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Vila-Vives JM, Hidalgo-Mora JJ, Soler I, et al. Fetal arthrogryposis secondary to a giant maternal uterine leiomyoma. Case Rep Obstet Gynecol. 2012;2012:726732doi: 10.1155/2012/726732.
Vila-Vives, J. M., Hidalgo-Mora, J. J., Soler, I., Rubio, J., Quiroga, R., & Perales, A. (2012). Fetal arthrogryposis secondary to a giant maternal uterine leiomyoma. Case reports in obstetrics and gynecology, 2012726732. https://doi.org/10.1155/2012/726732
Vila-Vives, José María, et al. "Fetal arthrogryposis secondary to a giant maternal uterine leiomyoma." Case reports in obstetrics and gynecology vol. 2012 (2012): 726732. doi: https://doi.org/10.1155/2012/726732
Vila-Vives JM, Hidalgo-Mora JJ, Soler I, Rubio J, Quiroga R, Perales A. Fetal arthrogryposis secondary to a giant maternal uterine leiomyoma. Case Rep Obstet Gynecol. 2012;2012:726732. doi: 10.1155/2012/726732. Epub 2012 Nov 12. PMID: 23198198; PMCID: PMC3502792.
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Case Rep Obstet Gynecol. 2012;2012:726732. doi: 10.1155/2012/726732. Epub 2012 Nov 12.

Fetal arthrogryposis secondary to a giant maternal uterine leiomyoma.

Case reports in obstetrics and gynecology

José María Vila-Vives, Juan José Hidalgo-Mora, Inmaculada Soler, Juan Rubio, Ramiro Quiroga, Alfredo Perales

Affiliations

  1. Department of Obstetrics, La Fe University Hospital, Bulevar Sur s/n, 46026 Valencia, Spain.

PMID: 23198198 PMCID: PMC3502792 DOI: 10.1155/2012/726732

Abstract

Arthrogryposis multiplex congenital is a rare condition defined as contractures in multiple joints at birth due to disorders starting in fetal life. Its etiology is associated with many different conditions and in many instances remains unknown. The final common pathway to all of them is decreased fetal movement (fetal akinesia) due to an abnormal intrauterine environment. Causes of decreased fetal movements may be neuropathic abnormalities, abnormalities of connective tissue or muscle, intrauterine vascular compromise, maternal diseases, and space limitations within the uterus. When the cause of arthrogryposis is space limitations in uterus, the most common etiology is oligohydramnios. The same can result from intrauterine tumours as fibroids, although to our knowledge there are only two papers reporting cases of fetal deformities related to uterine leiomyomas. We describe a well-documented exceptional case of arthrogryposis associated with the presence of a large uterine fibroid. It could illustrate the importance of a careful and appropriate assessment of uterine fibroids before and in the course of a pregnancy considering that they can cause both serious maternal and fetal complications.

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