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Amino Acid Medical Foods Provide a High Dietary Acid Load and Increase Urinary Excretion of Renal Net Acid, Calcium, and Magnesium Compared with Glycomacropeptide Medical Foods in Phenylketonuria.

Journal of nutrition and metabolism

Stroup BM, Sawin EA, Murali SG, Binkley N, Hansen KE, Ney DM.
PMID: 28546877
J Nutr Metab. 2017;2017:1909101. doi: 10.1155/2017/1909101. Epub 2017 May 04.

No abstract available.

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Stroup BM, Sawin EA, Murali SG, et al. Amino Acid Medical Foods Provide a High Dietary Acid Load and Increase Urinary Excretion of Renal Net Acid, Calcium, and Magnesium Compared with Glycomacropeptide Medical Foods in Phenylketonuria. J Nutr Metab. 2017;2017:1909101doi: 10.1155/2017/1909101.
Stroup, B. M., Sawin, E. A., Murali, S. G., Binkley, N., Hansen, K. E., & Ney, D. M. (2017). Amino Acid Medical Foods Provide a High Dietary Acid Load and Increase Urinary Excretion of Renal Net Acid, Calcium, and Magnesium Compared with Glycomacropeptide Medical Foods in Phenylketonuria. Journal of nutrition and metabolism, 20171909101. https://doi.org/10.1155/2017/1909101
Stroup, Bridget M, et al. "Amino Acid Medical Foods Provide a High Dietary Acid Load and Increase Urinary Excretion of Renal Net Acid, Calcium, and Magnesium Compared with Glycomacropeptide Medical Foods in Phenylketonuria." Journal of nutrition and metabolism vol. 2017 (2017): 1909101. doi: https://doi.org/10.1155/2017/1909101
Stroup BM, Sawin EA, Murali SG, Binkley N, Hansen KE, Ney DM. Amino Acid Medical Foods Provide a High Dietary Acid Load and Increase Urinary Excretion of Renal Net Acid, Calcium, and Magnesium Compared with Glycomacropeptide Medical Foods in Phenylketonuria. J Nutr Metab. 2017;2017:1909101. doi: 10.1155/2017/1909101. Epub 2017 May 04. PMID: 28546877; PMCID: PMC5436062.
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Early to Late Endosome Trafficking Controls Secretion and Zymogen Activation in Rodent and Human Pancreatic Acinar Cells.

Cellular and molecular gastroenterology and hepatology

Messenger SW, Thomas DD, Cooley MM, Jones EK, Falkowski MA, August BK, Fernandez LA, Gorelick FS, Groblewski GE.
PMID: 26618189
Cell Mol Gastroenterol Hepatol. 2015 Nov 01;1(6):695-709. doi: 10.1016/j.jcmgh.2015.08.002.

BACKGROUND & AIMS: Pancreatic acinar cells have an expanded apical endosomal system, the physiological and pathophysiological significance of which is still emerging. Phosphatidylinositol-3,5-bisphosphate (PI(3,5)PMETHODS: Inhibition of EE to LE trafficking was achieved using pharmacological inhibitors of PIKfyve, expression of...

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Messenger SW, Thomas DD, Cooley MM, et al. Early to Late Endosome Trafficking Controls Secretion and Zymogen Activation in Rodent and Human Pancreatic Acinar Cells. Cell Mol Gastroenterol Hepatol. 2015;1(6):695-709doi: 10.1016/j.jcmgh.2015.08.002.
Messenger, S. W., Thomas, D. D., Cooley, M. M., Jones, E. K., Falkowski, M. A., August, B. K., Fernandez, L. A., Gorelick, F. S., & Groblewski, G. E. (2015). Early to Late Endosome Trafficking Controls Secretion and Zymogen Activation in Rodent and Human Pancreatic Acinar Cells. Cellular and molecular gastroenterology and hepatology, 1(6), 695-709. https://doi.org/10.1016/j.jcmgh.2015.08.002
Messenger, Scott W, et al. "Early to Late Endosome Trafficking Controls Secretion and Zymogen Activation in Rodent and Human Pancreatic Acinar Cells." Cellular and molecular gastroenterology and hepatology vol. 1,6 (2015): 695-709. doi: https://doi.org/10.1016/j.jcmgh.2015.08.002
Messenger SW, Thomas DD, Cooley MM, Jones EK, Falkowski MA, August BK, Fernandez LA, Gorelick FS, Groblewski GE. Early to Late Endosome Trafficking Controls Secretion and Zymogen Activation in Rodent and Human Pancreatic Acinar Cells. Cell Mol Gastroenterol Hepatol. 2015 Nov 01;1(6):695-709. doi: 10.1016/j.jcmgh.2015.08.002. PMID: 26618189; PMCID: PMC4657148.
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Roquin is a major mediator of iron-regulated changes to transferrin receptor-1 mRNA stability.

iScience

Corral VM, Schultz ER, Eisenstein RS, Connell GJ.
PMID: 33898949
iScience. 2021 Mar 26;24(4):102360. doi: 10.1016/j.isci.2021.102360. eCollection 2021 Apr 23.

