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Peripheral tissue homing receptor control of naïve, effector, and memory CD8 T cell localization in lymphoid and non-lymphoid tissues.

Frontiers in immunology

Brinkman CC, Peske JD, Engelhard VH.
PMID: 23966998
Front Immunol. 2013 Aug 19;4:241. doi: 10.3389/fimmu.2013.00241. eCollection 2013.

T cell activation induces homing receptors that bind ligands on peripheral tissue vasculature, programing movement to sites of infection and injury. There are three major types of CD8 effector T cells based on homing receptor expression, which arise in...

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Brinkman CC, Peske JD, Engelhard VH. Peripheral tissue homing receptor control of naïve, effector, and memory CD8 T cell localization in lymphoid and non-lymphoid tissues. Front Immunol. 2013;4:241doi: 10.3389/fimmu.2013.00241.
Brinkman, C. C., Peske, J. D., & Engelhard, V. H. (2013). Peripheral tissue homing receptor control of naïve, effector, and memory CD8 T cell localization in lymphoid and non-lymphoid tissues. Frontiers in immunology, 4241. https://doi.org/10.3389/fimmu.2013.00241
Brinkman, C Colin, et al. "Peripheral tissue homing receptor control of naïve, effector, and memory CD8 T cell localization in lymphoid and non-lymphoid tissues." Frontiers in immunology vol. 4 (2013): 241. doi: https://doi.org/10.3389/fimmu.2013.00241
Brinkman CC, Peske JD, Engelhard VH. Peripheral tissue homing receptor control of naïve, effector, and memory CD8 T cell localization in lymphoid and non-lymphoid tissues. Front Immunol. 2013 Aug 19;4:241. doi: 10.3389/fimmu.2013.00241. eCollection 2013. PMID: 23966998; PMCID: PMC3746678.
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Spontaneous mutations in .

Microbial cell (Graz, Austria)

Maqani N, Fine RD, Shahid M, Li M, Enriquez-Hesles E, Smith JS.
PMID: 29796388
Microb Cell. 2018 Feb 19;5(5):233-248. doi: 10.15698/mic2018.05.630.

Chronologically aging yeast cells are prone to adaptive regrowth, whereby mutants with a survival advantage spontaneously appear and re-enter the cell cycle in stationary phase cultures. Adaptive regrowth is especially noticeable with short-lived strains, including those defective for SNF1,...

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Maqani N, Fine RD, Shahid M, et al. Spontaneous mutations in . Microb Cell. 2018;5(5):233-248doi: 10.15698/mic2018.05.630.
Maqani, N., Fine, R. D., Shahid, M., Li, M., Enriquez-Hesles, E., & Smith, J. S. (2018). Spontaneous mutations in . Microbial cell (Graz, Austria), 5(5), 233-248. https://doi.org/10.15698/mic2018.05.630
Maqani, Nazif, et al. "Spontaneous mutations in ." Microbial cell (Graz, Austria) vol. 5,5 (2018): 233-248. doi: https://doi.org/10.15698/mic2018.05.630
Maqani N, Fine RD, Shahid M, Li M, Enriquez-Hesles E, Smith JS. Spontaneous mutations in . Microb Cell. 2018 Feb 19;5(5):233-248. doi: 10.15698/mic2018.05.630. PMID: 29796388; PMCID: PMC5961917.
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A specialized pathway for erythroid iron delivery through lysosomal trafficking of transferrin receptor 2.

Blood advances

Khalil S, Holy M, Grado S, Fleming R, Kurita R, Nakamura Y, Goldfarb A.
PMID: 29296759
Blood Adv. 2017 Jun 27;1(15):1181-1194. doi: 10.1182/bloodadvances.2016003772. eCollection 2017 Jun 27.

Erythroid progenitors are the largest consumers of iron in the human body. In these cells, a high flux of iron must reach the mitochondrial matrix to form sufficient heme to support hemoglobinization. Canonical erythroid iron trafficking occurs via the...

