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Chronic Unexplained Nausea and Vomiting or Gastric Neuromuscular Dysfunction (GND)? An Update on Nomenclature, Pathophysiology and Treatment, and Relationship to Gastroparesis.

Current treatment options in gastroenterology

Harer KN, Pasricha PJ.
PMID: 27722801
Curr Treat Options Gastroenterol. 2016 Dec;14(4):410-419. doi: 10.1007/s11938-016-0113-z.

OPINION STATEMENT: Chronic unexplained nausea and vomiting is a debilitating condition that dramatically decreases patient quality of life and creates diagnostic and treatment challenges for healthcare providers. Additionally, the significant overlap in symptoms between disorders such as chronic unexplained...

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Harer KN, Pasricha PJ. Chronic Unexplained Nausea and Vomiting or Gastric Neuromuscular Dysfunction (GND)? An Update on Nomenclature, Pathophysiology and Treatment, and Relationship to Gastroparesis. Curr Treat Options Gastroenterol. 2016;14(4):410-419doi: 10.1007/s11938-016-0113-z.
Harer, K. N., & Pasricha, P. J. (2016). Chronic Unexplained Nausea and Vomiting or Gastric Neuromuscular Dysfunction (GND)? An Update on Nomenclature, Pathophysiology and Treatment, and Relationship to Gastroparesis. Current treatment options in gastroenterology, 14(4), 410-419. https://doi.org/10.1007/s11938-016-0113-z
Harer, Kimberly N, and Pasricha, Pankaj J. "Chronic Unexplained Nausea and Vomiting or Gastric Neuromuscular Dysfunction (GND)? An Update on Nomenclature, Pathophysiology and Treatment, and Relationship to Gastroparesis." Current treatment options in gastroenterology vol. 14,4 (2016): 410-419. doi: https://doi.org/10.1007/s11938-016-0113-z
Harer KN, Pasricha PJ. Chronic Unexplained Nausea and Vomiting or Gastric Neuromuscular Dysfunction (GND)? An Update on Nomenclature, Pathophysiology and Treatment, and Relationship to Gastroparesis. Curr Treat Options Gastroenterol. 2016 Dec;14(4):410-419. doi: 10.1007/s11938-016-0113-z. PMID: 27722801; PMCID: PMC5102760.
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Candida esophagitis in a human immunodeficiency virus-1-positive elite controller with hepatitis C virus cirrhosis.

Open forum infectious diseases

Chen A, Shieh E, Brinkley S, Blankson JN.
PMID: 25734179
Open Forum Infect Dis. 2014 Dec 23;1(3):ofu111. doi: 10.1093/ofid/ofu111. eCollection 2014 Dec.

We describe a case of Candida esophagitis in a human immunodeficiency virus elite controller with a preserved CD4 count, a population in which opportunistic infections are almost never seen. The patient has hepatitis C virus coinfection and compensated cirrhosis,...

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Chen A, Shieh E, Brinkley S, et al. Candida esophagitis in a human immunodeficiency virus-1-positive elite controller with hepatitis C virus cirrhosis. Open Forum Infect Dis. 2014;1(3):ofu111doi: 10.1093/ofid/ofu111.
Chen, A., Shieh, E., Brinkley, S., & Blankson, J. N. (2014). Candida esophagitis in a human immunodeficiency virus-1-positive elite controller with hepatitis C virus cirrhosis. Open forum infectious diseases, 1(3), ofu111. https://doi.org/10.1093/ofid/ofu111
Chen, Anders, et al. "Candida esophagitis in a human immunodeficiency virus-1-positive elite controller with hepatitis C virus cirrhosis." Open forum infectious diseases vol. 1,3 (2014): ofu111. doi: https://doi.org/10.1093/ofid/ofu111
Chen A, Shieh E, Brinkley S, Blankson JN. Candida esophagitis in a human immunodeficiency virus-1-positive elite controller with hepatitis C virus cirrhosis. Open Forum Infect Dis. 2014 Dec 23;1(3):ofu111. doi: 10.1093/ofid/ofu111. eCollection 2014 Dec. PMID: 25734179; PMCID: PMC4324211.
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Microfluidics platform for measurement of volume changes in immobilized intestinal enteroids.

