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Showing 1 to 12 of 18 entries
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High concentrations of LH cause virilization in a postmenopausal woman.

Clinical case reports

Ahmad I, Anawalt BD.
PMID: 28265377
Clin Case Rep. 2017 Jan 27;5(3):225-228. doi: 10.1002/ccr3.803. eCollection 2017 Mar.

Some testosterone-producing adrenal tumors can be successfully treated with long-acting GnRH analogs and adrenal venous sampling can be useful to detect rare ectopic sex steroid-producing tumors or confirm bilateral adrenal androgen hyperproduction in female hyperandrogenism.

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Ahmad I, Anawalt BD. High concentrations of LH cause virilization in a postmenopausal woman. Clin Case Rep. 2017;5(3):225-228doi: 10.1002/ccr3.803.
Ahmad, I., & Anawalt, B. D. (2017). High concentrations of LH cause virilization in a postmenopausal woman. Clinical case reports, 5(3), 225-228. https://doi.org/10.1002/ccr3.803
Ahmad, Iram, and Anawalt, Bradley D. "High concentrations of LH cause virilization in a postmenopausal woman." Clinical case reports vol. 5,3 (2017): 225-228. doi: https://doi.org/10.1002/ccr3.803
Ahmad I, Anawalt BD. High concentrations of LH cause virilization in a postmenopausal woman. Clin Case Rep. 2017 Jan 27;5(3):225-228. doi: 10.1002/ccr3.803. eCollection 2017 Mar. PMID: 28265377; PMCID: PMC5331285.
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Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response.

Molecular metabolism

Rojas JM, Matsen ME, Mundinger TO, Morton GJ, Stefanovski D, Bergman RN, Kaiyala KJ, Taborsky GJ, Schwartz MW.
PMID: 26266088
Mol Metab. 2015 Jun 04;4(8):561-8. doi: 10.1016/j.molmet.2015.05.005. eCollection 2015 Aug.

OBJECTIVE: Central administration of ligands for fibroblast growth factor receptors (FGFRs) such as fibroblast growth factor-19 (FGF19) and FGF21 exert glucose-lowering effects in rodent models of obesity and type 2 diabetes (T2D). Conversely, intracerebroventricular (icv) administration of the non-selective...

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Rojas JM, Matsen ME, Mundinger TO, et al. Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response. Mol Metab. 2015;4(8):561-8doi: 10.1016/j.molmet.2015.05.005.
Rojas, J. M., Matsen, M. E., Mundinger, T. O., Morton, G. J., Stefanovski, D., Bergman, R. N., Kaiyala, K. J., Taborsky, G. J., & Schwartz, M. W. (2015). Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response. Molecular metabolism, 4(8), 561-8. https://doi.org/10.1016/j.molmet.2015.05.005
Rojas, Jennifer M, et al. "Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response." Molecular metabolism vol. 4,8 (2015): 561-8. doi: https://doi.org/10.1016/j.molmet.2015.05.005
Rojas JM, Matsen ME, Mundinger TO, Morton GJ, Stefanovski D, Bergman RN, Kaiyala KJ, Taborsky GJ, Schwartz MW. Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response. Mol Metab. 2015 Jun 04;4(8):561-8. doi: 10.1016/j.molmet.2015.05.005. eCollection 2015 Aug. PMID: 26266088; PMCID: PMC4529498.
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Leptin receptor neurons in the dorsomedial hypothalamus regulate diurnal patterns of feeding, locomotion, and metabolism.

eLife

Faber CL, Deem JD, Phan BA, Doan TP, Ogimoto K, Mirzadeh Z, Schwartz MW, Morton GJ.
PMID: 33527893
Elife. 2021 Feb 02;10. doi: 10.7554/eLife.63671.

The brain plays an essential role in driving daily rhythms of behavior and metabolism in harmony with environmental light-dark cycles. Within the brain, the dorsomedial hypothalamic nucleus (DMH) has been implicated in the integrative circadian control of feeding and...