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Khalil S, Holy M, Grado S, et al. A specialized pathway for erythroid iron delivery through lysosomal trafficking of transferrin receptor 2. Blood Adv. 2017;1(15):1181-1194doi: 10.1182/bloodadvances.2016003772.
Khalil, S., Holy, M., Grado, S., Fleming, R., Kurita, R., Nakamura, Y., & Goldfarb, A. (2017). A specialized pathway for erythroid iron delivery through lysosomal trafficking of transferrin receptor 2. Blood advances, 1(15), 1181-1194. https://doi.org/10.1182/bloodadvances.2016003772
Khalil, Shadi, et al. "A specialized pathway for erythroid iron delivery through lysosomal trafficking of transferrin receptor 2." Blood advances vol. 1,15 (2017): 1181-1194. doi: https://doi.org/10.1182/bloodadvances.2016003772
Khalil S, Holy M, Grado S, Fleming R, Kurita R, Nakamura Y, Goldfarb A. A specialized pathway for erythroid iron delivery through lysosomal trafficking of transferrin receptor 2. Blood Adv. 2017 Jun 27;1(15):1181-1194. doi: 10.1182/bloodadvances.2016003772. eCollection 2017 Jun 27. PMID: 29296759; PMCID: PMC5728310.
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Frequent somatic TET2 mutations in chronic NK-LGL leukemia with distinct patterns of cytopenias.

Blood

Olson TL, Cheon H, Xing JC, Olson KC, Paila U, Hamele CE, Neelamraju Y, Shemo BC, Schmachtenberg M, Sundararaman SK, Toro MF, Keller CA, Farber EA, Onengut-Gumuscu S, Garrett-Bakelman FE, Hardison RC, Feith DJ, Ratan A, Loughran TP.
PMID: 33786584
Blood. 2021 Aug 26;138(8):662-673. doi: 10.1182/blood.2020005831.

Chronic natural killer large granular lymphocyte (NK-LGL) leukemia, also referred to as chronic lymphoproliferative disorder of NK cells, is a rare disorder defined by prolonged expansion of clonal NK cells. Similar prevalence of STAT3 mutations in chronic T-LGL and...

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Olson TL, Cheon H, Xing JC, et al. Frequent somatic TET2 mutations in chronic NK-LGL leukemia with distinct patterns of cytopenias. Blood. 2021;138(8):662-673doi: 10.1182/blood.2020005831.
Olson, T. L., Cheon, H., Xing, J. C., Olson, K. C., Paila, U., Hamele, C. E., Neelamraju, Y., Shemo, B. C., Schmachtenberg, M., Sundararaman, S. K., Toro, M. F., Keller, C. A., Farber, E. A., Onengut-Gumuscu, S., Garrett-Bakelman, F. E., Hardison, R. C., Feith, D. J., Ratan, A., & Loughran, T. P. (2021). Frequent somatic TET2 mutations in chronic NK-LGL leukemia with distinct patterns of cytopenias. Blood, 138(8), 662-673. https://doi.org/10.1182/blood.2020005831
Olson, Thomas L, et al. "Frequent somatic TET2 mutations in chronic NK-LGL leukemia with distinct patterns of cytopenias." Blood vol. 138,8 (2021): 662-673. doi: https://doi.org/10.1182/blood.2020005831
Olson TL, Cheon H, Xing JC, Olson KC, Paila U, Hamele CE, Neelamraju Y, Shemo BC, Schmachtenberg M, Sundararaman SK, Toro MF, Keller CA, Farber EA, Onengut-Gumuscu S, Garrett-Bakelman FE, Hardison RC, Feith DJ, Ratan A, Loughran TP. Frequent somatic TET2 mutations in chronic NK-LGL leukemia with distinct patterns of cytopenias. Blood. 2021 Aug 26;138(8):662-673. doi: 10.1182/blood.2020005831. PMID: 33786584; PMCID: PMC8394905.
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ATP and large signaling metabolites flux through caspase-activated Pannexin 1 channels.

eLife

Narahari AK, Kreutzberger AJ, Gaete PS, Chiu YH, Leonhardt SA, Medina CB, Jin X, Oleniacz PW, Kiessling V, Barrett PQ, Ravichandran KS, Yeager M, Contreras JE, Tamm LK, Bayliss DA.
PMID: 33410749
Elife. 2021 Jan 07;10. doi: 10.7554/eLife.64787.

Pannexin 1 (Panx1) is a membrane channel implicated in numerous physiological and pathophysiological processes via its ability to support release of ATP and other cellular metabolites for local intercellular signaling. However, to date, there has been no direct demonstration...

