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A seq2seq model to forecast the COVID-19 cases, deaths and reproductive R numbers in US counties.

Research square

Zhang-James Y, Hess J, Salkin A, Wang D, Chen S, Winkelstein P, Morley CP, Faraone SV.
PMID: 34013251
Res Sq. 2021 Apr 26; doi: 10.21203/rs.3.rs-456641/v1.

The global pandemic of coronavirus disease 2019 (COVID-19) has killed almost two million people worldwide and over 400 thousand in the United States (US). As the pandemic evolves, informed policy-making and strategic resource allocation relies on accurate forecasts. To...

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Zhang-James Y, Hess J, Salkin A, et al. A seq2seq model to forecast the COVID-19 cases, deaths and reproductive R numbers in US counties. Res Sq. 2021;doi: 10.21203/rs.3.rs-456641/v1.
Zhang-James, Y., Hess, J., Salkin, A., Wang, D., Chen, S., Winkelstein, P., Morley, C. P., & Faraone, S. V. (2021). A seq2seq model to forecast the COVID-19 cases, deaths and reproductive R numbers in US counties. Research square, . https://doi.org/10.21203/rs.3.rs-456641/v1
Zhang-James, Yanli, et al. "A seq2seq model to forecast the COVID-19 cases, deaths and reproductive R numbers in US counties." Research square vol. (2021). doi: https://doi.org/10.21203/rs.3.rs-456641/v1
Zhang-James Y, Hess J, Salkin A, Wang D, Chen S, Winkelstein P, Morley CP, Faraone SV. A seq2seq model to forecast the COVID-19 cases, deaths and reproductive R numbers in US counties. Res Sq. 2021 Apr 26; doi: 10.21203/rs.3.rs-456641/v1. PMID: 34013251; PMCID: PMC8132245.
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Smaller total brain volume but not subcortical structure volume related to common genetic risk for ADHD.

Psychological medicine

Mooney MA, Bhatt P, Hermosillo RJM, Ryabinin P, Nikolas M, Faraone SV, Fair DA, Wilmot B, Nigg JT.
PMID: 31973781
Psychol Med. 2020 Jan 24;1-10. doi: 10.1017/S0033291719004148. Epub 2020 Jan 24.

BACKGROUND: Mechanistic endophenotypes can inform process models of psychopathology and aid interpretation of genetic risk factors. Smaller total brain and subcortical volumes are associated with attention-deficit hyperactivity disorder (ADHD) and provide clues to its development. This study evaluates whether...

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Mooney MA, Bhatt P, Hermosillo RJM, et al. Smaller total brain volume but not subcortical structure volume related to common genetic risk for ADHD. Psychol Med. 2020;1-10doi: 10.1017/S0033291719004148.
Mooney, M. A., Bhatt, P., Hermosillo, R. J. M., Ryabinin, P., Nikolas, M., Faraone, S. V., Fair, D. A., Wilmot, B., & Nigg, J. T. (2020). Smaller total brain volume but not subcortical structure volume related to common genetic risk for ADHD. Psychological medicine, 1-10. https://doi.org/10.1017/S0033291719004148
Mooney, Michael A, et al. "Smaller total brain volume but not subcortical structure volume related to common genetic risk for ADHD." Psychological medicine vol. (2020): 1-10. doi: https://doi.org/10.1017/S0033291719004148
Mooney MA, Bhatt P, Hermosillo RJM, Ryabinin P, Nikolas M, Faraone SV, Fair DA, Wilmot B, Nigg JT. Smaller total brain volume but not subcortical structure volume related to common genetic risk for ADHD. Psychol Med. 2020 Jan 24;1-10. doi: 10.1017/S0033291719004148. Epub 2020 Jan 24. PMID: 31973781; PMCID: PMC7461955.
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Genetic Overlap Between Alzheimer's Disease and Bipolar Disorder Implicates the MARK2 and VAC14 Genes.

Frontiers in neuroscience

Drange OK, Smeland OB, Shadrin AA, Finseth PI, Witoelar A, Frei O, Wang Y, Hassani S, Djurovic S, Dale AM, Andreassen OA.
PMID: 30930738
Front Neurosci. 2019 Mar 13;13:220. doi: 10.3389/fnins.2019.00220. eCollection 2019.

