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Showing 1 to 12 of 1485 entries
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Wogonoside alleviates inflammation induced by traumatic spinal cord injury by suppressing NF-κB and NLRP3 inflammasome activation.

Experimental and therapeutic medicine

Zhu Y, Zhu H, Wang Z, Gao F, Wang J, Zhang W.
PMID: 28966691
Exp Ther Med. 2017 Oct;14(4):3304-3308. doi: 10.3892/etm.2017.4904. Epub 2017 Aug 08.

Wogonoside possesses anti-oxidative, anti-inflammatory, anti-allergy and anti-tumor properties. The aim of the present study was to evaluate whether wogonoside alleviates spinal cord injury (SCI)-induced inflammation via nuclear factor (NF)-κB and nucleotide-binding oligomerization domain-like receptor family pyrin domain containing 3...

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Zhu Y, Zhu H, Wang Z, et al. Wogonoside alleviates inflammation induced by traumatic spinal cord injury by suppressing NF-κB and NLRP3 inflammasome activation. Exp Ther Med. 2017;14(4):3304-3308doi: 10.3892/etm.2017.4904.
Zhu, Y., Zhu, H., Wang, Z., Gao, F., Wang, J., & Zhang, W. (2017). Wogonoside alleviates inflammation induced by traumatic spinal cord injury by suppressing NF-κB and NLRP3 inflammasome activation. Experimental and therapeutic medicine, 14(4), 3304-3308. https://doi.org/10.3892/etm.2017.4904
Zhu, Yonglin, et al. "Wogonoside alleviates inflammation induced by traumatic spinal cord injury by suppressing NF-κB and NLRP3 inflammasome activation." Experimental and therapeutic medicine vol. 14,4 (2017): 3304-3308. doi: https://doi.org/10.3892/etm.2017.4904
Zhu Y, Zhu H, Wang Z, Gao F, Wang J, Zhang W. Wogonoside alleviates inflammation induced by traumatic spinal cord injury by suppressing NF-κB and NLRP3 inflammasome activation. Exp Ther Med. 2017 Oct;14(4):3304-3308. doi: 10.3892/etm.2017.4904. Epub 2017 Aug 08. PMID: 28966691; PMCID: PMC5613185.
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Antibody Production, Anaphylactic Signs, and T-Cell Responses Induced by Oral Sensitization With Ovalbumin in BALB/c and C3H/HeOuJ Mice.

Allergy, asthma & immunology research

Pablos-Tanarro A, López-Expósito I, Lozano-Ojalvo D, López-Fandiño R, Molina E.
PMID: 26922934
Allergy Asthma Immunol Res. 2016 May;8(3):239-45. doi: 10.4168/aair.2016.8.3.239.

PURPOSE: Two mouse strains, BALB/c and C3H/HeOuJ, broadly used in the field of food allergy, were compared for the evaluation of the allergenic potential of ovalbumin (OVA).METHODS: Sensitization was made by administering 2 different OVA doses (1 and 5...

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Pablos-Tanarro A, López-Expósito I, Lozano-Ojalvo D, et al. Antibody Production, Anaphylactic Signs, and T-Cell Responses Induced by Oral Sensitization With Ovalbumin in BALB/c and C3H/HeOuJ Mice. Allergy Asthma Immunol Res. 2016;8(3):239-45doi: 10.4168/aair.2016.8.3.239.
Pablos-Tanarro, A., López-Expósito, I., Lozano-Ojalvo, D., López-Fandiño, R., & Molina, E. (2016). Antibody Production, Anaphylactic Signs, and T-Cell Responses Induced by Oral Sensitization With Ovalbumin in BALB/c and C3H/HeOuJ Mice. Allergy, asthma & immunology research, 8(3), 239-45. https://doi.org/10.4168/aair.2016.8.3.239
Pablos-Tanarro, Alba, et al. "Antibody Production, Anaphylactic Signs, and T-Cell Responses Induced by Oral Sensitization With Ovalbumin in BALB/c and C3H/HeOuJ Mice." Allergy, asthma & immunology research vol. 8,3 (2016): 239-45. doi: https://doi.org/10.4168/aair.2016.8.3.239
Pablos-Tanarro A, López-Expósito I, Lozano-Ojalvo D, López-Fandiño R, Molina E. Antibody Production, Anaphylactic Signs, and T-Cell Responses Induced by Oral Sensitization With Ovalbumin in BALB/c and C3H/HeOuJ Mice. Allergy Asthma Immunol Res. 2016 May;8(3):239-45. doi: 10.4168/aair.2016.8.3.239. PMID: 26922934; PMCID: PMC4773212.
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Expression of TLR4 in Non-Small Cell Lung Cancer Is Associated with PD-L1 and Poor Prognosis in Patients Receiving Pulmonectomy.