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Narahari AK, Kreutzberger AJ, Gaete PS, et al. ATP and large signaling metabolites flux through caspase-activated Pannexin 1 channels. Elife. 2021;10doi: 10.7554/eLife.64787.
Narahari, A. K., Kreutzberger, A. J., Gaete, P. S., Chiu, Y. H., Leonhardt, S. A., Medina, C. B., Jin, X., Oleniacz, P. W., Kiessling, V., Barrett, P. Q., Ravichandran, K. S., Yeager, M., Contreras, J. E., Tamm, L. K., & Bayliss, D. A. (2021). ATP and large signaling metabolites flux through caspase-activated Pannexin 1 channels. eLife, 10. https://doi.org/10.7554/eLife.64787
Narahari, Adishesh K, et al. "ATP and large signaling metabolites flux through caspase-activated Pannexin 1 channels." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.64787
Narahari AK, Kreutzberger AJ, Gaete PS, Chiu YH, Leonhardt SA, Medina CB, Jin X, Oleniacz PW, Kiessling V, Barrett PQ, Ravichandran KS, Yeager M, Contreras JE, Tamm LK, Bayliss DA. ATP and large signaling metabolites flux through caspase-activated Pannexin 1 channels. Elife. 2021 Jan 07;10. doi: 10.7554/eLife.64787. PMID: 33410749; PMCID: PMC7806264.
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One Seizure Please, Hold the Sprouts: The Role of Hippocampal Mossy Fiber Sprouting in Epilepsy.

Epilepsy currents

Beenhakker M, Ritger M.
PMID: 31558042
Epilepsy Curr. 2019 Nov-Dec;19(6):414-416. doi: 10.1177/1535759719876556. Epub 2019 Sep 26.

[Box: see text].

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Beenhakker M, Ritger M. One Seizure Please, Hold the Sprouts: The Role of Hippocampal Mossy Fiber Sprouting in Epilepsy. Epilepsy Curr. 2019;19(6):414-416doi: 10.1177/1535759719876556.
Beenhakker, M., & Ritger, M. (2019). One Seizure Please, Hold the Sprouts: The Role of Hippocampal Mossy Fiber Sprouting in Epilepsy. Epilepsy currents, 19(6), 414-416. https://doi.org/10.1177/1535759719876556
Beenhakker, Mark, and Ritger, Matthew. "One Seizure Please, Hold the Sprouts: The Role of Hippocampal Mossy Fiber Sprouting in Epilepsy." Epilepsy currents vol. 19,6 (2019): 414-416. doi: https://doi.org/10.1177/1535759719876556
Beenhakker M, Ritger M. One Seizure Please, Hold the Sprouts: The Role of Hippocampal Mossy Fiber Sprouting in Epilepsy. Epilepsy Curr. 2019 Nov-Dec;19(6):414-416. doi: 10.1177/1535759719876556. Epub 2019 Sep 26. PMID: 31558042; PMCID: PMC6891178.
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Rapamycin up-regulation of autophagy reduces infarct size and improves outcomes in both permanent MCAL, and embolic MCAO, murine models of stroke.

Experimental & translational stroke medicine

Buckley KM, Hess DL, Sazonova IY, Periyasamy-Thandavan S, Barrett JR, Kirks R, Grace H, Kondrikova G, Johnson MH, Hess DC, Schoenlein PV, Hoda MN, Hill WD.
PMID: 24991402
Exp Transl Stroke Med. 2014 Jun 21;6:8. doi: 10.1186/2040-7378-6-8. eCollection 2014.

BACKGROUND AND PURPOSE: The role of autophagy in response to ischemic stroke has been confusing with reports that both enhancement and inhibition of autophagy decrease infarct size and improve post-stroke outcomes. We sought to clarify this by comparing pharmacologic...

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Buckley KM, Hess DL, Sazonova IY, et al. Rapamycin up-regulation of autophagy reduces infarct size and improves outcomes in both permanent MCAL, and embolic MCAO, murine models of stroke. Exp Transl Stroke Med. 2014;6:8doi: 10.1186/2040-7378-6-8.
Buckley, K. M., Hess, D. L., Sazonova, I. Y., Periyasamy-Thandavan, S., Barrett, J. R., Kirks, R., Grace, H., Kondrikova, G., Johnson, M. H., Hess, D. C., Schoenlein, P. V., Hoda, M. N., & Hill, W. D. (2014). Rapamycin up-regulation of autophagy reduces infarct size and improves outcomes in both permanent MCAL, and embolic MCAO, murine models of stroke. Experimental & translational stroke medicine, 68. https://doi.org/10.1186/2040-7378-6-8
Buckley, Kathleen M, et al. "Rapamycin up-regulation of autophagy reduces infarct size and improves outcomes in both permanent MCAL, and embolic MCAO, murine models of stroke." Experimental & translational stroke medicine vol. 6 (2014): 8. doi: https://doi.org/10.1186/2040-7378-6-8
Buckley KM, Hess DL, Sazonova IY, Periyasamy-Thandavan S, Barrett JR, Kirks R, Grace H, Kondrikova G, Johnson MH, Hess DC, Schoenlein PV, Hoda MN, Hill WD. Rapamycin up-regulation of autophagy reduces infarct size and improves outcomes in both permanent MCAL, and embolic MCAO, murine models of stroke. Exp Transl Stroke Med. 2014 Jun 21;6:8. doi: 10.1186/2040-7378-6-8. eCollection 2014. PMID: 24991402; PMCID: PMC4079187.
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Regulation of T-cell Tolerance by Lymphatic Endothelial Cells.

