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Active components from Radix Scrophulariae inhibits the ventricular remodeling induced by hypertension in rats.

SpringerPlus

Zhang CC, Gu WL, Wu XM, Li YM, Chen CX, Huang XY.
PMID: 27066371
Springerplus. 2016 Mar 22;5:358. doi: 10.1186/s40064-016-1985-z. eCollection 2016.

BACKGROUND: In the previous study, active extract of Radix Scrophularia (ACRS) demonstrated beneficial effects on ventricular remodeling induced by coronary artery ligation and lowered blood pressure in rats. And ACRS also exhibited the effect on lowering the blood pressure...

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Zhang CC, Gu WL, Wu XM, et al. Active components from Radix Scrophulariae inhibits the ventricular remodeling induced by hypertension in rats. Springerplus. 2016;5:358doi: 10.1186/s40064-016-1985-z.
Zhang, C. C., Gu, W. L., Wu, X. M., Li, Y. M., Chen, C. X., & Huang, X. Y. (2016). Active components from Radix Scrophulariae inhibits the ventricular remodeling induced by hypertension in rats. SpringerPlus, 5358. https://doi.org/10.1186/s40064-016-1985-z
Zhang, Chao Chao, et al. "Active components from Radix Scrophulariae inhibits the ventricular remodeling induced by hypertension in rats." SpringerPlus vol. 5 (2016): 358. doi: https://doi.org/10.1186/s40064-016-1985-z
Zhang CC, Gu WL, Wu XM, Li YM, Chen CX, Huang XY. Active components from Radix Scrophulariae inhibits the ventricular remodeling induced by hypertension in rats. Springerplus. 2016 Mar 22;5:358. doi: 10.1186/s40064-016-1985-z. eCollection 2016. PMID: 27066371; PMCID: PMC4801831.
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Protection of trigonelline on experimental diabetic peripheral neuropathy.

Evidence-based complementary and alternative medicine : eCAM

Zhou JY, Zhou SW.
PMID: 23304193
Evid Based Complement Alternat Med. 2012;2012:164219. doi: 10.1155/2012/164219. Epub 2012 Dec 06.

The mechanisms leading to diabetic peripheral neuropathy are complex and there is no effective drug to treat it. As an active component of several traditional Chinese medicines, trigonelline has beneficial effects on diabetes with hyperlipidemia. The protective effects and...

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Zhou JY, Zhou SW. Protection of trigonelline on experimental diabetic peripheral neuropathy. Evid Based Complement Alternat Med. 2012;2012:164219doi: 10.1155/2012/164219.
Zhou, J. Y., & Zhou, S. W. (2012). Protection of trigonelline on experimental diabetic peripheral neuropathy. Evidence-based complementary and alternative medicine : eCAM, 2012164219. https://doi.org/10.1155/2012/164219
Zhou, Ji-Yin, and Zhou, Shi-Wen. "Protection of trigonelline on experimental diabetic peripheral neuropathy." Evidence-based complementary and alternative medicine : eCAM vol. 2012 (2012): 164219. doi: https://doi.org/10.1155/2012/164219
Zhou JY, Zhou SW. Protection of trigonelline on experimental diabetic peripheral neuropathy. Evid Based Complement Alternat Med. 2012;2012:164219. doi: 10.1155/2012/164219. Epub 2012 Dec 06. PMID: 23304193; PMCID: PMC3526196.
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Species Comparison of the Role of p38 MAP Kinase in the Female Reproductive System.

Journal of toxicologic pathology

Radi ZA, Marusak RA, Morris DL.
PMID: 22271984
J Toxicol Pathol. 2009 Jun;22(2):109-24. doi: 10.1293/tox.22.109. Epub 2009 Jul 07.

The p38 mitogen-activated protein kinases (MAPKs) are members of discrete signal transduction pathways that have significant regulatory roles in a variety of biological processes, depending on the cell, tissue and organ type. p38 MAPKs are involved in inflammation, cell...

