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Showing 1 to 12 of 19 entries
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Human uterine leiomyoma-derived fibroblasts stimulate uterine leiomyoma cell proliferation and collagen type I production, and activate RTKs and TGF beta receptor signaling in coculture.

Cell communication and signaling : CCS

Moore AB, Yu L, Swartz CD, Zheng X, Wang L, Castro L, Kissling GE, Walmer DK, Robboy SJ, Dixon D.
PMID: 20537183
Cell Commun Signal. 2010 Jun 10;8:10. doi: 10.1186/1478-811X-8-10.

BACKGROUND: Uterine leiomyomas (fibroids) are benign smooth muscle tumors that often contain an excessive extracellular matrix (ECM). In the present study, we investigated the interactions between human uterine leiomyoma (UtLM) cells and uterine leiomyoma-derived fibroblasts (FB), and their importance...

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Moore AB, Yu L, Swartz CD, et al. Human uterine leiomyoma-derived fibroblasts stimulate uterine leiomyoma cell proliferation and collagen type I production, and activate RTKs and TGF beta receptor signaling in coculture. Cell Commun Signal. 2010;8:10doi: 10.1186/1478-811X-8-10.
Moore, A. B., Yu, L., Swartz, C. D., Zheng, X., Wang, L., Castro, L., Kissling, G. E., Walmer, D. K., Robboy, S. J., & Dixon, D. (2010). Human uterine leiomyoma-derived fibroblasts stimulate uterine leiomyoma cell proliferation and collagen type I production, and activate RTKs and TGF beta receptor signaling in coculture. Cell communication and signaling : CCS, 810. https://doi.org/10.1186/1478-811X-8-10
Moore, Alicia B, et al. "Human uterine leiomyoma-derived fibroblasts stimulate uterine leiomyoma cell proliferation and collagen type I production, and activate RTKs and TGF beta receptor signaling in coculture." Cell communication and signaling : CCS vol. 8 (2010): 10. doi: https://doi.org/10.1186/1478-811X-8-10
Moore AB, Yu L, Swartz CD, Zheng X, Wang L, Castro L, Kissling GE, Walmer DK, Robboy SJ, Dixon D. Human uterine leiomyoma-derived fibroblasts stimulate uterine leiomyoma cell proliferation and collagen type I production, and activate RTKs and TGF beta receptor signaling in coculture. Cell Commun Signal. 2010 Jun 10;8:10. doi: 10.1186/1478-811X-8-10. PMID: 20537183; PMCID: PMC2897788.
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On-line hemodiafiltration modulates atherosclerosis signaling in peripheral lymphomonocytes of hemodialysis patients.

Journal of nephrology

Simone S, Chieti A, Pontrelli P, Rascio F, Castellano G, Stallone G, Infante B, Gesualdo L, Grandaliano G, Pertosa G.
PMID: 33761122
J Nephrol. 2021 Dec;34(6):1989-1997. doi: 10.1007/s40620-020-00958-z. Epub 2021 Mar 24.

BACKGROUND: Hemodialysis patients present a dramatic increase in cardiovascular morbidity/mortality. Circulating immune cells, activated by both uremic milieu and dialysis, play a key role in the pathogenesis of dialysis-related vascular disease. The aim of our study was to identify,...

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Simone S, Chieti A, Pontrelli P, et al. On-line hemodiafiltration modulates atherosclerosis signaling in peripheral lymphomonocytes of hemodialysis patients. J Nephrol. 2021;34(6):1989-1997doi: 10.1007/s40620-020-00958-z.
Simone, S., Chieti, A., Pontrelli, P., Rascio, F., Castellano, G., Stallone, G., Infante, B., Gesualdo, L., Grandaliano, G., & Pertosa, G. (2021). On-line hemodiafiltration modulates atherosclerosis signaling in peripheral lymphomonocytes of hemodialysis patients. Journal of nephrology, 34(6), 1989-1997. https://doi.org/10.1007/s40620-020-00958-z
Simone, Simona, et al. "On-line hemodiafiltration modulates atherosclerosis signaling in peripheral lymphomonocytes of hemodialysis patients." Journal of nephrology vol. 34,6 (2021): 1989-1997. doi: https://doi.org/10.1007/s40620-020-00958-z
Simone S, Chieti A, Pontrelli P, Rascio F, Castellano G, Stallone G, Infante B, Gesualdo L, Grandaliano G, Pertosa G. On-line hemodiafiltration modulates atherosclerosis signaling in peripheral lymphomonocytes of hemodialysis patients. J Nephrol. 2021 Dec;34(6):1989-1997. doi: 10.1007/s40620-020-00958-z. Epub 2021 Mar 24. PMID: 33761122; PMCID: PMC8610953.
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Expression and function of PDGF-C in development and stem cells.

