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Categorical versus dimensional approaches to autism-associated intermediate phenotypes in 22q11.2 microdeletion syndrome.

Biological psychiatry. Cognitive neuroscience and neuroimaging

Jalbrzikowski M, Ahmed KH, Patel A, Jonas R, Kushan L, Chow C, Bearden CE.
PMID: 28367513
Biol Psychiatry Cogn Neurosci Neuroimaging. 2017 Jan;2(1):53-65. doi: 10.1016/j.bpsc.2016.06.007.

BACKGROUND: 22q11.2 Microdeletion syndrome (22q11DS) is associated with elevated rates of autism spectrum disorders (ASDs), although the diagnosis is controversial. In order to determine whether there is a biological substrate of ASD in 22q11DS, we examined neurocognitive and structural...

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Jalbrzikowski M, Ahmed KH, Patel A, et al. Categorical versus dimensional approaches to autism-associated intermediate phenotypes in 22q11.2 microdeletion syndrome. Biol Psychiatry Cogn Neurosci Neuroimaging. 2017;2(1):53-65doi: 10.1016/j.bpsc.2016.06.007.
Jalbrzikowski, M., Ahmed, K. H., Patel, A., Jonas, R., Kushan, L., Chow, C., & Bearden, C. E. (2017). Categorical versus dimensional approaches to autism-associated intermediate phenotypes in 22q11.2 microdeletion syndrome. Biological psychiatry. Cognitive neuroscience and neuroimaging, 2(1), 53-65. https://doi.org/10.1016/j.bpsc.2016.06.007
Jalbrzikowski, Maria, et al. "Categorical versus dimensional approaches to autism-associated intermediate phenotypes in 22q11.2 microdeletion syndrome." Biological psychiatry. Cognitive neuroscience and neuroimaging vol. 2,1 (2017): 53-65. doi: https://doi.org/10.1016/j.bpsc.2016.06.007
Jalbrzikowski M, Ahmed KH, Patel A, Jonas R, Kushan L, Chow C, Bearden CE. Categorical versus dimensional approaches to autism-associated intermediate phenotypes in 22q11.2 microdeletion syndrome. Biol Psychiatry Cogn Neurosci Neuroimaging. 2017 Jan;2(1):53-65. doi: 10.1016/j.bpsc.2016.06.007. PMID: 28367513; PMCID: PMC5373800.
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Sex Differences in Functional Connectivity of the Salience, Default Mode, and Central Executive Networks in Youth with ASD.

Cerebral cortex (New York, N.Y. : 1991)

Lawrence KE, Hernandez LM, Bowman HC, Padgaonkar NT, Fuster E, Jack A, Aylward E, Gaab N, Van Horn JD, Bernier RA, Geschwind DH, McPartland JC, Nelson CA, Webb SJ, Pelphrey KA, Green SA, Bookheimer SY, Dapretto M.
PMID: 32350530
Cereb Cortex. 2020 Jul 30;30(9):5107-5120. doi: 10.1093/cercor/bhaa105.

Autism spectrum disorder (ASD) is associated with the altered functional connectivity of 3 neurocognitive networks that are hypothesized to be central to the symptomatology of ASD: the salience network (SN), default mode network (DMN), and central executive network (CEN)....

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Lawrence KE, Hernandez LM, Bowman HC, et al. Sex Differences in Functional Connectivity of the Salience, Default Mode, and Central Executive Networks in Youth with ASD. Cereb Cortex. 2020;30(9):5107-5120doi: 10.1093/cercor/bhaa105.
Lawrence, K. E., Hernandez, L. M., Bowman, H. C., Padgaonkar, N. T., Fuster, E., Jack, A., Aylward, E., Gaab, N., Van Horn, J. D., Bernier, R. A., Geschwind, D. H., McPartland, J. C., Nelson, C. A., Webb, S. J., Pelphrey, K. A., Green, S. A., Bookheimer, S. Y., Dapretto, M. (2020). Sex Differences in Functional Connectivity of the Salience, Default Mode, and Central Executive Networks in Youth with ASD. Cerebral cortex (New York, N.Y. : 1991), 30(9), 5107-5120. https://doi.org/10.1093/cercor/bhaa105
Lawrence, Katherine E, et al. "Sex Differences in Functional Connectivity of the Salience, Default Mode, and Central Executive Networks in Youth with ASD." Cerebral cortex (New York, N.Y. : 1991) vol. 30,9 (2020): 5107-5120. doi: https://doi.org/10.1093/cercor/bhaa105
Lawrence KE, Hernandez LM, Bowman HC, Padgaonkar NT, Fuster E, Jack A, Aylward E, Gaab N, Van Horn JD, Bernier RA, Geschwind DH, McPartland JC, Nelson CA, Webb SJ, Pelphrey KA, Green SA, Bookheimer SY, Dapretto M. Sex Differences in Functional Connectivity of the Salience, Default Mode, and Central Executive Networks in Youth with ASD. Cereb Cortex. 2020 Jul 30;30(9):5107-5120. doi: 10.1093/cercor/bhaa105. PMID: 32350530; PMCID: PMC7391269.
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Altered white matter microstructure is associated with social cognition and psychotic symptoms in 22q11.2 microdeletion syndrome.