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Radi ZA, Marusak RA, Morris DL. Species Comparison of the Role of p38 MAP Kinase in the Female Reproductive System. J Toxicol Pathol. 2009;22(2):109-24doi: 10.1293/tox.22.109.
Radi, Z. A., Marusak, R. A., & Morris, D. L. (2009). Species Comparison of the Role of p38 MAP Kinase in the Female Reproductive System. Journal of toxicologic pathology, 22(2), 109-24. https://doi.org/10.1293/tox.22.109
Radi, Zaher A, et al. "Species Comparison of the Role of p38 MAP Kinase in the Female Reproductive System." Journal of toxicologic pathology vol. 22,2 (2009): 109-24. doi: https://doi.org/10.1293/tox.22.109
Radi ZA, Marusak RA, Morris DL. Species Comparison of the Role of p38 MAP Kinase in the Female Reproductive System. J Toxicol Pathol. 2009 Jun;22(2):109-24. doi: 10.1293/tox.22.109. Epub 2009 Jul 07. PMID: 22271984; PMCID: PMC3246056.
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Recombinant Human Thioredoxin-1 Protects Macrophages from Oxidized Low-Density Lipoprotein-Induced Foam Cell Formation and Cell Apoptosis.

Biomolecules & therapeutics

Zhang H, Liu Q, Lin JL, Wang Y, Zhang RX, Hou JB, Yu B.
PMID: 28554199
Biomol Ther (Seoul). 2018 Mar 01;26(2):121-129. doi: 10.4062/biomolther.2016.275.

Oxidized low-density lipoprotein (ox-LDL)-induced macrophage foam cell formation and apoptosis play critical roles in the pathogenesis of atherosclerosis. Thioredoxin-1 (Trx) is an antioxidant that potently protects various cells from oxidative stress-induced cell death. However, the protective effect of Trx...

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Zhang H, Liu Q, Lin JL, et al. Recombinant Human Thioredoxin-1 Protects Macrophages from Oxidized Low-Density Lipoprotein-Induced Foam Cell Formation and Cell Apoptosis. Biomol Ther (Seoul). 2018;26(2):121-129doi: 10.4062/biomolther.2016.275.
Zhang, H., Liu, Q., Lin, J. L., Wang, Y., Zhang, R. X., Hou, J. B., & Yu, B. (2018). Recombinant Human Thioredoxin-1 Protects Macrophages from Oxidized Low-Density Lipoprotein-Induced Foam Cell Formation and Cell Apoptosis. Biomolecules & therapeutics, 26(2), 121-129. https://doi.org/10.4062/biomolther.2016.275
Zhang, Hui, et al. "Recombinant Human Thioredoxin-1 Protects Macrophages from Oxidized Low-Density Lipoprotein-Induced Foam Cell Formation and Cell Apoptosis." Biomolecules & therapeutics vol. 26,2 (2018): 121-129. doi: https://doi.org/10.4062/biomolther.2016.275
Zhang H, Liu Q, Lin JL, Wang Y, Zhang RX, Hou JB, Yu B. Recombinant Human Thioredoxin-1 Protects Macrophages from Oxidized Low-Density Lipoprotein-Induced Foam Cell Formation and Cell Apoptosis. Biomol Ther (Seoul). 2018 Mar 01;26(2):121-129. doi: 10.4062/biomolther.2016.275. PMID: 28554199; PMCID: PMC5839490.
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MicroRNA-543 promotes ovariectomy-induced osteoporosis through inhibition of AKT/p38 MAPK signaling pathway by targeting YAF2.

Journal of cellular biochemistry

Li X, Ning L, Zhao X, Wan S.
PMID: 30506950
J Cell Biochem. 2018 Dec 02; doi: 10.1002/jcb.28143. Epub 2018 Dec 02.

The present study aimed to determine the roles of miRNA-543 in osteoporosis in rats induced by ovariectomy. The osteoporosis rat model was established by ovariectomy induction. MiRNA-543 expression in osteoblasts was measured by quantitative real-time polymerase chain reaction. The...

