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Showing 1 to 9 of 9 entries
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Regulation and function of the peg3 imprinted domain.

Genomics & informatics

He H, Kim J.
PMID: 25317109
Genomics Inform. 2014 Sep;12(3):105-13. doi: 10.5808/GI.2014.12.3.105. Epub 2014 Sep 30.

A subset of mammalian genes differ functionally between two alleles due to genomic imprinting, and seven such genes (Peg3, Usp29, APeg3, Zfp264, Zim1, Zim2, Zim3) are localized within the 500-kb genomic interval of the human and mouse genomes, constituting...

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He H, Kim J. Regulation and function of the peg3 imprinted domain. Genomics Inform. 2014;12(3):105-13doi: 10.5808/GI.2014.12.3.105.
He, H., & Kim, J. (2014). Regulation and function of the peg3 imprinted domain. Genomics & informatics, 12(3), 105-13. https://doi.org/10.5808/GI.2014.12.3.105
He, Hongzhi, and Kim, Joomyeong. "Regulation and function of the peg3 imprinted domain." Genomics & informatics vol. 12,3 (2014): 105-13. doi: https://doi.org/10.5808/GI.2014.12.3.105
He H, Kim J. Regulation and function of the peg3 imprinted domain. Genomics Inform. 2014 Sep;12(3):105-13. doi: 10.5808/GI.2014.12.3.105. Epub 2014 Sep 30. PMID: 25317109; PMCID: PMC4196374.
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LINC00667/miR-449b-5p/YY1 axis promotes cell proliferation and migration in colorectal cancer.

Cancer cell international

Yu J, Wang F, Zhang J, Li J, Chen X, Han G.
PMID: 32694944
Cancer Cell Int. 2020 Jul 17;20:322. doi: 10.1186/s12935-020-01377-7. eCollection 2020.

BACKGROUND: Long non-coding RNAs (lncRNAs) have been defined as vital regulators in the progression of human cancers, including colorectal cancer (CRC). Long intergenic non-protein coding RNA 667 (LINC00667) is a tumor promoter in several cancer types, while its role...

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Yu J, Wang F, Zhang J, et al. LINC00667/miR-449b-5p/YY1 axis promotes cell proliferation and migration in colorectal cancer. Cancer Cell Int. 2020;20:322doi: 10.1186/s12935-020-01377-7.
Yu, J., Wang, F., Zhang, J., Li, J., Chen, X., & Han, G. (2020). LINC00667/miR-449b-5p/YY1 axis promotes cell proliferation and migration in colorectal cancer. Cancer cell international, 20322. https://doi.org/10.1186/s12935-020-01377-7
Yu, Juan, et al. "LINC00667/miR-449b-5p/YY1 axis promotes cell proliferation and migration in colorectal cancer." Cancer cell international vol. 20 (2020): 322. doi: https://doi.org/10.1186/s12935-020-01377-7
Yu J, Wang F, Zhang J, Li J, Chen X, Han G. LINC00667/miR-449b-5p/YY1 axis promotes cell proliferation and migration in colorectal cancer. Cancer Cell Int. 2020 Jul 17;20:322. doi: 10.1186/s12935-020-01377-7. eCollection 2020. PMID: 32694944; PMCID: PMC7368754.
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A p53-stabilizing agent, CP-31398, induces p21 expression with increased G2/M phase through the YY1 transcription factor in esophageal carcinoma defective of the p53 pathway.

American journal of cancer research

Zhong B, Shingyoji M, Hanazono M, Nguyễn TTT, Morinaga T, Tada Y, Hiroshima K, Shimada H, Tagawa M.
PMID: 30755813
Am J Cancer Res. 2019 Jan 01;9(1):79-93. eCollection 2019.

Restoration of p53 functions is one of the therapeutic strategies for esophageal carcinoma which is often defective of the p53 pathway. We examined effects of CP-31398 which potentially increased expression of wild-type p53 or converted mutated p53 to the...