Frontiers in behavioral neuroscience

Jalbrzikowski M, Villalon-Reina JE, Karlsgodt KH, Senturk D, Chow C, Thompson PM, Bearden CE.
PMID: 25426042
Front Behav Neurosci. 2014 Nov 11;8:393. doi: 10.3389/fnbeh.2014.00393. eCollection 2014.

22q11.2 Microdeletion Syndrome (22q11DS) is a highly penetrant genetic mutation associated with a significantly increased risk for psychosis. Aberrant neurodevelopment may lead to inappropriate neural circuit formation and cerebral dysconnectivity in 22q11DS, which may contribute to symptom development. Here...

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Jalbrzikowski M, Villalon-Reina JE, Karlsgodt KH, et al. Altered white matter microstructure is associated with social cognition and psychotic symptoms in 22q11.2 microdeletion syndrome. Front Behav Neurosci. 2014;8:393doi: 10.3389/fnbeh.2014.00393.
Jalbrzikowski, M., Villalon-Reina, J. E., Karlsgodt, K. H., Senturk, D., Chow, C., Thompson, P. M., & Bearden, C. E. (2014). Altered white matter microstructure is associated with social cognition and psychotic symptoms in 22q11.2 microdeletion syndrome. Frontiers in behavioral neuroscience, 8393. https://doi.org/10.3389/fnbeh.2014.00393
Jalbrzikowski, Maria, et al. "Altered white matter microstructure is associated with social cognition and psychotic symptoms in 22q11.2 microdeletion syndrome." Frontiers in behavioral neuroscience vol. 8 (2014): 393. doi: https://doi.org/10.3389/fnbeh.2014.00393
Jalbrzikowski M, Villalon-Reina JE, Karlsgodt KH, Senturk D, Chow C, Thompson PM, Bearden CE. Altered white matter microstructure is associated with social cognition and psychotic symptoms in 22q11.2 microdeletion syndrome. Front Behav Neurosci. 2014 Nov 11;8:393. doi: 10.3389/fnbeh.2014.00393. eCollection 2014. PMID: 25426042; PMCID: PMC4227518.
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The C677T variant in .

Biological psychiatry. Cognitive neuroscience and neuroimaging

Roussotte FF, Hua X, Narr KL, Small GW, Thompson PM.
PMID: 28435933
Biol Psychiatry Cogn Neurosci Neuroimaging. 2017 Apr;2(3):280-288. doi: 10.1016/j.bpsc.2016.09.005.

INTRODUCTION: The C677T functional variant in the methylene-tetrahydrofolate reductase (METHODS AND MATERIALS: Here, 3D magnetic resonance imaging data was analyzed from 738 individuals (age: 75.5 ± 6.8 years; 438 men/300 women) including 173 Alzheimer's patients, 359 subjects with mild...

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Roussotte FF, Hua X, Narr KL, et al. The C677T variant in . Biol Psychiatry Cogn Neurosci Neuroimaging. 2017;2(3):280-288doi: 10.1016/j.bpsc.2016.09.005.
Roussotte, F. F., Hua, X., Narr, K. L., Small, G. W., Thompson, P. M. (2017). The C677T variant in . Biological psychiatry. Cognitive neuroscience and neuroimaging, 2(3), 280-288. https://doi.org/10.1016/j.bpsc.2016.09.005
Roussotte, Florence F, et al. "The C677T variant in ." Biological psychiatry. Cognitive neuroscience and neuroimaging vol. 2,3 (2017): 280-288. doi: https://doi.org/10.1016/j.bpsc.2016.09.005
Roussotte FF, Hua X, Narr KL, Small GW, Thompson PM. The C677T variant in . Biol Psychiatry Cogn Neurosci Neuroimaging. 2017 Apr;2(3):280-288. doi: 10.1016/j.bpsc.2016.09.005. PMID: 28435933; PMCID: PMC5395287.
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Neural control of affiliative touch in prosocial interaction.

Nature

Wu YE, Dang J, Kingsbury L, Zhang M, Sun F, Hu RK, Hong W.
PMID: 34646019
Nature. 2021 Nov;599(7884):262-267. doi: 10.1038/s41586-021-03962-w. Epub 2021 Oct 13.