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Li X, Ning L, Zhao X, et al. MicroRNA-543 promotes ovariectomy-induced osteoporosis through inhibition of AKT/p38 MAPK signaling pathway by targeting YAF2. J Cell Biochem. 2018;doi: 10.1002/jcb.28143.
Li, X., Ning, L., Zhao, X., & Wan, S. (2018). MicroRNA-543 promotes ovariectomy-induced osteoporosis through inhibition of AKT/p38 MAPK signaling pathway by targeting YAF2. Journal of cellular biochemistry, . https://doi.org/10.1002/jcb.28143
Li, Xiang, et al. "MicroRNA-543 promotes ovariectomy-induced osteoporosis through inhibition of AKT/p38 MAPK signaling pathway by targeting YAF2." Journal of cellular biochemistry vol. (2018). doi: https://doi.org/10.1002/jcb.28143
Li X, Ning L, Zhao X, Wan S. MicroRNA-543 promotes ovariectomy-induced osteoporosis through inhibition of AKT/p38 MAPK signaling pathway by targeting YAF2. J Cell Biochem. 2018 Dec 02; doi: 10.1002/jcb.28143. Epub 2018 Dec 02. PMID: 30506950.
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Binding Immunoglobulin Protein (BIP) Inhibits TNF-α-Induced Osteoclast Differentiation and Systemic Bone Loss in an Erosive Arthritis Model.

ACR open rheumatology

Zaiss MM, Hall C, McGowan NWA, Babb R, Devlia V, Lucas S, Meghji S, Henderson B, Bozec A, Schett G, David JP, Panayi GS, Grigoriadis AE, Corrigall VM.
PMID: 31777818
ACR Open Rheumatol. 2019 Aug 03;1(6):382-393. doi: 10.1002/acr2.11060. eCollection 2019 Aug.

RESULTS: BiP-treated mice showed reduced inflammation and cartilage destruction, and histomorphometric analysis revealed a decrease in osteoclast number with protection from systemic bone loss. Abrogation of osteoclast function was also observed in an ex vivo murine calvarial model. BiP...

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Zaiss MM, Hall C, McGowan NWA, et al. Binding Immunoglobulin Protein (BIP) Inhibits TNF-α-Induced Osteoclast Differentiation and Systemic Bone Loss in an Erosive Arthritis Model. ACR Open Rheumatol. 2019;1(6):382-393doi: 10.1002/acr2.11060.
Zaiss, M. M., Hall, C., McGowan, N. W. A., Babb, R., Devlia, V., Lucas, S., Meghji, S., Henderson, B., Bozec, A., Schett, G., David, J. P., Panayi, G. S., Grigoriadis, A. E., & Corrigall, V. M. (2019). Binding Immunoglobulin Protein (BIP) Inhibits TNF-α-Induced Osteoclast Differentiation and Systemic Bone Loss in an Erosive Arthritis Model. ACR open rheumatology, 1(6), 382-393. https://doi.org/10.1002/acr2.11060
Zaiss, Mario M, et al. "Binding Immunoglobulin Protein (BIP) Inhibits TNF-α-Induced Osteoclast Differentiation and Systemic Bone Loss in an Erosive Arthritis Model." ACR open rheumatology vol. 1,6 (2019): 382-393. doi: https://doi.org/10.1002/acr2.11060
Zaiss MM, Hall C, McGowan NWA, Babb R, Devlia V, Lucas S, Meghji S, Henderson B, Bozec A, Schett G, David JP, Panayi GS, Grigoriadis AE, Corrigall VM. Binding Immunoglobulin Protein (BIP) Inhibits TNF-α-Induced Osteoclast Differentiation and Systemic Bone Loss in an Erosive Arthritis Model. ACR Open Rheumatol. 2019 Aug 03;1(6):382-393. doi: 10.1002/acr2.11060. eCollection 2019 Aug. PMID: 31777818; PMCID: PMC6857990.
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p38 Mitogen-Activated Protein Kinase Is Involved in Interleukin-6 Secretion from Human Ligamentum Flavum-Derived Cells Stimulated by Tumor Necrosis Factor-α.

Asian spine journal

Yagi K, Goto Y, Kato K, Suzuki N, Kondo A, Waseda Y, Mizutani J, Kawaguchi Y, Joyo Y, Waguri-Nagaya Y, Murakami H.
PMID: 33355843
Asian Spine J. 2021 Dec;15(6):713-720. doi: 10.31616/asj.2020.0425. Epub 2020 Dec 28.

STUDY DESIGN: Human ligamentum flavum-derived cells (HFCs) were obtained from surgical samples for a basic experimental study.PURPOSE: We sought to evaluate the inflammatory response of human ligamentum flavum cells to investigate hypertrophic changes occurring in the ligamentum flavum.OVERVIEW OF...

