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Kimmelstiel CD, Salem DN. The Open-Artery Hypothesis: An Overview. J Thromb Thrombolysis. 1997;4(2):227-237doi: 10.1023/a:1008842917403.
Kimmelstiel, C. D., & Salem, D. N. (1997). The Open-Artery Hypothesis: An Overview. Journal of thrombosis and thrombolysis, 4(2), 227-237. https://doi.org/10.1023/a:1008842917403
Kimmelstiel, and Salem. "The Open-Artery Hypothesis: An Overview." Journal of thrombosis and thrombolysis vol. 4,2 (1997): 227-237. doi: https://doi.org/10.1023/a:1008842917403
Kimmelstiel CD, Salem DN. The Open-Artery Hypothesis: An Overview. J Thromb Thrombolysis. 1997;4(2):227-237. doi: 10.1023/a:1008842917403. PMID: 10639257.
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J Thromb Thrombolysis. 1997;4(2):227-237. doi: 10.1023/a:1008842917403.

The Open-Artery Hypothesis: An Overview.

Journal of thrombosis and thrombolysis

Kimmelstiel, Salem

Affiliations

  1. Tufts University School of Medicine; Adult Cardiac Catheterization, Laboratory and Ambulatory Cardiology, New England Medical Center Boston, Massachusetts, USA.

PMID: 10639257 DOI: 10.1023/a:1008842917403

Abstract

The open artery hypothesis postulates salutary effects distinct from myocardial salvage referable to infarct related arterial patency. Short and long term clinical trials, as well as studies in animal models have suggested clinical benefit in patients with sustained infarct related arterial patency. Mechanisms of benefit are not entirely clear, but probably relate to mitigation of post infarction left ventricular remodeling and improvement in several electrophysiologic parameters. The effect of infarct related arterial patency on post infarction prognosis, presumed mechanisms of benefit and noninvasive assessment of arterial patency are summarized in this review.

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