Transferrin receptor-1 (TfR1) has essential iron transport and proposed signal transduction functions. Proper TfR1 regulation is a requirement for hematopoiesis, neurological development, and the homeostasis of tissues including the intestine and muscle, while dysregulation is associated with cancers and...

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Corral VM, Schultz ER, Eisenstein RS, et al. Roquin is a major mediator of iron-regulated changes to transferrin receptor-1 mRNA stability. iScience. 2021;24(4):102360doi: 10.1016/j.isci.2021.102360.
Corral, V. M., Schultz, E. R., Eisenstein, R. S., & Connell, G. J. (2021). Roquin is a major mediator of iron-regulated changes to transferrin receptor-1 mRNA stability. iScience, 24(4), 102360. https://doi.org/10.1016/j.isci.2021.102360
Corral, Victor M, et al. "Roquin is a major mediator of iron-regulated changes to transferrin receptor-1 mRNA stability." iScience vol. 24,4 (2021): 102360. doi: https://doi.org/10.1016/j.isci.2021.102360
Corral VM, Schultz ER, Eisenstein RS, Connell GJ. Roquin is a major mediator of iron-regulated changes to transferrin receptor-1 mRNA stability. iScience. 2021 Mar 26;24(4):102360. doi: 10.1016/j.isci.2021.102360. eCollection 2021 Apr 23. PMID: 33898949; PMCID: PMC8058555.
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Deficient Endoplasmic Reticulum Acetyl-CoA Import in Pancreatic Acinar Cells Leads to Chronic Pancreatitis.

Cellular and molecular gastroenterology and hepatology

Cooley MM, Thomas DDH, Deans K, Peng Y, Lugea A, Pandol SJ, Puglielli L, Groblewski GE.
PMID: 33080365
Cell Mol Gastroenterol Hepatol. 2021;11(3):725-738. doi: 10.1016/j.jcmgh.2020.10.008. Epub 2020 Oct 17.

BACKGROUND & AIMS: Maintaining endoplasmic reticulum (ER) proteostasis is essential for pancreatic acinar cell function. Under conditions of severe ER stress, activation of pathogenic unfolded protein response pathways plays a central role in the development and progression of pancreatitis....

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Cooley MM, Thomas DDH, Deans K, et al. Deficient Endoplasmic Reticulum Acetyl-CoA Import in Pancreatic Acinar Cells Leads to Chronic Pancreatitis. Cell Mol Gastroenterol Hepatol. 2020;11(3):725-738doi: 10.1016/j.jcmgh.2020.10.008.
Cooley, M. M., Thomas, D. D. H., Deans, K., Peng, Y., Lugea, A., Pandol, S. J., Puglielli, L., & Groblewski, G. E. (2021). Deficient Endoplasmic Reticulum Acetyl-CoA Import in Pancreatic Acinar Cells Leads to Chronic Pancreatitis. Cellular and molecular gastroenterology and hepatology, 11(3), 725-738. https://doi.org/10.1016/j.jcmgh.2020.10.008
Cooley, Michelle M, et al. "Deficient Endoplasmic Reticulum Acetyl-CoA Import in Pancreatic Acinar Cells Leads to Chronic Pancreatitis." Cellular and molecular gastroenterology and hepatology vol. 11,3 (2021): 725-738. doi: https://doi.org/10.1016/j.jcmgh.2020.10.008
Cooley MM, Thomas DDH, Deans K, Peng Y, Lugea A, Pandol SJ, Puglielli L, Groblewski GE. Deficient Endoplasmic Reticulum Acetyl-CoA Import in Pancreatic Acinar Cells Leads to Chronic Pancreatitis. Cell Mol Gastroenterol Hepatol. 2021;11(3):725-738. doi: 10.1016/j.jcmgh.2020.10.008. Epub 2020 Oct 17. PMID: 33080365; PMCID: PMC7841443.
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Fasting drives the metabolic, molecular and geroprotective effects of a calorie-restricted diet in mice.