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Faber CL, Deem JD, Phan BA, et al. Leptin receptor neurons in the dorsomedial hypothalamus regulate diurnal patterns of feeding, locomotion, and metabolism. Elife. 2021;10doi: 10.7554/eLife.63671.
Faber, C. L., Deem, J. D., Phan, B. A., Doan, T. P., Ogimoto, K., Mirzadeh, Z., Schwartz, M. W., & Morton, G. J. (2021). Leptin receptor neurons in the dorsomedial hypothalamus regulate diurnal patterns of feeding, locomotion, and metabolism. eLife, 10. https://doi.org/10.7554/eLife.63671
Faber, Chelsea L, et al. "Leptin receptor neurons in the dorsomedial hypothalamus regulate diurnal patterns of feeding, locomotion, and metabolism." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.63671
Faber CL, Deem JD, Phan BA, Doan TP, Ogimoto K, Mirzadeh Z, Schwartz MW, Morton GJ. Leptin receptor neurons in the dorsomedial hypothalamus regulate diurnal patterns of feeding, locomotion, and metabolism. Elife. 2021 Feb 02;10. doi: 10.7554/eLife.63671. PMID: 33527893; PMCID: PMC7880681.
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Muscle mass, not radiodensity, predicts physical function in cancer patients with or without cachexia.

Oncotarget

Anderson LJ, Chong N, Migula D, Sauer A, Garrison M, Wu P, Dash A, Garcia JM.
PMID: 32499874
Oncotarget. 2020 May 19;11(20):1911-1921. doi: 10.18632/oncotarget.27594. eCollection 2020 May 19.

BACKGROUND: There is a need to better understand the relationship between functional impairment and muscle mass in cancer cachexia. This study aimed to establish the relationship between computed tomography (CT)-derived muscle cross-sectional area (CSA), radiodensity, and skeletal muscle index...

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Anderson LJ, Chong N, Migula D, et al. Muscle mass, not radiodensity, predicts physical function in cancer patients with or without cachexia. Oncotarget. 2020;11(20):1911-1921doi: 10.18632/oncotarget.27594.
Anderson, L. J., Chong, N., Migula, D., Sauer, A., Garrison, M., Wu, P., Dash, A., & Garcia, J. M. (2020). Muscle mass, not radiodensity, predicts physical function in cancer patients with or without cachexia. Oncotarget, 11(20), 1911-1921. https://doi.org/10.18632/oncotarget.27594
Anderson, Lindsey J, et al. "Muscle mass, not radiodensity, predicts physical function in cancer patients with or without cachexia." Oncotarget vol. 11,20 (2020): 1911-1921. doi: https://doi.org/10.18632/oncotarget.27594
Anderson LJ, Chong N, Migula D, Sauer A, Garrison M, Wu P, Dash A, Garcia JM. Muscle mass, not radiodensity, predicts physical function in cancer patients with or without cachexia. Oncotarget. 2020 May 19;11(20):1911-1921. doi: 10.18632/oncotarget.27594. eCollection 2020 May 19. PMID: 32499874; PMCID: PMC7244015.
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Leptin, diabetes, and the brain.

Indian journal of endocrinology and metabolism

Meek TH, Morton GJ.
PMID: 23565487
Indian J Endocrinol Metab. 2012 Dec;16:S534-42. doi: 10.4103/2230-8210.105568.

Diabetes is a major worldwide problem. Despite some progress in the development of new antidiabetic agents, the ability to maintain tight glycemic control in order to prevent renal, retinal, and neuropathic complications of diabetes without adverse complications still remains...

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Meek TH, Morton GJ. Leptin, diabetes, and the brain. Indian J Endocrinol Metab. 2012;16:S534-42doi: 10.4103/2230-8210.105568.
Meek, T. H., & Morton, G. J. (2012). Leptin, diabetes, and the brain. Indian journal of endocrinology and metabolism, 16S534-42. https://doi.org/10.4103/2230-8210.105568
Meek, Thomas H, and Morton, Gregory J. "Leptin, diabetes, and the brain." Indian journal of endocrinology and metabolism vol. 16 (2012): S534-42. doi: https://doi.org/10.4103/2230-8210.105568
Meek TH, Morton GJ. Leptin, diabetes, and the brain. Indian J Endocrinol Metab. 2012 Dec;16:S534-42. doi: 10.4103/2230-8210.105568. PMID: 23565487; PMCID: PMC3602981.
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Genetically encoded biosensors for evaluating NAD.