Journal of clinical & cellular immunology

Rouhani SJ, Eccles JD, Tewalt EF, Engelhard VH.
PMID: 25580369
J Clin Cell Immunol. 2014;5. doi: 10.4172/2155-9899.1000242.

Lymphatic endothelial cells are most often thought of as structural cells that form the lymphatic vasculature, which transports fluid out of peripheral tissues and transports antigens and antigen presenting cells to lymph nodes. Recently, it has been shown that...

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Rouhani SJ, Eccles JD, Tewalt EF, et al. Regulation of T-cell Tolerance by Lymphatic Endothelial Cells. J Clin Cell Immunol. 2014;5doi: 10.4172/2155-9899.1000242.
Rouhani, S. J., Eccles, J. D., Tewalt, E. F., & Engelhard, V. H. (2014). Regulation of T-cell Tolerance by Lymphatic Endothelial Cells. Journal of clinical & cellular immunology, 5. https://doi.org/10.4172/2155-9899.1000242
Rouhani, Sherin J, et al. "Regulation of T-cell Tolerance by Lymphatic Endothelial Cells." Journal of clinical & cellular immunology vol. 5 (2014). doi: https://doi.org/10.4172/2155-9899.1000242
Rouhani SJ, Eccles JD, Tewalt EF, Engelhard VH. Regulation of T-cell Tolerance by Lymphatic Endothelial Cells. J Clin Cell Immunol. 2014;5. doi: 10.4172/2155-9899.1000242. PMID: 25580369; PMCID: PMC4286360.
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Single-cell mapping of focused ultrasound-transfected brain.

Gene therapy

Mathew AS, Gorick CM, Price RJ.
PMID: 33526842
Gene Ther. 2021 Feb 01; doi: 10.1038/s41434-021-00226-0. Epub 2021 Feb 01.

Gene delivery via focused ultrasound (FUS) mediated blood-brain barrier (BBB) opening is a disruptive therapeutic modality. Unlocking its full potential will require an understanding of how FUS parameters (e.g., peak-negative pressure (PNP)) affect transfected cell populations. Following plasmid (mRuby)...

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Mathew AS, Gorick CM, Price RJ. Single-cell mapping of focused ultrasound-transfected brain. Gene Ther. 2021;doi: 10.1038/s41434-021-00226-0.
Mathew, A. S., Gorick, C. M., & Price, R. J. (2021). Single-cell mapping of focused ultrasound-transfected brain. Gene therapy, . https://doi.org/10.1038/s41434-021-00226-0
Mathew, A S, et al. "Single-cell mapping of focused ultrasound-transfected brain." Gene therapy vol. (2021). doi: https://doi.org/10.1038/s41434-021-00226-0
Mathew AS, Gorick CM, Price RJ. Single-cell mapping of focused ultrasound-transfected brain. Gene Ther. 2021 Feb 01; doi: 10.1038/s41434-021-00226-0. Epub 2021 Feb 01. PMID: 33526842; PMCID: PMC8325700.
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Fractalkine is a "find-me" signal released by neurons undergoing ethanol-induced apoptosis.

Frontiers in cellular neuroscience

Sokolowski JD, Chabanon-Hicks CN, Han CZ, Heffron DS, Mandell JW.
PMID: 25426022
Front Cell Neurosci. 2014 Nov 07;8:360. doi: 10.3389/fncel.2014.00360. eCollection 2014.

Apoptotic neurons generated during normal brain development or secondary to pathologic insults are efficiently cleared from the central nervous system. Several soluble factors, including nucleotides, cytokines, and chemokines are released from injured neurons, signaling microglia to find and clear...

