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Gene expression markers in circulating tumor cells may predict bone metastasis and response to hormonal treatment in breast cancer.

Molecular and clinical oncology

Wang H, Molina J, Jiang J, Ferber M, Pruthi S, Jatkoe T, Derecho C, Rajpurohit Y, Zheng J, Wang Y.
PMID: 24649289
Mol Clin Oncol. 2013 Nov;1(6):1031-1038. doi: 10.3892/mco.2013.163. Epub 2013 Aug 02.

Circulating tumor cells (CTCs) have recently attracted attention due to their potential as prognostic and predictive markers for the clinical management of metastatic breast cancer patients. The isolation of CTCs from patients may enable the molecular characterization of these...

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Wang H, Molina J, Jiang J, et al. Gene expression markers in circulating tumor cells may predict bone metastasis and response to hormonal treatment in breast cancer. Mol Clin Oncol. 2013;1(6):1031-1038doi: 10.3892/mco.2013.163.
Wang, H., Molina, J., Jiang, J., Ferber, M., Pruthi, S., Jatkoe, T., Derecho, C., Rajpurohit, Y., Zheng, J., & Wang, Y. (2013). Gene expression markers in circulating tumor cells may predict bone metastasis and response to hormonal treatment in breast cancer. Molecular and clinical oncology, 1(6), 1031-1038. https://doi.org/10.3892/mco.2013.163
Wang, Haiying, et al. "Gene expression markers in circulating tumor cells may predict bone metastasis and response to hormonal treatment in breast cancer." Molecular and clinical oncology vol. 1,6 (2013): 1031-1038. doi: https://doi.org/10.3892/mco.2013.163
Wang H, Molina J, Jiang J, Ferber M, Pruthi S, Jatkoe T, Derecho C, Rajpurohit Y, Zheng J, Wang Y. Gene expression markers in circulating tumor cells may predict bone metastasis and response to hormonal treatment in breast cancer. Mol Clin Oncol. 2013 Nov;1(6):1031-1038. doi: 10.3892/mco.2013.163. Epub 2013 Aug 02. PMID: 24649289; PMCID: PMC3915691.
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Circ_0061825 Acts as a miR-593-3p Sponge to Promote Breast Cancer Progression by Regulating FGFR3 Expression.

Cancer management and research

Xie J, Wan Y, Zhang M, Jin Z, Yao Y.
PMID: 33177875
Cancer Manag Res. 2020 Nov 04;12:11243-11255. doi: 10.2147/CMAR.S269128. eCollection 2020.

BACKGROUND: Breast cancer (BC) remains the most common malignancy among women. Circular RNAs (circRNAs) have been demonstrated to play important roles in human cancers, including BC. In this study, we sought to identify the precise parts of circ_0061825 (circRNA...

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Xie J, Wan Y, Zhang M, et al. Circ_0061825 Acts as a miR-593-3p Sponge to Promote Breast Cancer Progression by Regulating FGFR3 Expression. Cancer Manag Res. 2020;12:11243-11255doi: 10.2147/CMAR.S269128.
Xie, J., Wan, Y., Zhang, M., Jin, Z., & Yao, Y. (2020). Circ_0061825 Acts as a miR-593-3p Sponge to Promote Breast Cancer Progression by Regulating FGFR3 Expression. Cancer management and research, 1211243-11255. https://doi.org/10.2147/CMAR.S269128
Xie, Jiping, et al. "Circ_0061825 Acts as a miR-593-3p Sponge to Promote Breast Cancer Progression by Regulating FGFR3 Expression." Cancer management and research vol. 12 (2020): 11243-11255. doi: https://doi.org/10.2147/CMAR.S269128
Xie J, Wan Y, Zhang M, Jin Z, Yao Y. Circ_0061825 Acts as a miR-593-3p Sponge to Promote Breast Cancer Progression by Regulating FGFR3 Expression. Cancer Manag Res. 2020 Nov 04;12:11243-11255. doi: 10.2147/CMAR.S269128. eCollection 2020. PMID: 33177875; PMCID: PMC7649251.
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7-ketocholesterol and 27-hydroxycholesterol decreased doxorubicin sensitivity in breast cancer cells: estrogenic activity and mTOR pathway.

