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Showing 1 to 12 of 63 entries
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Neurogenic inflammation in lung disease: burnt out?.

Inflammopharmacology

Rogers DF.
PMID: 17657611
Inflammopharmacology. 1997;5(4):319-29. doi: 10.1007/s10787-997-0029-2.

Neurogenic inflammation results from activation of sensory nerves which, acting in an 'efferent' manner, release sensory neuropeptides to induce a wide variety of physiological and immunological responses. This process is easy to demonstrate experimentally in the airways of small...

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Rogers DF. Neurogenic inflammation in lung disease: burnt out?. Inflammopharmacology. 1997;5(4):319-29doi: 10.1007/s10787-997-0029-2.
Rogers, D. F. (1997). Neurogenic inflammation in lung disease: burnt out?. Inflammopharmacology, 5(4), 319-29. https://doi.org/10.1007/s10787-997-0029-2
Rogers, D F. "Neurogenic inflammation in lung disease: burnt out?." Inflammopharmacology vol. 5,4 (1997): 319-29. doi: https://doi.org/10.1007/s10787-997-0029-2
Rogers DF. Neurogenic inflammation in lung disease: burnt out?. Inflammopharmacology. 1997;5(4):319-29. doi: 10.1007/s10787-997-0029-2. PMID: 17657611.
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mGluR(1) Receptors Contribute to Non-Purinergic Slow Excitatory Transmission to Submucosal VIP Neurons of Guinea-Pig Ileum.

Frontiers in neuroscience

Foong JP, Bornstein JC.
PMID: 20582273
Front Neurosci. 2009 May 29;3:46. doi: 10.3389/neuro.21.001.2009. eCollection 2009.

Vasoactive intestinal peptide (VIP) immunoreactive secretomotor neurons in the submucous plexus are involved in mediating bacterial toxin-induced hypersecretion leading to diarrhoea. VIP neurons become hyperexcitable after the mucosa is exposed to cholera toxin, which suggests that the manipulation of...

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Foong JP, Bornstein JC. mGluR(1) Receptors Contribute to Non-Purinergic Slow Excitatory Transmission to Submucosal VIP Neurons of Guinea-Pig Ileum. Front Neurosci. 2009;3:46doi: 10.3389/neuro.21.001.2009.
Foong, J. P., & Bornstein, J. C. (2009). mGluR(1) Receptors Contribute to Non-Purinergic Slow Excitatory Transmission to Submucosal VIP Neurons of Guinea-Pig Ileum. Frontiers in neuroscience, 346. https://doi.org/10.3389/neuro.21.001.2009
Foong, Jaime Pei Pei, and Bornstein, Joel C. "mGluR(1) Receptors Contribute to Non-Purinergic Slow Excitatory Transmission to Submucosal VIP Neurons of Guinea-Pig Ileum." Frontiers in neuroscience vol. 3 (2009): 46. doi: https://doi.org/10.3389/neuro.21.001.2009
Foong JP, Bornstein JC. mGluR(1) Receptors Contribute to Non-Purinergic Slow Excitatory Transmission to Submucosal VIP Neurons of Guinea-Pig Ileum. Front Neurosci. 2009 May 29;3:46. doi: 10.3389/neuro.21.001.2009. eCollection 2009. PMID: 20582273; PMCID: PMC2695390.
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Tachykinins and kinins in airway allergy.

Expert opinion on investigational drugs

Geppetti P, Tognetto M, Trevisani M, Amadesi S, Bertrand C.
PMID: 15992096
Expert Opin Investig Drugs. 1999 Jul;8(7):947-56. doi: 10.1517/13543784.8.7.947.

Using models of airway diseases, our understanding of the role of tachykinins and kinins in airway pathophysiology has been greatly enhanced by the recent development of a large series of peptide, peptoid and non-peptide antagonists for tachykinin and kinin...

