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Miller KA, Tan TY, Welfare MF, et al. A mouse splice-site mutant and individuals with atypical chromosome 22q11.2 deletions demonstrate the crucial role for crkl in craniofacial and pharyngeal development. Mol Syndromol. 2014;5(6):276-86doi: 10.1159/000368865.
Miller, K. A., Tan, T. Y., Welfare, M. F., White, S. M., Stark, Z., Savarirayan, R., Burgess, T., Heggie, A. A., Caruana, G., Bertram, J. F., Bateman, J. F., & Farlie, P. G. (2014). A mouse splice-site mutant and individuals with atypical chromosome 22q11.2 deletions demonstrate the crucial role for crkl in craniofacial and pharyngeal development. Molecular syndromology, 5(6), 276-86. https://doi.org/10.1159/000368865
Miller, Kerry A, et al. "A mouse splice-site mutant and individuals with atypical chromosome 22q11.2 deletions demonstrate the crucial role for crkl in craniofacial and pharyngeal development." Molecular syndromology vol. 5,6 (2014): 276-86. doi: https://doi.org/10.1159/000368865
Miller KA, Tan TY, Welfare MF, White SM, Stark Z, Savarirayan R, Burgess T, Heggie AA, Caruana G, Bertram JF, Bateman JF, Farlie PG. A mouse splice-site mutant and individuals with atypical chromosome 22q11.2 deletions demonstrate the crucial role for crkl in craniofacial and pharyngeal development. Mol Syndromol. 2014 Dec;5(6):276-86. doi: 10.1159/000368865. Epub 2014 Nov 08. PMID: 25565927; PMCID: PMC4281577.
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