Oncotarget

Wang CW, Huang CC, Chou PH, Chang YP, Wei S, Guengerich FP, Chou YC, Wang SF, Lai PS, Souček P, Ueng YF.
PMID: 29029490
Oncotarget. 2017 Aug 02;8(39):66033-66050. doi: 10.18632/oncotarget.19789. eCollection 2017 Sep 12.

Hypercholesterolemia is one of the risk factors for poor outcome in breast cancer therapy. To elucidate the influence of the main circulating oxysterols, cholesterol oxidation products, on the cell-killing effect of doxorubicin, cells were exposed to oxysterols at a...

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Wang CW, Huang CC, Chou PH, et al. 7-ketocholesterol and 27-hydroxycholesterol decreased doxorubicin sensitivity in breast cancer cells: estrogenic activity and mTOR pathway. Oncotarget. 2017;8(39):66033-66050doi: 10.18632/oncotarget.19789.
Wang, C. W., Huang, C. C., Chou, P. H., Chang, Y. P., Wei, S., Guengerich, F. P., Chou, Y. C., Wang, S. F., Lai, P. S., Souček, P., & Ueng, Y. F. (2017). 7-ketocholesterol and 27-hydroxycholesterol decreased doxorubicin sensitivity in breast cancer cells: estrogenic activity and mTOR pathway. Oncotarget, 8(39), 66033-66050. https://doi.org/10.18632/oncotarget.19789
Wang, Chun-Wei, et al. "7-ketocholesterol and 27-hydroxycholesterol decreased doxorubicin sensitivity in breast cancer cells: estrogenic activity and mTOR pathway." Oncotarget vol. 8,39 (2017): 66033-66050. doi: https://doi.org/10.18632/oncotarget.19789
Wang CW, Huang CC, Chou PH, Chang YP, Wei S, Guengerich FP, Chou YC, Wang SF, Lai PS, Souček P, Ueng YF. 7-ketocholesterol and 27-hydroxycholesterol decreased doxorubicin sensitivity in breast cancer cells: estrogenic activity and mTOR pathway. Oncotarget. 2017 Aug 02;8(39):66033-66050. doi: 10.18632/oncotarget.19789. eCollection 2017 Sep 12. PMID: 29029490; PMCID: PMC5630390.
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NF-kB-dependent activation of STAT3 by H. pylori is suppressed by TFF1.

Cancer cell international

Soutto M, Bhat N, Khalafi S, Zhu S, Poveda J, Garcia-Buitrago M, Zaika A, El-Rifai W.
PMID: 34419066
Cancer Cell Int. 2021 Aug 21;21(1):444. doi: 10.1186/s12935-021-02140-2.

BACKGROUND: H. pylori infection is the main risk factor for gastric cancer. In this study, we investigated H. pylori-mediated activation of STAT3 and NF-κB in gastric cancer, using in vitro and in vivo models.METHODS: To investigate the activation of...

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Soutto M, Bhat N, Khalafi S, et al. NF-kB-dependent activation of STAT3 by H. pylori is suppressed by TFF1. Cancer Cell Int. 2021;21(1):444doi: 10.1186/s12935-021-02140-2.
Soutto, M., Bhat, N., Khalafi, S., Zhu, S., Poveda, J., Garcia-Buitrago, M., Zaika, A., & El-Rifai, W. (2021). NF-kB-dependent activation of STAT3 by H. pylori is suppressed by TFF1. Cancer cell international, 21(1), 444. https://doi.org/10.1186/s12935-021-02140-2
Soutto, Mohammed, et al. "NF-kB-dependent activation of STAT3 by H. pylori is suppressed by TFF1." Cancer cell international vol. 21,1 (2021): 444. doi: https://doi.org/10.1186/s12935-021-02140-2
Soutto M, Bhat N, Khalafi S, Zhu S, Poveda J, Garcia-Buitrago M, Zaika A, El-Rifai W. NF-kB-dependent activation of STAT3 by H. pylori is suppressed by TFF1. Cancer Cell Int. 2021 Aug 21;21(1):444. doi: 10.1186/s12935-021-02140-2. PMID: 34419066; PMCID: PMC8380333.
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Trefoil Factor 1 Suppresses Epithelial-mesenchymal Transition through Inhibition of TGF-beta Signaling in Gastric Cancer Cells.