Biomicrofluidics

Jin BJ, Battula S, Zachos N, Kovbasnjuk O, Fawlke-Abel J, In J, Donowitz M, Verkman AS.
PMID: 24738013
Biomicrofluidics. 2014 Apr 01;8(2):024106. doi: 10.1063/1.4870400. eCollection 2014 Mar.

Intestinal enteroids are ex vivo primary cultured single-layer epithelial cell spheroids of average diameter ∼150 μm with luminal surface facing inward. Measurement of enteroid swelling in response to secretagogues has been applied to genetic testing in cystic fibrosis and...

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Jin BJ, Battula S, Zachos N, et al. Microfluidics platform for measurement of volume changes in immobilized intestinal enteroids. Biomicrofluidics. 2014;8(2):024106doi: 10.1063/1.4870400.
Jin, B. J., Battula, S., Zachos, N., Kovbasnjuk, O., Fawlke-Abel, J., In, J., Donowitz, M., & Verkman, A. S. (2014). Microfluidics platform for measurement of volume changes in immobilized intestinal enteroids. Biomicrofluidics, 8(2), 024106. https://doi.org/10.1063/1.4870400
Jin, Byung-Ju, et al. "Microfluidics platform for measurement of volume changes in immobilized intestinal enteroids." Biomicrofluidics vol. 8,2 (2014): 024106. doi: https://doi.org/10.1063/1.4870400
Jin BJ, Battula S, Zachos N, Kovbasnjuk O, Fawlke-Abel J, In J, Donowitz M, Verkman AS. Microfluidics platform for measurement of volume changes in immobilized intestinal enteroids. Biomicrofluidics. 2014 Apr 01;8(2):024106. doi: 10.1063/1.4870400. eCollection 2014 Mar. PMID: 24738013; PMCID: PMC3976466.
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Enterohemorrhagic .

Cellular and molecular gastroenterology and hepatology

In J, Foulke-Abel J, Zachos NC, Hansen AM, Kaper JB, Bernstein HD, Halushka M, Blutt S, Estes MK, Donowitz M, Kovbasnjuk O.
PMID: 26855967
Cell Mol Gastroenterol Hepatol. 2016 Jan 01;2(1):48-62.e3. doi: 10.1016/j.jcmgh.2015.10.001.

BACKGROUND AND AIMS: Enterohemorrhagic METHODS: Human colonoids derived from adult proximal colonic stem cells were developed into monolayers to study EHEC-epithelial interactions. Monolayer confluency and differentiation were monitored by transepithelial electrical resistance (TER) measurements. The monolayers were apically infected...

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In J, Foulke-Abel J, Zachos NC, et al. Enterohemorrhagic . Cell Mol Gastroenterol Hepatol. 2016;2(1):48-62.e3doi: 10.1016/j.jcmgh.2015.10.001.
In, J., Foulke-Abel, J., Zachos, N. C., Hansen, A. M., Kaper, J. B., Bernstein, H. D., Halushka, M., Blutt, S., Estes, M. K., Donowitz, M., & Kovbasnjuk, O. (2016). Enterohemorrhagic . Cellular and molecular gastroenterology and hepatology, 2(1), 48-62.e3. https://doi.org/10.1016/j.jcmgh.2015.10.001
In, Julie, et al. "Enterohemorrhagic ." Cellular and molecular gastroenterology and hepatology vol. 2,1 (2016): 48-62.e3. doi: https://doi.org/10.1016/j.jcmgh.2015.10.001
In J, Foulke-Abel J, Zachos NC, Hansen AM, Kaper JB, Bernstein HD, Halushka M, Blutt S, Estes MK, Donowitz M, Kovbasnjuk O. Enterohemorrhagic . Cell Mol Gastroenterol Hepatol. 2016 Jan 01;2(1):48-62.e3. doi: 10.1016/j.jcmgh.2015.10.001. PMID: 26855967; PMCID: PMC4740923.
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New insights in the development of pancreatic cancer.