Nature metabolism

Pak HH, Haws SA, Green CL, Koller M, Lavarias MT, Richardson NE, Yang SE, Dumas SN, Sonsalla M, Bray L, Johnson M, Barnes S, Darley-Usmar V, Zhang J, Yen CE, Denu JM, Lamming DW.
PMID: 34663973
Nat Metab. 2021 Oct;3(10):1327-1341. doi: 10.1038/s42255-021-00466-9. Epub 2021 Oct 18.

Calorie restriction (CR) promotes healthy ageing in diverse species. Recently, it has been shown that fasting for a portion of each day has metabolic benefits and promotes lifespan. These findings complicate the interpretation of rodent CR studies, in which...

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Pak HH, Haws SA, Green CL, et al. Fasting drives the metabolic, molecular and geroprotective effects of a calorie-restricted diet in mice. Nat Metab. 2021;3(10):1327-1341doi: 10.1038/s42255-021-00466-9.
Pak, H. H., Haws, S. A., Green, C. L., Koller, M., Lavarias, M. T., Richardson, N. E., Yang, S. E., Dumas, S. N., Sonsalla, M., Bray, L., Johnson, M., Barnes, S., Darley-Usmar, V., Zhang, J., Yen, C. E., Denu, J. M., & Lamming, D. W. (2021). Fasting drives the metabolic, molecular and geroprotective effects of a calorie-restricted diet in mice. Nature metabolism, 3(10), 1327-1341. https://doi.org/10.1038/s42255-021-00466-9
Pak, Heidi H, et al. "Fasting drives the metabolic, molecular and geroprotective effects of a calorie-restricted diet in mice." Nature metabolism vol. 3,10 (2021): 1327-1341. doi: https://doi.org/10.1038/s42255-021-00466-9
Pak HH, Haws SA, Green CL, Koller M, Lavarias MT, Richardson NE, Yang SE, Dumas SN, Sonsalla M, Bray L, Johnson M, Barnes S, Darley-Usmar V, Zhang J, Yen CE, Denu JM, Lamming DW. Fasting drives the metabolic, molecular and geroprotective effects of a calorie-restricted diet in mice. Nat Metab. 2021 Oct;3(10):1327-1341. doi: 10.1038/s42255-021-00466-9. Epub 2021 Oct 18. PMID: 34663973; PMCID: PMC8544824.
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Complement Blockade in Recipients Prevents Delayed Graft Function and Delays Antibody-mediated Rejection in a Nonhuman Primate Model of Kidney Transplantation.

Transplantation

Eerhart MJ, Reyes JA, Blanton CL, Danobeitia JS, Chlebeck PJ, Zitur LJ, Springer M, Polyak E, Coonen J, Capuano S, D'Alessandro AM, Torrealba J, van Amersfoort E, Ponstein Y, van Kooten C, Burlingham W, Sullivan J, Pozniak M, Zhong W, Yankol Y, Fernandez LA.
PMID: 34905763
Transplantation. 2022 Jan 01;106(1):60-71. doi: 10.1097/TP.0000000000003754.

BACKGROUND: Complement activation in kidney transplantation is implicated in the pathogenesis of delayed graft function (DGF). This study evaluated the therapeutic efficacy of high-dose recombinant human C1 esterase inhibitor (rhC1INH) to prevent DGF in a nonhuman primate model of...