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Geppetti P, Tognetto M, Trevisani M, et al. Tachykinins and kinins in airway allergy. Expert Opin Investig Drugs. 1999;8(7):947-56doi: 10.1517/13543784.8.7.947.
Geppetti, P., Tognetto, M., Trevisani, M., Amadesi, S., & Bertrand, C. (1999). Tachykinins and kinins in airway allergy. Expert opinion on investigational drugs, 8(7), 947-56. https://doi.org/10.1517/13543784.8.7.947
Geppetti, P, et al. "Tachykinins and kinins in airway allergy." Expert opinion on investigational drugs vol. 8,7 (1999): 947-56. doi: https://doi.org/10.1517/13543784.8.7.947
Geppetti P, Tognetto M, Trevisani M, Amadesi S, Bertrand C. Tachykinins and kinins in airway allergy. Expert Opin Investig Drugs. 1999 Jul;8(7):947-56. doi: 10.1517/13543784.8.7.947. PMID: 15992096.
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Porcine bladder urothelial, myofibroblast, and detrusor muscle cells: characterization and ATP release.

Frontiers in pharmacology

Cheng Y, Mansfield KJ, Sandow SL, Sadananda P, Burcher E, Moore KH.
PMID: 21713125
Front Pharmacol. 2011 Jun 07;2:27. doi: 10.3389/fphar.2011.00027. eCollection 2011.

ATP is released from the bladder mucosa in response to stretch, but the cell types responsible are unclear. Our aim was to isolate and characterize individual populations of urothelial, myofibroblast, and detrusor muscle cells in culture, and to examine...

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Cheng Y, Mansfield KJ, Sandow SL, et al. Porcine bladder urothelial, myofibroblast, and detrusor muscle cells: characterization and ATP release. Front Pharmacol. 2011;2:27doi: 10.3389/fphar.2011.00027.
Cheng, Y., Mansfield, K. J., Sandow, S. L., Sadananda, P., Burcher, E., & Moore, K. H. (2011). Porcine bladder urothelial, myofibroblast, and detrusor muscle cells: characterization and ATP release. Frontiers in pharmacology, 227. https://doi.org/10.3389/fphar.2011.00027
Cheng, Ying, et al. "Porcine bladder urothelial, myofibroblast, and detrusor muscle cells: characterization and ATP release." Frontiers in pharmacology vol. 2 (2011): 27. doi: https://doi.org/10.3389/fphar.2011.00027
Cheng Y, Mansfield KJ, Sandow SL, Sadananda P, Burcher E, Moore KH. Porcine bladder urothelial, myofibroblast, and detrusor muscle cells: characterization and ATP release. Front Pharmacol. 2011 Jun 07;2:27. doi: 10.3389/fphar.2011.00027. eCollection 2011. PMID: 21713125; PMCID: PMC3113165.
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Endothelin and neonatal capsaicin regulate gastric resistance to injury in BDL rats.

World journal of gastrointestinal pathophysiology

Câmara PR, Ferraz GJ, Velloso LA, Zeitune JM, Suassuna FA, Ferraz JG.
PMID: 23293745
World J Gastrointest Pathophysiol. 2012 Aug 15;3(4):85-91. doi: 10.4291/wjgp.v3.i4.85.

AIM: To investigate the relationship between primary afferent neurons, endothelin (ET) and the role of its receptors on ethanol-induced gastric damage in cirrhotic rats.METHODS: Cirrhosis and portal hypertension were induced in rats by bile duct ligation (BDL) while controls...

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Câmara PR, Ferraz GJ, Velloso LA, et al. Endothelin and neonatal capsaicin regulate gastric resistance to injury in BDL rats. World J Gastrointest Pathophysiol. 2012;3(4):85-91doi: 10.4291/wjgp.v3.i4.85.
Câmara, P. R., Ferraz, G. J., Velloso, L. A., Zeitune, J. M., Suassuna, F. A., & Ferraz, J. G. (2012). Endothelin and neonatal capsaicin regulate gastric resistance to injury in BDL rats. World journal of gastrointestinal pathophysiology, 3(4), 85-91. https://doi.org/10.4291/wjgp.v3.i4.85
Câmara, Paula Rs, et al. "Endothelin and neonatal capsaicin regulate gastric resistance to injury in BDL rats." World journal of gastrointestinal pathophysiology vol. 3,4 (2012): 85-91. doi: https://doi.org/10.4291/wjgp.v3.i4.85
Câmara PR, Ferraz GJ, Velloso LA, Zeitune JM, Suassuna FA, Ferraz JG. Endothelin and neonatal capsaicin regulate gastric resistance to injury in BDL rats. World J Gastrointest Pathophysiol. 2012 Aug 15;3(4):85-91. doi: 10.4291/wjgp.v3.i4.85. PMID: 23293745; PMCID: PMC3536844.
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Patients with high serum substance P levels previously to liver transplantation for hepatocellular carcinoma have higher risk of one-year liver transplantation mortality.