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Yagi K, Goto Y, Kato K, et al. p38 Mitogen-Activated Protein Kinase Is Involved in Interleukin-6 Secretion from Human Ligamentum Flavum-Derived Cells Stimulated by Tumor Necrosis Factor-α. Asian Spine J. 2020;15(6):713-720doi: 10.31616/asj.2020.0425.
Yagi, K., Goto, Y., Kato, K., Suzuki, N., Kondo, A., Waseda, Y., Mizutani, J., Kawaguchi, Y., Joyo, Y., Waguri-Nagaya, Y., & Murakami, H. (2021). p38 Mitogen-Activated Protein Kinase Is Involved in Interleukin-6 Secretion from Human Ligamentum Flavum-Derived Cells Stimulated by Tumor Necrosis Factor-α. Asian spine journal, 15(6), 713-720. https://doi.org/10.31616/asj.2020.0425
Yagi, Kiyoshi, et al. "p38 Mitogen-Activated Protein Kinase Is Involved in Interleukin-6 Secretion from Human Ligamentum Flavum-Derived Cells Stimulated by Tumor Necrosis Factor-α." Asian spine journal vol. 15,6 (2021): 713-720. doi: https://doi.org/10.31616/asj.2020.0425
Yagi K, Goto Y, Kato K, Suzuki N, Kondo A, Waseda Y, Mizutani J, Kawaguchi Y, Joyo Y, Waguri-Nagaya Y, Murakami H. p38 Mitogen-Activated Protein Kinase Is Involved in Interleukin-6 Secretion from Human Ligamentum Flavum-Derived Cells Stimulated by Tumor Necrosis Factor-α. Asian Spine J. 2021 Dec;15(6):713-720. doi: 10.31616/asj.2020.0425. Epub 2020 Dec 28. PMID: 33355843; PMCID: PMC8696066.
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Sex differences in the effects of whole-life, low-dose cadmium exposure on postweaning high-fat diet-induced cardiac pathogeneses.

The Science of the total environment

Zhou W, Young JL, Men H, Zhang H, Yu H, Lin Q, Xu H, Xu J, Tan Y, Zheng Y, Cai L.
PMID: 34875320
Sci Total Environ. 2021 Dec 04;152176. doi: 10.1016/j.scitotenv.2021.152176. Epub 2021 Dec 04.

We previously showed the development of cardiac remodeling (hypertrophy or fibrosis) in mice with either post-weaning high-fat diet (HFD, 60% kcal fat) feeding or exposure to chronic low-dose cadmium. Here, we determined whether whole-life exposure to environmentally relevant, low-dose...

Cite
Zhou W, Young JL, Men H, et al. Sex differences in the effects of whole-life, low-dose cadmium exposure on postweaning high-fat diet-induced cardiac pathogeneses. Sci Total Environ. 2021;152176doi: 10.1016/j.scitotenv.2021.152176.
Zhou, W., Young, J. L., Men, H., Zhang, H., Yu, H., Lin, Q., Xu, H., Xu, J., Tan, Y., Zheng, Y., & Cai, L. (2021). Sex differences in the effects of whole-life, low-dose cadmium exposure on postweaning high-fat diet-induced cardiac pathogeneses. The Science of the total environment, 152176. https://doi.org/10.1016/j.scitotenv.2021.152176
Zhou, Wenqian, et al. "Sex differences in the effects of whole-life, low-dose cadmium exposure on postweaning high-fat diet-induced cardiac pathogeneses." The Science of the total environment vol. (2021): 152176. doi: https://doi.org/10.1016/j.scitotenv.2021.152176
Zhou W, Young JL, Men H, Zhang H, Yu H, Lin Q, Xu H, Xu J, Tan Y, Zheng Y, Cai L. Sex differences in the effects of whole-life, low-dose cadmium exposure on postweaning high-fat diet-induced cardiac pathogeneses. Sci Total Environ. 2021 Dec 04;152176. doi: 10.1016/j.scitotenv.2021.152176. Epub 2021 Dec 04. PMID: 34875320.
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p38 Mitogen-Activated Protein Kinase Is Involved in Interleukin-6 Secretion from Human Ligamentum Flavum-Derived Cells Stimulated by Tumor Necrosis Factor-α.

