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Pathogenesis of neointima formation following vascular injury.

Cardiovascular & hematological disorders drug targets

O' Brien ER, Ma X, Simard T, Pourdjabbar A, Hibbert B.
PMID: 21446903
Cardiovasc Hematol Disord Drug Targets. 2011 Mar 01;11(1):30-9. doi: 10.2174/187152911795945169.

Revascularization remains the cornerstone of managing obstructive coronary artery disease. Although percutaneous coronary interventions involving the insertion of metal scaffolds, known as stents, has emerged as the preferred method of restoring vessel patency, as many as 30% of patients...

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O' Brien ER, Ma X, Simard T, et al. Pathogenesis of neointima formation following vascular injury. Cardiovasc Hematol Disord Drug Targets. 2011;11(1):30-9doi: 10.2174/187152911795945169.
O' Brien, E. R., Ma, X., Simard, T., Pourdjabbar, A., & Hibbert, B. (2011). Pathogenesis of neointima formation following vascular injury. Cardiovascular & hematological disorders drug targets, 11(1), 30-9. https://doi.org/10.2174/187152911795945169
O' Brien, Edward R, et al. "Pathogenesis of neointima formation following vascular injury." Cardiovascular & hematological disorders drug targets vol. 11,1 (2011): 30-9. doi: https://doi.org/10.2174/187152911795945169
O' Brien ER, Ma X, Simard T, Pourdjabbar A, Hibbert B. Pathogenesis of neointima formation following vascular injury. Cardiovasc Hematol Disord Drug Targets. 2011 Mar 01;11(1):30-9. doi: 10.2174/187152911795945169. PMID: 21446903.
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