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Sokolowski JD, Chabanon-Hicks CN, Han CZ, et al. Fractalkine is a "find-me" signal released by neurons undergoing ethanol-induced apoptosis. Front Cell Neurosci. 2014;8:360doi: 10.3389/fncel.2014.00360.
Sokolowski, J. D., Chabanon-Hicks, C. N., Han, C. Z., Heffron, D. S., & Mandell, J. W. (2014). Fractalkine is a "find-me" signal released by neurons undergoing ethanol-induced apoptosis. Frontiers in cellular neuroscience, 8360. https://doi.org/10.3389/fncel.2014.00360
Sokolowski, Jennifer D, et al. "Fractalkine is a "find-me" signal released by neurons undergoing ethanol-induced apoptosis." Frontiers in cellular neuroscience vol. 8 (2014): 360. doi: https://doi.org/10.3389/fncel.2014.00360
Sokolowski JD, Chabanon-Hicks CN, Han CZ, Heffron DS, Mandell JW. Fractalkine is a "find-me" signal released by neurons undergoing ethanol-induced apoptosis. Front Cell Neurosci. 2014 Nov 07;8:360. doi: 10.3389/fncel.2014.00360. eCollection 2014. PMID: 25426022; PMCID: PMC4224129.
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Lymphatic endothelial cells - key players in regulation of tolerance and immunity.

Frontiers in immunology

Tewalt EF, Cohen JN, Rouhani SJ, Engelhard VH.
PMID: 23060883
Front Immunol. 2012 Sep 28;3:305. doi: 10.3389/fimmu.2012.00305. eCollection 2012.

The lymphatic vasculature provides routes for dendritic cell and lymphocyte migration into and out of lymph nodes. Lymphatic endothelial cells (LEC) control these processes by expression of CCL21, sphingosine-1-phosphate, and adhesion molecules. LEC express MHC-I and MHC-II, but not...

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Tewalt EF, Cohen JN, Rouhani SJ, et al. Lymphatic endothelial cells - key players in regulation of tolerance and immunity. Front Immunol. 2012;3:305doi: 10.3389/fimmu.2012.00305.
Tewalt, E. F., Cohen, J. N., Rouhani, S. J., & Engelhard, V. H. (2012). Lymphatic endothelial cells - key players in regulation of tolerance and immunity. Frontiers in immunology, 3305. https://doi.org/10.3389/fimmu.2012.00305
Tewalt, Eric F, et al. "Lymphatic endothelial cells - key players in regulation of tolerance and immunity." Frontiers in immunology vol. 3 (2012): 305. doi: https://doi.org/10.3389/fimmu.2012.00305
Tewalt EF, Cohen JN, Rouhani SJ, Engelhard VH. Lymphatic endothelial cells - key players in regulation of tolerance and immunity. Front Immunol. 2012 Sep 28;3:305. doi: 10.3389/fimmu.2012.00305. eCollection 2012. PMID: 23060883; PMCID: PMC3460259.
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Exploring Non-Metabolic Functions of Glycolytic Enzymes in Immunity.

Frontiers in immunology

Seki SM, Gaultier A.
PMID: 29213268
Front Immunol. 2017 Nov 22;8:1549. doi: 10.3389/fimmu.2017.01549. eCollection 2017.

At the beginning of the twentieth century, discoveries in cancer research began to elucidate the idiosyncratic metabolic proclivities of tumor cells (1). Investigators postulated that revealing the distinct nutritional requirements of cells with unchecked growth potential would reveal targetable...

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Seki SM, Gaultier A. Exploring Non-Metabolic Functions of Glycolytic Enzymes in Immunity. Front Immunol. 2017;8:1549doi: 10.3389/fimmu.2017.01549.
Seki, S. M., & Gaultier, A. (2017). Exploring Non-Metabolic Functions of Glycolytic Enzymes in Immunity. Frontiers in immunology, 81549. https://doi.org/10.3389/fimmu.2017.01549
Seki, Scott M, and Gaultier, Alban. "Exploring Non-Metabolic Functions of Glycolytic Enzymes in Immunity." Frontiers in immunology vol. 8 (2017): 1549. doi: https://doi.org/10.3389/fimmu.2017.01549
Seki SM, Gaultier A. Exploring Non-Metabolic Functions of Glycolytic Enzymes in Immunity. Front Immunol. 2017 Nov 22;8:1549. doi: 10.3389/fimmu.2017.01549. eCollection 2017. PMID: 29213268; PMCID: PMC5702622.
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Showing 1 to 12 of 21 entries