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Eerhart MJ, Reyes JA, Blanton CL, et al. Complement Blockade in Recipients Prevents Delayed Graft Function and Delays Antibody-mediated Rejection in a Nonhuman Primate Model of Kidney Transplantation. Transplantation. 2022;106(1):60-71doi: 10.1097/TP.0000000000003754.
Eerhart, M. J., Reyes, J. A., Blanton, C. L., Danobeitia, J. S., Chlebeck, P. J., Zitur, L. J., Springer, M., Polyak, E., Coonen, J., Capuano, S., D'Alessandro, A. M., Torrealba, J., van Amersfoort, E., Ponstein, Y., van Kooten, C., Burlingham, W., Sullivan, J., Pozniak, M., Zhong, W., Yankol, Y., & Fernandez, L. A. (2022). Complement Blockade in Recipients Prevents Delayed Graft Function and Delays Antibody-mediated Rejection in a Nonhuman Primate Model of Kidney Transplantation. Transplantation, 106(1), 60-71. https://doi.org/10.1097/TP.0000000000003754
Eerhart, Michael J, et al. "Complement Blockade in Recipients Prevents Delayed Graft Function and Delays Antibody-mediated Rejection in a Nonhuman Primate Model of Kidney Transplantation." Transplantation vol. 106,1 (2022): 60-71. doi: https://doi.org/10.1097/TP.0000000000003754
Eerhart MJ, Reyes JA, Blanton CL, Danobeitia JS, Chlebeck PJ, Zitur LJ, Springer M, Polyak E, Coonen J, Capuano S, D'Alessandro AM, Torrealba J, van Amersfoort E, Ponstein Y, van Kooten C, Burlingham W, Sullivan J, Pozniak M, Zhong W, Yankol Y, Fernandez LA. Complement Blockade in Recipients Prevents Delayed Graft Function and Delays Antibody-mediated Rejection in a Nonhuman Primate Model of Kidney Transplantation. Transplantation. 2022 Jan 01;106(1):60-71. doi: 10.1097/TP.0000000000003754. PMID: 34905763; PMCID: PMC8674492.
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Exposure to dietary lipid leads to rapid production of cytosolic lipid droplets near the brush border membrane.

Nutrition & metabolism

Soayfane Z, Tercé F, Cantiello M, Robenek H, Nauze M, Bézirard V, Allart S, Payré B, Capilla F, Cartier C, Peres C, Al Saati T, Théodorou V, Nelson DW, Yen CE, Collet X, Coméra C.
PMID: 27478484
Nutr Metab (Lond). 2016 Jul 28;13:48. doi: 10.1186/s12986-016-0107-9. eCollection 2016.

BACKGROUND: Intestinal absorption of dietary lipids involves their hydrolysis in the lumen of proximal intestine as well as uptake, intracellular transport and re-assembly of hydrolyzed lipids in enterocytes, leading to the formation and secretion of the lipoproteins chylomicrons and...

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Soayfane Z, Tercé F, Cantiello M, et al. Exposure to dietary lipid leads to rapid production of cytosolic lipid droplets near the brush border membrane. Nutr Metab (Lond). 2016;13:48doi: 10.1186/s12986-016-0107-9.
Soayfane, Z., Tercé, F., Cantiello, M., Robenek, H., Nauze, M., Bézirard, V., Allart, S., Payré, B., Capilla, F., Cartier, C., Peres, C., Al Saati, T., Théodorou, V., Nelson, D. W., Yen, C. E., Collet, X., & Coméra, C. (2016). Exposure to dietary lipid leads to rapid production of cytosolic lipid droplets near the brush border membrane. Nutrition & metabolism, 1348. https://doi.org/10.1186/s12986-016-0107-9
Soayfane, Zeina, et al. "Exposure to dietary lipid leads to rapid production of cytosolic lipid droplets near the brush border membrane." Nutrition & metabolism vol. 13 (2016): 48. doi: https://doi.org/10.1186/s12986-016-0107-9
Soayfane Z, Tercé F, Cantiello M, Robenek H, Nauze M, Bézirard V, Allart S, Payré B, Capilla F, Cartier C, Peres C, Al Saati T, Théodorou V, Nelson DW, Yen CE, Collet X, Coméra C. Exposure to dietary lipid leads to rapid production of cytosolic lipid droplets near the brush border membrane. Nutr Metab (Lond). 2016 Jul 28;13:48. doi: 10.1186/s12986-016-0107-9. eCollection 2016. PMID: 27478484; PMCID: PMC4965885.
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Complement Blockade in Recipients Prevents Delayed Graft Function and Delays Antibody-mediated Rejection in a Nonhuman Primate Model of Kidney Transplantation.

Transplantation

Eerhart MJ, Reyes JA, Blanton CL, Danobeitia JS, Chlebeck PJ, Zitur LJ, Springer M, Polyak E, Coonen J, Capuano S, D'Alessandro AM, Torrealba J, van Amersfoort E, Ponstein Y, van Kooten C, Burlingham W, Sullivan J, Pozniak M, Zhong W, Yankol Y, Fernandez LA.
PMID: 34905763
Transplantation. 2022 Jan 01;106(1):60-71. doi: 10.1097/TP.0000000000003754.

BACKGROUND: Complement activation in kidney transplantation is implicated in the pathogenesis of delayed graft function (DGF). This study evaluated the therapeutic efficacy of high-dose recombinant human C1 esterase inhibitor (rhC1INH) to prevent DGF in a nonhuman primate model of...