Asian spine journal

Yagi K, Goto Y, Kato K, Suzuki N, Kondo A, Waseda Y, Mizutani J, Kawaguchi Y, Joyo Y, Waguri-Nagaya Y, Murakami H.
PMID: 33355843
Asian Spine J. 2021 Dec;15(6):713-720. doi: 10.31616/asj.2020.0425. Epub 2020 Dec 28.

STUDY DESIGN: Human ligamentum flavum-derived cells (HFCs) were obtained from surgical samples for a basic experimental study.PURPOSE: We sought to evaluate the inflammatory response of human ligamentum flavum cells to investigate hypertrophic changes occurring in the ligamentum flavum.OVERVIEW OF...

Cite
Yagi K, Goto Y, Kato K, et al. p38 Mitogen-Activated Protein Kinase Is Involved in Interleukin-6 Secretion from Human Ligamentum Flavum-Derived Cells Stimulated by Tumor Necrosis Factor-α. Asian Spine J. 2020;15(6):713-720doi: 10.31616/asj.2020.0425.
Yagi, K., Goto, Y., Kato, K., Suzuki, N., Kondo, A., Waseda, Y., Mizutani, J., Kawaguchi, Y., Joyo, Y., Waguri-Nagaya, Y., & Murakami, H. (2021). p38 Mitogen-Activated Protein Kinase Is Involved in Interleukin-6 Secretion from Human Ligamentum Flavum-Derived Cells Stimulated by Tumor Necrosis Factor-α. Asian spine journal, 15(6), 713-720. https://doi.org/10.31616/asj.2020.0425
Yagi, Kiyoshi, et al. "p38 Mitogen-Activated Protein Kinase Is Involved in Interleukin-6 Secretion from Human Ligamentum Flavum-Derived Cells Stimulated by Tumor Necrosis Factor-α." Asian spine journal vol. 15,6 (2021): 713-720. doi: https://doi.org/10.31616/asj.2020.0425
Yagi K, Goto Y, Kato K, Suzuki N, Kondo A, Waseda Y, Mizutani J, Kawaguchi Y, Joyo Y, Waguri-Nagaya Y, Murakami H. p38 Mitogen-Activated Protein Kinase Is Involved in Interleukin-6 Secretion from Human Ligamentum Flavum-Derived Cells Stimulated by Tumor Necrosis Factor-α. Asian Spine J. 2021 Dec;15(6):713-720. doi: 10.31616/asj.2020.0425. Epub 2020 Dec 28. PMID: 33355843.
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Sex differences in the effects of whole-life, low-dose cadmium exposure on postweaning high-fat diet-induced cardiac pathogeneses.

The Science of the total environment

Zhou W, Young JL, Men H, Zhang H, Yu H, Lin Q, Xu H, Xu J, Tan Y, Zheng Y, Cai L.
PMID: 34875320
Sci Total Environ. 2021 Dec 05;809:152176. doi: 10.1016/j.scitotenv.2021.152176. Epub 2021 Dec 05.

We previously showed the development of cardiac remodeling (hypertrophy or fibrosis) in mice with either post-weaning high-fat diet (HFD, 60% kcal fat) feeding or exposure to chronic low-dose cadmium. Here, we determined whether whole-life exposure to environmentally relevant, low-dose...

Cite
Zhou W, Young JL, Men H, et al. Sex differences in the effects of whole-life, low-dose cadmium exposure on postweaning high-fat diet-induced cardiac pathogeneses. Sci Total Environ. 2021;809:152176doi: 10.1016/j.scitotenv.2021.152176.
Zhou, W., Young, J. L., Men, H., Zhang, H., Yu, H., Lin, Q., Xu, H., Xu, J., Tan, Y., Zheng, Y., & Cai, L. (2021). Sex differences in the effects of whole-life, low-dose cadmium exposure on postweaning high-fat diet-induced cardiac pathogeneses. The Science of the total environment, 809152176. https://doi.org/10.1016/j.scitotenv.2021.152176
Zhou, Wenqian, et al. "Sex differences in the effects of whole-life, low-dose cadmium exposure on postweaning high-fat diet-induced cardiac pathogeneses." The Science of the total environment vol. 809 (2021): 152176. doi: https://doi.org/10.1016/j.scitotenv.2021.152176
Zhou W, Young JL, Men H, Zhang H, Yu H, Lin Q, Xu H, Xu J, Tan Y, Zheng Y, Cai L. Sex differences in the effects of whole-life, low-dose cadmium exposure on postweaning high-fat diet-induced cardiac pathogeneses. Sci Total Environ. 2021 Dec 05;809:152176. doi: 10.1016/j.scitotenv.2021.152176. Epub 2021 Dec 05. PMID: 34875320.
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Role of MKP-5-p38/MAPK pathway in Clopidogrel-induced gastric mucosal epithelial cells apoptosis and tight junction dysfunction.