Open biology

Tian Y, Zhan Y, Jiang Q, Lu W, Li X.
PMID: 34847773
Open Biol. 2021 Dec;11(12):210268. doi: 10.1098/rsob.210268. Epub 2021 Dec 01.

Platelet-derived growth factor C (PDGF-C) is a relatively new member of the PDGF family, discovered nearly 20 years after the finding of platelet-derived growth factor A (PDGF-A) and platelet-derived growth factor B (PDGF-B). PDGF-C is generally expressed in most...

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Tian Y, Zhan Y, Jiang Q, et al. Expression and function of PDGF-C in development and stem cells. Open Biol. 2021;11(12):210268doi: 10.1098/rsob.210268.
Tian, Y., Zhan, Y., Jiang, Q., Lu, W., & Li, X. (2021). Expression and function of PDGF-C in development and stem cells. Open biology, 11(12), 210268. https://doi.org/10.1098/rsob.210268
Tian, Yi, et al. "Expression and function of PDGF-C in development and stem cells." Open biology vol. 11,12 (2021): 210268. doi: https://doi.org/10.1098/rsob.210268
Tian Y, Zhan Y, Jiang Q, Lu W, Li X. Expression and function of PDGF-C in development and stem cells. Open Biol. 2021 Dec;11(12):210268. doi: 10.1098/rsob.210268. Epub 2021 Dec 01. PMID: 34847773; PMCID: PMC8633783.
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Expression and function of PDGF-C in development and stem cells.

Open biology

Tian Y, Zhan Y, Jiang Q, Lu W, Li X.
PMID: 34847773
Open Biol. 2021 Dec;11(12):210268. doi: 10.1098/rsob.210268. Epub 2021 Dec 01.

Platelet-derived growth factor C (PDGF-C) is a relatively new member of the PDGF family, discovered nearly 20 years after the finding of platelet-derived growth factor A (PDGF-A) and platelet-derived growth factor B (PDGF-B). PDGF-C is generally expressed in most...

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Tian Y, Zhan Y, Jiang Q, et al. Expression and function of PDGF-C in development and stem cells. Open Biol. 2021;11(12):210268doi: 10.1098/rsob.210268.
Tian, Y., Zhan, Y., Jiang, Q., Lu, W., & Li, X. (2021). Expression and function of PDGF-C in development and stem cells. Open biology, 11(12), 210268. https://doi.org/10.1098/rsob.210268
Tian, Yi, et al. "Expression and function of PDGF-C in development and stem cells." Open biology vol. 11,12 (2021): 210268. doi: https://doi.org/10.1098/rsob.210268
Tian Y, Zhan Y, Jiang Q, Lu W, Li X. Expression and function of PDGF-C in development and stem cells. Open Biol. 2021 Dec;11(12):210268. doi: 10.1098/rsob.210268. Epub 2021 Dec 01. PMID: 34847773.
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Corrigendum to: effect of methylene methylimino linkage of antisense oligonucleotide to the platelet-derived growth factor A-chain on growth of vascular smooth muscle cells from spontaneously hypertensive rats.

European journal of pharmacology

Kishioka H, Fukuda N, Nakayama M, Hu W, Satoh C, Kanmatsuse K, Bhat B, Symons AM, Manoharan M.
PMID: 10996605
Eur J Pharmacol. 2000 Sep 22;404(3):395. doi: 10.1016/s0014-2999(00)00614-2.

No abstract available.

