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Molecular Dependency Impacts on the Compensating Ability of Paralogs: A Response to Veitia.

Trends in genetics : TIG

Landry CR, Diss G.
PMID: 28870654
Trends Genet. 2017 Oct;33(10):657-658. doi: 10.1016/j.tig.2017.07.012. Epub 2017 Sep 01.

No abstract available.

Cite
Landry CR, Diss G. Molecular Dependency Impacts on the Compensating Ability of Paralogs: A Response to Veitia. Trends Genet. 2017;33(10):657-658doi: 10.1016/j.tig.2017.07.012.
Landry, C. R., & Diss, G. (2017). Molecular Dependency Impacts on the Compensating Ability of Paralogs: A Response to Veitia. Trends in genetics : TIG, 33(10), 657-658. https://doi.org/10.1016/j.tig.2017.07.012
Landry, Christian R, and Diss, Guillaume. "Molecular Dependency Impacts on the Compensating Ability of Paralogs: A Response to Veitia." Trends in genetics : TIG vol. 33,10 (2017): 657-658. doi: https://doi.org/10.1016/j.tig.2017.07.012
Landry CR, Diss G. Molecular Dependency Impacts on the Compensating Ability of Paralogs: A Response to Veitia. Trends Genet. 2017 Oct;33(10):657-658. doi: 10.1016/j.tig.2017.07.012. Epub 2017 Sep 01. PMID: 28870654.
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Digest: Gene duplication and social evolution-Using big, open data to answer big, open questions.

Evolution; international journal of organic evolution

Harpur BA, Smith NMA.
PMID: 29105748
Evolution. 2017 Dec;71(12):2952-2953. doi: 10.1111/evo.13390. Epub 2017 Nov 24.

No abstract available.

Cite
Harpur BA, Smith NMA. Digest: Gene duplication and social evolution-Using big, open data to answer big, open questions. Evolution. 2017;71(12):2952-2953doi: 10.1111/evo.13390.
Harpur, B. A., & Smith, N. M. A. (2017). Digest: Gene duplication and social evolution-Using big, open data to answer big, open questions. Evolution; international journal of organic evolution, 71(12), 2952-2953. https://doi.org/10.1111/evo.13390
Harpur, Brock A, and Smith, Nicholas M A. "Digest: Gene duplication and social evolution-Using big, open data to answer big, open questions." Evolution; international journal of organic evolution vol. 71,12 (2017): 2952-2953. doi: https://doi.org/10.1111/evo.13390
Harpur BA, Smith NMA. Digest: Gene duplication and social evolution-Using big, open data to answer big, open questions. Evolution. 2017 Dec;71(12):2952-2953. doi: 10.1111/evo.13390. Epub 2017 Nov 24. PMID: 29105748.
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EqualTDRL: illustrating equivalent tandem duplication random loss rearrangements.

BMC bioinformatics

Hartmann T, Bernt M, Middendorf M.
PMID: 29843612
BMC Bioinformatics. 2018 May 30;19(1):192. doi: 10.1186/s12859-018-2170-x.

BACKGROUND: To study the differences between two unichromosomal circular genomes, e.g., mitochondrial genomes, under the tandem duplication random loss (TDRL) rearrangement it is important to consider the whole set of potential TDRL rearrangement events that could have taken place....

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Hartmann T, Bernt M, Middendorf M. EqualTDRL: illustrating equivalent tandem duplication random loss rearrangements. BMC Bioinformatics. 2018;19(1):192doi: 10.1186/s12859-018-2170-x.
Hartmann, T., Bernt, M., & Middendorf, M. (2018). EqualTDRL: illustrating equivalent tandem duplication random loss rearrangements. BMC bioinformatics, 19(1), 192. https://doi.org/10.1186/s12859-018-2170-x
Hartmann, Tom, et al. "EqualTDRL: illustrating equivalent tandem duplication random loss rearrangements." BMC bioinformatics vol. 19,1 (2018): 192. doi: https://doi.org/10.1186/s12859-018-2170-x
Hartmann T, Bernt M, Middendorf M. EqualTDRL: illustrating equivalent tandem duplication random loss rearrangements. BMC Bioinformatics. 2018 May 30;19(1):192. doi: 10.1186/s12859-018-2170-x. PMID: 29843612; PMCID: PMC5975268.
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Duplication of the 22q11.2 region associated with congenital cardiac disease.

Cardiology in the young

Sparkes R, Chernos J, Dicke F.
PMID: 15845171
Cardiol Young. 2005 Apr;15(2):229-31. doi: 10.1017/S1047951105000466.

The DiGeorge, or velocardiofacial, syndrome has been aetiologically linked to heterozygous deletion of the q11.2 region of chromosome 22. It is the most common of the microdeletion syndromes, and is associated with malformations involving the ventricular outflow tracts. Duplication...

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Sparkes R, Chernos J, Dicke F. Duplication of the 22q11.2 region associated with congenital cardiac disease. Cardiol Young. 2005;15(2):229-31doi: 10.1017/S1047951105000466.
Sparkes, R., Chernos, J., & Dicke, F. (2005). Duplication of the 22q11.2 region associated with congenital cardiac disease. Cardiology in the young, 15(2), 229-31. https://doi.org/10.1017/S1047951105000466
Sparkes, Rebecca, et al. "Duplication of the 22q11.2 region associated with congenital cardiac disease." Cardiology in the young vol. 15,2 (2005): 229-31. doi: https://doi.org/10.1017/S1047951105000466
Sparkes R, Chernos J, Dicke F. Duplication of the 22q11.2 region associated with congenital cardiac disease. Cardiol Young. 2005 Apr;15(2):229-31. doi: 10.1017/S1047951105000466. PMID: 15845171.
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Gene Duplicates: Agents of Fragility? - A Reply to Landry and Diss.

Trends in genetics : TIG

Veitia RA.
PMID: 28823576
Trends Genet. 2017 Oct;33(10):658-660. doi: 10.1016/j.tig.2017.07.013. Epub 2017 Aug 17.

No abstract available.

Cite
Veitia RA. Gene Duplicates: Agents of Fragility? - A Reply to Landry and Diss. Trends Genet. 2017;33(10):658-660doi: 10.1016/j.tig.2017.07.013.
Veitia, R. A. (2017). Gene Duplicates: Agents of Fragility? - A Reply to Landry and Diss. Trends in genetics : TIG, 33(10), 658-660. https://doi.org/10.1016/j.tig.2017.07.013
Veitia, Reiner A. "Gene Duplicates: Agents of Fragility? - A Reply to Landry and Diss." Trends in genetics : TIG vol. 33,10 (2017): 658-660. doi: https://doi.org/10.1016/j.tig.2017.07.013
Veitia RA. Gene Duplicates: Agents of Fragility? - A Reply to Landry and Diss. Trends Genet. 2017 Oct;33(10):658-660. doi: 10.1016/j.tig.2017.07.013. Epub 2017 Aug 17. PMID: 28823576.
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Showing 1 to 5 of 5 entries