American journal of translational research

Wu H, Ming W, Tan J, Lu Q, Mulmi Shrestha S, Li N, Wu G, Zhang Z, Shi R.
PMID: 32509173
Am J Transl Res. 2020 May 15;12(5):1741-1753. eCollection 2020.

Bleeding and delayed healing of gastric ulcer are well-recognized in patients following Clopidorgrel treatment. Our previous studies have shown that endoplasmic reticulum stress (ER) is involved in Clopidogrel-induced gastric mucosal damage through activating p38 mitogen-activated protein kinases (MAPK) pathway....

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Wu H, Ming W, Tan J, et al. Role of MKP-5-p38/MAPK pathway in Clopidogrel-induced gastric mucosal epithelial cells apoptosis and tight junction dysfunction. Am J Transl Res. 2020;12(5):1741-1753
Wu, H., Ming, W., Tan, J., Lu, Q., Mulmi Shrestha, S., Li, N., Wu, G., Zhang, Z., & Shi, R. (2020). Role of MKP-5-p38/MAPK pathway in Clopidogrel-induced gastric mucosal epithelial cells apoptosis and tight junction dysfunction. American journal of translational research, 12(5), 1741-1753.
Wu, Hailu, et al. "Role of MKP-5-p38/MAPK pathway in Clopidogrel-induced gastric mucosal epithelial cells apoptosis and tight junction dysfunction." American journal of translational research vol. 12,5 (2020): 1741-1753.
Wu H, Ming W, Tan J, Lu Q, Mulmi Shrestha S, Li N, Wu G, Zhang Z, Shi R. Role of MKP-5-p38/MAPK pathway in Clopidogrel-induced gastric mucosal epithelial cells apoptosis and tight junction dysfunction. Am J Transl Res. 2020 May 15;12(5):1741-1753. eCollection 2020. PMID: 32509173; PMCID: PMC7269986.
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p38α Mitogen-Activated Protein Kinase Is a Druggable Target in Pancreatic Adenocarcinoma.

Frontiers in oncology

Yang L, Sun X, Ye Y, Lu Y, Zuo J, Liu W, Elcock A, Zhu S.
PMID: 31828036
Front Oncol. 2019 Nov 26;9:1294. doi: 10.3389/fonc.2019.01294. eCollection 2019.

p38 mitogen-activated protein kinases are signaling molecules with major involvement in cancer. A detailed mechanistic understanding of how p38 MAPK family members function is urgently warranted for cancer targeted therapy. The conformational dynamics of the most common member of...

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Yang L, Sun X, Ye Y, et al. p38α Mitogen-Activated Protein Kinase Is a Druggable Target in Pancreatic Adenocarcinoma. Front Oncol. 2019;9:1294doi: 10.3389/fonc.2019.01294.
Yang, L., Sun, X., Ye, Y., Lu, Y., Zuo, J., Liu, W., Elcock, A., & Zhu, S. (2019). p38α Mitogen-Activated Protein Kinase Is a Druggable Target in Pancreatic Adenocarcinoma. Frontiers in oncology, 91294. https://doi.org/10.3389/fonc.2019.01294
Yang, Ling, et al. "p38α Mitogen-Activated Protein Kinase Is a Druggable Target in Pancreatic Adenocarcinoma." Frontiers in oncology vol. 9 (2019): 1294. doi: https://doi.org/10.3389/fonc.2019.01294
Yang L, Sun X, Ye Y, Lu Y, Zuo J, Liu W, Elcock A, Zhu S. p38α Mitogen-Activated Protein Kinase Is a Druggable Target in Pancreatic Adenocarcinoma. Front Oncol. 2019 Nov 26;9:1294. doi: 10.3389/fonc.2019.01294. eCollection 2019. PMID: 31828036; PMCID: PMC6890821.
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