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Kishioka H, Fukuda N, Nakayama M, et al. Corrigendum to: effect of methylene methylimino linkage of antisense oligonucleotide to the platelet-derived growth factor A-chain on growth of vascular smooth muscle cells from spontaneously hypertensive rats. Eur J Pharmacol. 2000;404(3):395doi: 10.1016/s0014-2999(00)00614-2.
Kishioka, H., Fukuda, N., Nakayama, M., Hu, W., Satoh, C., Kanmatsuse, K., Bhat, B., Symons, A. M., & Manoharan, M. (2000). Corrigendum to: effect of methylene methylimino linkage of antisense oligonucleotide to the platelet-derived growth factor A-chain on growth of vascular smooth muscle cells from spontaneously hypertensive rats. European journal of pharmacology, 404(3), 395. https://doi.org/10.1016/s0014-2999(00)00614-2
Kishioka, et al. "Corrigendum to: effect of methylene methylimino linkage of antisense oligonucleotide to the platelet-derived growth factor A-chain on growth of vascular smooth muscle cells from spontaneously hypertensive rats." European journal of pharmacology vol. 404,3 (2000): 395. doi: https://doi.org/10.1016/s0014-2999(00)00614-2
Kishioka H, Fukuda N, Nakayama M, Hu W, Satoh C, Kanmatsuse K, Bhat B, Symons AM, Manoharan M. Corrigendum to: effect of methylene methylimino linkage of antisense oligonucleotide to the platelet-derived growth factor A-chain on growth of vascular smooth muscle cells from spontaneously hypertensive rats. Eur J Pharmacol. 2000 Sep 22;404(3):395. doi: 10.1016/s0014-2999(00)00614-2. PMID: 10996605.
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Statement of Retraction. Kristina Blaslov, Višjna Kokić Maleš, Slaven Kokić, and Ivan Kruljac. 1909-P: The Platelet-Derived Growth Factor a-An Additional Pathophysiological Factor in Hepatic Steatosis and Fibrosis Development Both in Patients with Hepatitis C and Patients with Prediabetes. Diabetes 2019;68(Suppl. 1). https://doi.org/10.2337/db19-1909-P.

Diabetes

[No authors listed]
PMID: 32718935
Diabetes. 2020 Jul 27; doi: 10.2337/db20-rt10a. Epub 2020 Jul 27.

No abstract available.

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Statement of Retraction. Kristina Blaslov, Višjna Kokić Maleš, Slaven Kokić, and Ivan Kruljac. 1909-P: The Platelet-Derived Growth Factor a-An Additional Pathophysiological Factor in Hepatic Steatosis and Fibrosis Development Both in Patients with Hepatitis C and Patients with Prediabetes. Diabetes 2019;68(Suppl. 1). https://doi.org/10.2337/db19-1909-P. Diabetes. 2020;doi: 10.2337/db20-rt10a.
(2020). Statement of Retraction. Kristina Blaslov, Višjna Kokić Maleš, Slaven Kokić, and Ivan Kruljac. 1909-P: The Platelet-Derived Growth Factor a-An Additional Pathophysiological Factor in Hepatic Steatosis and Fibrosis Development Both in Patients with Hepatitis C and Patients with Prediabetes. Diabetes 2019;68(Suppl. 1). https://doi.org/10.2337/db19-1909-P. Diabetes, . https://doi.org/10.2337/db20-rt10a
"Statement of Retraction. Kristina Blaslov, Višjna Kokić Maleš, Slaven Kokić, and Ivan Kruljac. 1909-P: The Platelet-Derived Growth Factor a-An Additional Pathophysiological Factor in Hepatic Steatosis and Fibrosis Development Both in Patients with Hepatitis C and Patients with Prediabetes. Diabetes 2019;68(Suppl. 1). https://doi.org/10.2337/db19-1909-P." Diabetes vol. (2020). doi: https://doi.org/10.2337/db20-rt10a
Statement of Retraction. Kristina Blaslov, Višjna Kokić Maleš, Slaven Kokić, and Ivan Kruljac. 1909-P: The Platelet-Derived Growth Factor a-An Additional Pathophysiological Factor in Hepatic Steatosis and Fibrosis Development Both in Patients with Hepatitis C and Patients with Prediabetes. Diabetes 2019;68(Suppl. 1). https://doi.org/10.2337/db19-1909-P. Diabetes. 2020 Jul 27; doi: 10.2337/db20-rt10a. Epub 2020 Jul 27. PMID: 32718935.
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PDGFA in Cashmere Goat: A Motivation for the Hair Follicle Stem Cells to Activate.