Frontiers in immunology

Wang K, Wang J, Wei F, Zhao N, Yang F, Ren X.
PMID: 28484456
Front Immunol. 2017 Apr 21;8:456. doi: 10.3389/fimmu.2017.00456. eCollection 2017.

Currently, the effect of inflammation on tumorigenesis and progression has been widely noted. As a member of pattern recognition receptors, toll-like receptor 4 (TLR4) plays a pivotal role in tumor immune microenvironment and has been increasingly investigated. In the...

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Wang K, Wang J, Wei F, et al. Expression of TLR4 in Non-Small Cell Lung Cancer Is Associated with PD-L1 and Poor Prognosis in Patients Receiving Pulmonectomy. Front Immunol. 2017;8:456doi: 10.3389/fimmu.2017.00456.
Wang, K., Wang, J., Wei, F., Zhao, N., Yang, F., & Ren, X. (2017). Expression of TLR4 in Non-Small Cell Lung Cancer Is Associated with PD-L1 and Poor Prognosis in Patients Receiving Pulmonectomy. Frontiers in immunology, 8456. https://doi.org/10.3389/fimmu.2017.00456
Wang, Kaiyuan, et al. "Expression of TLR4 in Non-Small Cell Lung Cancer Is Associated with PD-L1 and Poor Prognosis in Patients Receiving Pulmonectomy." Frontiers in immunology vol. 8 (2017): 456. doi: https://doi.org/10.3389/fimmu.2017.00456
Wang K, Wang J, Wei F, Zhao N, Yang F, Ren X. Expression of TLR4 in Non-Small Cell Lung Cancer Is Associated with PD-L1 and Poor Prognosis in Patients Receiving Pulmonectomy. Front Immunol. 2017 Apr 21;8:456. doi: 10.3389/fimmu.2017.00456. eCollection 2017. PMID: 28484456; PMCID: PMC5399072.
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Toll-like receptor 4 gene Asp299Gly and Thr399Ile polymorphisms in type 2 diabetes mellitus: A meta-analysis of 15,059 subjects: Need for clarification of data in a recent meta-analysis.

Diabetes research and clinical practice

Chang WW, Zhang L, Jin YL, Yao YS.
PMID: 26371398
Diabetes Res Clin Pract. 2015 Dec;110(3):e31-2. doi: 10.1016/j.diabres.2015.08.001. Epub 2015 Aug 22.

No abstract available.

