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p53 Acetylation: Regulation and Consequences.

Cancers

Reed SM, Quelle DE.
PMID: 25545885
Cancers (Basel). 2014 Dec 23;7(1):30-69. doi: 10.3390/cancers7010030.

Post-translational modifications of p53 are critical in modulating its tumor suppressive functions. Ubiquitylation, for example, plays a major role in dictating p53 stability, subcellular localization and transcriptional vs. non-transcriptional activities. Less is known about p53 acetylation. It has been...

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Reed SM, Quelle DE. p53 Acetylation: Regulation and Consequences. Cancers (Basel). 2014;7(1):30-69doi: 10.3390/cancers7010030.
Reed, S. M., & Quelle, D. E. (2014). p53 Acetylation: Regulation and Consequences. Cancers, 7(1), 30-69. https://doi.org/10.3390/cancers7010030
Reed, Sara M, and Quelle, Dawn E. "p53 Acetylation: Regulation and Consequences." Cancers vol. 7,1 (2014): 30-69. doi: https://doi.org/10.3390/cancers7010030
Reed SM, Quelle DE. p53 Acetylation: Regulation and Consequences. Cancers (Basel). 2014 Dec 23;7(1):30-69. doi: 10.3390/cancers7010030. PMID: 25545885; PMCID: PMC4381250.
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Paracrine WNT5A signaling in healthy and neoplastic mammary tissue.

Molecular & cellular oncology

Kusner D, Borcherding N, Zhang W.
PMID: 27308558
Mol Cell Oncol. 2015 Jul 28;3(1):e1040145. doi: 10.1080/23723556.2015.1040145. eCollection 2016 Jan.

Paracrine signaling between mammary epithelial cells has long been appreciated. Recently, we found that Wnt5a, a novel noncanonical Wnt ligand of luminal origin, counteracts canonical Wnt signaling in basal mammary epithelial cells through a paracrine pathway, inhibits the expansion...

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Kusner D, Borcherding N, Zhang W. Paracrine WNT5A signaling in healthy and neoplastic mammary tissue. Mol Cell Oncol. 2015;3(1):e1040145doi: 10.1080/23723556.2015.1040145.
Kusner, D., Borcherding, N., & Zhang, W. (2016). Paracrine WNT5A signaling in healthy and neoplastic mammary tissue. Molecular & cellular oncology, 3(1), e1040145. https://doi.org/10.1080/23723556.2015.1040145
Kusner, David, et al. "Paracrine WNT5A signaling in healthy and neoplastic mammary tissue." Molecular & cellular oncology vol. 3,1 (2016): e1040145. doi: https://doi.org/10.1080/23723556.2015.1040145
Kusner D, Borcherding N, Zhang W. Paracrine WNT5A signaling in healthy and neoplastic mammary tissue. Mol Cell Oncol. 2015 Jul 28;3(1):e1040145. doi: 10.1080/23723556.2015.1040145. eCollection 2016 Jan. PMID: 27308558; PMCID: PMC4845198.
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RABL6A Promotes Oxaliplatin Resistance in Tumor Cells and Is a New Marker of Survival for Resected Pancreatic Ductal Adenocarcinoma Patients.

Genes & cancer

Muniz VP, Askeland RW, Zhang X, Reed SM, Tompkins VS, Hagen J, McDowell BD, Button A, Smith BJ, Weydert JA, Mezhir JJ, Quelle DE.
PMID: 24167655
Genes Cancer. 2013 Jul;4(7):273-84. doi: 10.1177/1947601913501074.

Pancreatic ductal adenocarcinoma (PDAC) is characterized by early recurrence following pancreatectomy, rapid progression, and chemoresistance. Novel prognostic and predictive biomarkers are urgently needed to both stratify patients for clinical trials and select patients for adjuvant therapy regimens. This study...

