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Resistin induces cardiac fibroblast-myofibroblast differentiation through JAK/STAT3 and JNK/c-Jun signaling.

Pharmacological research

Singh R, Kaundal RK, Zhao B, Bouchareb R, Lebeche D.
PMID: 33524540
Pharmacol Res. 2021 May;167:105414. doi: 10.1016/j.phrs.2020.105414. Epub 2021 Jan 29.

Cardiac fibrosis is characterized by excessive deposition of extracellular matrix proteins and myofibroblast differentiation. Our previous findings have implicated resistin in cardiac fibrosis; however, the molecular mechanisms underlying this process are still unclear. Here we investigated the role of...

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Singh R, Kaundal RK, Zhao B, et al. Resistin induces cardiac fibroblast-myofibroblast differentiation through JAK/STAT3 and JNK/c-Jun signaling. Pharmacol Res. 2021;167:105414doi: 10.1016/j.phrs.2020.105414.
Singh, R., Kaundal, R. K., Zhao, B., Bouchareb, R., & Lebeche, D. (2021). Resistin induces cardiac fibroblast-myofibroblast differentiation through JAK/STAT3 and JNK/c-Jun signaling. Pharmacological research, 167105414. https://doi.org/10.1016/j.phrs.2020.105414
Singh, Rajvir, et al. "Resistin induces cardiac fibroblast-myofibroblast differentiation through JAK/STAT3 and JNK/c-Jun signaling." Pharmacological research vol. 167 (2021): 105414. doi: https://doi.org/10.1016/j.phrs.2020.105414
Singh R, Kaundal RK, Zhao B, Bouchareb R, Lebeche D. Resistin induces cardiac fibroblast-myofibroblast differentiation through JAK/STAT3 and JNK/c-Jun signaling. Pharmacol Res. 2021 May;167:105414. doi: 10.1016/j.phrs.2020.105414. Epub 2021 Jan 29. PMID: 33524540; PMCID: PMC8085100.
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Regulation of the Methylation and Expression Levels of the BMPR2 Gene by SIN3a as a Novel Therapeutic Mechanism in Pulmonary Arterial Hypertension.

Circulation

Bisserier M, Mathiyalagan P, Zhang S, Elmastour F, Dorfmüller P, Humbert M, David G, Tarzami S, Weber T, Perros F, Sassi Y, Sahoo S, Hadri L.
PMID: 34078089
Circulation. 2021 Jul 06;144(1):52-73. doi: 10.1161/CIRCULATIONAHA.120.047978. Epub 2021 Jun 03.

BACKGROUND: Epigenetic mechanisms are critical in the pathogenesis of pulmonary arterial hypertension (PAH). Previous studies have suggested that hypermethylation of the BMPR2 (bone morphogenetic protein receptor type 2) promoter is associated with BMPR2 downregulation and progression of PAH. Here,...

Cite
Bisserier M, Mathiyalagan P, Zhang S, et al. Regulation of the Methylation and Expression Levels of the BMPR2 Gene by SIN3a as a Novel Therapeutic Mechanism in Pulmonary Arterial Hypertension. Circulation. 2021;144(1):52-73doi: 10.1161/CIRCULATIONAHA.120.047978.
Bisserier, M., Mathiyalagan, P., Zhang, S., Elmastour, F., Dorfmüller, P., Humbert, M., David, G., Tarzami, S., Weber, T., Perros, F., Sassi, Y., Sahoo, S., & Hadri, L. (2021). Regulation of the Methylation and Expression Levels of the BMPR2 Gene by SIN3a as a Novel Therapeutic Mechanism in Pulmonary Arterial Hypertension. Circulation, 144(1), 52-73. https://doi.org/10.1161/CIRCULATIONAHA.120.047978
Bisserier, Malik, et al. "Regulation of the Methylation and Expression Levels of the BMPR2 Gene by SIN3a as a Novel Therapeutic Mechanism in Pulmonary Arterial Hypertension." Circulation vol. 144,1 (2021): 52-73. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.047978
Bisserier M, Mathiyalagan P, Zhang S, Elmastour F, Dorfmüller P, Humbert M, David G, Tarzami S, Weber T, Perros F, Sassi Y, Sahoo S, Hadri L. Regulation of the Methylation and Expression Levels of the BMPR2 Gene by SIN3a as a Novel Therapeutic Mechanism in Pulmonary Arterial Hypertension. Circulation. 2021 Jul 06;144(1):52-73. doi: 10.1161/CIRCULATIONAHA.120.047978. Epub 2021 Jun 03. PMID: 34078089; PMCID: PMC8293289.
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Immune cell profiling in atherosclerosis: role in research and precision medicine.