Cell reports methods

Hu Q, Wu D, Walker M, Wang P, Tian R, Wang W.
PMID: 34901920
Cell Rep Methods. 2021 Nov 22;1(7). doi: 10.1016/j.crmeth.2021.100116. Epub 2021 Nov 15.

The ratio of oxidized to reduced NAD (NAD

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Hu Q, Wu D, Walker M, et al. Genetically encoded biosensors for evaluating NAD. Cell Rep Methods. 2021;1(7)doi: 10.1016/j.crmeth.2021.100116.
Hu, Q., Wu, D., Walker, M., Wang, P., Tian, R., & Wang, W. (2021). Genetically encoded biosensors for evaluating NAD. Cell reports methods, 1(7), . https://doi.org/10.1016/j.crmeth.2021.100116
Hu, Qingxun, et al. "Genetically encoded biosensors for evaluating NAD." Cell reports methods vol. 1,7 (2021). doi: https://doi.org/10.1016/j.crmeth.2021.100116
Hu Q, Wu D, Walker M, Wang P, Tian R, Wang W. Genetically encoded biosensors for evaluating NAD. Cell Rep Methods. 2021 Nov 22;1(7). doi: 10.1016/j.crmeth.2021.100116. Epub 2021 Nov 15. PMID: 34901920; PMCID: PMC8659198.
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Androgens and estrogens predict sexual function after autologous hematopoietic stem cell transplant in men.

Andrology

Anderson LJ, Migula D, Abay R, Crabtree S, Graf SA, Matsumoto AM, Chauncey TR, Garcia JM.
PMID: 34624176
Andrology. 2022 Feb;10(2):291-302. doi: 10.1111/andr.13117. Epub 2021 Nov 25.

BACKGROUND: Autologous hematopoietic stem cell transplantation (AHSCT) is associated with sexual dysfunction and hypogonadism. Androgens are associated with sexual function in healthy men, but the role of estrogens is less well-known, and the association of these sex steroids with...

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Anderson LJ, Migula D, Abay R, et al. Androgens and estrogens predict sexual function after autologous hematopoietic stem cell transplant in men. Andrology. 2021;10(2):291-302doi: 10.1111/andr.13117.
Anderson, L. J., Migula, D., Abay, R., Crabtree, S., Graf, S. A., Matsumoto, A. M., Chauncey, T. R., & Garcia, J. M. (2022). Androgens and estrogens predict sexual function after autologous hematopoietic stem cell transplant in men. Andrology, 10(2), 291-302. https://doi.org/10.1111/andr.13117
Anderson, Lindsey J, et al. "Androgens and estrogens predict sexual function after autologous hematopoietic stem cell transplant in men." Andrology vol. 10,2 (2022): 291-302. doi: https://doi.org/10.1111/andr.13117
Anderson LJ, Migula D, Abay R, Crabtree S, Graf SA, Matsumoto AM, Chauncey TR, Garcia JM. Androgens and estrogens predict sexual function after autologous hematopoietic stem cell transplant in men. Andrology. 2022 Feb;10(2):291-302. doi: 10.1111/andr.13117. Epub 2021 Nov 25. PMID: 34624176; PMCID: PMC8760151.
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Experience of a Pituitary Clinic for US Military Veterans With Traumatic Brain Injury.

Journal of the Endocrine Society

Lee J, Anderson LJ, Migula D, Yuen KCJ, McPeak L, Garcia JM.
PMID: 33655093
J Endocr Soc. 2021 Jan 25;5(4):bvab005. doi: 10.1210/jendso/bvab005. eCollection 2021 Apr 01.

CONTEXT: Traumatic brain injury (TBI) is considered the "signature" injury of veterans returning from wartime conflicts in Iraq and Afghanistan. While moderate/severe TBI is associated with pituitary dysfunction, this association has not been well established in the military setting...