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Muniz VP, Askeland RW, Zhang X, et al. RABL6A Promotes Oxaliplatin Resistance in Tumor Cells and Is a New Marker of Survival for Resected Pancreatic Ductal Adenocarcinoma Patients. Genes Cancer. 2013;4(7):273-84doi: 10.1177/1947601913501074.
Muniz, V. P., Askeland, R. W., Zhang, X., Reed, S. M., Tompkins, V. S., Hagen, J., McDowell, B. D., Button, A., Smith, B. J., Weydert, J. A., Mezhir, J. J., & Quelle, D. E. (2013). RABL6A Promotes Oxaliplatin Resistance in Tumor Cells and Is a New Marker of Survival for Resected Pancreatic Ductal Adenocarcinoma Patients. Genes & cancer, 4(7), 273-84. https://doi.org/10.1177/1947601913501074
Muniz, Viviane P, et al. "RABL6A Promotes Oxaliplatin Resistance in Tumor Cells and Is a New Marker of Survival for Resected Pancreatic Ductal Adenocarcinoma Patients." Genes & cancer vol. 4,7 (2013): 273-84. doi: https://doi.org/10.1177/1947601913501074
Muniz VP, Askeland RW, Zhang X, Reed SM, Tompkins VS, Hagen J, McDowell BD, Button A, Smith BJ, Weydert JA, Mezhir JJ, Quelle DE. RABL6A Promotes Oxaliplatin Resistance in Tumor Cells and Is a New Marker of Survival for Resected Pancreatic Ductal Adenocarcinoma Patients. Genes Cancer. 2013 Jul;4(7):273-84. doi: 10.1177/1947601913501074. PMID: 24167655; PMCID: PMC3807645.
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Regulation of human neutrophil apoptosis and lifespan in health and disease.

Journal of cell death

McCracken JM, Allen LA.
PMID: 25278783
J Cell Death. 2014 May 08;7:15-23. doi: 10.4137/JCD.S11038. eCollection 2014.

Neutrophils (also called polymorphonuclear leukocytes, PMNs) are the most abundant white blood cells in humans and play a central role in innate host defense. Another distinguishing feature of PMNs is their short lifespan. Specifically, these cells survive for less...

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McCracken JM, Allen LA. Regulation of human neutrophil apoptosis and lifespan in health and disease. J Cell Death. 2014;7:15-23doi: 10.4137/JCD.S11038.
McCracken, J. M., & Allen, L. A. (2014). Regulation of human neutrophil apoptosis and lifespan in health and disease. Journal of cell death, 715-23. https://doi.org/10.4137/JCD.S11038
McCracken, Jenna M, and Allen, Lee-Ann H. "Regulation of human neutrophil apoptosis and lifespan in health and disease." Journal of cell death vol. 7 (2014): 15-23. doi: https://doi.org/10.4137/JCD.S11038
McCracken JM, Allen LA. Regulation of human neutrophil apoptosis and lifespan in health and disease. J Cell Death. 2014 May 08;7:15-23. doi: 10.4137/JCD.S11038. eCollection 2014. PMID: 25278783; PMCID: PMC4167320.
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A semi-automatic framework of measuring pulmonary arterial metrics at anatomic airway locations using CT imaging.

Proceedings of SPIE--the International Society for Optical Engineering

Jin D, Guo J, Dougherty TM, Iyer KS, Hoffman EA, Saha PK.
PMID: 28250572
Proc SPIE Int Soc Opt Eng. 2016 Feb 27;9788. doi: 10.1117/12.2216558. Epub 2016 Mar 29.

Pulmonary vascular dysfunction has been implicated in smoking-related susceptibility to emphysema. With the growing interest in characterizing arterial morphology for early evaluation of the vascular role in pulmonary diseases, there is an increasing need for the standardization of a...