Animals : an open access journal from MDPI

Pazzaglia I, Mercati F, Antonini M, Capomaccio S, Cappelli K, Dall'Aglio C, La Terza A, Mozzicafreddo M, Nocelli C, Pallotti S, Pediconi D, Renieri C.
PMID: 30695990
Animals (Basel). 2019 Jan 28;9(2). doi: 10.3390/ani9020038.

The cashmere hair follicle (HF) perpetually goes through cycles of growth, involution and rest. The photoperiod is the main factor in the control of seasonal coat change in cashmere goats while stem cells play a crucial role in the...

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Pazzaglia I, Mercati F, Antonini M, et al. PDGFA in Cashmere Goat: A Motivation for the Hair Follicle Stem Cells to Activate. Animals (Basel). 2019;9(2)doi: 10.3390/ani9020038.
Pazzaglia, I., Mercati, F., Antonini, M., Capomaccio, S., Cappelli, K., Dall'Aglio, C., La Terza, A., Mozzicafreddo, M., Nocelli, C., Pallotti, S., Pediconi, D., & Renieri, C. (2019). PDGFA in Cashmere Goat: A Motivation for the Hair Follicle Stem Cells to Activate. Animals : an open access journal from MDPI, 9(2), . https://doi.org/10.3390/ani9020038
Pazzaglia, Irene, et al. "PDGFA in Cashmere Goat: A Motivation for the Hair Follicle Stem Cells to Activate." Animals : an open access journal from MDPI vol. 9,2 (2019). doi: https://doi.org/10.3390/ani9020038
Pazzaglia I, Mercati F, Antonini M, Capomaccio S, Cappelli K, Dall'Aglio C, La Terza A, Mozzicafreddo M, Nocelli C, Pallotti S, Pediconi D, Renieri C. PDGFA in Cashmere Goat: A Motivation for the Hair Follicle Stem Cells to Activate. Animals (Basel). 2019 Jan 28;9(2). doi: 10.3390/ani9020038. PMID: 30695990; PMCID: PMC6407032.
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Growth factor expression with different wound treatments after laser resurfacing.

Aesthetic surgery journal

Jae Lee C, Hee Whang J, Lazova R, Ciesielski TE, Thomson JG, McCarthy T, Persing JA.
PMID: 19341630
Aesthet Surg J. 2007 Jan-Feb;27(1):55-64. doi: 10.1016/j.asj.2006.12.002.

BACKGROUND: Many studies have demonstrated that the specific method of wound dressing used may affect the healing process. However, the effect of the method of wound dressings on the expression of growth factors is not well documented.OBJECTIVE: The aim...

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Jae Lee C, Hee Whang J, Lazova R, et al. Growth factor expression with different wound treatments after laser resurfacing. Aesthet Surg J. 2007;27(1):55-64doi: 10.1016/j.asj.2006.12.002.
Jae Lee, C., Hee Whang, J., Lazova, R., Ciesielski, T. E., Thomson, J. G., McCarthy, T., & Persing, J. A. (2007). Growth factor expression with different wound treatments after laser resurfacing. Aesthetic surgery journal, 27(1), 55-64. https://doi.org/10.1016/j.asj.2006.12.002
Jae Lee, Choong, et al. "Growth factor expression with different wound treatments after laser resurfacing." Aesthetic surgery journal vol. 27,1 (2007): 55-64. doi: https://doi.org/10.1016/j.asj.2006.12.002
Jae Lee C, Hee Whang J, Lazova R, Ciesielski TE, Thomson JG, McCarthy T, Persing JA. Growth factor expression with different wound treatments after laser resurfacing. Aesthet Surg J. 2007 Jan-Feb;27(1):55-64. doi: 10.1016/j.asj.2006.12.002. PMID: 19341630.
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Association and regulation of protein factors of field effect in prostate tissues.

International journal of oncology

Gabriel KN, Jones AC, Nguyen JP, Antillon KS, Janos SN, Overton HN, Jenkins SM, Frisch EH, Trujillo KA, Bisoffi M.
PMID: 27634112
Int J Oncol. 2016 Oct;49(4):1541-1552. doi: 10.3892/ijo.2016.3666. Epub 2016 Aug 22.

