell Res. 2022 Jan;1869(1):119139. doi: 10.1016/j.bbamcr.2021.119139. Epub 2021 Oct 06. PMID: 34624436.
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MCU Overexpression Rescues Inotropy and Reverses Heart Failure by Reducing SR Ca.

Circulation research

Liu T, Yang N, Sidor A, O'Rourke B.
PMID: 33522833
Circ Res. 2021 Apr 16;128(8):1191-1204. doi: 10.1161/CIRCRESAHA.120.318562. Epub 2021 Feb 01.

[Figure: see text].

Cite
Liu T, Yang N, Sidor A, et al. MCU Overexpression Rescues Inotropy and Reverses Heart Failure by Reducing SR Ca. Circ Res. 2021;128(8):1191-1204doi: 10.1161/CIRCRESAHA.120.318562.
Liu, T., Yang, N., Sidor, A., & O'Rourke, B. (2021). MCU Overexpression Rescues Inotropy and Reverses Heart Failure by Reducing SR Ca. Circulation research, 128(8), 1191-1204. https://doi.org/10.1161/CIRCRESAHA.120.318562
Liu, Ting, et al. "MCU Overexpression Rescues Inotropy and Reverses Heart Failure by Reducing SR Ca." Circulation research vol. 128,8 (2021): 1191-1204. doi: https://doi.org/10.1161/CIRCRESAHA.120.318562
Liu T, Yang N, Sidor A, O'Rourke B. MCU Overexpression Rescues Inotropy and Reverses Heart Failure by Reducing SR Ca. Circ Res. 2021 Apr 16;128(8):1191-1204. doi: 10.1161/CIRCRESAHA.120.318562. Epub 2021 Feb 01. PMID: 33522833; PMCID: PMC8049980.
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RyR2 Gain-of-Function and Not So Sudden Cardiac Death.

Circulation research

Hamilton S, Terentyev D.
PMID: 34292783
Circ Res. 2021 Jul 23;129(3):417-419. doi: 10.1161/CIRCRESAHA.121.319651. Epub 2021 Jul 22.

No abstract available.

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Hamilton S, Terentyev D. RyR2 Gain-of-Function and Not So Sudden Cardiac Death. Circ Res. 2021;129(3):417-419doi: 10.1161/CIRCRESAHA.121.319651.
Hamilton, S., & Terentyev, D. (2021). RyR2 Gain-of-Function and Not So Sudden Cardiac Death. Circulation research, 129(3), 417-419. https://doi.org/10.1161/CIRCRESAHA.121.319651
Hamilton, Shanna, and Terentyev, Dmitry. "RyR2 Gain-of-Function and Not So Sudden Cardiac Death." Circulation research vol. 129,3 (2021): 417-419. doi: https://doi.org/10.1161/CIRCRESAHA.121.319651
Hamilton S, Terentyev D. RyR2 Gain-of-Function and Not So Sudden Cardiac Death. Circ Res. 2021 Jul 23;129(3):417-419. doi: 10.1161/CIRCRESAHA.121.319651. Epub 2021 Jul 22. PMID: 34292783; PMCID: PMC8318380.
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Increased Reactive Oxygen Species-Mediated Ca.

Circulation

Liu X, Wang S, Guo X, Li Y, Ogurlu R, Lu F, Prondzynski M, de la Serna Buzon S, Ma Q, Zhang D, Wang G, Cotton J, Guo Y, Xiao L, Milan DJ, Xu Y, Schlame M, Bezzerides VJ, Pu WT.
PMID: 33793303
Circulation. 2021 May 11;143(19):1894-1911. doi: 10.1161/CIRCULATIONAHA.120.048698. Epub 2021 Apr 01.