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Chang WW, Zhang L, Jin YL, et al. Toll-like receptor 4 gene Asp299Gly and Thr399Ile polymorphisms in type 2 diabetes mellitus: A meta-analysis of 15,059 subjects: Need for clarification of data in a recent meta-analysis. Diabetes Res Clin Pract. 2015;110(3):e31-2doi: 10.1016/j.diabres.2015.08.001.
Chang, W. W., Zhang, L., Jin, Y. L., & Yao, Y. S. (2015). Toll-like receptor 4 gene Asp299Gly and Thr399Ile polymorphisms in type 2 diabetes mellitus: A meta-analysis of 15,059 subjects: Need for clarification of data in a recent meta-analysis. Diabetes research and clinical practice, 110(3), e31-2. https://doi.org/10.1016/j.diabres.2015.08.001
Chang, Wei-Wei, et al. "Toll-like receptor 4 gene Asp299Gly and Thr399Ile polymorphisms in type 2 diabetes mellitus: A meta-analysis of 15,059 subjects: Need for clarification of data in a recent meta-analysis." Diabetes research and clinical practice vol. 110,3 (2015): e31-2. doi: https://doi.org/10.1016/j.diabres.2015.08.001
Chang WW, Zhang L, Jin YL, Yao YS. Toll-like receptor 4 gene Asp299Gly and Thr399Ile polymorphisms in type 2 diabetes mellitus: A meta-analysis of 15,059 subjects: Need for clarification of data in a recent meta-analysis. Diabetes Res Clin Pract. 2015 Dec;110(3):e31-2. doi: 10.1016/j.diabres.2015.08.001. Epub 2015 Aug 22. PMID: 26371398.
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HBD-3 regulation of the immune response and the LPS/TLR4-mediated signaling pathway.

Experimental and therapeutic medicine

Zhu C, Bao NR, Chen S, Zhao JN.
PMID: 27703496
Exp Ther Med. 2016 Oct;12(4):2150-2154. doi: 10.3892/etm.2016.3579. Epub 2016 Aug 09.

The aim of the study was to investigate the mechanisms of human β-defensin 3 (HBD-3) regulation of the immune response and the lipopolysaccharide/Toll-like receptor-4 (LPS/TLR4)-mediated signaling pathway. A TLR4 extracellular gene fragment was cloned into the pET32a plasmid to...

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Zhu C, Bao NR, Chen S, et al. HBD-3 regulation of the immune response and the LPS/TLR4-mediated signaling pathway. Exp Ther Med. 2016;12(4):2150-2154doi: 10.3892/etm.2016.3579.
Zhu, C., Bao, N. R., Chen, S., & Zhao, J. N. (2016). HBD-3 regulation of the immune response and the LPS/TLR4-mediated signaling pathway. Experimental and therapeutic medicine, 12(4), 2150-2154. https://doi.org/10.3892/etm.2016.3579
Zhu, Chen, et al. "HBD-3 regulation of the immune response and the LPS/TLR4-mediated signaling pathway." Experimental and therapeutic medicine vol. 12,4 (2016): 2150-2154. doi: https://doi.org/10.3892/etm.2016.3579
Zhu C, Bao NR, Chen S, Zhao JN. HBD-3 regulation of the immune response and the LPS/TLR4-mediated signaling pathway. Exp Ther Med. 2016 Oct;12(4):2150-2154. doi: 10.3892/etm.2016.3579. Epub 2016 Aug 09. PMID: 27703496; PMCID: PMC5038901.
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Expression and clinical significance of SATB1 and TLR4 in breast cancer.

Oncology letters

Wang X, Yu X, Wang Q, Lu Y, Chen H.
PMID: 28927120
Oncol Lett. 2017 Sep;14(3):3611-3615. doi: 10.3892/ol.2017.6571. Epub 2017 Jul 12.

This study investigated the expression of special AT-rich sequence-binding protein 1 (SATB1) and toll-like receptor 4 (TLR4) protein in breast cancer and its clinical significance. We collected breast cancer tissues from 120 patients and adjacent non-cancerous tissue from 53...