Oncotarget

Lorente L, Rodriguez ST, Sanz P, Pérez-Cejas A, Padilla J, Díaz D, González A, Martín MM, Jiménez A, Cerro P, Barrera MA.
PMID: 29765559
Oncotarget. 2018 Apr 20;9(30):21552-21559. doi: 10.18632/oncotarget.25097. eCollection 2018 Apr 20.

PURPOSE: Substance P is a tachykinins family member with inflammatory effects. Higher circulating levels of substance P have been found in patients with liver diseases and in patients with higher severity of liver diseases. The objective of this study...

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Lorente L, Rodriguez ST, Sanz P, et al. Patients with high serum substance P levels previously to liver transplantation for hepatocellular carcinoma have higher risk of one-year liver transplantation mortality. Oncotarget. 2018;9(30):21552-21559doi: 10.18632/oncotarget.25097.
Lorente, L., Rodriguez, S. T., Sanz, P., Pérez-Cejas, A., Padilla, J., Díaz, D., González, A., Martín, M. M., Jiménez, A., Cerro, P., & Barrera, M. A. (2018). Patients with high serum substance P levels previously to liver transplantation for hepatocellular carcinoma have higher risk of one-year liver transplantation mortality. Oncotarget, 9(30), 21552-21559. https://doi.org/10.18632/oncotarget.25097
Lorente, Leonardo, et al. "Patients with high serum substance P levels previously to liver transplantation for hepatocellular carcinoma have higher risk of one-year liver transplantation mortality." Oncotarget vol. 9,30 (2018): 21552-21559. doi: https://doi.org/10.18632/oncotarget.25097
Lorente L, Rodriguez ST, Sanz P, Pérez-Cejas A, Padilla J, Díaz D, González A, Martín MM, Jiménez A, Cerro P, Barrera MA. Patients with high serum substance P levels previously to liver transplantation for hepatocellular carcinoma have higher risk of one-year liver transplantation mortality. Oncotarget. 2018 Apr 20;9(30):21552-21559. doi: 10.18632/oncotarget.25097. eCollection 2018 Apr 20. PMID: 29765559; PMCID: PMC5940410.
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Both exogenous 5-HT and endogenous 5-HT, released by fluoxetine, enhance distension evoked propulsion in guinea-pig ileum in vitro.

Frontiers in neuroscience

Gwynne RM, Clarke AJ, Furness JB, Bornstein JC.
PMID: 25285066
Front Neurosci. 2014 Sep 19;8:301. doi: 10.3389/fnins.2014.00301. eCollection 2014.

The roles of 5-HT3 and 5-HT4 receptors in the modulation of intestinal propulsion by luminal application of 5-HT and augmentation of endogenous 5-HT effects were studied in segments of guinea-pig ileum in vitro. Persistent propulsive contractions evoked by saline...

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Gwynne RM, Clarke AJ, Furness JB, et al. Both exogenous 5-HT and endogenous 5-HT, released by fluoxetine, enhance distension evoked propulsion in guinea-pig ileum in vitro. Front Neurosci. 2014;8:301doi: 10.3389/fnins.2014.00301.
Gwynne, R. M., Clarke, A. J., Furness, J. B., & Bornstein, J. C. (2014). Both exogenous 5-HT and endogenous 5-HT, released by fluoxetine, enhance distension evoked propulsion in guinea-pig ileum in vitro. Frontiers in neuroscience, 8301. https://doi.org/10.3389/fnins.2014.00301
Gwynne, Rachel M, et al. "Both exogenous 5-HT and endogenous 5-HT, released by fluoxetine, enhance distension evoked propulsion in guinea-pig ileum in vitro." Frontiers in neuroscience vol. 8 (2014): 301. doi: https://doi.org/10.3389/fnins.2014.00301
Gwynne RM, Clarke AJ, Furness JB, Bornstein JC. Both exogenous 5-HT and endogenous 5-HT, released by fluoxetine, enhance distension evoked propulsion in guinea-pig ileum in vitro. Front Neurosci. 2014 Sep 19;8:301. doi: 10.3389/fnins.2014.00301. eCollection 2014. PMID: 25285066; PMCID: PMC4168689.
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Carcinoid Syndrome: A Review.