Nature reviews. Cardiology

Fernandez DM, Giannarelli C.
PMID: 34267377
Nat Rev Cardiol. 2022 Jan;19(1):43-58. doi: 10.1038/s41569-021-00589-2. Epub 2021 Jul 15.

Inflammation is intimately involved at all stages of atherosclerosis and remains a substantial residual cardiovascular risk factor in optimally treated patients. The proof of concept that targeting inflammation reduces cardiovascular events in patients with a history of myocardial infarction...

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Fernandez DM, Giannarelli C. Immune cell profiling in atherosclerosis: role in research and precision medicine. Nat Rev Cardiol. 2021;19(1):43-58doi: 10.1038/s41569-021-00589-2.
Fernandez, D. M., & Giannarelli, C. (2022). Immune cell profiling in atherosclerosis: role in research and precision medicine. Nature reviews. Cardiology, 19(1), 43-58. https://doi.org/10.1038/s41569-021-00589-2
Fernandez, Dawn M, and Giannarelli, Chiara. "Immune cell profiling in atherosclerosis: role in research and precision medicine." Nature reviews. Cardiology vol. 19,1 (2022): 43-58. doi: https://doi.org/10.1038/s41569-021-00589-2
Fernandez DM, Giannarelli C. Immune cell profiling in atherosclerosis: role in research and precision medicine. Nat Rev Cardiol. 2022 Jan;19(1):43-58. doi: 10.1038/s41569-021-00589-2. Epub 2021 Jul 15. PMID: 34267377; PMCID: PMC8280607.
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Extraction and analysis of signatures from the Gene Expression Omnibus by the crowd.

Nature communications

Wang Z, Monteiro CD, Jagodnik KM, Fernandez NF, Gundersen GW, Rouillard AD, Jenkins SL, Feldmann AS, Hu KS, McDermott MG, Duan Q, Clark NR, Jones MR, Kou Y, Goff T, Woodland H, Amaral FMR, Szeto GL, Fuchs O, Schüssler-Fiorenza Rose SM, Sharma S, Schwartz U, Bausela XB, Szymkiewicz M, Maroulis V, Salykin A, Barra CM, Kruth CD, Bongio NJ, Mathur V, Todoric RD, Rubin UE, Malatras A, Fulp CT, Galindo JA, Motiejunaite R, Jüschke C, Dishuck PC, Lahl K, Jafari M, Aibar S, Zaravinos A, Steenhuizen LH, Allison LR, Gamallo P, de Andres Segura F, Dae Devlin T, Pérez-García V, Ma'ayan A.
PMID: 27667448
Nat Commun. 2016 Sep 26;7:12846. doi: 10.1038/ncomms12846.

Gene expression data are accumulating exponentially in public repositories. Reanalysis and integration of themed collections from these studies may provide new insights, but requires further human curation. Here we report a crowdsourcing project to annotate and reanalyse a large...