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Zhong B, Shingyoji M, Hanazono M, et al. A p53-stabilizing agent, CP-31398, induces p21 expression with increased G2/M phase through the YY1 transcription factor in esophageal carcinoma defective of the p53 pathway. Am J Cancer Res. 2019;9(1):79-93
Zhong, B., Shingyoji, M., Hanazono, M., Nguyễn, T. T. T., Morinaga, T., Tada, Y., Hiroshima, K., Shimada, H., & Tagawa, M. (2019). A p53-stabilizing agent, CP-31398, induces p21 expression with increased G2/M phase through the YY1 transcription factor in esophageal carcinoma defective of the p53 pathway. American journal of cancer research, 9(1), 79-93.
Zhong, Boya, et al. "A p53-stabilizing agent, CP-31398, induces p21 expression with increased G2/M phase through the YY1 transcription factor in esophageal carcinoma defective of the p53 pathway." American journal of cancer research vol. 9,1 (2019): 79-93.
Zhong B, Shingyoji M, Hanazono M, Nguyễn TTT, Morinaga T, Tada Y, Hiroshima K, Shimada H, Tagawa M. A p53-stabilizing agent, CP-31398, induces p21 expression with increased G2/M phase through the YY1 transcription factor in esophageal carcinoma defective of the p53 pathway. Am J Cancer Res. 2019 Jan 01;9(1):79-93. eCollection 2019. PMID: 30755813; PMCID: PMC6356922.
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YY1-mediated up-regulation of lncRNA LINC00466 facilitates glioma progression via miR-508/CHEK1.

The journal of gene medicine

Li F, Shen ZZ, Xiao CM, Sha QK.
PMID: 33037684
J Gene Med. 2021 Jan;23(1):e3287. doi: 10.1002/jgm.3287. Epub 2020 Nov 24.

BACKGROUND: The abnormal expression of lncRNA LINC00466 (LINC00466) has been demonstrated in several tumor types. However, the expression pattern and functions of LINC00466 in glioma remain uninvestigated.METHODS: A reverse transcriptase-polymerase chain reaction (RT-PCR) was utilized to analyze LINC00466 in...

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Li F, Shen ZZ, Xiao CM, et al. YY1-mediated up-regulation of lncRNA LINC00466 facilitates glioma progression via miR-508/CHEK1. J Gene Med. 2020;23(1):e3287doi: 10.1002/jgm.3287.
Li, F., Shen, Z. Z., Xiao, C. M., & Sha, Q. K. (2021). YY1-mediated up-regulation of lncRNA LINC00466 facilitates glioma progression via miR-508/CHEK1. The journal of gene medicine, 23(1), e3287. https://doi.org/10.1002/jgm.3287
Li, Fei, et al. "YY1-mediated up-regulation of lncRNA LINC00466 facilitates glioma progression via miR-508/CHEK1." The journal of gene medicine vol. 23,1 (2021): e3287. doi: https://doi.org/10.1002/jgm.3287
Li F, Shen ZZ, Xiao CM, Sha QK. YY1-mediated up-regulation of lncRNA LINC00466 facilitates glioma progression via miR-508/CHEK1. J Gene Med. 2021 Jan;23(1):e3287. doi: 10.1002/jgm.3287. Epub 2020 Nov 24. PMID: 33037684.
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A Positive Feedback Loop of lncRNA MIR31HG-miR-361-3p -YY1 Accelerates Colorectal Cancer Progression Through Modulating Proliferation, Angiogenesis, and Glycolysis.

Frontiers in oncology

Guo T, Liu D, Peng S, Wang M, Li Y.
PMID: 34485123
Front Oncol. 2021 Aug 17;11:684984. doi: 10.3389/fonc.2021.684984. eCollection 2021.

BACKGROUND: Colorectal cancer (CRC) is a common malignant tumor with high metastatic and recurrent rates. This study probes the effect and mechanism of long non-coding RNA MIR31HG on the progression of CRC cells.MATERIALS AND METHODS: Quantitative real-time PCR (qRT-PCR)...