No abstract available.

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Drange OK, Smeland OB, Shadrin AA, et al. Genetic Overlap Between Alzheimer's Disease and Bipolar Disorder Implicates the MARK2 and VAC14 Genes. Front Neurosci. 2019;13:220doi: 10.3389/fnins.2019.00220.
Drange, O. K., Smeland, O. B., Shadrin, A. A., Finseth, P. I., Witoelar, A., Frei, O., Wang, Y., Hassani, S., Djurovic, S., Dale, A. M., & Andreassen, O. A. (2019). Genetic Overlap Between Alzheimer's Disease and Bipolar Disorder Implicates the MARK2 and VAC14 Genes. Frontiers in neuroscience, 13220. https://doi.org/10.3389/fnins.2019.00220
Drange, Ole Kristian, et al. "Genetic Overlap Between Alzheimer's Disease and Bipolar Disorder Implicates the MARK2 and VAC14 Genes." Frontiers in neuroscience vol. 13 (2019): 220. doi: https://doi.org/10.3389/fnins.2019.00220
Drange OK, Smeland OB, Shadrin AA, Finseth PI, Witoelar A, Frei O, Wang Y, Hassani S, Djurovic S, Dale AM, Andreassen OA. Genetic Overlap Between Alzheimer's Disease and Bipolar Disorder Implicates the MARK2 and VAC14 Genes. Front Neurosci. 2019 Mar 13;13:220. doi: 10.3389/fnins.2019.00220. eCollection 2019. PMID: 30930738; PMCID: PMC6425305.
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Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders.

Cell

[No authors listed]
PMID: 31835028
Cell. 2019 Dec 12;179(7):1469-1482.e11. doi: 10.1016/j.cell.2019.11.020.

Genetic influences on psychiatric disorders transcend diagnostic boundaries, suggesting substantial pleiotropy of contributing loci. However, the nature and mechanisms of these pleiotropic effects remain unclear. We performed analyses of 232,964 cases and 494,162 controls from genome-wide studies of anorexia...

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Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell. 2019;179(7):1469-1482.e11doi: 10.1016/j.cell.2019.11.020.
(2019). Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell, 179(7), 1469-1482.e11. https://doi.org/10.1016/j.cell.2019.11.020
"Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders." Cell vol. 179,7 (2019): 1469-1482.e11. doi: https://doi.org/10.1016/j.cell.2019.11.020
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell. 2019 Dec 12;179(7):1469-1482.e11. doi: 10.1016/j.cell.2019.11.020. PMID: 31835028; PMCID: PMC7077032.
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Applying polygenic risk scoring for psychiatric disorders to a large family with bipolar disorder and major depressive disorder.

Communications biology

de Jong S, Diniz MJA, Saloma A, Gadelha A, Santoro ML, Ota VK, Noto C, Curtis C, Newhouse SJ, Patel H, Hall LS, O Reilly PF, Belangero SI, Bressan RA, Breen G.
PMID: 30320231
Commun Biol. 2018 Oct 08;1:163. doi: 10.1038/s42003-018-0155-y. eCollection 2018.

Psychiatric disorders are thought to have a complex genetic pathology consisting of interplay of common and rare variation. Traditionally, pedigrees are used to shed light on the latter only, while here we discuss the application of polygenic risk scores...