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Jin D, Guo J, Dougherty TM, et al. A semi-automatic framework of measuring pulmonary arterial metrics at anatomic airway locations using CT imaging. Proc SPIE Int Soc Opt Eng. 2016;9788doi: 10.1117/12.2216558.
Jin, D., Guo, J., Dougherty, T. M., Iyer, K. S., Hoffman, E. A., & Saha, P. K. (2016). A semi-automatic framework of measuring pulmonary arterial metrics at anatomic airway locations using CT imaging. Proceedings of SPIE--the International Society for Optical Engineering, 9788. https://doi.org/10.1117/12.2216558
Jin, Dakai, et al. "A semi-automatic framework of measuring pulmonary arterial metrics at anatomic airway locations using CT imaging." Proceedings of SPIE--the International Society for Optical Engineering vol. 9788 (2016). doi: https://doi.org/10.1117/12.2216558
Jin D, Guo J, Dougherty TM, Iyer KS, Hoffman EA, Saha PK. A semi-automatic framework of measuring pulmonary arterial metrics at anatomic airway locations using CT imaging. Proc SPIE Int Soc Opt Eng. 2016 Feb 27;9788. doi: 10.1117/12.2216558. Epub 2016 Mar 29. PMID: 28250572; PMCID: PMC5327735.
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Correction: Recessive coding and regulatory mutations in FBLIM1 underlie the pathogenesis of chronic recurrent multifocal osteomyelitis (CRMO).

PloS one

Cox AJ, Darbro BW, Laxer RM, Velez G, Bing X, Finer AL, Erives A, Mahajan VB, Bassuk AG, Ferguson PJ.
PMID: 28686717
PLoS One. 2017 Jul 07;12(7):e0181222. doi: 10.1371/journal.pone.0181222. eCollection 2017.

[This corrects the article DOI: 10.1371/journal.pone.0169687.].

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Cox AJ, Darbro BW, Laxer RM, et al. Correction: Recessive coding and regulatory mutations in FBLIM1 underlie the pathogenesis of chronic recurrent multifocal osteomyelitis (CRMO). PLoS One. 2017;12(7):e0181222doi: 10.1371/journal.pone.0181222.
Cox, A. J., Darbro, B. W., Laxer, R. M., Velez, G., Bing, X., Finer, A. L., Erives, A., Mahajan, V. B., Bassuk, A. G., & Ferguson, P. J. (2017). Correction: Recessive coding and regulatory mutations in FBLIM1 underlie the pathogenesis of chronic recurrent multifocal osteomyelitis (CRMO). PloS one, 12(7), e0181222. https://doi.org/10.1371/journal.pone.0181222
Cox, Allison J, et al. "Correction: Recessive coding and regulatory mutations in FBLIM1 underlie the pathogenesis of chronic recurrent multifocal osteomyelitis (CRMO)." PloS one vol. 12,7 (2017): e0181222. doi: https://doi.org/10.1371/journal.pone.0181222
Cox AJ, Darbro BW, Laxer RM, Velez G, Bing X, Finer AL, Erives A, Mahajan VB, Bassuk AG, Ferguson PJ. Correction: Recessive coding and regulatory mutations in FBLIM1 underlie the pathogenesis of chronic recurrent multifocal osteomyelitis (CRMO). PLoS One. 2017 Jul 07;12(7):e0181222. doi: 10.1371/journal.pone.0181222. eCollection 2017. PMID: 28686717; PMCID: PMC5501673.
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O.

Cancer cell

Schoenfeld JD, Sibenaller ZA, Mapuskar KA, Wagner BA, Cramer-Morales KL, Furqan M, Sandhu S, Carlisle TL, Smith MC, Abu Hejleh T, Berg DJ, Zhang J, Keech J, Parekh KR, Bhatia S, Monga V, Bodeker KL, Ahmann L, Vollstedt S, Brown H, Kauffman EPS, Schall ME, Hohl RJ, Clamon GH, Greenlee JD, Howard MA, Schultz MK, Smith BJ, Riley DP, Domann FE, Cullen JJ, Buettner GR, Buatti JM, Spitz DR, Allen BG.
PMID: 28810149
Cancer Cell. 2017 Aug 14;32(2):268. doi: 10.1016/j.ccell.2017.07.008.

No abstract available.