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Lee J, Anderson LJ, Migula D, et al. Experience of a Pituitary Clinic for US Military Veterans With Traumatic Brain Injury. J Endocr Soc. 2021;5(4):bvab005doi: 10.1210/jendso/bvab005.
Lee, J., Anderson, L. J., Migula, D., Yuen, K. C. J., McPeak, L., & Garcia, J. M. (2021). Experience of a Pituitary Clinic for US Military Veterans With Traumatic Brain Injury. Journal of the Endocrine Society, 5(4), bvab005. https://doi.org/10.1210/jendso/bvab005
Lee, Jonathan, et al. "Experience of a Pituitary Clinic for US Military Veterans With Traumatic Brain Injury." Journal of the Endocrine Society vol. 5,4 (2021): bvab005. doi: https://doi.org/10.1210/jendso/bvab005
Lee J, Anderson LJ, Migula D, Yuen KCJ, McPeak L, Garcia JM. Experience of a Pituitary Clinic for US Military Veterans With Traumatic Brain Injury. J Endocr Soc. 2021 Jan 25;5(4):bvab005. doi: 10.1210/jendso/bvab005. eCollection 2021 Apr 01. PMID: 33655093; PMCID: PMC7903186.
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Rapid glutamate release in the mediobasal hypothalamus accompanies feeding and is exaggerated by an obesogenic food.

Molecular metabolism

Guyenet SJ, Matsen ME, Morton GJ, Kaiyala KJ, Schwartz MW.
PMID: 24199157
Mol Metab. 2013 Feb 11;2(2):116-22. doi: 10.1016/j.molmet.2013.02.001. eCollection 2013.

The mediobasal hypothalamus (MBH) plays a central role in the regulation of food intake and energy balance. Although the excitatory neurotransmitter glutamate is implicated in energy balance regulation by the MBH, the hypothesis that feeding elicits local glutamate release...

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Guyenet SJ, Matsen ME, Morton GJ, et al. Rapid glutamate release in the mediobasal hypothalamus accompanies feeding and is exaggerated by an obesogenic food. Mol Metab. 2013;2(2):116-22doi: 10.1016/j.molmet.2013.02.001.
Guyenet, S. J., Matsen, M. E., Morton, G. J., Kaiyala, K. J., & Schwartz, M. W. (2013). Rapid glutamate release in the mediobasal hypothalamus accompanies feeding and is exaggerated by an obesogenic food. Molecular metabolism, 2(2), 116-22. https://doi.org/10.1016/j.molmet.2013.02.001
Guyenet, Stephan J, et al. "Rapid glutamate release in the mediobasal hypothalamus accompanies feeding and is exaggerated by an obesogenic food." Molecular metabolism vol. 2,2 (2013): 116-22. doi: https://doi.org/10.1016/j.molmet.2013.02.001
Guyenet SJ, Matsen ME, Morton GJ, Kaiyala KJ, Schwartz MW. Rapid glutamate release in the mediobasal hypothalamus accompanies feeding and is exaggerated by an obesogenic food. Mol Metab. 2013 Feb 11;2(2):116-22. doi: 10.1016/j.molmet.2013.02.001. eCollection 2013. PMID: 24199157; PMCID: PMC3817387.
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Treatment of Hypogonadism: Current and Future Therapies.

F1000Research

Thirumalai A, Berkseth KE, Amory JK.
PMID: 28149506
F1000Res. 2017 Jan 23;6:68. doi: 10.12688/f1000research.10102.1. eCollection 2017.

The treatment of hypogonadism in men is of great interest to both patients and providers. There are a number of testosterone formulations currently available and several additional formulations under development. In addition, there are some lesser-used alternative therapies for...