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de Jong S, Diniz MJA, Saloma A, et al. Applying polygenic risk scoring for psychiatric disorders to a large family with bipolar disorder and major depressive disorder. Commun Biol. 2018;1:163doi: 10.1038/s42003-018-0155-y.
de Jong, S., Diniz, M. J. A., Saloma, A., Gadelha, A., Santoro, M. L., Ota, V. K., Noto, C., Curtis, C., Newhouse, S. J., Patel, H., Hall, L. S., O Reilly, P. F., Belangero, S. I., Bressan, R. A., & Breen, G. (2018). Applying polygenic risk scoring for psychiatric disorders to a large family with bipolar disorder and major depressive disorder. Communications biology, 1163. https://doi.org/10.1038/s42003-018-0155-y
de Jong, Simone, et al. "Applying polygenic risk scoring for psychiatric disorders to a large family with bipolar disorder and major depressive disorder." Communications biology vol. 1 (2018): 163. doi: https://doi.org/10.1038/s42003-018-0155-y
de Jong S, Diniz MJA, Saloma A, Gadelha A, Santoro ML, Ota VK, Noto C, Curtis C, Newhouse SJ, Patel H, Hall LS, O Reilly PF, Belangero SI, Bressan RA, Breen G. Applying polygenic risk scoring for psychiatric disorders to a large family with bipolar disorder and major depressive disorder. Commun Biol. 2018 Oct 08;1:163. doi: 10.1038/s42003-018-0155-y. eCollection 2018. PMID: 30320231; PMCID: PMC6175827.
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Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders.

Cell

[No authors listed]
PMID: 31835028
Cell. 2019 Dec 12;179(7):1469-1482.e11. doi: 10.1016/j.cell.2019.11.020.

Genetic influences on psychiatric disorders transcend diagnostic boundaries, suggesting substantial pleiotropy of contributing loci. However, the nature and mechanisms of these pleiotropic effects remain unclear. We performed analyses of 232,964 cases and 494,162 controls from genome-wide studies of anorexia...

Cite
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell. 2019;179(7):1469-1482.e11doi: 10.1016/j.cell.2019.11.020.
(2019). Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell, 179(7), 1469-1482.e11. https://doi.org/10.1016/j.cell.2019.11.020
"Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders." Cell vol. 179,7 (2019): 1469-1482.e11. doi: https://doi.org/10.1016/j.cell.2019.11.020
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell. 2019 Dec 12;179(7):1469-1482.e11. doi: 10.1016/j.cell.2019.11.020. PMID: 31835028; PMCID: PMC7077032.
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Further evidence of high level of persistence of pediatric bipolar-I disorder from childhood onto young adulthood: a five-year follow up.

Scandinavian journal of child and adolescent psychiatry and psychology

Wozniak J, Wolenski R, Fitzgerald M, Faraone SV, Joshi G, Uchida M, Biederman J.
PMID: 33520750
Scand J Child Adolesc Psychiatr Psychol. 2018 Jul 10;6(1):40-51. doi: 10.21307/sjcapp-2018-005. eCollection 2018.

BACKGROUND: Pediatric bipolar (BP)-I disorder affects a sizeable minority of children and is associated with high levels of morbidity. Relatively few studies have assessed the persistence of the disorder over time.OBJECTIVE: The main aim of this study was to...

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Wozniak J, Wolenski R, Fitzgerald M, et al. Further evidence of high level of persistence of pediatric bipolar-I disorder from childhood onto young adulthood: a five-year follow up. Scand J Child Adolesc Psychiatr Psychol. 2018;6(1):40-51doi: 10.21307/sjcapp-2018-005.
Wozniak, J., Wolenski, R., Fitzgerald, M., Faraone, S. V., Joshi, G., Uchida, M., & Biederman, J. (2018). Further evidence of high level of persistence of pediatric bipolar-I disorder from childhood onto young adulthood: a five-year follow up. Scandinavian journal of child and adolescent psychiatry and psychology, 6(1), 40-51. https://doi.org/10.21307/sjcapp-2018-005
Wozniak, Janet, et al. "Further evidence of high level of persistence of pediatric bipolar-I disorder from childhood onto young adulthood: a five-year follow up." Scandinavian journal of child and adolescent psychiatry and psychology vol. 6,1 (2018): 40-51. doi: https://doi.org/10.21307/sjcapp-2018-005
Wozniak J, Wolenski R, Fitzgerald M, Faraone SV, Joshi G, Uchida M, Biederman J. Further evidence of high level of persistence of pediatric bipolar-I disorder from childhood onto young adulthood: a five-year follow up. Scand J Child Adolesc Psychiatr Psychol. 2018 Jul 10;6(1):40-51. doi: 10.21307/sjcapp-2018-005. eCollection 2018. PMID: 33520750; PMCID: PMC7750699.
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Showing 1 to 7 of 7 entries