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Schoenfeld JD, Sibenaller ZA, Mapuskar KA, et al. O. Cancer Cell. 2017;32(2):268doi: 10.1016/j.ccell.2017.07.008.
Schoenfeld, J. D., Sibenaller, Z. A., Mapuskar, K. A., Wagner, B. A., Cramer-Morales, K. L., Furqan, M., Sandhu, S., Carlisle, T. L., Smith, M. C., Abu Hejleh, T., Berg, D. J., Zhang, J., Keech, J., Parekh, K. R., Bhatia, S., Monga, V., Bodeker, K. L., Ahmann, L., Vollstedt, S., Brown, H., Kauffman, E. P. S., Schall, M. E., Hohl, R. J., Clamon, G. H., Greenlee, J. D., Howard, M. A., Schultz, M. K., Smith, B. J., Riley, D. P., Domann, F. E., Cullen, J. J., Buettner, G. R., Buatti, J. M., Spitz, D. R., & Allen, B. G. (2017). O. Cancer cell, 32(2), 268. https://doi.org/10.1016/j.ccell.2017.07.008
Schoenfeld, Joshua D, et al. "O." Cancer cell vol. 32,2 (2017): 268. doi: https://doi.org/10.1016/j.ccell.2017.07.008
Schoenfeld JD, Sibenaller ZA, Mapuskar KA, Wagner BA, Cramer-Morales KL, Furqan M, Sandhu S, Carlisle TL, Smith MC, Abu Hejleh T, Berg DJ, Zhang J, Keech J, Parekh KR, Bhatia S, Monga V, Bodeker KL, Ahmann L, Vollstedt S, Brown H, Kauffman EPS, Schall ME, Hohl RJ, Clamon GH, Greenlee JD, Howard MA, Schultz MK, Smith BJ, Riley DP, Domann FE, Cullen JJ, Buettner GR, Buatti JM, Spitz DR, Allen BG. O. Cancer Cell. 2017 Aug 14;32(2):268. doi: 10.1016/j.ccell.2017.07.008. PMID: 28810149.
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Assessment of Whole-Exome Sequence Data in Attempted Suicide within a Bipolar Disorder Cohort.

Molecular neuropsychiatry

Monson ET, Pirooznia M, Parla J, Kramer M, Goes FS, Gaine ME, Gaynor SC, de Klerk K, Jancic D, Karchin R, McCombie WR, Zandi PP, Potash JB, Willour VL.
PMID: 28879196
Mol Neuropsychiatry. 2017 Jul;3(1):1-11. doi: 10.1159/000454773. Epub 2017 Jan 18.

Suicidal behavior is a complex and devastating phenotype with a heritable component that has not been fully explained by existing common genetic variant analyses. This study represents the first large-scale DNA sequencing project designed to assess the role of...

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Monson ET, Pirooznia M, Parla J, et al. Assessment of Whole-Exome Sequence Data in Attempted Suicide within a Bipolar Disorder Cohort. Mol Neuropsychiatry. 2017;3(1):1-11doi: 10.1159/000454773.
Monson, E. T., Pirooznia, M., Parla, J., Kramer, M., Goes, F. S., Gaine, M. E., Gaynor, S. C., de Klerk, K., Jancic, D., Karchin, R., McCombie, W. R., Zandi, P. P., Potash, J. B., & Willour, V. L. (2017). Assessment of Whole-Exome Sequence Data in Attempted Suicide within a Bipolar Disorder Cohort. Molecular neuropsychiatry, 3(1), 1-11. https://doi.org/10.1159/000454773
Monson, Eric T, et al. "Assessment of Whole-Exome Sequence Data in Attempted Suicide within a Bipolar Disorder Cohort." Molecular neuropsychiatry vol. 3,1 (2017): 1-11. doi: https://doi.org/10.1159/000454773
Monson ET, Pirooznia M, Parla J, Kramer M, Goes FS, Gaine ME, Gaynor SC, de Klerk K, Jancic D, Karchin R, McCombie WR, Zandi PP, Potash JB, Willour VL. Assessment of Whole-Exome Sequence Data in Attempted Suicide within a Bipolar Disorder Cohort. Mol Neuropsychiatry. 2017 Jul;3(1):1-11. doi: 10.1159/000454773. Epub 2017 Jan 18. PMID: 28879196; PMCID: PMC5582499.
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Rapid and Sensitive Detection of Breast Cancer Cells in Patient Blood with Nuclease-Activated Probe Technology.