Cureus

Gade AK, Olariu E, Douthit NT.
PMID: 32257725
Cureus. 2020 Mar 05;12(3):e7186. doi: 10.7759/cureus.7186.

Carcinoid syndrome (CS) is a paraneoplastic syndrome caused by the release of serotonin and other substances from well-differentiated neuroendocrine tumors (NETs). The hallmark symptoms of carcinoid syndrome are flushing and diarrhea; atypical signs and symptoms can include wheezing, abdominal...

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Gade AK, Olariu E, Douthit NT. Carcinoid Syndrome: A Review. Cureus. 2020;12(3):e7186doi: 10.7759/cureus.7186.
Gade, A. K., Olariu, E., & Douthit, N. T. (2020). Carcinoid Syndrome: A Review. Cureus, 12(3), e7186. https://doi.org/10.7759/cureus.7186
Gade, Ajay K, et al. "Carcinoid Syndrome: A Review." Cureus vol. 12,3 (2020): e7186. doi: https://doi.org/10.7759/cureus.7186
Gade AK, Olariu E, Douthit NT. Carcinoid Syndrome: A Review. Cureus. 2020 Mar 05;12(3):e7186. doi: 10.7759/cureus.7186. PMID: 32257725; PMCID: PMC7124884.
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Overexpression of Substance P in pig airways increases MUC5AC through an NF-kβ pathway.

Physiological reports

Sponchiado M, Liao YS, Atanasova KR, Collins EN, Schurmann V, Bravo L, Reznikov LR.
PMID: 33580593
Physiol Rep. 2021 Feb;9(3):e14749. doi: 10.14814/phy2.14749.

Substance P (SP) is a tachykinin that regulates airway mucous secretion in both health and disease. Our study aimed to determine whether overexpression of SP without pre-existing inflammation was sufficient to induce changes in mucin secretion and transport in...

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Sponchiado M, Liao YS, Atanasova KR, et al. Overexpression of Substance P in pig airways increases MUC5AC through an NF-kβ pathway. Physiol Rep. 2021;9(3):e14749doi: 10.14814/phy2.14749.
Sponchiado, M., Liao, Y. S., Atanasova, K. R., Collins, E. N., Schurmann, V., Bravo, L., & Reznikov, L. R. (2021). Overexpression of Substance P in pig airways increases MUC5AC through an NF-kβ pathway. Physiological reports, 9(3), e14749. https://doi.org/10.14814/phy2.14749
Sponchiado, Mariana, et al. "Overexpression of Substance P in pig airways increases MUC5AC through an NF-kβ pathway." Physiological reports vol. 9,3 (2021): e14749. doi: https://doi.org/10.14814/phy2.14749
Sponchiado M, Liao YS, Atanasova KR, Collins EN, Schurmann V, Bravo L, Reznikov LR. Overexpression of Substance P in pig airways increases MUC5AC through an NF-kβ pathway. Physiol Rep. 2021 Feb;9(3):e14749. doi: 10.14814/phy2.14749. PMID: 33580593; PMCID: PMC7881348.
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Tachykinin receptor modulators: novel therapeutics for rheumatoid arthritis.

Expert opinion on investigational drugs

Brunelleschi S.
PMID: 11139831
Expert Opin Investig Drugs. 1999 Nov;8(11):1873-1891. doi: 10.1517/13543784.8.11.1873.

The activation of a cellular immune response in a genetically susceptible individual is widely recognised as a main step in triggering rheumatoid arthritis (RA). The tachykinins, substance P (SP) and neurokinin A (NKA), can play a major role in...