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Wang X, Yu X, Wang Q, et al. Expression and clinical significance of SATB1 and TLR4 in breast cancer. Oncol Lett. 2017;14(3):3611-3615doi: 10.3892/ol.2017.6571.
Wang, X., Yu, X., Wang, Q., Lu, Y., & Chen, H. (2017). Expression and clinical significance of SATB1 and TLR4 in breast cancer. Oncology letters, 14(3), 3611-3615. https://doi.org/10.3892/ol.2017.6571
Wang, Xuebo, et al. "Expression and clinical significance of SATB1 and TLR4 in breast cancer." Oncology letters vol. 14,3 (2017): 3611-3615. doi: https://doi.org/10.3892/ol.2017.6571
Wang X, Yu X, Wang Q, Lu Y, Chen H. Expression and clinical significance of SATB1 and TLR4 in breast cancer. Oncol Lett. 2017 Sep;14(3):3611-3615. doi: 10.3892/ol.2017.6571. Epub 2017 Jul 12. PMID: 28927120; PMCID: PMC5587979.
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Involvement of Toll-like receptor 4 in vinorelbine-induced vascular endothelial injury.

Experimental and therapeutic medicine

Qian W, Gao L, Chen C, Tan Y, Zhou Y, Li Z.
PMID: 26170913
Exp Ther Med. 2015 Jul;10(1):62-66. doi: 10.3892/etm.2015.2494. Epub 2015 May 14.

Vinorelbine (VIN) is a semi-synthetic vinca alkaloid and is one of the most active agents for the treatment of solid tumors. The drug is commonly administered through a peripheral vein. Although VIN is known to cause local venous toxicity,...

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Qian W, Gao L, Chen C, et al. Involvement of Toll-like receptor 4 in vinorelbine-induced vascular endothelial injury. Exp Ther Med. 2015;10(1):62-66doi: 10.3892/etm.2015.2494.
Qian, W., Gao, L., Chen, C., Tan, Y., Zhou, Y., & Li, Z. (2015). Involvement of Toll-like receptor 4 in vinorelbine-induced vascular endothelial injury. Experimental and therapeutic medicine, 10(1), 62-66. https://doi.org/10.3892/etm.2015.2494
Qian, Weiwei, et al. "Involvement of Toll-like receptor 4 in vinorelbine-induced vascular endothelial injury." Experimental and therapeutic medicine vol. 10,1 (2015): 62-66. doi: https://doi.org/10.3892/etm.2015.2494
Qian W, Gao L, Chen C, Tan Y, Zhou Y, Li Z. Involvement of Toll-like receptor 4 in vinorelbine-induced vascular endothelial injury. Exp Ther Med. 2015 Jul;10(1):62-66. doi: 10.3892/etm.2015.2494. Epub 2015 May 14. PMID: 26170913; PMCID: PMC4486887.
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Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages.

Chinese medicine

Peng XX, Zhang SH, Wang XL, Ye TJ, Li H, Yan XF, Wei L, Wu ZP, Hu J, Zou CP, Wang YH, Hu XD.
PMID: 26155304
Chin Med. 2015 Jul 01;10:15. doi: 10.1186/s13020-015-0045-x. eCollection 2015.

BACKGROUND: Panax Notoginseng flower saponins (PNFS) are the main active component of Panax notoginseng (Burk) F. H. Chen flower bud (PNF) and possess significant anti-inflammatory efficacy. This study aims to explore the mechanisms underlying PNFS' antiflammatory action in RAW264.7...

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Peng XX, Zhang SH, Wang XL, et al. Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages. Chin Med. 2015;10:15doi: 10.1186/s13020-015-0045-x.
Peng, X. X., Zhang, S. H., Wang, X. L., Ye, T. J., Li, H., Yan, X. F., Wei, L., Wu, Z. P., Hu, J., Zou, C. P., Wang, Y. H., & Hu, X. D. (2015). Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages. Chinese medicine, 1015. https://doi.org/10.1186/s13020-015-0045-x
Peng, Xiao-Xu, et al. "Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages." Chinese medicine vol. 10 (2015): 15. doi: https://doi.org/10.1186/s13020-015-0045-x
Peng XX, Zhang SH, Wang XL, Ye TJ, Li H, Yan XF, Wei L, Wu ZP, Hu J, Zou CP, Wang YH, Hu XD. Panax Notoginseng flower saponins (PNFS) inhibit LPS-stimulated NO overproduction and iNOS gene overexpression via the suppression of TLR4-mediated MAPK/NF-kappa B signaling pathways in RAW264.7 macrophages. Chin Med. 2015 Jul 01;10:15. doi: 10.1186/s13020-015-0045-x. eCollection 2015. PMID: 26155304; PMCID: PMC4493833.
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Involvement of Cbl-b-mediated macrophage inactivation in insulin resistance.