Field effect or field cancerization denotes the presence of molecular aberrations in structurally intact cells residing in histologically normal tissues adjacent to solid tumors. Currently, the etiology of prostate field‑effect formation is unknown and there is a prominent lack...

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Gabriel KN, Jones AC, Nguyen JP, et al. Association and regulation of protein factors of field effect in prostate tissues. Int J Oncol. 2016;49(4):1541-1552doi: 10.3892/ijo.2016.3666.
Gabriel, K. N., Jones, A. C., Nguyen, J. P., Antillon, K. S., Janos, S. N., Overton, H. N., Jenkins, S. M., Frisch, E. H., Trujillo, K. A., & Bisoffi, M. (2016). Association and regulation of protein factors of field effect in prostate tissues. International journal of oncology, 49(4), 1541-1552. https://doi.org/10.3892/ijo.2016.3666
Gabriel, Kristin N, et al. "Association and regulation of protein factors of field effect in prostate tissues." International journal of oncology vol. 49,4 (2016): 1541-1552. doi: https://doi.org/10.3892/ijo.2016.3666
Gabriel KN, Jones AC, Nguyen JP, Antillon KS, Janos SN, Overton HN, Jenkins SM, Frisch EH, Trujillo KA, Bisoffi M. Association and regulation of protein factors of field effect in prostate tissues. Int J Oncol. 2016 Oct;49(4):1541-1552. doi: 10.3892/ijo.2016.3666. Epub 2016 Aug 22. PMID: 27634112; PMCID: PMC5021247.
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Chotosan (Diaoteng San)-induced improvement of cognitive deficits in senescence-accelerated mouse (SAMP8) involves the amelioration of angiogenic/neurotrophic factors and neuroplasticity systems in the brain.

Chinese medicine

Zhao Q, Yokozawa T, Tsuneyama K, Tanaka K, Miyata T, Shibahara N, Matsumoto K.
PMID: 21943225
Chin Med. 2011 Sep 23;6:33. doi: 10.1186/1749-8546-6-33.

BACKGROUND: Chotosan (CTS, Diaoteng San), a Kampo medicine (ie Chinese medicine) formula, is reportedly effective in the treatment of patients with cerebral ischemic insults. This study aims to evaluate the therapeutic potential of CTS in cognitive deficits and investigates...

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Zhao Q, Yokozawa T, Tsuneyama K, et al. Chotosan (Diaoteng San)-induced improvement of cognitive deficits in senescence-accelerated mouse (SAMP8) involves the amelioration of angiogenic/neurotrophic factors and neuroplasticity systems in the brain. Chin Med. 2011;6:33doi: 10.1186/1749-8546-6-33.
Zhao, Q., Yokozawa, T., Tsuneyama, K., Tanaka, K., Miyata, T., Shibahara, N., & Matsumoto, K. (2011). Chotosan (Diaoteng San)-induced improvement of cognitive deficits in senescence-accelerated mouse (SAMP8) involves the amelioration of angiogenic/neurotrophic factors and neuroplasticity systems in the brain. Chinese medicine, 633. https://doi.org/10.1186/1749-8546-6-33
Zhao, Qi, et al. "Chotosan (Diaoteng San)-induced improvement of cognitive deficits in senescence-accelerated mouse (SAMP8) involves the amelioration of angiogenic/neurotrophic factors and neuroplasticity systems in the brain." Chinese medicine vol. 6 (2011): 33. doi: https://doi.org/10.1186/1749-8546-6-33
Zhao Q, Yokozawa T, Tsuneyama K, Tanaka K, Miyata T, Shibahara N, Matsumoto K. Chotosan (Diaoteng San)-induced improvement of cognitive deficits in senescence-accelerated mouse (SAMP8) involves the amelioration of angiogenic/neurotrophic factors and neuroplasticity systems in the brain. Chin Med. 2011 Sep 23;6:33. doi: 10.1186/1749-8546-6-33. PMID: 21943225; PMCID: PMC3189182.
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GSDMB is increased in IBD and regulates epithelial restitution/repair independent of pyroptosis.

