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Leyer F, Nallet O, Cattan S. Le syndrome de takotsubo ou ballonisation apicale transitoire du ventricule gauche. [Takotsubo cardiomyopathy or transient left ventricular apical ballooning syndrome]. Ann Cardiol Angeiol (Paris). 2008;57(5):284-9doi: 10.1016/j.ancard.2008.08.004.
Leyer, F., Nallet, O., & Cattan, S. (2008). Le syndrome de takotsubo ou ballonisation apicale transitoire du ventricule gauche. [Takotsubo cardiomyopathy or transient left ventricular apical ballooning syndrome]. Annales de cardiologie et d'angeiologie, 57(5), 284-9. https://doi.org/10.1016/j.ancard.2008.08.004
Leyer, F, et al. "Le syndrome de takotsubo ou ballonisation apicale transitoire du ventricule gauche." [Takotsubo cardiomyopathy or transient left ventricular apical ballooning syndrome]. Annales de cardiologie et d'angeiologie vol. 57,5 (2008): 284-9. doi: https://doi.org/10.1016/j.ancard.2008.08.004
Leyer F, Nallet O, Cattan S. Le syndrome de takotsubo ou ballonisation apicale transitoire du ventricule gauche. [Takotsubo cardiomyopathy or transient left ventricular apical ballooning syndrome]. Ann Cardiol Angeiol (Paris). 2008 Nov;57(5):284-9. doi: 10.1016/j.ancard.2008.08.004. Epub 2008 Sep 18. French. PMID: 18937923.
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Ann Cardiol Angeiol (Paris). 2008 Nov;57(5):284-9. doi: 10.1016/j.ancard.2008.08.004. Epub 2008 Sep 18.

[Takotsubo cardiomyopathy or transient left ventricular apical ballooning syndrome].

Annales de cardiologie et d'angeiologie

[Article in French]
F Leyer, O Nallet, S Cattan

Affiliations

  1. Service de cardiologie, CHI Le-Raincy-Montfermeil, 10, rue du Général-Leclerc, 93370 Montfermeil, France. [email protected]

PMID: 18937923 DOI: 10.1016/j.ancard.2008.08.004

Abstract

Takotsubo is a reversible cardiomyopathy, often triggered by a stressful event. It combines clinical features mimicking a myocardial infarction, transient apical ballooning of the left ventricle, normal coronary arteries and a small rise in troponin level. There is a striking female predominance with mean age ranging from 65 to 76 years among series. Preceding stressful event is documented in 50 to 100% of patients. The most common clinical presentation is an angor-like chest pain with ST-segment elevation on the electrocardiogram (70%). The prognosis is excellent even if serious complications may occur: pulmonary oedema, cardiogenic shock, transient dynamic intraventricular gradient, life-threatening arrhythmias. In-hospital mortality is in the range of 0 to 10%. The recurrence rate is low. The precise physiopathology of the syndrome remains unknown but catecholamine mediated myocardial stunning is the most favored explanation.

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