The ability to help and care for others fosters social cohesiveness and is vital to the physical and emotional well-being of social species, including humans

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Wu YE, Dang J, Kingsbury L, et al. Neural control of affiliative touch in prosocial interaction. Nature. 2021;599(7884):262-267doi: 10.1038/s41586-021-03962-w.
Wu, Y. E., Dang, J., Kingsbury, L., Zhang, M., Sun, F., Hu, R. K., & Hong, W. (2021). Neural control of affiliative touch in prosocial interaction. Nature, 599(7884), 262-267. https://doi.org/10.1038/s41586-021-03962-w
Wu, Ye Emily, et al. "Neural control of affiliative touch in prosocial interaction." Nature vol. 599,7884 (2021): 262-267. doi: https://doi.org/10.1038/s41586-021-03962-w
Wu YE, Dang J, Kingsbury L, Zhang M, Sun F, Hu RK, Hong W. Neural control of affiliative touch in prosocial interaction. Nature. 2021 Nov;599(7884):262-267. doi: 10.1038/s41586-021-03962-w. Epub 2021 Oct 13. PMID: 34646019; PMCID: PMC8605624.
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Impact of autism genetic risk on brain connectivity: a mechanism for the female protective effect.

Brain : a journal of neurology

Lawrence KE, Hernandez LM, Fuster E, Padgaonkar NT, Patterson G, Jung J, Okada NJ, Lowe JK, Hoekstra JN, Jack A, Aylward E, Gaab N, Van Horn JD, Bernier RA, McPartland JC, Webb SJ, Pelphrey KA, Green SA, Bookheimer SY, Geschwind DH, Dapretto M.
PMID: 34050743
Brain. 2021 May 29; doi: 10.1093/brain/awab204. Epub 2021 May 29.

The biological mechanisms underlying the greater prevalence of autism spectrum disorder in males than females remain poorly understood. One hypothesis posits that this female protective effect arises from genetic load for autism spectrum disorder differentially impacting male and female...

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Lawrence KE, Hernandez LM, Fuster E, et al. Impact of autism genetic risk on brain connectivity: a mechanism for the female protective effect. Brain. 2021;doi: 10.1093/brain/awab204.
Lawrence, K. E., Hernandez, L. M., Fuster, E., Padgaonkar, N. T., Patterson, G., Jung, J., Okada, N. J., Lowe, J. K., Hoekstra, J. N., Jack, A., Aylward, E., Gaab, N., Van Horn, J. D., Bernier, R. A., McPartland, J. C., Webb, S. J., Pelphrey, K. A., Green, S. A., Bookheimer, S. Y., Geschwind, D. H., Dapretto, M. (2021). Impact of autism genetic risk on brain connectivity: a mechanism for the female protective effect. Brain : a journal of neurology, . https://doi.org/10.1093/brain/awab204
Lawrence, Katherine E, et al. "Impact of autism genetic risk on brain connectivity: a mechanism for the female protective effect." Brain : a journal of neurology vol. (2021). doi: https://doi.org/10.1093/brain/awab204
Lawrence KE, Hernandez LM, Fuster E, Padgaonkar NT, Patterson G, Jung J, Okada NJ, Lowe JK, Hoekstra JN, Jack A, Aylward E, Gaab N, Van Horn JD, Bernier RA, McPartland JC, Webb SJ, Pelphrey KA, Green SA, Bookheimer SY, Geschwind DH, Dapretto M. Impact of autism genetic risk on brain connectivity: a mechanism for the female protective effect. Brain. 2021 May 29; doi: 10.1093/brain/awab204. Epub 2021 May 29. PMID: 34050743.
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Neural control of affiliative touch in prosocial interaction.

Nature

Wu YE, Dang J, Kingsbury L, Zhang M, Sun F, Hu RK, Hong W.
PMID: 34646019
Nature. 2021 Nov;599(7884):262-267. doi: 10.1038/s41586-021-03962-w. Epub 2021 Oct 13.

The ability to help and care for others fosters social cohesiveness and is vital to the physical and emotional well-being of social species, including humans

Cite
Wu YE, Dang J, Kingsbury L, et al. Neural control of affiliative touch in prosocial interaction. Nature. 2021;599(7884):262-267doi: 10.1038/s41586-021-03962-w.
Wu, Y. E., Dang, J., Kingsbury, L., Zhang, M., Sun, F., Hu, R. K., & Hong, W. (2021). Neural control of affiliative touch in prosocial interaction. Nature, 599(7884), 262-267. https://doi.org/10.1038/s41586-021-03962-w
Wu, Ye Emily, et al. "Neural control of affiliative touch in prosocial interaction." Nature vol. 599,7884 (2021): 262-267. doi: https://doi.org/10.1038/s41586-021-03962-w
Wu YE, Dang J, Kingsbury L, Zhang M, Sun F, Hu RK, Hong W. Neural control of affiliative touch in prosocial interaction. Nature. 2021 Nov;599(7884):262-267. doi: 10.1038/s41586-021-03962-w. Epub 2021 Oct 13. PMID: 34646019; PMCID: PMC8605624.
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Showing 1 to 7 of 7 entries