Journal of cancer prevention

Lee DY, Song MY, Kim EH.
PMID: 34258252
J Cancer Prev. 2021 Jun 30;26(2):137-144. doi: 10.15430/JCP.2021.26.2.137.

Gastric cancer is a malignancy with high incidence and mortality worldwide. In gastric cancer, epithelial-mesenchymal transition (EMT) and metastasis further increase the mortality rate. Trefoil factor 1 (TFF1) has been reported as a protective factor in the gastric mucosa....

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Lee DY, Song MY, Kim EH. Trefoil Factor 1 Suppresses Epithelial-mesenchymal Transition through Inhibition of TGF-beta Signaling in Gastric Cancer Cells. J Cancer Prev. 2021;26(2):137-144doi: 10.15430/JCP.2021.26.2.137.
Lee, D. Y., Song, M. Y., & Kim, E. H. (2021). Trefoil Factor 1 Suppresses Epithelial-mesenchymal Transition through Inhibition of TGF-beta Signaling in Gastric Cancer Cells. Journal of cancer prevention, 26(2), 137-144. https://doi.org/10.15430/JCP.2021.26.2.137
Lee, Da-Young, et al. "Trefoil Factor 1 Suppresses Epithelial-mesenchymal Transition through Inhibition of TGF-beta Signaling in Gastric Cancer Cells." Journal of cancer prevention vol. 26,2 (2021): 137-144. doi: https://doi.org/10.15430/JCP.2021.26.2.137
Lee DY, Song MY, Kim EH. Trefoil Factor 1 Suppresses Epithelial-mesenchymal Transition through Inhibition of TGF-beta Signaling in Gastric Cancer Cells. J Cancer Prev. 2021 Jun 30;26(2):137-144. doi: 10.15430/JCP.2021.26.2.137. PMID: 34258252; PMCID: PMC8249209.
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Humulene Inhibits Acute Gastric Mucosal Injury by Enhancing Mucosal Integrity.

Antioxidants (Basel, Switzerland)

Yeo D, Hwang SJ, Song YS, Lee HJ.
PMID: 34064830
Antioxidants (Basel). 2021 May 11;10(5). doi: 10.3390/antiox10050761.

This study was designed to determine whether α-humulene, a major constituent in many plants used in fragrances, has a protective role against gastric injury in vivo and in vitro. A rat model of hydrochloric acid (HCl)/ethanol-induced gastritis and human...

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Yeo D, Hwang SJ, Song YS, et al. Humulene Inhibits Acute Gastric Mucosal Injury by Enhancing Mucosal Integrity. Antioxidants (Basel). 2021;10(5)doi: 10.3390/antiox10050761.
Yeo, D., Hwang, S. J., Song, Y. S., & Lee, H. J. (2021). Humulene Inhibits Acute Gastric Mucosal Injury by Enhancing Mucosal Integrity. Antioxidants (Basel, Switzerland), 10(5), . https://doi.org/10.3390/antiox10050761
Yeo, Dahee, et al. "Humulene Inhibits Acute Gastric Mucosal Injury by Enhancing Mucosal Integrity." Antioxidants (Basel, Switzerland) vol. 10,5 (2021). doi: https://doi.org/10.3390/antiox10050761
Yeo D, Hwang SJ, Song YS, Lee HJ. Humulene Inhibits Acute Gastric Mucosal Injury by Enhancing Mucosal Integrity. Antioxidants (Basel). 2021 May 11;10(5). doi: 10.3390/antiox10050761. PMID: 34064830; PMCID: PMC8150829.
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Immunoexpression of Trefoil Factor 1 in Non-Neoplastic and Neoplastic Canine Gastric Tissues.