Cite
Eerhart MJ, Reyes JA, Blanton CL, et al. Complement Blockade in Recipients Prevents Delayed Graft Function and Delays Antibody-mediated Rejection in a Nonhuman Primate Model of Kidney Transplantation. Transplantation. 2022;106(1):60-71doi: 10.1097/TP.0000000000003754.
Eerhart, M. J., Reyes, J. A., Blanton, C. L., Danobeitia, J. S., Chlebeck, P. J., Zitur, L. J., Springer, M., Polyak, E., Coonen, J., Capuano, S., D'Alessandro, A. M., Torrealba, J., van Amersfoort, E., Ponstein, Y., van Kooten, C., Burlingham, W., Sullivan, J., Pozniak, M., Zhong, W., Yankol, Y., & Fernandez, L. A. (2022). Complement Blockade in Recipients Prevents Delayed Graft Function and Delays Antibody-mediated Rejection in a Nonhuman Primate Model of Kidney Transplantation. Transplantation, 106(1), 60-71. https://doi.org/10.1097/TP.0000000000003754
Eerhart, Michael J, et al. "Complement Blockade in Recipients Prevents Delayed Graft Function and Delays Antibody-mediated Rejection in a Nonhuman Primate Model of Kidney Transplantation." Transplantation vol. 106,1 (2022): 60-71. doi: https://doi.org/10.1097/TP.0000000000003754
Eerhart MJ, Reyes JA, Blanton CL, Danobeitia JS, Chlebeck PJ, Zitur LJ, Springer M, Polyak E, Coonen J, Capuano S, D'Alessandro AM, Torrealba J, van Amersfoort E, Ponstein Y, van Kooten C, Burlingham W, Sullivan J, Pozniak M, Zhong W, Yankol Y, Fernandez LA. Complement Blockade in Recipients Prevents Delayed Graft Function and Delays Antibody-mediated Rejection in a Nonhuman Primate Model of Kidney Transplantation. Transplantation. 2022 Jan 01;106(1):60-71. doi: 10.1097/TP.0000000000003754. PMID: 34905763; PMCID: PMC8674492.
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Special Considerations for Measuring Energy Expenditure with Doubly Labeled Water under Atypical Conditions.

Journal of obesity & weight loss therapy

Bhutani S, Racine N, Shriver T, Schoeller DA.
PMID: 26962472
J Obes Weight Loss Ther. 2015 Jul;5. doi: 10.4172/2165-7904.S5-002. Epub 2015 Jul 30.

The global increase in the prevalence of obesity has dramatically increased interest in understanding the factors that influence human total energy expenditure (TEE). This in turn has increased interest in the doubly labeled water (DLW) method, a technique for...

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Bhutani S, Racine N, Shriver T, et al. Special Considerations for Measuring Energy Expenditure with Doubly Labeled Water under Atypical Conditions. J Obes Weight Loss Ther. 2015;5doi: 10.4172/2165-7904.S5-002.
Bhutani, S., Racine, N., Shriver, T., & Schoeller, D. A. (2015). Special Considerations for Measuring Energy Expenditure with Doubly Labeled Water under Atypical Conditions. Journal of obesity & weight loss therapy, 5. https://doi.org/10.4172/2165-7904.S5-002
Bhutani, Surabhi, et al. "Special Considerations for Measuring Energy Expenditure with Doubly Labeled Water under Atypical Conditions." Journal of obesity & weight loss therapy vol. 5 (2015). doi: https://doi.org/10.4172/2165-7904.S5-002
Bhutani S, Racine N, Shriver T, Schoeller DA. Special Considerations for Measuring Energy Expenditure with Doubly Labeled Water under Atypical Conditions. J Obes Weight Loss Ther. 2015 Jul;5. doi: 10.4172/2165-7904.S5-002. Epub 2015 Jul 30. PMID: 26962472; PMCID: PMC4780846.
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Evaluation of a multi-year policy-focused intervention to increase physical activity and related behaviors in lower-resourced early care and education settings: .

Preventive medicine reports

Tomayko EJ, Prince RJ, Hoiting J, Braun A, LaRowe TL, Adams AK.
PMID: 28936391
Prev Med Rep. 2017 Sep 06;8:93-100. doi: 10.1016/j.pmedr.2017.08.008. eCollection 2017 Dec.