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Guo T, Liu D, Peng S, et al. A Positive Feedback Loop of lncRNA MIR31HG-miR-361-3p -YY1 Accelerates Colorectal Cancer Progression Through Modulating Proliferation, Angiogenesis, and Glycolysis. Front Oncol. 2021;11:684984doi: 10.3389/fonc.2021.684984.
Guo, T., Liu, D., Peng, S., Wang, M., & Li, Y. (2021). A Positive Feedback Loop of lncRNA MIR31HG-miR-361-3p -YY1 Accelerates Colorectal Cancer Progression Through Modulating Proliferation, Angiogenesis, and Glycolysis. Frontiers in oncology, 11684984. https://doi.org/10.3389/fonc.2021.684984
Guo, Tao, et al. "A Positive Feedback Loop of lncRNA MIR31HG-miR-361-3p -YY1 Accelerates Colorectal Cancer Progression Through Modulating Proliferation, Angiogenesis, and Glycolysis." Frontiers in oncology vol. 11 (2021): 684984. doi: https://doi.org/10.3389/fonc.2021.684984
Guo T, Liu D, Peng S, Wang M, Li Y. A Positive Feedback Loop of lncRNA MIR31HG-miR-361-3p -YY1 Accelerates Colorectal Cancer Progression Through Modulating Proliferation, Angiogenesis, and Glycolysis. Front Oncol. 2021 Aug 17;11:684984. doi: 10.3389/fonc.2021.684984. eCollection 2021. PMID: 34485123; PMCID: PMC8416113.
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YY1-inudced activation of lncRNA DUXAP8 promotes proliferation and suppresses apoptosis of triple negative breast cancer cells through upregulating SAPCD2.

Cancer biology & therapy

Yang Z, Ding H, Pan Z, Li H, Ding J, Chen Q.
PMID: 33683171
Cancer Biol Ther. 2021 Mar 04;22(3):216-224. doi: 10.1080/15384047.2021.1881201. Epub 2021 Mar 08.

Double homeobox A pseudogene 8 (DUXAP8) belongs to long non-coding RNAs (lncRNAs), which has been proven to promote the biological processes of multiple human cancers. Triple-negative breast cancer (TNBC) is the leading cause of cancer-related death in women worldwide....

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Yang Z, Ding H, Pan Z, et al. YY1-inudced activation of lncRNA DUXAP8 promotes proliferation and suppresses apoptosis of triple negative breast cancer cells through upregulating SAPCD2. Cancer Biol Ther. 2021;22(3):216-224doi: 10.1080/15384047.2021.1881201.
Yang, Z., Ding, H., Pan, Z., Li, H., Ding, J., & Chen, Q. (2021). YY1-inudced activation of lncRNA DUXAP8 promotes proliferation and suppresses apoptosis of triple negative breast cancer cells through upregulating SAPCD2. Cancer biology & therapy, 22(3), 216-224. https://doi.org/10.1080/15384047.2021.1881201
Yang, Zhen, et al. "YY1-inudced activation of lncRNA DUXAP8 promotes proliferation and suppresses apoptosis of triple negative breast cancer cells through upregulating SAPCD2." Cancer biology & therapy vol. 22,3 (2021): 216-224. doi: https://doi.org/10.1080/15384047.2021.1881201
Yang Z, Ding H, Pan Z, Li H, Ding J, Chen Q. YY1-inudced activation of lncRNA DUXAP8 promotes proliferation and suppresses apoptosis of triple negative breast cancer cells through upregulating SAPCD2. Cancer Biol Ther. 2021 Mar 04;22(3):216-224. doi: 10.1080/15384047.2021.1881201. Epub 2021 Mar 08. PMID: 33683171; PMCID: PMC8043174.
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Screening of Transcription Factors Involved in Fetal Hemoglobin Regulation Using Phylogenetic Footprinting.

Evolutionary bioinformatics online

de Souza Carrocini GC, Venancio LP, Bonini-Domingos CR.
PMID: 26543346
Evol Bioinform Online. 2015 Oct 27;11:239-44. doi: 10.4137/EBO.S15364. eCollection 2015.

Fetal hemoglobin (Hb F) is an important genetic modulator of the beta-hemoglobinopathies. The regulation of Hb F levels is influenced by transcription factors. We used phylogenetic footprinting to screen transcription factors that have binding sites in HBG1 and HBG2...