Current opinion in gastroenterology

Sinha S, Leach SD.
PMID: 27454028
Curr Opin Gastroenterol. 2016 Sep;32(5):394-400. doi: 10.1097/MOG.0000000000000295.

PURPOSE OF REVIEW: The review intends to describe recent studies on the development of pancreatic cancer from a genetic, molecular, and microenvironment perspective.RECENT FINDINGS: Pancreatic cancer has been discovered to have distinct molecular subtypes based on transcriptome analyses that...

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Sinha S, Leach SD. New insights in the development of pancreatic cancer. Curr Opin Gastroenterol. 2016;32(5):394-400doi: 10.1097/MOG.0000000000000295.
Sinha, S., & Leach, S. D. (2016). New insights in the development of pancreatic cancer. Current opinion in gastroenterology, 32(5), 394-400. https://doi.org/10.1097/MOG.0000000000000295
Sinha, Smrita, and Leach, Steven D. "New insights in the development of pancreatic cancer." Current opinion in gastroenterology vol. 32,5 (2016): 394-400. doi: https://doi.org/10.1097/MOG.0000000000000295
Sinha S, Leach SD. New insights in the development of pancreatic cancer. Curr Opin Gastroenterol. 2016 Sep;32(5):394-400. doi: 10.1097/MOG.0000000000000295. PMID: 27454028; PMCID: PMC6028298.
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Chemokine (C-C Motif) Receptor 2 Mediates Dendritic Cell Recruitment to the Human Colon but Is Not Responsible for Differences Observed in Dendritic Cell Subsets, Phenotype, and Function Between the Proximal and Distal Colon.

Cellular and molecular gastroenterology and hepatology

Bernardo D, Durant L, Mann ER, Bassity E, Montalvillo E, Man R, Vora R, Reddi D, Bayiroglu F, Fernández-Salazar L, English NR, Peake ST, Landy J, Lee GH, Malietzis G, Siaw YH, Murugananthan AU, Hendy P, Sánchez-Recio E, Phillips RK, Garrote JA, Scott P, Parkhill J, Paulsen M, Hart AL, Al-Hassi HO, Arranz E, Walker AW, Carding SR, Knight SC.
PMID: 26866054
Cell Mol Gastroenterol Hepatol. 2015 Sep 03;2(1):22-39.e5. doi: 10.1016/j.jcmgh.2015.08.006. eCollection 2016 Jan.

BACKGROUND & AIMS: Most knowledge about gastrointestinal (GI)-tract dendritic cells (DC) relies on murine studies where CD103METHODS: Paired proximal (right/ascending) and distal (left/descending) human colonic biopsies from 95 healthy subjects were taken; DC were assessed by flow cytometry and...

