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Nagasawa S, Kawanishi M, Kondoh S, et al. Normal perfusion pressure hyperperfusion in cerebral arteriovenous malformation surgery: model study on the hemodynamics and mechanisms. J Clin Neurosci. 1998;5:30-2doi: 10.1016/s0967-5868(98)90007-8.
Nagasawa, S., Kawanishi, M., Kondoh, S., Yamaguchi, K., Kajimoto, S., Tada, Y., & Ohta, T. (1998). Normal perfusion pressure hyperperfusion in cerebral arteriovenous malformation surgery: model study on the hemodynamics and mechanisms. Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia, 530-2. https://doi.org/10.1016/s0967-5868(98)90007-8
Nagasawa, S, et al. "Normal perfusion pressure hyperperfusion in cerebral arteriovenous malformation surgery: model study on the hemodynamics and mechanisms." Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia vol. 5 (1998): 30-2. doi: https://doi.org/10.1016/s0967-5868(98)90007-8
Nagasawa S, Kawanishi M, Kondoh S, Yamaguchi K, Kajimoto S, Tada Y, Ohta T. Normal perfusion pressure hyperperfusion in cerebral arteriovenous malformation surgery: model study on the hemodynamics and mechanisms. J Clin Neurosci. 1998 Mar;5:30-2. doi: 10.1016/s0967-5868(98)90007-8. PMID: 18639096.
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J Clin Neurosci. 1998 Mar;5:30-2. doi: 10.1016/s0967-5868(98)90007-8.

Normal perfusion pressure hyperperfusion in cerebral arteriovenous malformation surgery: model study on the hemodynamics and mechanisms.

Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia

S Nagasawa, M Kawanishi, S Kondoh, K Yamaguchi, S Kajimoto, Y Tada, T Ohta

Affiliations

  1. Department of Neurosurgery, Osaka Medical College, Takatsuki, Japan.

PMID: 18639096 DOI: 10.1016/s0967-5868(98)90007-8

Abstract

A simulation study was undertaken using a compartmental flow model of a large high-flow cerebral arteriovenous malformation to investigate the hemodynamic changes during obliteration procedures. Under certain autoregulatory conditions, marked hyperperfusion (92 ml/100 g/min) could be induced in association with increased wall stress of the arterioles. Narrowing of the autoregulatory pressure range and its shift to a low pressure level are suspected to be among the possible causes of normal perfusion pressure breakthrough phenomenon.

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