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Wang Z, Monteiro CD, Jagodnik KM, et al. Extraction and analysis of signatures from the Gene Expression Omnibus by the crowd. Nat Commun. 2016;7:12846doi: 10.1038/ncomms12846.
Wang, Z., Monteiro, C. D., Jagodnik, K. M., Fernandez, N. F., Gundersen, G. W., Rouillard, A. D., Jenkins, S. L., Feldmann, A. S., Hu, K. S., McDermott, M. G., Duan, Q., Clark, N. R., Jones, M. R., Kou, Y., Goff, T., Woodland, H., Amaral, F. M. R., Szeto, G. L., Fuchs, O., Schüssler-Fiorenza Rose, S. M., Sharma, S., Schwartz, U., Bausela, X. B., Szymkiewicz, M., Maroulis, V., Salykin, A., Barra, C. M., Kruth, C. D., Bongio, N. J., Mathur, V., Todoric, R. D., Rubin, U. E., Malatras, A., Fulp, C. T., Galindo, J. A., Motiejunaite, R., Jüschke, C., Dishuck, P. C., Lahl, K., Jafari, M., Aibar, S., Zaravinos, A., Steenhuizen, L. H., Allison, L. R., Gamallo, P., de Andres Segura, F., Dae Devlin, T., Pérez-García, V., & Ma'ayan, A. (2016). Extraction and analysis of signatures from the Gene Expression Omnibus by the crowd. Nature communications, 712846. https://doi.org/10.1038/ncomms12846
Wang, Zichen, et al. "Extraction and analysis of signatures from the Gene Expression Omnibus by the crowd." Nature communications vol. 7 (2016): 12846. doi: https://doi.org/10.1038/ncomms12846
Wang Z, Monteiro CD, Jagodnik KM, Fernandez NF, Gundersen GW, Rouillard AD, Jenkins SL, Feldmann AS, Hu KS, McDermott MG, Duan Q, Clark NR, Jones MR, Kou Y, Goff T, Woodland H, Amaral FMR, Szeto GL, Fuchs O, Schüssler-Fiorenza Rose SM, Sharma S, Schwartz U, Bausela XB, Szymkiewicz M, Maroulis V, Salykin A, Barra CM, Kruth CD, Bongio NJ, Mathur V, Todoric RD, Rubin UE, Malatras A, Fulp CT, Galindo JA, Motiejunaite R, Jüschke C, Dishuck PC, Lahl K, Jafari M, Aibar S, Zaravinos A, Steenhuizen LH, Allison LR, Gamallo P, de Andres Segura F, Dae Devlin T, Pérez-García V, Ma'ayan A. Extraction and analysis of signatures from the Gene Expression Omnibus by the crowd. Nat Commun. 2016 Sep 26;7:12846. doi: 10.1038/ncomms12846. PMID: 27667448; PMCID: PMC5052684.
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L1000CDS.

NPJ systems biology and applications

Duan Q, Reid SP, Clark NR, Wang Z, Fernandez NF, Rouillard AD, Readhead B, Tritsch SR, Hodos R, Hafner M, Niepel M, Sorger PK, Dudley JT, Bavari S, Panchal RG, Ma'ayan A.
PMID: 28413689
NPJ Syst Biol Appl. 2016;2. doi: 10.1038/npjsba.2016.15. Epub 2016 Aug 04.

The library of integrated network-based cellular signatures (LINCS) L1000 data set currently comprises of over a million gene expression profiles of chemically perturbed human cell lines. Through unique several intrinsic and extrinsic benchmarking schemes, we demonstrate that processing the...

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Duan Q, Reid SP, Clark NR, et al. L1000CDS. NPJ Syst Biol Appl. 2016;2doi: 10.1038/npjsba.2016.15.
Duan, Q., Reid, S. P., Clark, N. R., Wang, Z., Fernandez, N. F., Rouillard, A. D., Readhead, B., Tritsch, S. R., Hodos, R., Hafner, M., Niepel, M., Sorger, P. K., Dudley, J. T., Bavari, S., Panchal, R. G., & Ma'ayan, A. (2016). L1000CDS. NPJ systems biology and applications, 2. https://doi.org/10.1038/npjsba.2016.15
Duan, Qiaonan, et al. "L1000CDS." NPJ systems biology and applications vol. 2 (2016). doi: https://doi.org/10.1038/npjsba.2016.15
Duan Q, Reid SP, Clark NR, Wang Z, Fernandez NF, Rouillard AD, Readhead B, Tritsch SR, Hodos R, Hafner M, Niepel M, Sorger PK, Dudley JT, Bavari S, Panchal RG, Ma'ayan A. L1000CDS. NPJ Syst Biol Appl. 2016;2. doi: 10.1038/npjsba.2016.15. Epub 2016 Aug 04. PMID: 28413689; PMCID: PMC5389891.
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Cell-Free Mitochondrial DNA as a Potential Biomarker for Astronauts' Health.