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Bernardo D, Durant L, Mann ER, et al. Chemokine (C-C Motif) Receptor 2 Mediates Dendritic Cell Recruitment to the Human Colon but Is Not Responsible for Differences Observed in Dendritic Cell Subsets, Phenotype, and Function Between the Proximal and Distal Colon. Cell Mol Gastroenterol Hepatol. 2015;2(1):22-39.e5doi: 10.1016/j.jcmgh.2015.08.006.
Bernardo, D., Durant, L., Mann, E. R., Bassity, E., Montalvillo, E., Man, R., Vora, R., Reddi, D., Bayiroglu, F., Fernández-Salazar, L., English, N. R., Peake, S. T., Landy, J., Lee, G. H., Malietzis, G., Siaw, Y. H., Murugananthan, A. U., Hendy, P., Sánchez-Recio, E., Phillips, R. K., Garrote, J. A., Scott, P., Parkhill, J., Paulsen, M., Hart, A. L., Al-Hassi, H. O., Arranz, E., Walker, A. W., Carding, S. R., & Knight, S. C. (2016). Chemokine (C-C Motif) Receptor 2 Mediates Dendritic Cell Recruitment to the Human Colon but Is Not Responsible for Differences Observed in Dendritic Cell Subsets, Phenotype, and Function Between the Proximal and Distal Colon. Cellular and molecular gastroenterology and hepatology, 2(1), 22-39.e5. https://doi.org/10.1016/j.jcmgh.2015.08.006
Bernardo, David, et al. "Chemokine (C-C Motif) Receptor 2 Mediates Dendritic Cell Recruitment to the Human Colon but Is Not Responsible for Differences Observed in Dendritic Cell Subsets, Phenotype, and Function Between the Proximal and Distal Colon." Cellular and molecular gastroenterology and hepatology vol. 2,1 (2016): 22-39.e5. doi: https://doi.org/10.1016/j.jcmgh.2015.08.006
Bernardo D, Durant L, Mann ER, Bassity E, Montalvillo E, Man R, Vora R, Reddi D, Bayiroglu F, Fernández-Salazar L, English NR, Peake ST, Landy J, Lee GH, Malietzis G, Siaw YH, Murugananthan AU, Hendy P, Sánchez-Recio E, Phillips RK, Garrote JA, Scott P, Parkhill J, Paulsen M, Hart AL, Al-Hassi HO, Arranz E, Walker AW, Carding SR, Knight SC. Chemokine (C-C Motif) Receptor 2 Mediates Dendritic Cell Recruitment to the Human Colon but Is Not Responsible for Differences Observed in Dendritic Cell Subsets, Phenotype, and Function Between the Proximal and Distal Colon. Cell Mol Gastroenterol Hepatol. 2015 Sep 03;2(1):22-39.e5. doi: 10.1016/j.jcmgh.2015.08.006. eCollection 2016 Jan. PMID: 26866054; PMCID: PMC4705905.
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Modulatory Effects of Gut Microbiota on the Central Nervous System: How Gut Could Play a Role in Neuropsychiatric Health and Diseases.

Journal of neurogastroenterology and motility

Yarandi SS, Peterson DA, Treisman GJ, Moran TH, Pasricha PJ.
PMID: 27032544
J Neurogastroenterol Motil. 2016 Apr 30;22(2):201-12. doi: 10.5056/jnm15146.

Gut microbiome is an integral part of the Gut-Brain axis. It is becoming increasingly recognized that the presence of a healthy and diverse gut microbiota is important to normal cognitive and emotional processing. It was known that altered emotional...

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Yarandi SS, Peterson DA, Treisman GJ, et al. Modulatory Effects of Gut Microbiota on the Central Nervous System: How Gut Could Play a Role in Neuropsychiatric Health and Diseases. J Neurogastroenterol Motil. 2016;22(2):201-12doi: 10.5056/jnm15146.
Yarandi, S. S., Peterson, D. A., Treisman, G. J., Moran, T. H., & Pasricha, P. J. (2016). Modulatory Effects of Gut Microbiota on the Central Nervous System: How Gut Could Play a Role in Neuropsychiatric Health and Diseases. Journal of neurogastroenterology and motility, 22(2), 201-12. https://doi.org/10.5056/jnm15146
Yarandi, Shadi S, et al. "Modulatory Effects of Gut Microbiota on the Central Nervous System: How Gut Could Play a Role in Neuropsychiatric Health and Diseases." Journal of neurogastroenterology and motility vol. 22,2 (2016): 201-12. doi: https://doi.org/10.5056/jnm15146
Yarandi SS, Peterson DA, Treisman GJ, Moran TH, Pasricha PJ. Modulatory Effects of Gut Microbiota on the Central Nervous System: How Gut Could Play a Role in Neuropsychiatric Health and Diseases. J Neurogastroenterol Motil. 2016 Apr 30;22(2):201-12. doi: 10.5056/jnm15146. PMID: 27032544; PMCID: PMC4819858.
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Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps.

Cancer prevention research (Philadelphia, Pa.)

Santiago CN, Rifkin S, Drewes J, Mullin G, Spence E, Hylind LM, Gills JJ, Kafonek D, Cromwell DM, La Luna L, Giardello F, Sears CL.
PMID: 33947705
Cancer Prev Res (Phila). 2021 Jul;14(7):697-708. doi: 10.1158/1940-6207.CAPR-20-0664. Epub 2021 May 04.

