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Ali A, Char G, Hanchard B. Chronic inflammatory demyelinating polyneuropathy in a patient infected with human T lymphotropic virus type I. BMJ Case Rep. 2009;2009doi: 10.1136/bcr.03.2009.1680.
Ali, A., Char, G., & Hanchard, B. (2009). Chronic inflammatory demyelinating polyneuropathy in a patient infected with human T lymphotropic virus type I. BMJ case reports, 2009. https://doi.org/10.1136/bcr.03.2009.1680
Ali, Amza, et al. "Chronic inflammatory demyelinating polyneuropathy in a patient infected with human T lymphotropic virus type I." BMJ case reports vol. 2009 (2009). doi: https://doi.org/10.1136/bcr.03.2009.1680
Ali A, Char G, Hanchard B. Chronic inflammatory demyelinating polyneuropathy in a patient infected with human T lymphotropic virus type I. BMJ Case Rep. 2009;2009. doi: 10.1136/bcr.03.2009.1680. Epub 2009 Aug 20. PMID: 21857879; PMCID: PMC3027780.
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BMJ Case Rep. 2009;2009. doi: 10.1136/bcr.03.2009.1680. Epub 2009 Aug 20.

Chronic inflammatory demyelinating polyneuropathy in a patient infected with human T lymphotropic virus type I.

BMJ case reports

Amza Ali, Gurendra Char, Barrie Hanchard

Affiliations

  1. University of the West Indies, c/o Epilepsy Centre of Jamaica, 27 Hope Rd, Kingston, 10, Jamaica.

PMID: 21857879 PMCID: PMC3027780 DOI: 10.1136/bcr.03.2009.1680

Abstract

A 32-year-old Afro-Caribbean woman presented with a 1-year history of slowly progressive sensory and motor symptoms initially affecting the legs and later involving the arms. Clinical examination demonstrated a mainly distal pattern of weakness with little objective sensory impairment. The clinical features suggested the possibility of chronic inflammatory demyelinating polyneuropathy. This diagnosis was supported by neurophysiological testing and examination of the cerebrospinal fluid and confirmed by sural nerve biopsy as well as by exclusion of other causes of neuropathy. Seropositivity for human T lymphotropic virus type I (HTLV-I) was demonstrated. The clinical significance of this finding in an area with a high HTLV-I endemicity, as well as its possible aetiological relevance to the diagnosis of chronic inflammatory demyelinating polyneuropathy, is discussed.

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