Journal of the American Heart Association

Bisserier M, Shanmughapriya S, Rai AK, Gonzalez C, Brojakowska A, Garikipati VNS, Madesh M, Mills PJ, Walsh K, Arakelyan A, Kishore R, Hadri L, Goukassian DA.
PMID: 34666498
J Am Heart Assoc. 2021 Nov 02;10(21):e022055. doi: 10.1161/JAHA.121.022055. Epub 2021 Oct 20.

Background Space travel-associated stressors such as microgravity or radiation exposure have been reported in astronauts after short- and long-duration missions aboard the International Space Station. Despite risk mitigation strategies, adverse health effects remain a concern. Thus, there is a...

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Bisserier M, Shanmughapriya S, Rai AK, et al. Cell-Free Mitochondrial DNA as a Potential Biomarker for Astronauts' Health. J Am Heart Assoc. 2021;10(21):e022055doi: 10.1161/JAHA.121.022055.
Bisserier, M., Shanmughapriya, S., Rai, A. K., Gonzalez, C., Brojakowska, A., Garikipati, V. N. S., Madesh, M., Mills, P. J., Walsh, K., Arakelyan, A., Kishore, R., Hadri, L., & Goukassian, D. A. (2021). Cell-Free Mitochondrial DNA as a Potential Biomarker for Astronauts' Health. Journal of the American Heart Association, 10(21), e022055. https://doi.org/10.1161/JAHA.121.022055
Bisserier, Malik, et al. "Cell-Free Mitochondrial DNA as a Potential Biomarker for Astronauts' Health." Journal of the American Heart Association vol. 10,21 (2021): e022055. doi: https://doi.org/10.1161/JAHA.121.022055
Bisserier M, Shanmughapriya S, Rai AK, Gonzalez C, Brojakowska A, Garikipati VNS, Madesh M, Mills PJ, Walsh K, Arakelyan A, Kishore R, Hadri L, Goukassian DA. Cell-Free Mitochondrial DNA as a Potential Biomarker for Astronauts' Health. J Am Heart Assoc. 2021 Nov 02;10(21):e022055. doi: 10.1161/JAHA.121.022055. Epub 2021 Oct 20. PMID: 34666498.
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Cell-Free Mitochondrial DNA as a Potential Biomarker for Astronauts' Health.

Journal of the American Heart Association

Bisserier M, Shanmughapriya S, Rai AK, Gonzalez C, Brojakowska A, Garikipati VNS, Madesh M, Mills PJ, Walsh K, Arakelyan A, Kishore R, Hadri L, Goukassian DA.
PMID: 34666498
J Am Heart Assoc. 2021 Nov 02;10(21):e022055. doi: 10.1161/JAHA.121.022055. Epub 2021 Oct 20.

Background Space travel-associated stressors such as microgravity or radiation exposure have been reported in astronauts after short- and long-duration missions aboard the International Space Station. Despite risk mitigation strategies, adverse health effects remain a concern. Thus, there is a...

Cite
Bisserier M, Shanmughapriya S, Rai AK, et al. Cell-Free Mitochondrial DNA as a Potential Biomarker for Astronauts' Health. J Am Heart Assoc. 2021;10(21):e022055doi: 10.1161/JAHA.121.022055.
Bisserier, M., Shanmughapriya, S., Rai, A. K., Gonzalez, C., Brojakowska, A., Garikipati, V. N. S., Madesh, M., Mills, P. J., Walsh, K., Arakelyan, A., Kishore, R., Hadri, L., & Goukassian, D. A. (2021). Cell-Free Mitochondrial DNA as a Potential Biomarker for Astronauts' Health. Journal of the American Heart Association, 10(21), e022055. https://doi.org/10.1161/JAHA.121.022055
Bisserier, Malik, et al. "Cell-Free Mitochondrial DNA as a Potential Biomarker for Astronauts' Health." Journal of the American Heart Association vol. 10,21 (2021): e022055. doi: https://doi.org/10.1161/JAHA.121.022055
Bisserier M, Shanmughapriya S, Rai AK, Gonzalez C, Brojakowska A, Garikipati VNS, Madesh M, Mills PJ, Walsh K, Arakelyan A, Kishore R, Hadri L, Goukassian DA. Cell-Free Mitochondrial DNA as a Potential Biomarker for Astronauts' Health. J Am Heart Assoc. 2021 Nov 02;10(21):e022055. doi: 10.1161/JAHA.121.022055. Epub 2021 Oct 20. PMID: 34666498; PMCID: PMC8751818.
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Immune cell profiling in atherosclerosis: role in research and precision medicine.