Molecular therapy. Nucleic acids

Kruspe S, Dickey DD, Urak KT, Blanco GN, Miller MJ, Clark KC, Burghardt E, Gutierrez WR, Phadke SD, Kamboj S, Ginader T, Smith BJ, Grimm SK, Schappet J, Ozer H, Thomas A, McNamara JO, Chan CH, Giangrande PH.
PMID: 28918054
Mol Ther Nucleic Acids. 2017 Sep 15;8:542-557. doi: 10.1016/j.omtn.2017.08.004. Epub 2017 Aug 12.

A challenge for circulating tumor cell (CTC)-based diagnostics is the development of simple and inexpensive methods that reliably detect the diverse cells that make up CTCs. CTC-derived nucleases are one category of proteins that could be exploited to meet...

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Kruspe S, Dickey DD, Urak KT, et al. Rapid and Sensitive Detection of Breast Cancer Cells in Patient Blood with Nuclease-Activated Probe Technology. Mol Ther Nucleic Acids. 2017;8:542-557doi: 10.1016/j.omtn.2017.08.004.
Kruspe, S., Dickey, D. D., Urak, K. T., Blanco, G. N., Miller, M. J., Clark, K. C., Burghardt, E., Gutierrez, W. R., Phadke, S. D., Kamboj, S., Ginader, T., Smith, B. J., Grimm, S. K., Schappet, J., Ozer, H., Thomas, A., McNamara, J. O., Chan, C. H., & Giangrande, P. H. (2017). Rapid and Sensitive Detection of Breast Cancer Cells in Patient Blood with Nuclease-Activated Probe Technology. Molecular therapy. Nucleic acids, 8542-557. https://doi.org/10.1016/j.omtn.2017.08.004
Kruspe, Sven, et al. "Rapid and Sensitive Detection of Breast Cancer Cells in Patient Blood with Nuclease-Activated Probe Technology." Molecular therapy. Nucleic acids vol. 8 (2017): 542-557. doi: https://doi.org/10.1016/j.omtn.2017.08.004
Kruspe S, Dickey DD, Urak KT, Blanco GN, Miller MJ, Clark KC, Burghardt E, Gutierrez WR, Phadke SD, Kamboj S, Ginader T, Smith BJ, Grimm SK, Schappet J, Ozer H, Thomas A, McNamara JO, Chan CH, Giangrande PH. Rapid and Sensitive Detection of Breast Cancer Cells in Patient Blood with Nuclease-Activated Probe Technology. Mol Ther Nucleic Acids. 2017 Sep 15;8:542-557. doi: 10.1016/j.omtn.2017.08.004. Epub 2017 Aug 12. PMID: 28918054; PMCID: PMC5577414.
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ProSave: an application for restoring quantitative data to manipulated subsets of protein lists.

Source code for biology and medicine

Machlab DA, Velez G, Bassuk AG, Mahajan VB.
PMID: 30459825
Source Code Biol Med. 2018 Nov 12;13:3. doi: 10.1186/s13029-018-0070-0. eCollection 2018.

BACKGROUND: In proteomics studies, liquid chromatography tandem mass spectrometry data (LC-MS/MS) is quantified by spectral counts or by some measure of ion abundance. Downstream comparative analysis of protein content (e.g. Venn diagrams and network analysis) typically does not include...

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Machlab DA, Velez G, Bassuk AG, et al. ProSave: an application for restoring quantitative data to manipulated subsets of protein lists. Source Code Biol Med. 2018;13:3doi: 10.1186/s13029-018-0070-0.
Machlab, D. A., Velez, G., Bassuk, A. G., & Mahajan, V. B. (2018). ProSave: an application for restoring quantitative data to manipulated subsets of protein lists. Source code for biology and medicine, 133. https://doi.org/10.1186/s13029-018-0070-0
Machlab, Daniel A, et al. "ProSave: an application for restoring quantitative data to manipulated subsets of protein lists." Source code for biology and medicine vol. 13 (2018): 3. doi: https://doi.org/10.1186/s13029-018-0070-0
Machlab DA, Velez G, Bassuk AG, Mahajan VB. ProSave: an application for restoring quantitative data to manipulated subsets of protein lists. Source Code Biol Med. 2018 Nov 12;13:3. doi: 10.1186/s13029-018-0070-0. eCollection 2018. PMID: 30459825; PMCID: PMC6233572.
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Differential role of "Signal 3" inflammatory cytokines in regulating CD8 T cell expansion and differentiation in vivo.