BACKGROUND: Mutations in tafazzin (METHODS: We performed in vivo electrophysiology to define arrhythmia vulnerability in cardiac-specific RESULTS: A subset of mice with cardiac-specific CONCLUSIONS: This study identified a molecular pathway that links

Cite
Liu X, Wang S, Guo X, et al. Increased Reactive Oxygen Species-Mediated Ca. Circulation. 2021;143(19):1894-1911doi: 10.1161/CIRCULATIONAHA.120.048698.
Liu, X., Wang, S., Guo, X., Li, Y., Ogurlu, R., Lu, F., Prondzynski, M., de la Serna Buzon, S., Ma, Q., Zhang, D., Wang, G., Cotton, J., Guo, Y., Xiao, L., Milan, D. J., Xu, Y., Schlame, M., Bezzerides, V. J., & Pu, W. T. (2021). Increased Reactive Oxygen Species-Mediated Ca. Circulation, 143(19), 1894-1911. https://doi.org/10.1161/CIRCULATIONAHA.120.048698
Liu, Xujie, et al. "Increased Reactive Oxygen Species-Mediated Ca." Circulation vol. 143,19 (2021): 1894-1911. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.048698
Liu X, Wang S, Guo X, Li Y, Ogurlu R, Lu F, Prondzynski M, de la Serna Buzon S, Ma Q, Zhang D, Wang G, Cotton J, Guo Y, Xiao L, Milan DJ, Xu Y, Schlame M, Bezzerides VJ, Pu WT. Increased Reactive Oxygen Species-Mediated Ca. Circulation. 2021 May 11;143(19):1894-1911. doi: 10.1161/CIRCULATIONAHA.120.048698. Epub 2021 Apr 01. PMID: 33793303.
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MCU Overexpression Rescues Inotropy and Reverses Heart Failure by Reducing SR Ca.

Circulation research

Liu T, Yang N, Sidor A, O'Rourke B.
PMID: 33522833
Circ Res. 2021 Apr 16;128(8):1191-1204. doi: 10.1161/CIRCRESAHA.120.318562. Epub 2021 Feb 01.

[Figure: see text].

Cite
Liu T, Yang N, Sidor A, et al. MCU Overexpression Rescues Inotropy and Reverses Heart Failure by Reducing SR Ca. Circ Res. 2021;128(8):1191-1204doi: 10.1161/CIRCRESAHA.120.318562.
Liu, T., Yang, N., Sidor, A., & O'Rourke, B. (2021). MCU Overexpression Rescues Inotropy and Reverses Heart Failure by Reducing SR Ca. Circulation research, 128(8), 1191-1204. https://doi.org/10.1161/CIRCRESAHA.120.318562
Liu, Ting, et al. "MCU Overexpression Rescues Inotropy and Reverses Heart Failure by Reducing SR Ca." Circulation research vol. 128,8 (2021): 1191-1204. doi: https://doi.org/10.1161/CIRCRESAHA.120.318562
Liu T, Yang N, Sidor A, O'Rourke B. MCU Overexpression Rescues Inotropy and Reverses Heart Failure by Reducing SR Ca. Circ Res. 2021 Apr 16;128(8):1191-1204. doi: 10.1161/CIRCRESAHA.120.318562. Epub 2021 Feb 01. PMID: 33522833; PMCID: PMC8049980.
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Ryanodine receptor calcium release channels in trophoblasts and their role in cell migration.

Biochimica et biophysica acta. Molecular cell research

Zheng L, Lindsay A, McSweeney K, Aplin J, Forbes K, Smith S, Tunwell R, Mackrill JJ.
PMID: 34624436
Biochim Biophys Acta Mol Cell Res. 2022 Jan;1869(1):119139. doi: 10.1016/j.bbamcr.2021.119139. Epub 2021 Oct 06.

Trophoblasts are specialized epithelial cells of the placenta that are involved in invasion, communication and the exchange of materials between the mother and fetus. Cytoplasmic Ca

Cite
Zheng L, Lindsay A, McSweeney K, et al. Ryanodine receptor calcium release channels in trophoblasts and their role in cell migration. Biochim Biophys Acta Mol Cell Res. 2021;1869(1):119139doi: 10.1016/j.bbamcr.2021.119139.
Zheng, L., Lindsay, A., McSweeney, K., Aplin, J., Forbes, K., Smith, S., Tunwell, R., & Mackrill, J. J. (2022). Ryanodine receptor calcium release channels in trophoblasts and their role in cell migration. Biochimica et biophysica acta. Molecular cell research, 1869(1), 119139. https://doi.org/10.1016/j.bbamcr.2021.119139
Zheng, Limian, et al. "Ryanodine receptor calcium release channels in trophoblasts and their role in cell migration." Biochimica et biophysica acta. Molecular cell research vol. 1869,1 (2022): 119139. doi: https://doi.org/10.1016/j.bbamcr.2021.119139
Zheng L, Lindsay A, McSweeney K, Aplin J, Forbes K, Smith S, Tunwell R, Mackrill JJ. Ryanodine receptor calcium release channels in trophoblasts and their role in cell migration. Biochim Biophys Acta Mol Cell Res. 2022 Jan;1869(1):119139. doi: 10.1016/j.bbamcr.2021.119139. Epub 2021 Oct 06. PMID: 34624436.
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Regulation of the ryanodine receptor calcium release channel: a molecular complex system.

