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Morimoto T, Yamada T, Nagata K, et al. Intramedullary gadolinium-DTPA enhancement in a patient with cervical spondylotic myelopathy and an associated vascular lesion. Case report. Neurosurg Focus. 1996;1(6):e3
Morimoto, T., Yamada, T., Nagata, K., Matsuyama, T., & Sakaki, T. (1996). Intramedullary gadolinium-DTPA enhancement in a patient with cervical spondylotic myelopathy and an associated vascular lesion. Case report. Neurosurgical focus, 1(6), e3.
Morimoto, T, et al. "Intramedullary gadolinium-DTPA enhancement in a patient with cervical spondylotic myelopathy and an associated vascular lesion. Case report." Neurosurgical focus vol. 1,6 (1996): e3.
Morimoto T, Yamada T, Nagata K, Matsuyama T, Sakaki T. Intramedullary gadolinium-DTPA enhancement in a patient with cervical spondylotic myelopathy and an associated vascular lesion. Case report. Neurosurg Focus. 1996 Dec 15;1(6):e3. PMID: 15096029.
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Neurosurg Focus. 1996 Dec 15;1(6):e3.

Intramedullary gadolinium-DTPA enhancement in a patient with cervical spondylotic myelopathy and an associated vascular lesion. Case report.

Neurosurgical focus

T Morimoto, T Yamada, K Nagata, T Matsuyama, T Sakaki

Affiliations

  1. Department of Neurosurgery, Nara Medical University, Kashihara, Japan.

PMID: 15096029

Abstract

Intramedullary enhancement of the cervical spinal cord is rare in chronic compression disease. An accompanying vascular lesion should be considered in such a case. A 59-year-old man presented with severe cervical spondylotic myelopathy. Gadolinium-diethylenetriamine pentaacetic acid (Gd-DTPA) enhanced magnetic resonance imaging showed intramedullary enhancement at the C5-6 level, which was the most severely compressed level. A right ascending cervical arteriogram demonstrated a spinal arteriovenous fistula (AVF) fed mainly by the C-6 radicular artery and draining from the posterior medullary vein. Surgery was performed to decompress the myelopathy and to obliterate the AVF. Postoperative MR imaging with Gd-DTPA enhancement showed immediate and complete disappearance of the previous enhancement. The intramedullary enhancement presumably resulted from the intraparenchymal hemodynamics due to the AVF.

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