Animals : an open access journal from MDPI

Flores AR, Castro M, Rêma A, Mesquita JR, Taulescu M, Gärtner F, Seixas F, Amorim I.
PMID: 34679875
Animals (Basel). 2021 Sep 29;11(10). doi: 10.3390/ani11102855.

TFF1 expression is markedly reduced in human GCs, suggesting that TFF1 is a tumor suppressor for human gastric cancer. The present study evaluated the expression and distribution pattern of TFF1 in paraffin-embedded canine gastric tissue samples, including normal mucosa...

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Flores AR, Castro M, Rêma A, et al. Immunoexpression of Trefoil Factor 1 in Non-Neoplastic and Neoplastic Canine Gastric Tissues. Animals (Basel). 2021;11(10)doi: 10.3390/ani11102855.
Flores, A. R., Castro, M., Rêma, A., Mesquita, J. R., Taulescu, M., Gärtner, F., Seixas, F., & Amorim, I. (2021). Immunoexpression of Trefoil Factor 1 in Non-Neoplastic and Neoplastic Canine Gastric Tissues. Animals : an open access journal from MDPI, 11(10), . https://doi.org/10.3390/ani11102855
Flores, Ana R, et al. "Immunoexpression of Trefoil Factor 1 in Non-Neoplastic and Neoplastic Canine Gastric Tissues." Animals : an open access journal from MDPI vol. 11,10 (2021). doi: https://doi.org/10.3390/ani11102855
Flores AR, Castro M, Rêma A, Mesquita JR, Taulescu M, Gärtner F, Seixas F, Amorim I. Immunoexpression of Trefoil Factor 1 in Non-Neoplastic and Neoplastic Canine Gastric Tissues. Animals (Basel). 2021 Sep 29;11(10). doi: 10.3390/ani11102855. PMID: 34679875; PMCID: PMC8532865.
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Bile proteomics for differentiation of malignant from benign biliary strictures: a pilot study.

Gastroenterology report

Navaneethan U, Lourdusamy V, Gk Venkatesh P, Willard B, Sanaka MR, Parsi MA.
PMID: 25304323
Gastroenterol Rep (Oxf). 2015 May;3(2):136-43. doi: 10.1093/gastro/gou066. Epub 2014 Oct 09.

BACKGROUND: Determining the etiology of biliary strictures is challenging, and the sensitivities of the current tests to diagnose them are low. Protein biomarkers in bile, in combination with other tests, may improve sensitivity in diagnosing biliary strictures.OBJECTIVE: To analyse...

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Navaneethan U, Lourdusamy V, Gk Venkatesh P, et al. Bile proteomics for differentiation of malignant from benign biliary strictures: a pilot study. Gastroenterol Rep (Oxf). 2014;3(2):136-43doi: 10.1093/gastro/gou066.
Navaneethan, U., Lourdusamy, V., Gk Venkatesh, P., Willard, B., Sanaka, M. R., & Parsi, M. A. (2015). Bile proteomics for differentiation of malignant from benign biliary strictures: a pilot study. Gastroenterology report, 3(2), 136-43. https://doi.org/10.1093/gastro/gou066
Navaneethan, Udayakumar, et al. "Bile proteomics for differentiation of malignant from benign biliary strictures: a pilot study." Gastroenterology report vol. 3,2 (2015): 136-43. doi: https://doi.org/10.1093/gastro/gou066
Navaneethan U, Lourdusamy V, Gk Venkatesh P, Willard B, Sanaka MR, Parsi MA. Bile proteomics for differentiation of malignant from benign biliary strictures: a pilot study. Gastroenterol Rep (Oxf). 2015 May;3(2):136-43. doi: 10.1093/gastro/gou066. Epub 2014 Oct 09. PMID: 25304323; PMCID: PMC4423458.
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Trefoil factor 1 and gastrokine 2 inhibit .