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Thirumalai A, Berkseth KE, Amory JK. Treatment of Hypogonadism: Current and Future Therapies. F1000Res. 2017;6:68doi: 10.12688/f1000research.10102.1.
Thirumalai, A., Berkseth, K. E., & Amory, J. K. (2017). Treatment of Hypogonadism: Current and Future Therapies. F1000Research, 668. https://doi.org/10.12688/f1000research.10102.1
Thirumalai, Arthi, et al. "Treatment of Hypogonadism: Current and Future Therapies." F1000Research vol. 6 (2017): 68. doi: https://doi.org/10.12688/f1000research.10102.1
Thirumalai A, Berkseth KE, Amory JK. Treatment of Hypogonadism: Current and Future Therapies. F1000Res. 2017 Jan 23;6:68. doi: 10.12688/f1000research.10102.1. eCollection 2017. PMID: 28149506; PMCID: PMC5265703.
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Acute sleep disruption- and high-fat diet-induced hypothalamic inflammation are not related to glucose tolerance in mice.

Neurobiology of sleep and circadian rhythms

Ho JM, Ducich NH, Nguyen NK, Opp MR.
PMID: 29732438
Neurobiol Sleep Circadian Rhythms. 2018 Jan;4:1-9. doi: 10.1016/j.nbscr.2017.09.003.

Chronic insufficient sleep is a major societal problem and is associated with increased risk of metabolic disease. Hypothalamic inflammation contributes to hyperphagia and weight gain in diet-induced obesity, but insufficient sleep-induced neuroinflammation has yet to be examined in relation...

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Ho JM, Ducich NH, Nguyen NK, et al. Acute sleep disruption- and high-fat diet-induced hypothalamic inflammation are not related to glucose tolerance in mice. Neurobiol Sleep Circadian Rhythms. 2018;4:1-9doi: 10.1016/j.nbscr.2017.09.003.
Ho, J. M., Ducich, N. H., Nguyen, N. K., & Opp, M. R. (2018). Acute sleep disruption- and high-fat diet-induced hypothalamic inflammation are not related to glucose tolerance in mice. Neurobiology of sleep and circadian rhythms, 41-9. https://doi.org/10.1016/j.nbscr.2017.09.003
Ho, Jacqueline M, et al. "Acute sleep disruption- and high-fat diet-induced hypothalamic inflammation are not related to glucose tolerance in mice." Neurobiology of sleep and circadian rhythms vol. 4 (2018): 1-9. doi: https://doi.org/10.1016/j.nbscr.2017.09.003
Ho JM, Ducich NH, Nguyen NK, Opp MR. Acute sleep disruption- and high-fat diet-induced hypothalamic inflammation are not related to glucose tolerance in mice. Neurobiol Sleep Circadian Rhythms. 2018 Jan;4:1-9. doi: 10.1016/j.nbscr.2017.09.003. PMID: 29732438; PMCID: PMC5931726.
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An update on testosterone, HDL and cardiovascular risk in men.

Clinical lipidology

Thirumalai A, Rubinow KB, Page ST.
PMID: 26257830
Clin Lipidol. 2015;10(3):251-258. doi: 10.2217/clp.15.10.

Testosterone prescriptions have risen steadily and sharply in the USA despite a lack of clear understanding of the relationship between androgens and cardiovascular disease. In men with increasing age, testosterone levels decline and cardiovascular disease risk goes up. Ties...

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Thirumalai A, Rubinow KB, Page ST. An update on testosterone, HDL and cardiovascular risk in men. Clin Lipidol. 2015;10(3):251-258doi: 10.2217/clp.15.10.
Thirumalai, A., Rubinow, K. B., & Page, S. T. (2015). An update on testosterone, HDL and cardiovascular risk in men. Clinical lipidology, 10(3), 251-258. https://doi.org/10.2217/clp.15.10
Thirumalai, Arthi, et al. "An update on testosterone, HDL and cardiovascular risk in men." Clinical lipidology vol. 10,3 (2015): 251-258. doi: https://doi.org/10.2217/clp.15.10
Thirumalai A, Rubinow KB, Page ST. An update on testosterone, HDL and cardiovascular risk in men. Clin Lipidol. 2015;10(3):251-258. doi: 10.2217/clp.15.10. PMID: 26257830; PMCID: PMC4527564.
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Showing 1 to 12 of 18 entries