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Brunelleschi S. Tachykinin receptor modulators: novel therapeutics for rheumatoid arthritis. Expert Opin Investig Drugs. 1999;8(11):1873-1891doi: 10.1517/13543784.8.11.1873.
Brunelleschi, S. (1999). Tachykinin receptor modulators: novel therapeutics for rheumatoid arthritis. Expert opinion on investigational drugs, 8(11), 1873-1891. https://doi.org/10.1517/13543784.8.11.1873
Brunelleschi. "Tachykinin receptor modulators: novel therapeutics for rheumatoid arthritis." Expert opinion on investigational drugs vol. 8,11 (1999): 1873-1891. doi: https://doi.org/10.1517/13543784.8.11.1873
Brunelleschi S. Tachykinin receptor modulators: novel therapeutics for rheumatoid arthritis. Expert Opin Investig Drugs. 1999 Nov;8(11):1873-1891. doi: 10.1517/13543784.8.11.1873. PMID: 11139831.
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Tachykinins amplify the action of capsaicin on central histaminergic neurons.

Peptides

Sergeeva OA, Mazur K, Kernder A, Haas HL, De Luca R.
PMID: 34958850
Peptides. 2021 Dec 24;170729. doi: 10.1016/j.peptides.2021.170729. Epub 2021 Dec 24.

Substance P (SP), a product of the tachykinin 1 (Tac1) gene, is expressed in many hypothalamic neurons. Its wake-promoting potential could be mediated through histaminergic (HA) neurons of the tuberomamillary nucleus (TMN), where functional expression of neurokinin receptors (NKRs)...

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Sergeeva OA, Mazur K, Kernder A, et al. Tachykinins amplify the action of capsaicin on central histaminergic neurons. Peptides. 2021;170729doi: 10.1016/j.peptides.2021.170729.
Sergeeva, O. A., Mazur, K., Kernder, A., Haas, H. L., & De Luca, R. (2021). Tachykinins amplify the action of capsaicin on central histaminergic neurons. Peptides, 170729. https://doi.org/10.1016/j.peptides.2021.170729
Sergeeva, O A, et al. "Tachykinins amplify the action of capsaicin on central histaminergic neurons." Peptides vol. (2021): 170729. doi: https://doi.org/10.1016/j.peptides.2021.170729
Sergeeva OA, Mazur K, Kernder A, Haas HL, De Luca R. Tachykinins amplify the action of capsaicin on central histaminergic neurons. Peptides. 2021 Dec 24;170729. doi: 10.1016/j.peptides.2021.170729. Epub 2021 Dec 24. PMID: 34958850.
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Tachykinins: Neuropeptides That Are Ancient, Diverse, Widespread and Functionally Pleiotropic.

Frontiers in neuroscience

Nässel DR, Zandawala M, Kawada T, Satake H.
PMID: 31824255
Front Neurosci. 2019 Nov 20;13:1262. doi: 10.3389/fnins.2019.01262. eCollection 2019.

Tachykinins (TKs) are ancient neuropeptides present throughout the bilaterians and are, with some exceptions, characterized by a conserved FX

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Nässel DR, Zandawala M, Kawada T, et al. Tachykinins: Neuropeptides That Are Ancient, Diverse, Widespread and Functionally Pleiotropic. Front Neurosci. 2019;13:1262doi: 10.3389/fnins.2019.01262.
Nässel, D. R., Zandawala, M., Kawada, T., & Satake, H. (2019). Tachykinins: Neuropeptides That Are Ancient, Diverse, Widespread and Functionally Pleiotropic. Frontiers in neuroscience, 131262. https://doi.org/10.3389/fnins.2019.01262
Nässel, Dick R, et al. "Tachykinins: Neuropeptides That Are Ancient, Diverse, Widespread and Functionally Pleiotropic." Frontiers in neuroscience vol. 13 (2019): 1262. doi: https://doi.org/10.3389/fnins.2019.01262
Nässel DR, Zandawala M, Kawada T, Satake H. Tachykinins: Neuropeptides That Are Ancient, Diverse, Widespread and Functionally Pleiotropic. Front Neurosci. 2019 Nov 20;13:1262. doi: 10.3389/fnins.2019.01262. eCollection 2019. PMID: 31824255; PMCID: PMC6880623.
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Showing 1 to 12 of 63 entries