Physical activity is a critical component of obesity prevention, but few interventions targeting early childhood have been described. The

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Tomayko EJ, Prince RJ, Hoiting J, et al. Evaluation of a multi-year policy-focused intervention to increase physical activity and related behaviors in lower-resourced early care and education settings: . Prev Med Rep. 2017;8:93-100doi: 10.1016/j.pmedr.2017.08.008.
Tomayko, E. J., Prince, R. J., Hoiting, J., Braun, A., LaRowe, T. L., & Adams, A. K. (2017). Evaluation of a multi-year policy-focused intervention to increase physical activity and related behaviors in lower-resourced early care and education settings: . Preventive medicine reports, 893-100. https://doi.org/10.1016/j.pmedr.2017.08.008
Tomayko, Emily J, et al. "Evaluation of a multi-year policy-focused intervention to increase physical activity and related behaviors in lower-resourced early care and education settings: ." Preventive medicine reports vol. 8 (2017): 93-100. doi: https://doi.org/10.1016/j.pmedr.2017.08.008
Tomayko EJ, Prince RJ, Hoiting J, Braun A, LaRowe TL, Adams AK. Evaluation of a multi-year policy-focused intervention to increase physical activity and related behaviors in lower-resourced early care and education settings: . Prev Med Rep. 2017 Sep 06;8:93-100. doi: 10.1016/j.pmedr.2017.08.008. eCollection 2017 Dec. PMID: 28936391; PMCID: PMC5602886.
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Zinc-responsive coactivator recruitment by the yeast Zap1 transcription factor.

MicrobiologyOpen

Frey AG, Eide DJ.
PMID: 22950018
Microbiologyopen. 2012 Jun;1(2):105-14. doi: 10.1002/mbo3.8.

The zinc-responsive Zap1 transcription factor of Saccharomyces cerevisiae controls many genes involved in zinc homeostasis. Zap1 has two activation domains, AD1 and AD2, which are independently regulated by zinc. While AD1 can fully activate most Zap1 target genes, AD2...

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Frey AG, Eide DJ. Zinc-responsive coactivator recruitment by the yeast Zap1 transcription factor. Microbiologyopen. 2012;1(2):105-14doi: 10.1002/mbo3.8.
Frey, A. G., & Eide, D. J. (2012). Zinc-responsive coactivator recruitment by the yeast Zap1 transcription factor. MicrobiologyOpen, 1(2), 105-14. https://doi.org/10.1002/mbo3.8
Frey, Avery G, and Eide, David J. "Zinc-responsive coactivator recruitment by the yeast Zap1 transcription factor." MicrobiologyOpen vol. 1,2 (2012): 105-14. doi: https://doi.org/10.1002/mbo3.8
Frey AG, Eide DJ. Zinc-responsive coactivator recruitment by the yeast Zap1 transcription factor. Microbiologyopen. 2012 Jun;1(2):105-14. doi: 10.1002/mbo3.8. PMID: 22950018; PMCID: PMC3426420.
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Mitochondrial regulator PGC-1a-Modulating the modulator.

Current opinion in endocrine and metabolic research

Miller KN, Clark JP, Anderson RM.
PMID: 31406949
Curr Opin Endocr Metab Res. 2019 Mar;5:37-44. doi: 10.1016/j.coemr.2019.02.002. Epub 2019 Feb 27.

Peroxisome proliferator-activated receptor gamma coactivator-1 alpha (PGC-1a) is a central regulator of metabolism that is poised at the intersection of myriad intracellular signaling pathways. In this brief update, we discuss regulation of PGC-1a at multiple levels, including transcriptional, post-transcriptional,...

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Miller KN, Clark JP, Anderson RM. Mitochondrial regulator PGC-1a-Modulating the modulator. Curr Opin Endocr Metab Res. 2019;5:37-44doi: 10.1016/j.coemr.2019.02.002.
Miller, K. N., Clark, J. P., & Anderson, R. M. (2019). Mitochondrial regulator PGC-1a-Modulating the modulator. Current opinion in endocrine and metabolic research, 537-44. https://doi.org/10.1016/j.coemr.2019.02.002
Miller, Karl N, et al. "Mitochondrial regulator PGC-1a-Modulating the modulator." Current opinion in endocrine and metabolic research vol. 5 (2019): 37-44. doi: https://doi.org/10.1016/j.coemr.2019.02.002
Miller KN, Clark JP, Anderson RM. Mitochondrial regulator PGC-1a-Modulating the modulator. Curr Opin Endocr Metab Res. 2019 Mar;5:37-44. doi: 10.1016/j.coemr.2019.02.002. Epub 2019 Feb 27. PMID: 31406949; PMCID: PMC6690794.
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