Oncology letters

Liu W, Li J, Zhang D, Chen B, Wang X, Zhang X, Xue L.
PMID: 33133254
Oncol Lett. 2020 Dec;20(6):318. doi: 10.3892/ol.2020.12181. Epub 2020 Oct 01.

No abstract available.

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Liu W, Li J, Zhang D, et al. Trefoil factor 1 and gastrokine 2 inhibit . Oncol Lett. 2020;20(6):318doi: 10.3892/ol.2020.12181.
Liu, W., Li, J., Zhang, D., Chen, B., Wang, X., Zhang, X., & Xue, L. (2020). Trefoil factor 1 and gastrokine 2 inhibit . Oncology letters, 20(6), 318. https://doi.org/10.3892/ol.2020.12181
Liu, Wenjing, et al. "Trefoil factor 1 and gastrokine 2 inhibit ." Oncology letters vol. 20,6 (2020): 318. doi: https://doi.org/10.3892/ol.2020.12181
Liu W, Li J, Zhang D, Chen B, Wang X, Zhang X, Xue L. Trefoil factor 1 and gastrokine 2 inhibit . Oncol Lett. 2020 Dec;20(6):318. doi: 10.3892/ol.2020.12181. Epub 2020 Oct 01. PMID: 33133254; PMCID: PMC7590425.
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Correction: Chemical synthesis of human trefoil factor 1 (TFF1) and its homodimer provides novel insights into their mechanisms of action.

Chemical communications (Cambridge, England)

Braga Emidio N, Baik H, Lee D, Stürmer R, Heuer J, Elliott AG, Blaskovich MAT, Haupenthal K, Tegtmeyer N, Hoffmann W, Schroeder CI, Muttenthaler M.
PMID: 32555797
Chem Commun (Camb). 2020 Jun 25;56(51):7049. doi: 10.1039/d0cc90250k.

Correction for 'Chemical synthesis of human trefoil factor 1 (TFF1) and its homodimer provides novel insights into their mechanisms of action' by Nayara Braga Emidio et al., Chem. Commun., 2020, DOI: 10.1039/D0CC02321C.

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Braga Emidio N, Baik H, Lee D, et al. Correction: Chemical synthesis of human trefoil factor 1 (TFF1) and its homodimer provides novel insights into their mechanisms of action. Chem Commun (Camb). 2020;56(51):7049doi: 10.1039/d0cc90250k.
Braga Emidio, N., Baik, H., Lee, D., Stürmer, R., Heuer, J., Elliott, A. G., Blaskovich, M. A. T., Haupenthal, K., Tegtmeyer, N., Hoffmann, W., Schroeder, C. I., & Muttenthaler, M. (2020). Correction: Chemical synthesis of human trefoil factor 1 (TFF1) and its homodimer provides novel insights into their mechanisms of action. Chemical communications (Cambridge, England), 56(51), 7049. https://doi.org/10.1039/d0cc90250k
Braga Emidio, Nayara, et al. "Correction: Chemical synthesis of human trefoil factor 1 (TFF1) and its homodimer provides novel insights into their mechanisms of action." Chemical communications (Cambridge, England) vol. 56,51 (2020): 7049. doi: https://doi.org/10.1039/d0cc90250k
Braga Emidio N, Baik H, Lee D, Stürmer R, Heuer J, Elliott AG, Blaskovich MAT, Haupenthal K, Tegtmeyer N, Hoffmann W, Schroeder CI, Muttenthaler M. Correction: Chemical synthesis of human trefoil factor 1 (TFF1) and its homodimer provides novel insights into their mechanisms of action. Chem Commun (Camb). 2020 Jun 25;56(51):7049. doi: 10.1039/d0cc90250k. PMID: 32555797.
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Role of NELF-B in supporting epithelial-mesenchymal transition and cell proliferation during hepatocellular carcinoma progression.

Oncology letters

Ghouraba MH, Masad RJ, Mpingirika EZ, Abdelraheem OM, Zeghlache R, Alserw AM, Amleh A.
PMID: 34539865
Oncol Lett. 2021 Nov;22(5):761. doi: 10.3892/ol.2021.13022. Epub 2021 Sep 06.