Frontiers in immunology

Pham NL, Badovinac VP, Harty JT.
PMID: 22566795
Front Immunol. 2011 Feb 11;2:4. doi: 10.3389/fimmu.2011.00004. eCollection 2011.

Following an infection, naïve CD8 T cells are stimulated by dendritic cells (DC) displaying pathogen-derived peptides on MHC class I molecules (signal 1) and costimulatory molecules (signal 2). Additionally, pathogen-induced inflammatory cytokines also act directly on the responding CD8...

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Pham NL, Badovinac VP, Harty JT. Differential role of "Signal 3" inflammatory cytokines in regulating CD8 T cell expansion and differentiation in vivo. Front Immunol. 2011;2:4doi: 10.3389/fimmu.2011.00004.
Pham, N. L., Badovinac, V. P., & Harty, J. T. (2011). Differential role of "Signal 3" inflammatory cytokines in regulating CD8 T cell expansion and differentiation in vivo. Frontiers in immunology, 24. https://doi.org/10.3389/fimmu.2011.00004
Pham, Nhat-Long L, et al. "Differential role of "Signal 3" inflammatory cytokines in regulating CD8 T cell expansion and differentiation in vivo." Frontiers in immunology vol. 2 (2011): 4. doi: https://doi.org/10.3389/fimmu.2011.00004
Pham NL, Badovinac VP, Harty JT. Differential role of "Signal 3" inflammatory cytokines in regulating CD8 T cell expansion and differentiation in vivo. Front Immunol. 2011 Feb 11;2:4. doi: 10.3389/fimmu.2011.00004. eCollection 2011. PMID: 22566795; PMCID: PMC3342074.
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Expeditious recruitment of circulating memory CD8 T cells to the liver facilitates control of malaria.

Cell reports

Lefebvre MN, Surette FA, Anthony SM, Vijay R, Jensen IJ, Pewe LL, Hancox LS, Van Braeckel-Budimir N, van de Wall S, Urban SL, Mix MR, Kurup SP, Badovinac VP, Butler NS, Harty JT.
PMID: 34731605
Cell Rep. 2021 Nov 02;37(5):109956. doi: 10.1016/j.celrep.2021.109956.

Circulating memory CD8 T cell trafficking and protective capacity during liver-stage malaria infection remains undefined. We find that effector memory CD8 T cells (Tem) infiltrate the liver within 6 hours after malarial or bacterial infections and mediate pathogen clearance....

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Lefebvre MN, Surette FA, Anthony SM, et al. Expeditious recruitment of circulating memory CD8 T cells to the liver facilitates control of malaria. Cell Rep. 2021;37(5):109956doi: 10.1016/j.celrep.2021.109956.
Lefebvre, M. N., Surette, F. A., Anthony, S. M., Vijay, R., Jensen, I. J., Pewe, L. L., Hancox, L. S., Van Braeckel-Budimir, N., van de Wall, S., Urban, S. L., Mix, M. R., Kurup, S. P., Badovinac, V. P., Butler, N. S., & Harty, J. T. (2021). Expeditious recruitment of circulating memory CD8 T cells to the liver facilitates control of malaria. Cell reports, 37(5), 109956. https://doi.org/10.1016/j.celrep.2021.109956
Lefebvre, Mitchell N, et al. "Expeditious recruitment of circulating memory CD8 T cells to the liver facilitates control of malaria." Cell reports vol. 37,5 (2021): 109956. doi: https://doi.org/10.1016/j.celrep.2021.109956
Lefebvre MN, Surette FA, Anthony SM, Vijay R, Jensen IJ, Pewe LL, Hancox LS, Van Braeckel-Budimir N, van de Wall S, Urban SL, Mix MR, Kurup SP, Badovinac VP, Butler NS, Harty JT. Expeditious recruitment of circulating memory CD8 T cells to the liver facilitates control of malaria. Cell Rep. 2021 Nov 02;37(5):109956. doi: 10.1016/j.celrep.2021.109956. PMID: 34731605; PMCID: PMC8628427.
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