Nature reviews. Cardiology

Fernandez DM, Giannarelli C.
PMID: 34267377
Nat Rev Cardiol. 2021 Jul 15; doi: 10.1038/s41569-021-00589-2. Epub 2021 Jul 15.

Inflammation is intimately involved at all stages of atherosclerosis and remains a substantial residual cardiovascular risk factor in optimally treated patients. The proof of concept that targeting inflammation reduces cardiovascular events in patients with a history of myocardial infarction...

Cite
Fernandez DM, Giannarelli C. Immune cell profiling in atherosclerosis: role in research and precision medicine. Nat Rev Cardiol. 2021;doi: 10.1038/s41569-021-00589-2.
Fernandez, D. M., & Giannarelli, C. (2021). Immune cell profiling in atherosclerosis: role in research and precision medicine. Nature reviews. Cardiology, . https://doi.org/10.1038/s41569-021-00589-2
Fernandez, Dawn M, and Giannarelli, Chiara. "Immune cell profiling in atherosclerosis: role in research and precision medicine." Nature reviews. Cardiology vol. (2021). doi: https://doi.org/10.1038/s41569-021-00589-2
Fernandez DM, Giannarelli C. Immune cell profiling in atherosclerosis: role in research and precision medicine. Nat Rev Cardiol. 2021 Jul 15; doi: 10.1038/s41569-021-00589-2. Epub 2021 Jul 15. PMID: 34267377; PMCID: PMC8280607.
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Regulation of the Methylation and Expression Levels of the BMPR2 Gene by SIN3a as a Novel Therapeutic Mechanism in Pulmonary Arterial Hypertension.

Circulation

Bisserier M, Mathiyalagan P, Zhang S, Elmastour F, Dorfmüller P, Humbert M, David G, Tarzami S, Weber T, Perros F, Sassi Y, Sahoo S, Hadri L.
PMID: 34078089
Circulation. 2021 Jul 06;144(1):52-73. doi: 10.1161/CIRCULATIONAHA.120.047978. Epub 2021 Jun 03.

BACKGROUND: Epigenetic mechanisms are critical in the pathogenesis of pulmonary arterial hypertension (PAH). Previous studies have suggested that hypermethylation of the BMPR2 (bone morphogenetic protein receptor type 2) promoter is associated with BMPR2 downregulation and progression of PAH. Here,...

Cite
Bisserier M, Mathiyalagan P, Zhang S, et al. Regulation of the Methylation and Expression Levels of the BMPR2 Gene by SIN3a as a Novel Therapeutic Mechanism in Pulmonary Arterial Hypertension. Circulation. 2021;144(1):52-73doi: 10.1161/CIRCULATIONAHA.120.047978.
Bisserier, M., Mathiyalagan, P., Zhang, S., Elmastour, F., Dorfmüller, P., Humbert, M., David, G., Tarzami, S., Weber, T., Perros, F., Sassi, Y., Sahoo, S., & Hadri, L. (2021). Regulation of the Methylation and Expression Levels of the BMPR2 Gene by SIN3a as a Novel Therapeutic Mechanism in Pulmonary Arterial Hypertension. Circulation, 144(1), 52-73. https://doi.org/10.1161/CIRCULATIONAHA.120.047978
Bisserier, Malik, et al. "Regulation of the Methylation and Expression Levels of the BMPR2 Gene by SIN3a as a Novel Therapeutic Mechanism in Pulmonary Arterial Hypertension." Circulation vol. 144,1 (2021): 52-73. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.047978
Bisserier M, Mathiyalagan P, Zhang S, Elmastour F, Dorfmüller P, Humbert M, David G, Tarzami S, Weber T, Perros F, Sassi Y, Sahoo S, Hadri L. Regulation of the Methylation and Expression Levels of the BMPR2 Gene by SIN3a as a Novel Therapeutic Mechanism in Pulmonary Arterial Hypertension. Circulation. 2021 Jul 06;144(1):52-73. doi: 10.1161/CIRCULATIONAHA.120.047978. Epub 2021 Jun 03. PMID: 34078089; PMCID: PMC8293289.
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Targeting epigenetic mechanisms as an emerging therapeutic strategy in pulmonary hypertension disease.

