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Furusato E, Cameron JD, Chan CC. Evolution of Cellular Inclusions in Bietti's Crystalline Dystrophy. Ophthalmol Eye Dis. 2010;2010(2):9-15doi: 10.4137/OED.S2821.
Furusato, E., Cameron, J. D., & Chan, C. C. (2010). Evolution of Cellular Inclusions in Bietti's Crystalline Dystrophy. Ophthalmology and eye diseases, 2010(2), 9-15. https://doi.org/10.4137/OED.S2821
Furusato, Emiko, et al. "Evolution of Cellular Inclusions in Bietti's Crystalline Dystrophy." Ophthalmology and eye diseases vol. 2010,2 (2010): 9-15. doi: https://doi.org/10.4137/OED.S2821
Furusato E, Cameron JD, Chan CC. Evolution of Cellular Inclusions in Bietti's Crystalline Dystrophy. Ophthalmol Eye Dis. 2010 Mar 09;2010(2):9-15. doi: 10.4137/OED.S2821. PMID: 21359135; PMCID: PMC3045089.
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Ophthalmol Eye Dis. 2010 Mar 09;2010(2):9-15. doi: 10.4137/OED.S2821.

Evolution of Cellular Inclusions in Bietti's Crystalline Dystrophy.

Ophthalmology and eye diseases

Emiko Furusato, J Douglas Cameron, Chi-Chao Chan

Affiliations

  1. Immunopathology Section, Laboratory of Immunology, National Eye Institute, National Institutes of Health, Bethesda, MD.

PMID: 21359135 PMCID: PMC3045089 DOI: 10.4137/OED.S2821

Abstract

Bietti's crystalline dystrophy (BCD) consists of small, yellow-white, glistening intraretinal crystals in the posterior pole, tapetoretinal degeneration with atrophy of the retinal pigment epithelium (RPE) and "sclerosis" of the choroid; in addition, sparking yellow crystals in the superficial marginal cornea are also found in many patients. BCD is inherited as an autosomal-recessive trait (4q35-tel) and usually has its onset in the third decade of life. This review focuses on the ultrastructure of cellular crystals and lipid inclusions of BCD.

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