Negative elongation factor-B (NELF-B), also known as cofactor of BRCA1 (COBRA1), is one of the four subunits of the NELF complex. It interacts with BRCA1, in addition to other transcription complexes in various tissues. The NELF complex represses the...

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Ghouraba MH, Masad RJ, Mpingirika EZ, et al. Role of NELF-B in supporting epithelial-mesenchymal transition and cell proliferation during hepatocellular carcinoma progression. Oncol Lett. 2021;22(5):761doi: 10.3892/ol.2021.13022.
Ghouraba, M. H., Masad, R. J., Mpingirika, E. Z., Abdelraheem, O. M., Zeghlache, R., Alserw, A. M., & Amleh, A. (2021). Role of NELF-B in supporting epithelial-mesenchymal transition and cell proliferation during hepatocellular carcinoma progression. Oncology letters, 22(5), 761. https://doi.org/10.3892/ol.2021.13022
Ghouraba, Mennatallah Hani, et al. "Role of NELF-B in supporting epithelial-mesenchymal transition and cell proliferation during hepatocellular carcinoma progression." Oncology letters vol. 22,5 (2021): 761. doi: https://doi.org/10.3892/ol.2021.13022
Ghouraba MH, Masad RJ, Mpingirika EZ, Abdelraheem OM, Zeghlache R, Alserw AM, Amleh A. Role of NELF-B in supporting epithelial-mesenchymal transition and cell proliferation during hepatocellular carcinoma progression. Oncol Lett. 2021 Nov;22(5):761. doi: 10.3892/ol.2021.13022. Epub 2021 Sep 06. PMID: 34539865; PMCID: PMC8436359.
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Long non-coding RNA FOXD3 antisense RNA 1 augments anti-estrogen resistance in breast cancer cells through the microRNA-363/ trefoil factor 1/ phosphatidylinositol 3-kinase/protein kinase B axis.

Bioengineered

Ren L, Zhou H, Lei L, Zhang Y, Cai H, Wang X.
PMID: 34424807
Bioengineered. 2021 Dec;12(1):5266-5278. doi: 10.1080/21655979.2021.1962694.

Long non-coding RNA (lncRNA) FOXD3 antisense RNA 1 (FOXD3-AS1) has been reported to participate in multiple processes that contribute toward the development of cancer. The present study aimed to explore the effect of lncRNA FOXD3-AS1 on anti-estrogen resistance in...

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Ren L, Zhou H, Lei L, et al. Long non-coding RNA FOXD3 antisense RNA 1 augments anti-estrogen resistance in breast cancer cells through the microRNA-363/ trefoil factor 1/ phosphatidylinositol 3-kinase/protein kinase B axis. Bioengineered. 2021;12(1):5266-5278doi: 10.1080/21655979.2021.1962694.
Ren, L., Zhou, H., Lei, L., Zhang, Y., Cai, H., & Wang, X. (2021). Long non-coding RNA FOXD3 antisense RNA 1 augments anti-estrogen resistance in breast cancer cells through the microRNA-363/ trefoil factor 1/ phosphatidylinositol 3-kinase/protein kinase B axis. Bioengineered, 12(1), 5266-5278. https://doi.org/10.1080/21655979.2021.1962694
Ren, Lili, et al. "Long non-coding RNA FOXD3 antisense RNA 1 augments anti-estrogen resistance in breast cancer cells through the microRNA-363/ trefoil factor 1/ phosphatidylinositol 3-kinase/protein kinase B axis." Bioengineered vol. 12,1 (2021): 5266-5278. doi: https://doi.org/10.1080/21655979.2021.1962694
Ren L, Zhou H, Lei L, Zhang Y, Cai H, Wang X. Long non-coding RNA FOXD3 antisense RNA 1 augments anti-estrogen resistance in breast cancer cells through the microRNA-363/ trefoil factor 1/ phosphatidylinositol 3-kinase/protein kinase B axis. Bioengineered. 2021 Dec;12(1):5266-5278. doi: 10.1080/21655979.2021.1962694. PMID: 34424807.
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