Vascular biology (Bristol, England)

Bisserier M, Janostiak R, Lezoualc'h F, Hadri L.
PMID: 32161845
Vasc Biol. 2020 Jan;2(1):R17-R34. doi: 10.1530/vb-19-0030. Epub 2020 Jan 09.

Pulmonary arterial hypertension (PAH) is a multifactorial cardiopulmonary disease characterized by an elevation of pulmonary artery pressure (PAP) and pulmonary vascular resistance (PVR), which can lead to right ventricular (RV) failure, multi-organ dysfunction, and ultimately to premature death. Despite...

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Bisserier M, Janostiak R, Lezoualc'h F, et al. Targeting epigenetic mechanisms as an emerging therapeutic strategy in pulmonary hypertension disease. Vasc Biol. 2020;2(1):R17-R34doi: 10.1530/vb-19-0030.
Bisserier, M., Janostiak, R., Lezoualc'h, F., & Hadri, L. (2020). Targeting epigenetic mechanisms as an emerging therapeutic strategy in pulmonary hypertension disease. Vascular biology (Bristol, England), 2(1), R17-R34. https://doi.org/10.1530/vb-19-0030
Bisserier, Malik, et al. "Targeting epigenetic mechanisms as an emerging therapeutic strategy in pulmonary hypertension disease." Vascular biology (Bristol, England) vol. 2,1 (2020): R17-R34. doi: https://doi.org/10.1530/vb-19-0030
Bisserier M, Janostiak R, Lezoualc'h F, Hadri L. Targeting epigenetic mechanisms as an emerging therapeutic strategy in pulmonary hypertension disease. Vasc Biol. 2020 Jan;2(1):R17-R34. doi: 10.1530/vb-19-0030. Epub 2020 Jan 09. PMID: 32161845; PMCID: PMC7065685.
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Rare loss-of-function mutations of PTGIR are enriched in fibromuscular dysplasia.

Cardiovascular research

Georges A, Albuisson J, Berrandou T, Dupré D, Lorthioir A, D'Escamard V, Di Narzo AF, Kadian-Dodov D, Olin JW, Warchol-Celinska E, Prejbisz A, Januszewicz A, Bruneval P, Baranowska AA, Webb TR, Hamby SE, Samani NJ, Adlam D, Fendrikova-Mahlay N, Hazen S, Wang Y, Yang ML, Hunker K, Combaret N, Motreff P, Chédid A, Fiquet B, Plouin PF, Mousseaux E, Azarine A, Amar L, Azizi M, Gornik HL, Ganesh SK, Kovacic JC, Jeunemaitre X, Bouatia-Naji N.
PMID: 32531060
Cardiovasc Res. 2021 Mar 21;117(4):1154-1165. doi: 10.1093/cvr/cvaa161.

AIMS: Fibromuscular dysplasia (FMD) and spontaneous coronary artery dissection (SCAD) are related, non-atherosclerotic arterial diseases mainly affecting middle-aged women. Little is known about their physiopathological mechanisms. We aimed to identify rare genetic causes to elucidate molecular mechanisms implicated in...