Biophysical chemistry

Kasai M, Kawasaki T, Yamaguchi N.
PMID: 17030344
Biophys Chem. 1999 Dec 13;82(2):173-81. doi: 10.1016/s0301-4622(99)00117-9.

Skeletal muscle contraction is regulated by Ca(2+) released from the sarcoplasmic reticulum (SR). The Ca(2+) release channel in the SR has been identified as the ryanodine receptor (RyR). Recently, it was found that the RyR is a large transmembrane...

Cite
Kasai M, Kawasaki T, Yamaguchi N. Regulation of the ryanodine receptor calcium release channel: a molecular complex system. Biophys Chem. 1999;82(2):173-81doi: 10.1016/s0301-4622(99)00117-9.
Kasai, M., Kawasaki, T., & Yamaguchi, N. (1999). Regulation of the ryanodine receptor calcium release channel: a molecular complex system. Biophysical chemistry, 82(2), 173-81. https://doi.org/10.1016/s0301-4622(99)00117-9
Kasai, M, et al. "Regulation of the ryanodine receptor calcium release channel: a molecular complex system." Biophysical chemistry vol. 82,2 (1999): 173-81. doi: https://doi.org/10.1016/s0301-4622(99)00117-9
Kasai M, Kawasaki T, Yamaguchi N. Regulation of the ryanodine receptor calcium release channel: a molecular complex system. Biophys Chem. 1999 Dec 13;82(2):173-81. doi: 10.1016/s0301-4622(99)00117-9. PMID: 17030344.
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Increased Reactive Oxygen Species-Mediated Ca.

Circulation

Liu X, Wang S, Guo X, Li Y, Ogurlu R, Lu F, Prondzynski M, de la Serna Buzon S, Ma Q, Zhang D, Wang G, Cotton J, Guo Y, Xiao L, Milan DJ, Xu Y, Schlame M, Bezzerides VJ, Pu WT.
PMID: 33793303
Circulation. 2021 May 11;143(19):1894-1911. doi: 10.1161/CIRCULATIONAHA.120.048698. Epub 2021 Apr 01.

BACKGROUND: Mutations in tafazzin (METHODS: We performed in vivo electrophysiology to define arrhythmia vulnerability in cardiac-specific RESULTS: A subset of mice with cardiac-specific CONCLUSIONS: This study identified a molecular pathway that links

Cite
Liu X, Wang S, Guo X, et al. Increased Reactive Oxygen Species-Mediated Ca. Circulation. 2021;143(19):1894-1911doi: 10.1161/CIRCULATIONAHA.120.048698.
Liu, X., Wang, S., Guo, X., Li, Y., Ogurlu, R., Lu, F., Prondzynski, M., de la Serna Buzon, S., Ma, Q., Zhang, D., Wang, G., Cotton, J., Guo, Y., Xiao, L., Milan, D. J., Xu, Y., Schlame, M., Bezzerides, V. J., & Pu, W. T. (2021). Increased Reactive Oxygen Species-Mediated Ca. Circulation, 143(19), 1894-1911. https://doi.org/10.1161/CIRCULATIONAHA.120.048698
Liu, Xujie, et al. "Increased Reactive Oxygen Species-Mediated Ca." Circulation vol. 143,19 (2021): 1894-1911. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.048698
Liu X, Wang S, Guo X, Li Y, Ogurlu R, Lu F, Prondzynski M, de la Serna Buzon S, Ma Q, Zhang D, Wang G, Cotton J, Guo Y, Xiao L, Milan DJ, Xu Y, Schlame M, Bezzerides VJ, Pu WT. Increased Reactive Oxygen Species-Mediated Ca. Circulation. 2021 May 11;143(19):1894-1911. doi: 10.1161/CIRCULATIONAHA.120.048698. Epub 2021 Apr 01. PMID: 33793303; PMCID: PMC8691127.
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Showing 1 to 12 of 17 entries