World journal of diabetes

Abe T, Hirasaka K, Nikawa T.
PMID: 28344752
World J Diabetes. 2017 Mar 15;8(3):97-103. doi: 10.4239/wjd.v8.i3.97.

Aging and overnutrition cause obesity in rodents and humans. It is well-known that obesity causes various diseases by producing insulin resistance (IR). Macrophages infiltrate the adipose tissue (AT) of obese individuals and cause chronic low-level inflammation associated with IR....

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Abe T, Hirasaka K, Nikawa T. Involvement of Cbl-b-mediated macrophage inactivation in insulin resistance. World J Diabetes. 2017;8(3):97-103doi: 10.4239/wjd.v8.i3.97.
Abe, T., Hirasaka, K., & Nikawa, T. (2017). Involvement of Cbl-b-mediated macrophage inactivation in insulin resistance. World journal of diabetes, 8(3), 97-103. https://doi.org/10.4239/wjd.v8.i3.97
Abe, Tomoki, et al. "Involvement of Cbl-b-mediated macrophage inactivation in insulin resistance." World journal of diabetes vol. 8,3 (2017): 97-103. doi: https://doi.org/10.4239/wjd.v8.i3.97
Abe T, Hirasaka K, Nikawa T. Involvement of Cbl-b-mediated macrophage inactivation in insulin resistance. World J Diabetes. 2017 Mar 15;8(3):97-103. doi: 10.4239/wjd.v8.i3.97. PMID: 28344752; PMCID: PMC5348625.
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Vitamin D3 alters Toll-like receptor 4 signaling in monocytes of pregnant women at risk for preeclampsia.

International journal of clinical and experimental medicine

Qian L, Wang H, Wu F, Li M, Chen W, Lv L.
PMID: 26770399
Int J Clin Exp Med. 2015 Oct 15;8(10):18041-9. eCollection 2015.

Vitamin D deficiency during pregnancy is thought to play a role in the development of preeclampsia; however, the underlying mechanism is not fully understood. In this study, a randomized double-blind placebo-controlled clinical trial was performed among 60 pregnant women...

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Qian L, Wang H, Wu F, et al. Vitamin D3 alters Toll-like receptor 4 signaling in monocytes of pregnant women at risk for preeclampsia. Int J Clin Exp Med. 2015;8(10):18041-9
Qian, L., Wang, H., Wu, F., Li, M., Chen, W., & Lv, L. (2015). Vitamin D3 alters Toll-like receptor 4 signaling in monocytes of pregnant women at risk for preeclampsia. International journal of clinical and experimental medicine, 8(10), 18041-9.
Qian, Lei, et al. "Vitamin D3 alters Toll-like receptor 4 signaling in monocytes of pregnant women at risk for preeclampsia." International journal of clinical and experimental medicine vol. 8,10 (2015): 18041-9.
Qian L, Wang H, Wu F, Li M, Chen W, Lv L. Vitamin D3 alters Toll-like receptor 4 signaling in monocytes of pregnant women at risk for preeclampsia. Int J Clin Exp Med. 2015 Oct 15;8(10):18041-9. eCollection 2015. PMID: 26770399; PMCID: PMC4694299.
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Toll-like receptor 4 confers inflammatory response to Suilysin.