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de Souza Carrocini GC, Venancio LP, Bonini-Domingos CR. Screening of Transcription Factors Involved in Fetal Hemoglobin Regulation Using Phylogenetic Footprinting. Evol Bioinform Online. 2015;11:239-44doi: 10.4137/EBO.S15364.
de Souza Carrocini, G. C., Venancio, L. P., & Bonini-Domingos, C. R. (2015). Screening of Transcription Factors Involved in Fetal Hemoglobin Regulation Using Phylogenetic Footprinting. Evolutionary bioinformatics online, 11239-44. https://doi.org/10.4137/EBO.S15364
de Souza Carrocini, Gisele Cristine, et al. "Screening of Transcription Factors Involved in Fetal Hemoglobin Regulation Using Phylogenetic Footprinting." Evolutionary bioinformatics online vol. 11 (2015): 239-44. doi: https://doi.org/10.4137/EBO.S15364
de Souza Carrocini GC, Venancio LP, Bonini-Domingos CR. Screening of Transcription Factors Involved in Fetal Hemoglobin Regulation Using Phylogenetic Footprinting. Evol Bioinform Online. 2015 Oct 27;11:239-44. doi: 10.4137/EBO.S15364. eCollection 2015. PMID: 26543346; PMCID: PMC4624090.
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Enhancer LncRNAs Influence Chromatin Interactions in Different Ways.

Frontiers in genetics

Hou Y, Zhang R, Sun X.
PMID: 31681405
Front Genet. 2019 Oct 16;10:936. doi: 10.3389/fgene.2019.00936. eCollection 2019.

More than 98% of the human genome does not encode proteins, and the vast majority of the noncoding regions have not been well studied. Some of these regions contain enhancers and functional non-coding RNAs. Previous research suggested that enhancer...

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Hou Y, Zhang R, Sun X. Enhancer LncRNAs Influence Chromatin Interactions in Different Ways. Front Genet. 2019;10:936doi: 10.3389/fgene.2019.00936.
Hou, Y., Zhang, R., & Sun, X. (2019). Enhancer LncRNAs Influence Chromatin Interactions in Different Ways. Frontiers in genetics, 10936. https://doi.org/10.3389/fgene.2019.00936
Hou, Yue, et al. "Enhancer LncRNAs Influence Chromatin Interactions in Different Ways." Frontiers in genetics vol. 10 (2019): 936. doi: https://doi.org/10.3389/fgene.2019.00936
Hou Y, Zhang R, Sun X. Enhancer LncRNAs Influence Chromatin Interactions in Different Ways. Front Genet. 2019 Oct 16;10:936. doi: 10.3389/fgene.2019.00936. eCollection 2019. PMID: 31681405; PMCID: PMC6807612.
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Puerarin inhibits apoptosis and inflammation in myocardial cells via PPARα expression in rats with chronic heart failure.

Experimental and therapeutic medicine

He L, Wang T, Chen BW, Lu FM, Xu J.
PMID: 31602208
Exp Ther Med. 2019 Nov;18(5):3347-3356. doi: 10.3892/etm.2019.7984. Epub 2019 Sep 06.

Chronic heart failure affects myocardial energy metabolism and cardiac function. Puerarin has been reported to improve cardiac function through regulation of energy metabolism in mice with myocardial infarction. The aim of the current study was to determine whether puerarin...

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He L, Wang T, Chen BW, et al. Puerarin inhibits apoptosis and inflammation in myocardial cells via PPARα expression in rats with chronic heart failure. Exp Ther Med. 2019;18(5):3347-3356doi: 10.3892/etm.2019.7984.
He, L., Wang, T., Chen, B. W., Lu, F. M., & Xu, J. (2019). Puerarin inhibits apoptosis and inflammation in myocardial cells via PPARα expression in rats with chronic heart failure. Experimental and therapeutic medicine, 18(5), 3347-3356. https://doi.org/10.3892/etm.2019.7984
He, Le, et al. "Puerarin inhibits apoptosis and inflammation in myocardial cells via PPARα expression in rats with chronic heart failure." Experimental and therapeutic medicine vol. 18,5 (2019): 3347-3356. doi: https://doi.org/10.3892/etm.2019.7984
He L, Wang T, Chen BW, Lu FM, Xu J. Puerarin inhibits apoptosis and inflammation in myocardial cells via PPARα expression in rats with chronic heart failure. Exp Ther Med. 2019 Nov;18(5):3347-3356. doi: 10.3892/etm.2019.7984. Epub 2019 Sep 06. PMID: 31602208; PMCID: PMC6777288.
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Showing 1 to 9 of 9 entries