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Meyer J J, Erbel R, Pop T, et al. Balloon coronary angioplasty in patients with acute myocardial infarction. Tex Heart Inst J. 1986;13(4):393-400
Meyer J, J., Erbel, R., Pop, T., von Olshausen, K., Schuster, C. J., Treese, N., Rupprecht, H. J., Henrichs, K. J., Diefenbach, C., Eissner, D., & Hahn, K. (1986). Balloon coronary angioplasty in patients with acute myocardial infarction. Texas Heart Institute journal, 13(4), 393-400.
Meyer J, J, et al. "Balloon coronary angioplasty in patients with acute myocardial infarction." Texas Heart Institute journal vol. 13,4 (1986): 393-400.
Meyer J J, Erbel R, Pop T, von Olshausen K, Schuster CJ, Treese N, Rupprecht HJ, Henrichs KJ, Diefenbach C, Eissner D, Hahn K. Balloon coronary angioplasty in patients with acute myocardial infarction. Tex Heart Inst J. 1986 Dec;13(4):393-400. PMID: 15227348; PMCID: PMC324670.
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Tex Heart Inst J. 1986 Dec;13(4):393-400.

Balloon coronary angioplasty in patients with acute myocardial infarction.

Texas Heart Institute journal

J Meyer J, R Erbel, T Pop, K von Olshausen, C J Schuster, N Treese, H J Rupprecht, K J Henrichs, C Diefenbach, D Eissner, K Hahn

Affiliations

  1. Second Medical Clinic and the Department of Nuclear Medicine, Johannes Gutenberg University, Mainz, Germany.

PMID: 15227348 PMCID: PMC324670

Abstract

After successful thrombolysis, approximately 75% of all patients will have significant coronary stenosis, which can be dilated by means of percutaneous transluminal balloon angioplasty (PTCA). In a randomized control study, 95 of our patients (Group I) had thrombolysis alone, whereas 95 others (Group II) had thrombolysis and PTCA. Both groups were comparable with respect to age, sex, infarct location, and maximal creatine kinase (CK) value. The clinical outcome during the hospital phase was better in Group II, which had a reocclusion rate of 13%, a reinfarction rate of 5%, a lethal reinfarction rate of 2%, and a cardiac death rate of 7%, compared with respective rates of 20%, 13%, 7%, and 13% in Group I. Furthermore, in Group I, residual coronary stenosis immediately after thrombolysis (75% +/- 20%) did not improve significantly until the end of the hospital phase, when it decreased to 69% +/- 21%. In Group II, stenosis (78% +/- 16%) was improved by PTCA to 33% +/- 21%, and this improvement remained constant during the hospital phase (30% +/- 26%). In Group-II patients who had an unsuccessful PTCA, stenosis was approximately the same before dilatation (83% +/- 12%), after dilatation (80% +/- 17%), and at the control study (83% +/- 17%). The end-diastolic, end-systolic, and stroke volume indices, as well as the ejection fraction, also remained unchanged. In Group I, the number of pathologic wall segments (12.2 +/- 5.0) did not improve during the hospital phase (12.2 +/- 7.9), but in Group II, the improvement was significant (14.0 +/- 5.7 vs. 10.9 +/- 8.2) (p < 0.05). PTCA seems to improve the clinical outcome, reduce the infarction and mortality rates, and enhance myocardial perfusion and performance.

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