Studies have found a positive association between metabolic risk factors, such as obesity and diabetes, and adenomatous polyps (AP). However, fewer studies have assessed the association between sessile serrated polyps (SSP) or synchronous diagnosis of APs and SSPs (synch...

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Santiago CN, Rifkin S, Drewes J, et al. Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps. Cancer Prev Res (Phila). 2021;14(7):697-708doi: 10.1158/1940-6207.CAPR-20-0664.
Santiago, C. N., Rifkin, S., Drewes, J., Mullin, G., Spence, E., Hylind, L. M., Gills, J. J., Kafonek, D., Cromwell, D. M., La Luna, L., Giardello, F., Sears, C. L. (2021). Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps. Cancer prevention research (Philadelphia, Pa.), 14(7), 697-708. https://doi.org/10.1158/1940-6207.CAPR-20-0664
Santiago, Celina N, et al. "Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps." Cancer prevention research (Philadelphia, Pa.) vol. 14,7 (2021): 697-708. doi: https://doi.org/10.1158/1940-6207.CAPR-20-0664
Santiago CN, Rifkin S, Drewes J, Mullin G, Spence E, Hylind LM, Gills JJ, Kafonek D, Cromwell DM, La Luna L, Giardello F, Sears CL. Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps. Cancer Prev Res (Phila). 2021 Jul;14(7):697-708. doi: 10.1158/1940-6207.CAPR-20-0664. Epub 2021 May 04. PMID: 33947705; PMCID: PMC8295232.
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Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps.

Cancer prevention research (Philadelphia, Pa.)

Santiago CN, Rifkin S, Drewes J, Mullin G, Spence E, Hylind LM, Gills JJ, Kafonek D, Cromwell DM, La Luna L, Giardello F, Sears CL.
PMID: 33947705
Cancer Prev Res (Phila). 2021 Jul;14(7):697-708. doi: 10.1158/1940-6207.CAPR-20-0664. Epub 2021 May 04.

Studies have found a positive association between metabolic risk factors, such as obesity and diabetes, and adenomatous polyps (AP). However, fewer studies have assessed the association between sessile serrated polyps (SSP) or synchronous diagnosis of APs and SSPs (synch...

Cite
Santiago CN, Rifkin S, Drewes J, et al. Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps. Cancer Prev Res (Phila). 2021;14(7):697-708doi: 10.1158/1940-6207.CAPR-20-0664.
Santiago, C. N., Rifkin, S., Drewes, J., Mullin, G., Spence, E., Hylind, L. M., Gills, J. J., Kafonek, D., Cromwell, D. M., La Luna, L., Giardello, F., Sears, C. L. (2021). Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps. Cancer prevention research (Philadelphia, Pa.), 14(7), 697-708. https://doi.org/10.1158/1940-6207.CAPR-20-0664
Santiago, Celina N, et al. "Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps." Cancer prevention research (Philadelphia, Pa.) vol. 14,7 (2021): 697-708. doi: https://doi.org/10.1158/1940-6207.CAPR-20-0664
Santiago CN, Rifkin S, Drewes J, Mullin G, Spence E, Hylind LM, Gills JJ, Kafonek D, Cromwell DM, La Luna L, Giardello F, Sears CL. Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps. Cancer Prev Res (Phila). 2021 Jul;14(7):697-708. doi: 10.1158/1940-6207.CAPR-20-0664. Epub 2021 May 04. PMID: 33947705; PMCID: PMC8295232.
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Corrigendum: Functional Coupling of Human Microphysiology Systems: Intestine, Liver, Kidney Proximal Tubule, Blood-Brain Barrier and Skeletal Muscle.

Scientific reports

Vernetti L, Gough A, Baetz N, Blutt S, Broughman JR, Brown JA, Foulke-Abel J, Hasan N, In J, Kelly E, Kovbasnjuk O, Repper J, Senutovitch N, Stabb J, Yeung C, Zachos NC, Donowitz M, Estes M, Himmelfarb J, Truskey G, Wikswo JP, Taylor DL.
PMID: 28300206
Sci Rep. 2017 Mar 16;7:44517. doi: 10.1038/srep44517.

No abstract available.