Frontiers in microbiology

Bi L, Pian Y, Chen S, Ren Z, Liu P, Lv Q, Zheng Y, Zhang S, Hao H, Yuan Y, Jiang Y.
PMID: 26167160
Front Microbiol. 2015 Jun 26;6:644. doi: 10.3389/fmicb.2015.00644. eCollection 2015.

Streptococcus suis serotype 2 (SS2) is an emerging human pathogen worldwide. A large outbreak occurred in the summer of 2005 in China. Serum samples from this outbreak revealed that levels of the main proinflammatory cytokines were significantly higher in...

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Bi L, Pian Y, Chen S, et al. Toll-like receptor 4 confers inflammatory response to Suilysin. Front Microbiol. 2015;6:644doi: 10.3389/fmicb.2015.00644.
Bi, L., Pian, Y., Chen, S., Ren, Z., Liu, P., Lv, Q., Zheng, Y., Zhang, S., Hao, H., Yuan, Y., & Jiang, Y. (2015). Toll-like receptor 4 confers inflammatory response to Suilysin. Frontiers in microbiology, 6644. https://doi.org/10.3389/fmicb.2015.00644
Bi, Lili, et al. "Toll-like receptor 4 confers inflammatory response to Suilysin." Frontiers in microbiology vol. 6 (2015): 644. doi: https://doi.org/10.3389/fmicb.2015.00644
Bi L, Pian Y, Chen S, Ren Z, Liu P, Lv Q, Zheng Y, Zhang S, Hao H, Yuan Y, Jiang Y. Toll-like receptor 4 confers inflammatory response to Suilysin. Front Microbiol. 2015 Jun 26;6:644. doi: 10.3389/fmicb.2015.00644. eCollection 2015. PMID: 26167160; PMCID: PMC4481166.
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Effect of age on pro-inflammatory miRNAs contained in mesenchymal stem cell-derived extracellular vesicles.

Scientific reports

Fafián-Labora J, Lesende-Rodriguez I, Fernández-Pernas P, Sangiao-Alvarellos S, Monserrat L, Arntz OJ, van de Loo FJ, Mateos J, Arufe MC.
PMID: 28262816
Sci Rep. 2017 Mar 06;7:43923. doi: 10.1038/srep43923.

Stem cells possess significant age-dependent differences in their immune-response profile. These differences were analysed by Next-Generation Sequencing of six age groups from bone marrow mesenchymal stem cells. A total of 9,628 genes presenting differential expression between age groups were...

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Fafián-Labora J, Lesende-Rodriguez I, Fernández-Pernas P, et al. Effect of age on pro-inflammatory miRNAs contained in mesenchymal stem cell-derived extracellular vesicles. Sci Rep. 2017;7:43923doi: 10.1038/srep43923.
Fafián-Labora, J., Lesende-Rodriguez, I., Fernández-Pernas, P., Sangiao-Alvarellos, S., Monserrat, L., Arntz, O. J., van de Loo, F. J., Mateos, J., & Arufe, M. C. (2017). Effect of age on pro-inflammatory miRNAs contained in mesenchymal stem cell-derived extracellular vesicles. Scientific reports, 743923. https://doi.org/10.1038/srep43923
Fafián-Labora, J, et al. "Effect of age on pro-inflammatory miRNAs contained in mesenchymal stem cell-derived extracellular vesicles." Scientific reports vol. 7 (2017): 43923. doi: https://doi.org/10.1038/srep43923
Fafián-Labora J, Lesende-Rodriguez I, Fernández-Pernas P, Sangiao-Alvarellos S, Monserrat L, Arntz OJ, van de Loo FJ, Mateos J, Arufe MC. Effect of age on pro-inflammatory miRNAs contained in mesenchymal stem cell-derived extracellular vesicles. Sci Rep. 2017 Mar 06;7:43923. doi: 10.1038/srep43923. PMID: 28262816; PMCID: PMC5338265.
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Showing 1 to 12 of 1485 entries