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Georges A, Albuisson J, Berrandou T, et al. Rare loss-of-function mutations of PTGIR are enriched in fibromuscular dysplasia. Cardiovasc Res. 2021;117(4):1154-1165doi: 10.1093/cvr/cvaa161.
Georges, A., Albuisson, J., Berrandou, T., Dupré, D., Lorthioir, A., D'Escamard, V., Di Narzo, A. F., Kadian-Dodov, D., Olin, J. W., Warchol-Celinska, E., Prejbisz, A., Januszewicz, A., Bruneval, P., Baranowska, A. A., Webb, T. R., Hamby, S. E., Samani, N. J., Adlam, D., Fendrikova-Mahlay, N., Hazen, S., Wang, Y., Yang, M. L., Hunker, K., Combaret, N., Motreff, P., Chédid, A., Fiquet, B., Plouin, P. F., Mousseaux, E., Azarine, A., Amar, L., Azizi, M., Gornik, H. L., Ganesh, S. K., Kovacic, J. C., Jeunemaitre, X., & Bouatia-Naji, N. (2021). Rare loss-of-function mutations of PTGIR are enriched in fibromuscular dysplasia. Cardiovascular research, 117(4), 1154-1165. https://doi.org/10.1093/cvr/cvaa161
Georges, Adrien, et al. "Rare loss-of-function mutations of PTGIR are enriched in fibromuscular dysplasia." Cardiovascular research vol. 117,4 (2021): 1154-1165. doi: https://doi.org/10.1093/cvr/cvaa161
Georges A, Albuisson J, Berrandou T, Dupré D, Lorthioir A, D'Escamard V, Di Narzo AF, Kadian-Dodov D, Olin JW, Warchol-Celinska E, Prejbisz A, Januszewicz A, Bruneval P, Baranowska AA, Webb TR, Hamby SE, Samani NJ, Adlam D, Fendrikova-Mahlay N, Hazen S, Wang Y, Yang ML, Hunker K, Combaret N, Motreff P, Chédid A, Fiquet B, Plouin PF, Mousseaux E, Azarine A, Amar L, Azizi M, Gornik HL, Ganesh SK, Kovacic JC, Jeunemaitre X, Bouatia-Naji N. Rare loss-of-function mutations of PTGIR are enriched in fibromuscular dysplasia. Cardiovasc Res. 2021 Mar 21;117(4):1154-1165. doi: 10.1093/cvr/cvaa161. PMID: 32531060; PMCID: PMC7983006.
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Datasets2Tools, repository and search engine for bioinformatics datasets, tools and canned analyses.

Scientific data

Torre D, Krawczuk P, Jagodnik KM, Lachmann A, Wang Z, Wang L, Kuleshov MV, Ma'ayan A.
PMID: 29485625
Sci Data. 2018 Feb 27;5:180023. doi: 10.1038/sdata.2018.23.

Biomedical data repositories such as the Gene Expression Omnibus (GEO) enable the search and discovery of relevant biomedical digital data objects. Similarly, resources such as OMICtools, index bioinformatics tools that can extract knowledge from these digital data objects. However,...

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Torre D, Krawczuk P, Jagodnik KM, et al. Datasets2Tools, repository and search engine for bioinformatics datasets, tools and canned analyses. Sci Data. 2018;5:180023doi: 10.1038/sdata.2018.23.
Torre, D., Krawczuk, P., Jagodnik, K. M., Lachmann, A., Wang, Z., Wang, L., Kuleshov, M. V., & Ma'ayan, A. (2018). Datasets2Tools, repository and search engine for bioinformatics datasets, tools and canned analyses. Scientific data, 5180023. https://doi.org/10.1038/sdata.2018.23
Torre, Denis, et al. "Datasets2Tools, repository and search engine for bioinformatics datasets, tools and canned analyses." Scientific data vol. 5 (2018): 180023. doi: https://doi.org/10.1038/sdata.2018.23
Torre D, Krawczuk P, Jagodnik KM, Lachmann A, Wang Z, Wang L, Kuleshov MV, Ma'ayan A. Datasets2Tools, repository and search engine for bioinformatics datasets, tools and canned analyses. Sci Data. 2018 Feb 27;5:180023. doi: 10.1038/sdata.2018.23. PMID: 29485625; PMCID: PMC5827688.
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Showing 1 to 12 of 22 entries