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Vernetti L, Gough A, Baetz N, et al. Corrigendum: Functional Coupling of Human Microphysiology Systems: Intestine, Liver, Kidney Proximal Tubule, Blood-Brain Barrier and Skeletal Muscle. Sci Rep. 2017;7:44517doi: 10.1038/srep44517.
Vernetti, L., Gough, A., Baetz, N., Blutt, S., Broughman, J. R., Brown, J. A., Foulke-Abel, J., Hasan, N., In, J., Kelly, E., Kovbasnjuk, O., Repper, J., Senutovitch, N., Stabb, J., Yeung, C., Zachos, N. C., Donowitz, M., Estes, M., Himmelfarb, J., Truskey, G., Wikswo, J. P., & Taylor, D. L. (2017). Corrigendum: Functional Coupling of Human Microphysiology Systems: Intestine, Liver, Kidney Proximal Tubule, Blood-Brain Barrier and Skeletal Muscle. Scientific reports, 744517. https://doi.org/10.1038/srep44517
Vernetti, Lawrence, et al. "Corrigendum: Functional Coupling of Human Microphysiology Systems: Intestine, Liver, Kidney Proximal Tubule, Blood-Brain Barrier and Skeletal Muscle." Scientific reports vol. 7 (2017): 44517. doi: https://doi.org/10.1038/srep44517
Vernetti L, Gough A, Baetz N, Blutt S, Broughman JR, Brown JA, Foulke-Abel J, Hasan N, In J, Kelly E, Kovbasnjuk O, Repper J, Senutovitch N, Stabb J, Yeung C, Zachos NC, Donowitz M, Estes M, Himmelfarb J, Truskey G, Wikswo JP, Taylor DL. Corrigendum: Functional Coupling of Human Microphysiology Systems: Intestine, Liver, Kidney Proximal Tubule, Blood-Brain Barrier and Skeletal Muscle. Sci Rep. 2017 Mar 16;7:44517. doi: 10.1038/srep44517. PMID: 28300206; PMCID: PMC5353746.
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Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps.

Cancer prevention research (Philadelphia, Pa.)

Santiago CN, Rifkin S, Drewes J, Mullin G, Spence E, Hylind LM, Gills JJ, Kafonek D, Cromwell DM, La Luna L, Giardello F, Sears CL.
PMID: 33947705
Cancer Prev Res (Phila). 2021 Jul;14(7):697-708. doi: 10.1158/1940-6207.CAPR-20-0664. Epub 2021 May 04.

Studies have found a positive association between metabolic risk factors, such as obesity and diabetes, and adenomatous polyps (AP). However, fewer studies have assessed the association between sessile serrated polyps (SSP) or synchronous diagnosis of APs and SSPs (synch...

Cite
Santiago CN, Rifkin S, Drewes J, et al. Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps. Cancer Prev Res (Phila). 2021;14(7):697-708doi: 10.1158/1940-6207.CAPR-20-0664.
Santiago, C. N., Rifkin, S., Drewes, J., Mullin, G., Spence, E., Hylind, L. M., Gills, J. J., Kafonek, D., Cromwell, D. M., La Luna, L., Giardello, F., Sears, C. L. (2021). Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps. Cancer prevention research (Philadelphia, Pa.), 14(7), 697-708. https://doi.org/10.1158/1940-6207.CAPR-20-0664
Santiago, Celina N, et al. "Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps." Cancer prevention research (Philadelphia, Pa.) vol. 14,7 (2021): 697-708. doi: https://doi.org/10.1158/1940-6207.CAPR-20-0664
Santiago CN, Rifkin S, Drewes J, Mullin G, Spence E, Hylind LM, Gills JJ, Kafonek D, Cromwell DM, La Luna L, Giardello F, Sears CL. Self-reported Metabolic Risk Factor Associations with Adenomatous, Sessile Serrated, and Synchronous Adenomatous and Sessile Serrated Polyps. Cancer Prev Res (Phila). 2021 Jul;14(7):697-708. doi: 10.1158/1940-6207.CAPR-20-0664. Epub 2021 May 04. PMID: 33947705; PMCID: PMC8295232.
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