Cell

Rana N, Privitera G, Kondolf HC, Bulek K, Lechuga S, De Salvo C, Corridoni D, Antanaviciute A, Maywald RL, Hurtado AM, Zhao J, Huang EH, Li X, Chan ER, Simmons A, Bamias G, Abbott DW, Heaney JD, Ivanov AI, Pizarro TT.
PMID: 35021065
Cell. 2022 Jan 07; doi: 10.1016/j.cell.2021.12.024. Epub 2022 Jan 07.

Gasdermins are a family of structurally related proteins originally described for their role in pyroptosis. Gasdermin B (GSDMB) is currently the least studied, and while its association with genetic susceptibility to chronic mucosal inflammatory disorders is well established, little...

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Rana N, Privitera G, Kondolf HC, et al. GSDMB is increased in IBD and regulates epithelial restitution/repair independent of pyroptosis. Cell. 2022;doi: 10.1016/j.cell.2021.12.024.
Rana, N., Privitera, G., Kondolf, H. C., Bulek, K., Lechuga, S., De Salvo, C., Corridoni, D., Antanaviciute, A., Maywald, R. L., Hurtado, A. M., Zhao, J., Huang, E. H., Li, X., Chan, E. R., Simmons, A., Bamias, G., Abbott, D. W., Heaney, J. D., Ivanov, A. I., & Pizarro, T. T. (2022). GSDMB is increased in IBD and regulates epithelial restitution/repair independent of pyroptosis. Cell, . https://doi.org/10.1016/j.cell.2021.12.024
Rana, Nitish, et al. "GSDMB is increased in IBD and regulates epithelial restitution/repair independent of pyroptosis." Cell vol. (2022). doi: https://doi.org/10.1016/j.cell.2021.12.024
Rana N, Privitera G, Kondolf HC, Bulek K, Lechuga S, De Salvo C, Corridoni D, Antanaviciute A, Maywald RL, Hurtado AM, Zhao J, Huang EH, Li X, Chan ER, Simmons A, Bamias G, Abbott DW, Heaney JD, Ivanov AI, Pizarro TT. GSDMB is increased in IBD and regulates epithelial restitution/repair independent of pyroptosis. Cell. 2022 Jan 07; doi: 10.1016/j.cell.2021.12.024. Epub 2022 Jan 07. PMID: 35021065.
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The Effect of Sunitinib Treatment in Human Melanoma Xenografts: Associations with Angiogenic Profiles.

Translational oncology

Gaustad JV, Simonsen TG, Andersen LM, Rofstad EK.
PMID: 28167241
Transl Oncol. 2017 Apr;10(2):158-167. doi: 10.1016/j.tranon.2016.12.007. Epub 2017 Feb 03.

The effect of antiangiogenic agents targeting the vascular endothelial growth factor A (VEGF-A) pathway has been reported to vary substantially in preclinical studies. The purpose of this study was to investigate the effect of sunitinib treatment on tumor vasculature...

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Gaustad JV, Simonsen TG, Andersen LM, et al. The Effect of Sunitinib Treatment in Human Melanoma Xenografts: Associations with Angiogenic Profiles. Transl Oncol. 2017;10(2):158-167doi: 10.1016/j.tranon.2016.12.007.
Gaustad, J. V., Simonsen, T. G., Andersen, L. M., & Rofstad, E. K. (2017). The Effect of Sunitinib Treatment in Human Melanoma Xenografts: Associations with Angiogenic Profiles. Translational oncology, 10(2), 158-167. https://doi.org/10.1016/j.tranon.2016.12.007
Gaustad, Jon-Vidar, et al. "The Effect of Sunitinib Treatment in Human Melanoma Xenografts: Associations with Angiogenic Profiles." Translational oncology vol. 10,2 (2017): 158-167. doi: https://doi.org/10.1016/j.tranon.2016.12.007
Gaustad JV, Simonsen TG, Andersen LM, Rofstad EK. The Effect of Sunitinib Treatment in Human Melanoma Xenografts: Associations with Angiogenic Profiles. Transl Oncol. 2017 Apr;10(2):158-167. doi: 10.1016/j.tranon.2016.12.007. Epub 2017 Feb 03. PMID: 28167241; PMCID: PMC5293738.
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