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Bai X, Fermandez E, Gould G, et al. Homozygous Deletion of Glutathione Peroxidase 1 and Aldehyde Dehydrogenase 1a1 Genes Is Not Associated with Schizophrenia-Like Behavior in Mice. J Biochem Pharmacol Res. 2013;1(4):228-235
Bai, X., Fermandez, E., Gould, G., & Strong, R. (2013). Homozygous Deletion of Glutathione Peroxidase 1 and Aldehyde Dehydrogenase 1a1 Genes Is Not Associated with Schizophrenia-Like Behavior in Mice. Journal of biochemical and pharmacological research, 1(4), 228-235.
Bai, Xiang, et al. "Homozygous Deletion of Glutathione Peroxidase 1 and Aldehyde Dehydrogenase 1a1 Genes Is Not Associated with Schizophrenia-Like Behavior in Mice." Journal of biochemical and pharmacological research vol. 1,4 (2013): 228-235.
Bai X, Fermandez E, Gould G, Strong R. Homozygous Deletion of Glutathione Peroxidase 1 and Aldehyde Dehydrogenase 1a1 Genes Is Not Associated with Schizophrenia-Like Behavior in Mice. J Biochem Pharmacol Res. 2013 Dec 01;1(4):228-235. PMID: 24494173; PMCID: PMC3909525.
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J Biochem Pharmacol Res. 2013 Dec 01;1(4):228-235.

Homozygous Deletion of Glutathione Peroxidase 1 and Aldehyde Dehydrogenase 1a1 Genes Is Not Associated with Schizophrenia-Like Behavior in Mice.

Journal of biochemical and pharmacological research

Xiang Bai, Elizabeth Fermandez, Georgianna Gould, Randy Strong

Affiliations

  1. Department of Pharmacology, University of Texas Health Science Center, San Antonio, TX USA ; Barshop Institute for Longevity and Aging Studies, University of Texas Health Science Center, San Antonio, TX USA.
  2. Department of Pharmacology, University of Texas Health Science Center, San Antonio, TX USA ; Barshop Institute for Longevity and Aging Studies, University of Texas Health Science Center, San Antonio, TX USA ; Geriatric Research, Education and Clinical Center, South Texas Veterans Health Care Network, San Antonio, TX USA.
  3. Department of Physiology, South Texas Veterans Health Care Network, San Antonio, TX USA.

PMID: 24494173 PMCID: PMC3909525

Abstract

Much evidence suggests that oxidative stress plays a role in schizophrenia pathogenesis. Major oxidative stress sources include hydrogen peroxide and biogenic aldehydes that are mainly cleared

Keywords: aldehyde dehydrogenase 1a1 (Aldh1a1); gene deletion; glutathione peroxidase 1 (Gpx1); oxidative stress; schizophrenia

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